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1 and calf vascular resistance (determined by plethysmography).
2 nce tomography and six respiratory inductive plethysmography).
3 ined by the forced oscillation technique and plethysmography.
4 0% O2) with 5 min intervals, was measured by plethysmography.
5 nction was determined by peripheral arterial plethysmography.
6 ion reflex responses to ETS were examined by plethysmography.
7 composition was measured by air-displacement plethysmography.
8 choline challenge was measured by whole-body plethysmography.
9 russide were assessed using venous occlusion plethysmography.
10 ssessed by standard forearm venous occlusion plethysmography.
11 Respiration was examined by plethysmography.
12 sensitivity were assessed during LBNP using plethysmography.
13 ition was measured by using air-displacement plethysmography.
14 omposition was measured via air-displacement plethysmography.
15 week-old unanaesthetized rats via whole-body plethysmography.
16 re, and pulmonary function was determined by plethysmography.
17 oronasal airflow and respiratory inductance plethysmography.
18 elivery as measured by respiratory inductive plethysmography.
19 onchoconstriction was assessed by whole body plethysmography.
20 al pulse wave velocity, and venous occlusion plethysmography.
21 easured using venous-occlusion, strain-gauge plethysmography.
22 ted for every subject using venous occlusion plethysmography.
23 nitroprusside were measured by strain-gauge plethysmography.
24 CAD) using a robust, modified form of volume plethysmography.
25 re airway hyperreactivity through whole-body plethysmography.
26 ow responses were measured with strain-gauge plethysmography.
27 as measured using bilateral venous occlusion plethysmography.
28 (0.3 and 1 nmol/min) using venous occlusion plethysmography.
29 il) vasodilators were measured using forearm plethysmography.
30 coronary artery disease by venous occlusion plethysmography.
31 nitroprusside were measured by strain-gauge plethysmography.
32 and during reactive hyperemia by venous air plethysmography.
33 in 10 cirrhotic patients by venous occlusion plethysmography.
34 ults with concentrations of exhaled NO after plethysmography.
35 n at 2 wk was determined by air-displacement plethysmography.
36 whites and 18 blacks by use of strain-gauge plethysmography.
37 ng, and body composition by air-displacement plethysmography.
38 lood flow was determined by venous occlusion plethysmography.
39 Forearm blood flow was determined by plethysmography.
40 g non-dominant forearm with venous occlusion plethysmography.
41 ntilation (VE) was measured using whole-body plethysmography.
42 ery and correlated well with measurements at plethysmography.
43 lood flow was determined by venous occlusion plethysmography.
44 y hyperresponsiveness was evaluated by using plethysmography.
45 oxide production by forearm venous occlusion plethysmography.
46 lood flow was determined by venous occlusion plethysmography.
47 d flow (FBF was measured by venous occlusion plethysmography.
48 ume measurements using respiratory inductive plethysmography.
49 nce in live anesthetized mice using invasive plethysmography.
50 ation, as measured by respiratory inductance plethysmography.
51 position was measured using air displacement plethysmography.
52 m vasodilator responses to isoprenaline with plethysmography.
53 pedance tomography and respiratory inductive plethysmography.
54 zoelectric sensor, and inductive respiratory plethysmography.
55 rements were taken by using air displacement plethysmography.
56 measured lung function using spirometry and plethysmography.
57 Ventilation was measured by whole-body plethysmography.
58 using invasive tracheostomy and unrestrained plethysmography.
59 l arm blood flow as assessed by strain-gauge plethysmography.
60 h COPD underwent MR imaging, spirometry, and plethysmography.
61 tion was measured in nonanesthetized mice by plethysmography.
62 Breathing was monitored with plethysmography.
63 by monitoring their sniffing with whole-body plethysmography.
64 s, left/right limbs 14/9, using strain gauge plethysmography; 7/16 of these had thrombosis extending
65 sodilation, by both forearm venous occlusion plethysmography (93 +/- 67% vs. 145 +/- 106%, p = 0.006)
66 re, electrocardiogram, and oxygen saturation plethysmography activity were recorded and reviewed for
67 to determine the ability of air-displacement plethysmography (ADP) and formulas based on triceps skin
68 l age (GA), with the use of air-displacement plethysmography (ADP) and the lambda-mu-sigma method for
69 mposition measurement using air displacement plethysmography (ADP) that was used to calculate a refer
70 , 4.5, and 6 mo of age with air-displacement plethysmography (ADP) validated against a stable isotope
72 tokine profile from reactivated splenocytes, plethysmography after aerosol challenge to pollen, cell,
74 ng forearm blood flow (FBF; venous occlusion plethysmography) after 5 minutes of arterial occlusion.
75 analysed in unanaesthetised animals by body plethysmography along with rate of O2 consumption (VO2))
77 3 months corrected age with air displacement plethysmography and at 1-year-corrected age with skin-fo
78 factors were assessed with venous occlusion plethysmography and blood sampling during intra-arterial
79 espiratory circuits, we performed whole-body plethysmography and electrophysiological recordings from
84 tion was assessed through the use of forearm plethysmography and related to clinical disease activity
87 ed forearm blood flow (FBF; venous occlusion plethysmography) and calculated the vascular conductance
88 easured forearm blood flow (venous occlusion plethysmography) and calculated vascular conductance (FV
89 vascular conductance (FVC, venous occlusion plethysmography) and cutaneous vascular conductance (CVC
90 rm blood flow (measured via venous occlusion plethysmography) and intra-arterial blood pressure to qu
91 ulmonary function studies (spirometry and/or plethysmography), and a cardiology evaluation which incl
93 lood flow was determined by venous occlusion plethysmography, and dose-response curves were generated
95 methacholine was assessed by barometric body plethysmography, and numbers of lung eosinophils and lev
96 methacholine was assessed by barometric body plethysmography, and numbers of lung eosinophils and pro
99 graphy, graded treadmill exercise, tail cuff plethysmography, and telemetry (heart rate, activity, te
100 Blood flow was measured by venous occlusion plethysmography, and the percentage of HbO(2) perfusing
101 illary lactate (the primary endpoint), thumb plethysmography, and ulnar frame count to investigate th
102 ibrated and validated a new air-displacement plethysmography (AP) method for measuring body compositi
104 ycemia, we studied forearm blood flow (FBF) (plethysmography), arteriovenous glucose difference (AV-d
105 ed as enhanced pause (Penh) with noninvasive plethysmography at Day 16 of age), and did not cause inf
108 od flow, was measured noninvasively by using plethysmography at rest, after breathing supplemental ox
109 ial occlusion was determined by strain gauge plethysmography before and 4 h and six weeks after combi
110 ial acetylcholine) was assessed with forearm plethysmography before and after (1) 15-minute reperfusi
111 earm blood flow (FBF) using venous occlusion plethysmography before and after intra-arterial infusion
112 .2 microg/min) were assessed by strain-gauge plethysmography before and after nonselective blockade o
113 al voluntary contraction by venous occlusion plethysmography before and after regional inhibition of
114 rip and cold pressor test were determined by plethysmography before and during adrenergic receptor bl
115 latation were assessed with venous occlusion plethysmography before and during intra-arterial infusio
116 te-macrophage colony-stimulating factor) and plethysmography, before and after methacholine, to asses
117 blood flow was measured by venous occlusion plethysmography, before, and 8 min after, completing inf
119 re measured using bilateral venous occlusion plethysmography, bioimpedance cardiography, transthoraci
120 to 10 minutes of forearm arterial occlusion (plethysmography), blood pressure, and muscle sympathetic
121 ing) constant bias, and the air displacement plethysmography body density and SFT methods showed posi
122 -compartment, Db(VRJ), BIA, air displacement plethysmography body density, and SFT ranging from a mea
123 e-macrophage colony-stimulating factor), and plethysmography, both before and after methacholine trea
124 sed at birth and at 3 mo by air-displacement plethysmography by using the Pea Pod system (Cosmed) and
125 of 10 healthy volunteers enclosed in a body plethysmography chamber mimicking the entrapment environ
126 pliant protocol and underwent spirometry and plethysmography, completed the St George's Respiratory Q
127 ume (both at airway opening and by inductive plethysmography) could be identified on the deflation li
128 with the reduction in respiratory inductive plethysmography-derived lung volume, high continuous dis
129 tive pulmonary disease underwent spirometry, plethysmography, diffusing capacity of carbon monoxide,
131 cardiac output were made by venous occlusion plethysmography, Doppler flow wire and quantitative coro
132 pedance tomography and respiratory inductive plethysmography during a stepwise recruitment procedure.
133 blood flow was measured by venous occlusion plethysmography during an intrabrachial infusion of brad
134 , freely behaving rat pups, using whole-body plethysmography during breathing of room air (RA), durin
135 blood flow was measured by venous occlusion plethysmography during intrabrachial infusions of apelin
136 of FM, and FFM measured by air-displacement plethysmography during the first 5 mo of life.Applying p
138 asurements were performed using strain-gauge plethysmography during transfusion, followed by testing
139 sing deuterium dilution and air-displacement plethysmography), eating behavior (measured by using a 3
141 ion was assessed by forearm venous occlusion plethysmography, flow-mediated dilation, and pulse wave
142 All three visits utilized ECG and finger plethysmography for haemodynamic measures, and the high
143 e (HR), forearm blood flow (venous occlusion plethysmography), FVR, and MSNA (obtained through direct
144 ne resorption in rat AIA were assessed using plethysmography, histopathologic analysis, and immunohis
145 y frequency (RF) was monitored by whole-body plethysmography immediately after the 4th challenge (ear
146 hma were characterized with spirometry, body plethysmography, impulse oscillometry, alveolar and bron
147 BQ-123 were assessed using venous occlusion plethysmography in 10 patients with syndrome X and 10 ma
148 Lung volumes were measured by spirometry and plethysmography in 109 healthy subjects aged 7-21 years.
149 lood flow was measured with venous occlusion plethysmography in 12 cigarette smokers and 12 age- and
150 blood flow was measured by venous occlusion plethysmography in 16 volunteers during infusion of SFLL
151 enous tone using radionuclide forearm venous plethysmography in 24 healthy subjects with no cardiovas
152 ) receptor blocker (BQ-123) were analyzed by plethysmography in 37 normotensive patients and 27 hyper
155 blood flow was measured by venous occlusion plethysmography in response to serial intra-arterial inf
156 viously determined by respiratory inductance plethysmography in that subject (i.e., particles inhaled
157 responses were assessed by venous occlusion plethysmography in the brachial circulation before and a
159 lf blood flow was measured with strain gauge plethysmography in the two higher dose treatment groups
160 (brachial catheter) and forearm blood flow (plethysmography) in 19 healthy subjects at baseline and
161 l improved peak blood flow (venous occlusion plethysmography) in arms (+24%, P=0.027) and legs (+23%,
163 ed by a combination of whole-body barometric plethysmography, invasive measurement of airway resistan
165 nhaled methacholine by barometric whole-body plethysmography is a valid indicator of airway hyperresp
166 ve surrogates for cystometry, such as penile plethysmography, lack sufficient evidence to allow recom
170 ficantly correlated to respiratory inductive plethysmography measurements in 12 patients (mean r = 0.
172 Parameters studied included strain-gauge plethysmography measures of peripheral circulatory funct
173 electroencephalography, electrocardiography, plethysmography, mechanical ventilation or pharmacologic
174 lood pressure data by the indirect tail-cuff plethysmography method consistently shows increased pres
175 lood flow were evaluated by venous occlusion plethysmography, MSNA by microneurography, and blood pre
176 control subjects underwent spirometry, body plethysmography, multiple-breath inert gas washout (with
177 otocols mimicking those imposed by tail-cuff plethysmography (novel environment, heat, restraint, inf
178 abdominal wall motion using opto-electronic plethysmography (OEP), (ii) intra-thoracic and intra-abd
182 linical Severity Scoring (VCSS) systems, air-plethysmography (outflow fraction [OF], venous filling i
183 es (deltaV(L)[t]) via respiratory inductance plethysmography over a range of P(aw) settings in five p
185 ung volume measured by respiratory inductive plethysmography, oxygen saturation, perfusion index, reg
189 monitoring was limited to blood pressure and plethysmography preoperatively and intraoperatively.
190 ) data available from respiratory inductance plethysmography provided important insight to changes in
191 t a constant level to acquire a pulse volume plethysmography (PVP) waveform and calibrate it to brach
193 l artery to measure forearm blood flow (FBF, plethysmography) responses to administration of isoprena
194 n, forearm blood flow (FBF; venous occlusion plethysmography) responses to brachial artery administra
195 Forearm blood flow (FBF, by strain-gauge plethysmography) responses to local intra-arterial infus
200 utility of calibrated respiratory inductance plethysmography (RIP) and esophageal manometry to identi
201 nd, to address whether respiratory inductive plethysmography (RIP) could be used to monitor changes i
202 ed home monitor using respiratory inductance plethysmography (RIP) with simultaneously recorded polys
205 ermination of cardiac function (by impedance plethysmography), serial pulmonary function tests (spiro
208 ultiple breath inert gas washout (MBW), body plethysmography, single-breath determination of carbon m
210 s measured by spirometry (FEV1; n = 521) and plethysmography (sRaw; n = 567), and AHR by methacholine
211 vity was measured by non-invasive whole-body plethysmography, Th2 response and airway inflammation by
212 kg (airway opening tidal volume), and 13.7% (plethysmography tidal volume) compared with total lung c
213 llary density (GCD); and 3) venous occlusion plethysmography to assess endothelium-dependent (% Hyper
214 to use the technique of abdominal inductance plethysmography to compare diurnal variation in girth in
215 ratory monitors using respiratory inductance plethysmography to detect apnea and obstructed breathing
216 Subjects were studied using strain gauge plethysmography to measure blood flow, P(v), and the ven
218 arm blood flow (FBF) responses (strain-gauge plethysmography) to intra-arterial infusion of a selecti
219 arm blood flow (FBF) responses (strain gauge plethysmography) to intraarterial infusion of a selectiv
220 We measured forearm blood flow responses (plethysmography) to isocapnic hypoxia (arterial saturati
222 The following measurements were taken: FM by plethysmography, total and specific immunoglobulin E (Ig
223 ecorded in newborn rat pups using whole-body plethysmography under normoxic and hypoxic conditions.
224 healthy control subjects, with strain-gauge plethysmography used to measure venous pressure (Pv), fo
225 sessed by bilateral forearm venous occlusion plethysmography using acetylcholine and N-monomethyl-L-a
226 d methacholine were determined by whole body plethysmography using changes in enhanced pause (Penh) a
227 including compression ultrasound, impedance plethysmography, ventilation-perfusion scanning, compute
228 ism of death in cases of viral encephalitis, plethysmography was evaluated in mice infected with 3 fl
232 sponse), and in 6 subjects, venous occlusion plethysmography was used to measure forearm blood flow d
233 To address this question, venous occlusion plethysmography was used to measure forearm blood flow r
236 Pulmonary airflow resistance, as measured by plethysmography, was detected 1 day postinoculation and
237 pathology is the need to combine whole-body plethysmography (WBP) to measure respiration with electr
238 ) or noninvasive techniques, like whole body plethysmography (WBP), assesses the severity of pulmonar
242 h the dorsal hand vein technique and forearm plethysmography, we studied the effects of PAR-2 activat
243 phageal manometry and respiratory inductance plethysmography were made preextubation during airway oc
244 spiration and expiration), and spirometry or plethysmography were performed during a 2-hour visit to
245 ial artery catheter) and forearm blood flow (plethysmography) were measured and vascular conductance
246 Subjects (n=157) underwent venous occlusion plethysmography with acetylcholine, bradykinin, glyceryl
247 ssessed by standard forearm venous occlusion plethysmography with acetylcholine, nitroprusside, and v
248 ultaneously in both arms by venous occlusion plethysmography with mercury-in-Silastic strain gauges w
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