ライフサイエンスコーパス: pleocytosis

1 (91%) of 11 children had cerebrospinal fluid pleocytosis.

2 s with and without cerebrospinal fluid (CSF) pleocytosis.

3 d lethargy, and lumbar puncture might reveal pleocytosis.

4 presenting to emergency departments with CSF pleocytosis.

5 sually shows modest elevation of protein and pleocytosis.

6 the diagnostic sensitivity or specificity of pleocytosis.

7 igns or symptoms despite cerebrospinal fluid pleocytosis.

8 repeated CSF culture, even in the absence of pleocytosis.

9 ging and 43 patients had cerebrospinal fluid pleocytosis.

10 ents with normal CSF cell counts, those with pleocytosis (1) more often reported radicular pain and m

11 Among 3295 patients with CSF pleocytosis, 121 (3.7%; 95% confidence interval [CI], 3.

12 Cerebrospinal fluid pleocytosis and rash are absent in about a third of case

13 rolipram (0-0.01 microgram/kg/h), inhibited pleocytosis and reduced the lipopolysaccharide-induced i

14 Degree of CSF pleocytosis and skin-lesion severity were estimated by t

15 Our objective was to determine if pleocytosis and/or elevated protein levels were predicti

16 All patients had pleocytosis, and 3 had severe prodromal diarrhea of unkn

17 ty (agitation, myoclonus, tremor, seizures), pleocytosis, and frequent diarrhea at symptom onset.

18 tutional symptoms, cerebrospinal fluid (CSF) pleocytosis, and hilar adenopathy more frequently than N

19 gs suggested that disease stage, lymphocytic pleocytosis, and HIV-1 RNA levels in plasma may influenc

20 titers, time to maximum titer, degree of CSF pleocytosis, and severity of skin lesions differed signi

21 Children with cerebrospinal fluid (CSF) pleocytosis are routinely admitted to the hospital and t

22 rebrospinal fluid (CSF) for parasites and/or pleocytosis are sensitive, but recent evidence suggests

23 of evaluable cerebrospinal fluid samples had pleocytosis, but HPeV was detected in 95%.

24 was strongly correlated to plasma RNA and to pleocytosis, but in AIDS, CSF and plasma RNA were indepe

25 January 1, 2001, and June 30, 2004, with CSF pleocytosis (CSF white blood cells > or =10 cells/microL

26 hotomous-stage diagnosis on the basis of CSF pleocytosis does not accurately reflect the biological c

27 CSF) analysis revealed lymphocytic/monocytic pleocytosis, elevated protein concentration, and intrath

28 CSF pleocytosis, elevated protein, or depressed glucose conc

29 The positive predictive value of pleocytosis for microbiologically proven borrelial infec

30 than 0.5, a neutrophilic cerebrospinal fluid pleocytosis (> 5 cells/muL), and fever.

31 ts in all patients; however, 23.3% still had pleocytosis (>10 x 10(6) cells/L).

32 Cerebrospinal fluid (CSF) pleocytosis (>5 white blood cells/microL) was common (81

33 45%) and cerebrospinal fluid analysis showed pleocytosis in 17 of 22 patients (77%).

34 -6, IL-8, chemokine ligand 2, and CXCL13 and pleocytosis in all infected animals, except dexamethason

35 All of the articles have shown CSF pleocytosis in MS.

36 m 8 had CSF abnormalities (n = 3 lymphocytic pleocytosis, n = 5 elevated protein, n = 4 increased ope

37 ients have a mononuclear cerebrospinal fluid pleocytosis, often with red blood cells.

38 CSF RNA levels were independent of CSF pleocytosis or antiretroviral exposure.

39 ry parameter, including fever, leukocytosis, pleocytosis, or CSF protein and glucose, could reliably

40 meningismus (P = 0.04), cerebrospinal fluid pleocytosis (P = 0.04) or multifocal enhancing magnetic

41 The CSF of all four patients contained a pleocytosis, predominantly mononuclear with elevated lev

42 All patients had abnormal CSF: six pleocytosis, six had increased protein concentration, an

43 solated from EM skin lesions (odds ratio for pleocytosis was 31 times higher in patients with establi

44 Cerebrospinal fluid pleocytosis was generally lacking.

45 p of patients aged >2 months, the absence of pleocytosis was highly predictive of a negative RT-PCR r

46 Severity of skin lesions and of CSF pleocytosis were inversely correlated (P=.005).

47 normalities with mildly elevated protein and pleocytosis with >90% lymphocytes, predominantly CD8, we

48 atients, cerebrospinal fluid analyses showed pleocytosis with elevated protein.

49 aboratory results (cerebrospinal fluid (CSF) pleocytosis with eosinophils and/or neutrophils, oligocl

50 evated protein concentration and lymphocytic pleocytosis with no malignant cells on cytological analy

51 Cerebrospinal fluid invariably shows a pleocytosis, with a predominance of neutrophils in up to

52 eadache, elevated intracranial pressure, and pleocytosis, with or without eosinophilia, particularly