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1  PEL, which arises in a hypoxic environment (pleural effusions).
2 nical characters, such as refractoriness and pleural effusion.
3 peutic strategy for the control of malignant pleural effusion.
4 e II (n = 88) studies for the development of pleural effusion.
5 schedule may result in a higher incidence of pleural effusion.
6  patient irritability, small pericardial +/- pleural effusion.
7 o be secondary to a decrease or absence of a pleural effusion.
8 they showed any mismatched V/Q defect or any pleural effusion.
9 l or reticulonodular pattern with or without pleural effusion.
10 graphy for PE with radiographic evidence for pleural effusion.
11 t upper lobe mass along with a moderate-size pleural effusion.
12 diffuse interstitial pattern with or without pleural effusion.
13 owed mild pulmonary edema with a small right pleural effusion.
14 rentially diagnose tuberculous and malignant pleural effusion.
15 aphy showed an enlarged heart with bilateral pleural effusion.
16 ostoperative AF or postoperative pericardial/pleural effusion.
17 nts were postoperative AF and pericardial or pleural effusion.
18 esis is performed to identify the cause of a pleural effusion.
19 ractory MPP children or in MPP children with pleural effusion.
20 s. 19+/-6 bpm, p<0.01) than those with lower pleural effusion.
21 anulomas as well as ascites and a left-sided pleural effusion.
22 tein, release of inflammatory cytokines, and pleural effusion.
23 xudative pleural effusions from transudative pleural effusions.
24 cy and the presence, size, and laterality of pleural effusions.
25 e may reduce the incidence of pneumonia with pleural effusions.
26 ed as factors associated with development of pleural effusions.
27 s Fontan morbidity, particularly duration of pleural effusions.
28 ain the prominent and persistent hemorrhagic pleural effusions.
29 ne, it induced transient production of large pleural effusions.
30 ty, thickened bronchial walls, and bilateral pleural effusions.
31 loped low cardiac output, and 3 patients had pleural effusions.
32 rodesis was successful in treating malignant pleural effusions.
33 evaluated; (c) unsuspected pneumonia; or (d) pleural effusions.
34 lycans mimicked the inhibition observed with pleural effusions.
35 hymal opacities, cavitation, adenopathy, and pleural effusions.
36 onventional inpatient treatment of malignant pleural effusions.
37 all thickening, nodules, Kerley B lines, and pleural effusions.
38 eft pulmonary artery thrombosis, and chronic pleural effusions.
39 ntional T2-weighted sequences revealed small pleural effusions.
40 airment were more likely to have significant pleural effusions.
41 PCs (13.2% vs 40.5%, P < 0.001), symptomatic pleural effusion (11.6% vs 26.4%, P = 0.003), pleural ef
42 -up data were available for 31 of 39 treated pleural effusions: 11 (35%) had CR, 18 (58%) had PR, and
43 ), thoracic duct dilatation (4% vs 0, P=.3), pleural effusion (12% vs 6%, P=.2), or ascites (10% vs 6
44  (20%), intrathoracic lymphadenopathy (16%), pleural effusion (12%), reticular infiltration (4%), and
45              Interstitial opacities (6%) and pleural effusions (12%) were uncommon.
46 ophic scar formation (35), atelectasis (12), pleural effusion (13), recurrent sternal depression (5),
47 %), oral ulcers (24.2%), joint pain (23.0%), pleural effusion (16.5%) and increase in abdominal girth
48 rapy was most effective in the resolution of pleural effusions (2 of 2), ascites (6 of 8), and hydrop
49 opathy (23%), mass-like consolidation (17%), pleural effusion (8.6%), and honey combing (5.7%).
50                                  Significant pleural effusions after the Fontan operation prolong hos
51 tients in the once-daily group experienced a pleural effusion (all grades, 20% vs 39% P < .001).
52 ily resulted in significantly lower rates of pleural effusion (all grades, 7% v 16%; P = .024) and gr
53 y-proven PE, 77 (47%) had at least one large pleural effusion and 86 (53%) had a small effusion; 33 (
54 ome features was strong (eg, alpha = .78 for pleural effusion and ascites) but was lower for others (
55 ded the use of ultrasonography for ruling-in pleural effusion and assisting its drainage, ascites dra
56 ersistent fever caused by pneumonia (n = 2), pleural effusion and atelectasis (n = 1), or liver absce
57 adiologically as interstitial pneumonia with pleural effusion and clinically as hypoxemic respiratory
58                                     CAP with pleural effusion and documented pneumococcal CAP were di
59 th dasatinib compared with imatinib, whereas pleural effusion and grade 3/4 thrombocytopenia were mor
60 ion in lung injury, as assessed by volume of pleural effusion and histological analyses (significant
61                                         When pleural effusion and intra-abdominal hypertension were s
62                Among patients with malignant pleural effusion and no previous pleurodesis, there was
63 unrelated to alectinib: acute renal failure; pleural effusion and pericardial effusion; and brain met
64 r talc slurry in the management of recurrent pleural effusion and pneumothorax.
65 urs in mouse models of malignant lung cancer pleural effusion and spontaneous colon cancer metastasis
66 lations of leukocytes and malignant cells in pleural effusions and accurately predict disease state i
67                              In both groups, pleural effusions and air bronchograms are common, and K
68       Two patients, including one with large pleural effusions and another with ventricular tachycard
69 n of peritoneal spread, presence and size of pleural effusions and ascites, lymphadenopathy, and dist
70 renchyma, which is sometimes associated with pleural effusions and diffuse alveolar hemorrhage.
71 nt has altered the traditional management of pleural effusions and empyema in children, resulting in
72 tered to improve the drainage of complicated pleural effusions and empyemas and it is the most effect
73                     Prominent serosanguinous pleural effusions and hemorrhagic mediastinitis were fou
74 hy of the chest demonstrated large bilateral pleural effusions and hemorrhagic mediastinitis.
75 ared to be related to significant, prolonged pleural effusions and longer hospitalizations.
76 gnificantly post antibiotic treatment in the pleural effusions and pleural macrophages up-regulated m
77 by progression of respiratory insufficiency, pleural effusions and pulmonary edema, and, ultimately,
78 s observed, in the other one, pneumonia with pleural effusion, and as a consequence of inflammatory i
79 primary tumor, pleural metastases, malignant pleural effusion, and ascites obtained during disease pr
80 arch for thickening of the gallbladder wall, pleural effusion, and ascites.
81 ata suggest a strong impact of PCV13 on CAP, pleural effusion, and documented pneumococcal pneumonia,
82 cul-de-sac, peritoneal implants, ipsilateral pleural effusion, and intratumoral hemorrhage.
83            Reported events included seizure, pleural effusion, and lymphocytopenia.
84 sults suggest that drainage of the malignant pleural effusion, and perhaps enzymatic pretreatment of
85 pic support, thoracostomy tube drainage of a pleural effusion, and prolonged antimicrobial therapy.
86 ts and included disease progression, anemia, pleural effusion, and thrombocytopenia.
87 required no further treatment), two cases of pleural effusion, and two cases of moderate pain (one ca
88 on with tamponade associated with pneumonia, pleural effusion, and urinary tract infection.
89 y, and most animals also developed leukemia, pleural effusion, and, in some cases, ascites associated
90 monstrated mediastinal widening, adenopathy, pleural effusions, and air-space disease.
91 norexia, and nausea), skin rash, cytopenias, pleural effusions, and fatigue.
92 samples (tumor and normal pleura), malignant pleural effusions, and in established mesothelioma cell
93 of thiourea, W/D increased to 5.5 and marked pleural effusions appeared, but there were no difference
94                               Hemorrhage and pleural effusion are prominent pathological features of
95                                  Over 200000 pleural effusions are attributable to cancer in the UK a
96                                              Pleural effusions are common in candidates for heart, li
97 heir presence in urine, chylous ascites, and pleural effusions are consistent with their ability to m
98                                              Pleural effusions are expected sequelae after lung trans
99 ts corresponding to radiographically-evident pleural effusions are of intermediate probability for PE
100 nvolvement-thoracic duct dilatation, chylous pleural effusion, ascites, and LALM-is more common in LA
101  that increased gall bladder wall thickness, pleural effusion, ascites, hepatomegaly, and splenomegal
102 ngs included enlarged abdominal lymph nodes, pleural effusions, ascites, and dilatation of the thorac
103 estigated the risk factors and management of pleural effusion associated with dasatinib therapy for c
104 sion may be instrumental to the treatment of pleural effusion-associated lung restriction and cyclica
105  was designed to determine the prevalence of pleural effusion at approximately 28 days after cardiac
106 ion, but 1 patient developed pericardial and pleural effusion attributed to pericardial instrumentati
107                            The presence of a pleural effusion before dasatanib therapy predicted the
108 liphatic analogues toward MCF-7 cells (human pleural effusion breast adenocarcinoma cell line).
109          Lung congestion and pericardial and pleural effusions can cause attenuation in the ECG poten
110 rom 41.3% to 29.7% (P < .001), the number of pleural effusion cases decreased by 53% (167 to 79; P <
111 e believe that this is the first report of a pleural effusion caused by HSV II, and suggest that this
112                                              Pleural effusions caused by herpes simplex viruses are r
113                                    Malignant pleural effusion causes disabling dyspnea in patients wi
114                                     RNA from pleural effusion cells was examined to determine TGF-bet
115 ntestinal and/or pulmonary lymphangiectasia, pleural effusions, chylothoraces and/or pericardial effu
116                                      Chylous pleural effusion (chylothorax) frequently accompanies ly
117                      Those with CNS disease, pleural effusion, circulating lymphoma cells > or = 5,00
118 -2.2% to 17.6%) or postoperative pericardial/pleural effusion (colchicine, 103 patients [57.2%]; plac
119  phosphorylation in MT cells, derived from a pleural effusion, compared with cells from the primary t
120 adiograph revealed hilar adenopathy and left pleural effusion; computed tomography scan showed a 2-cm
121 s predictive of CT outcome, although lack of pleural effusion correlated with clinical stability.
122 investigated whether fibulin-3 in plasma and pleural effusions could meet sensitivity and specificity
123     In cytology-negative suspected malignant pleural effusions, CT-guided pleural biopsy is a better
124                Among patients with malignant pleural effusion, daily drainage of pleural fluid via an
125                              Pericardial and pleural effusions developed in one patient after cycles
126 ever, higher morbidity of tachypnea/dyspnea, pleural effusion, diarrhea, hepatosplenomegaly, consciou
127 )CD16(-) NK cells isolated from inflammatory pleural effusions display a potent regulatory activity.
128 to improve overall survival in patients with pleural effusion due to malignant pleural mesothelioma,
129 tasis have a history of asbestos exposure or pleural effusion due to various causes.
130 ion defect(s), and (g) perfusion defect from pleural effusion equal to one third or more of the pleur
131 ential diagnosis of an unexplained exudative pleural effusion, especially in an immunocompromised hos
132 2S protein were observed in seven of 17 lung pleural effusion fluids of lung cancer patients.
133 es vascular permeability and is important in pleural effusion formation.
134 s immunosuppressive therapy, who had a large pleural effusion, found on a chest radiograph during the
135 nd primary cultures, as well as in a primary pleural effusion from a breast cancer patient.
136 Organ-specific pleural complications include pleural effusion from hepatic venoocclusive disease, spo
137 after bone marrow transplantation, and early pleural effusion from urinothorax and late effusion from
138 d we also detected CXCL12-gamma in malignant pleural effusions from patients with breast cancer.
139 values can help in differentiating exudative pleural effusions from transudative pleural effusions.
140 -172 mL vs. 80+/-87 mL, p<0.0001, for higher pleural effusion group vs. lower pleural effusion group)
141 /-124 mL vs. 23+/-29 mL, p<0.0001 for higher pleural effusion group vs. lower pleural effusion group,
142  for higher pleural effusion group vs. lower pleural effusion group) was greater than the estimated l
143  for higher pleural effusion group vs. lower pleural effusion group, respectively).
144  Patients were divided into higher and lower pleural effusion groups according to the median value (2
145 s following induction of an antigen-specific pleural effusion, guinea pigs were injected intrapleural
146           RATIONALE: Patients with malignant pleural effusions have significant dyspnea and shortened
147                DLT in later courses included pleural effusions, hemangiomatosis, and GI hemorrhage.
148 ommonly seen pulmonary complications include pleural effusion, hemothorax, atelectasis, pulmonary ede
149 n 480 (39.5%) patients including symptomatic pleural effusion in 366 (30.1%) patients, respiratory in
150  63 patients, CT demonstrated an increase in pleural effusion in 46 (73%), periaortic changes in 21 (
151 d JIMT-1 breast cancer cells, derived from a pleural effusion in a patient who displayed clinical res
152                                              Pleural effusion in acute lung injury or acute respirato
153                     To assess the effects of pleural effusion in patients with acute lung injury on l
154 ions in 13 group A versus 4 group B fetuses, pleural effusions in 2 group A versus 0 group B, ascites
155                        Readers 1 and 2 found pleural effusions in 40 and 41 stage III and 20 and 21 s
156   Small-bore catheter drainage of persistent pleural effusions in lung transplant recipients is usual
157                            The prevalence of pleural effusions in the patients undergoing only CABG s
158 sion defects and elimination of lungs with a pleural effusion indicate that triple matches with PE (r
159 -expiratory pressure 10 benefit in reversing pleural effusion-induced de-recruitment.
160                                   Unilateral pleural effusion instillation (13 mL/kg), intra-abdomina
161 s a primary target organ with serosanguinous pleural effusions, intra-alveolar edema, and hemorrhagic
162 al hypertension (15 mm Hg), and simultaneous pleural effusion/intra abdominal hypertension.
163  analysis revealed that histology, malignant pleural effusion, intraparenchymal liver metastasis, and
164                                              Pleural effusion is a frequent finding in patients with
165       Treatment of symptomatic carcinomatous pleural effusions is primarily directed at local palliat
166 areas of consolidation, areas of cavitation, pleural effusions, linear opacities, septal thickening,
167                   Proliferative responses of pleural effusion lymphocytes were examined in response t
168 ned as a stable episome in latently infected pleural effusion lymphoma (PEL) cells.
169 and host-cell genomes of a latently infected pleural effusion lymphoma cell line BCBL1.
170                                           In pleural effusion lymphoma cell lines that express variab
171 s constitutively expressed in HHV-8-positive pleural effusion lymphoma cell lines.
172 rantly accumulated in latently KSHV-infected pleural effusion lymphoma cells and results in increased
173 transcription in the latently infected BCBL1 pleural effusion lymphoma-derived cell line.
174 svirus 8 (HHV-8) causes Kaposi's sarcoma and pleural effusion lymphoma.
175 ecreased intracellular bacterial survival in pleural-effusion macrophages of the guinea pigs infected
176                                        Large pleural effusions may develop in a small proportion of p
177                  The lymphoma presented with pleural effusions, mediastinal and cervical adenopathy,
178                                    Malignant pleural effusion (MPE) is the lethal consequence of vari
179                                    Malignant pleural effusion (MPE) poses a significant clinical prob
180 us pleural effusion (TPE, n = 50), malignant pleural effusion (MPE, n = 41), other cases including pn
181 quantified MCs in human and murine malignant pleural effusions (MPEs) and evaluated the fate and func
182 revealed a globular dilated heart, bilateral pleural effusions, myocyte apoptosis, and transmural myo
183 areas of hazy increased attenuation (n = 1), pleural effusion (n = 1), and areas of cavitation (n = 1
184 e included patients with pneumonia (n = 28), pleural effusion (n = 13), pleural empyema (n = 4), lung
185 l thickening (n = 7), consolidation (n = 5), pleural effusion (n = 4), and solitary endobronchial les
186 onal lymph node invasion (n = 8), ascites or pleural effusion (n = 7), and metastases to liver (n = 5
187                       Complications included pleural effusion (n = 7), pneumothorax (n = 2), pericard
188    Adverse events included procedure-related pleural effusion (n=2), cholecystitis (n=1), and additio
189                   For treatment of malignant pleural effusion, nonsteroidal anti-inflammatory drugs (
190      Perfusion defects associated with small pleural effusions (obliteration of the costophrenic angl
191             In conclusion, the prevalence of pleural effusions occupying more than 25% of the hemitho
192                                              Pleural effusions occur during dasatinib therapy, partic
193                                              Pleural effusion occurred in 27% of patients (grade 3 to
194                                              Pleural effusion occurred in 48 patients (35%; grade 3/4
195       One case of thrombosis and one case of pleural effusion occurred.
196  mouse xenograft (Sparky-X) from a malignant pleural effusion of a 12-year-old Persian male with auto
197 a population established in culture from the pleural effusion of a breast cancer patient showed disti
198         Pulmonary emboli are associated with pleural effusions of all sizes.
199 cancer cell lines in vitro, or isolated from pleural effusions of mesothelioma patients triggered dow
200 els of activity were seen with exosomes from pleural effusions of mesothelioma patients.
201               Cells isolated from ascites or pleural effusions of patients with metastatic breast can
202 ictor of anthrax was mediastinal widening or pleural effusion on a chest radiograph.
203 re (HR, 2.55; 95% CI, 1.15 to 5.63; P=0.02), pleural effusion on chest radiograph (HR, 2.56; 95% CI,
204 lking status, and ascites, moderate-to-large pleural effusion on CT images was significantly associat
205 reasing periaortic hematoma, and hemorrhagic pleural effusion on imaging identify patients with compl
206                            Moderate-to-large pleural effusion on preoperative CT images in patients w
207  hematocrit <30% (OR, 2.0; 95% CI, 1.3-3.2), pleural effusion on presenting chest x-ray (OR, 1.6; 95%
208 L/One hundred and twenty eight patients with pleural effusions on thoracic CT who underwent thoracent
209 hospital admissions for bronchitis (one) and pleural effusion (one), grade 3 increase in aminotransfe
210 Methods Patients with NSCLC (stage IIIB with pleural effusion or stage IV according to American Joint
211          Patients with stage IIIB NSCLC with pleural effusion or stage IV NSCLC, who had Eastern Coop
212 l of patients with advanced (stage IIIB with pleural effusion or stage IV) non-small-cell lung cancer
213               Patients with stage IIIB (with pleural effusion or supraclavicular nodes) to IV NSCLC a
214    Radiographs were assessed for presence of pleural effusions or lymphadenopathy.
215 uce a pleurodesis in patients with recurrent pleural effusions or pneumothorax should be investigated
216 chemotherapy-naive patients with stage IIIB (pleural effusion) or IV NSCLC, without restrictions by h
217 chemotherapy-naive patients with stage IIIB (pleural effusion) or stage IV NSCLC of any histology or
218 her findings (mediastinal edema, adenopathy, pleural effusion, or a sternal or lung abnormality).
219      Nodular opacities, reticular opacities, pleural effusion, or lymphadenopathy were not observed i
220        Patients with stage IIIB disease with pleural effusion, or stage IV NSCLC and Eastern Cooperat
221 ned or recurrent pain (P<0.0001), increasing pleural effusion (P=0.0003), and both the maximum diamet
222  0.009), pulmonary hypertension (P < 0.001), pleural effusions (P = 0.01), myositis (P = 0.02), and a
223  that regulate the pathogenesis of malignant pleural effusion (PE) associated with advanced stage of
224 milar effect, achieving control of malignant pleural effusion (pleurodesis).
225 riables studied were the occurrence of PPCs, pleural effusion, pneumonia, and pulmonary embolism.
226 s (mediastinal bleeding, pneumothoraces, and pleural effusions), postdischarge complications, reopera
227                                 These larger pleural effusions produce dyspnea but not chest pain or
228    There were 4 independent risk factors for pleural effusion: prolonged surgery (OR = 1), surgery on
229                    For dyspnea and malignant pleural effusions, providers should offer thoracentesis,
230  correlated significantly with the volume of pleural effusions (r = 0.79, p < 0.00001).
231  of confirmed or suspected mesothelioma with pleural effusion, recruited from 12 hospitals in the UK.
232 leural effusion (11.6% vs 26.4%, P = 0.003), pleural effusion requiring drainage (1.7% vs 9.9%, P = 0
233  = 49, 12%), incisional hernia (n = 22, 6%), pleural effusion requiring intervention (n = 21, 5%), ne
234 cantly (P<0.0001) associated with pneumonia, pleural effusions requiring drainage, and maximum postop
235 l gene transfer to cancer cells in malignant pleural effusions revealed that transduction was markedl
236  other signs (ie, halo sign, hypodense sign, pleural effusion, reversed halo sign) is unknown.
237 pted method for the treatment of symptomatic pleural effusions secondary to mesotheliomas.
238                         HH is a transudative pleural effusion seen in 5%-10% of cirrhosis patients, i
239  none), dyspnoea (six [2%] vs one [1%]), and pleural effusion (six [2%] vs none).
240  cancer were thrombocytopenia (eight [11%]), pleural effusion (six [8%]), and increased lipase (five
241     This study was conducted to determine if pleural effusion size affects ventilation/perfusion (V/Q
242 l lung scan interpretive criteria based upon pleural effusion size is not warranted.
243           Bedside US can be used to detect a pleural effusion so well in critically ill patients that
244                                        Large pleural effusions sometimes occur after coronary artery
245 ine (SUM-52PE), originating from a malignant pleural effusion specimen, that can be cultured under se
246 assays and biochemical analyses of malignant pleural effusion specimens revealed CS in relevant conce
247                 Subcutaneous edema, ascites, pleural effusion, splenomegaly, varices, portal venous t
248 ociations among themselves and with ascites, pleural effusions, subcutaneous edema, and low mean seru
249 ion of the heart, increased lung weight, and pleural effusion, suggesting that they died of congestiv
250 B but increased risk of rapid progression to pleural effusion TB.
251                                              Pleural effusions that occupied more than 25% of the hem
252 sed included noncalcified pulmonary nodules, pleural effusion, thoracic duct dilatation, hepatic and
253 the selected conditions ranged from 0.22 for pleural effusion to 0.0004 for tension pneumothorax.
254 ndwelling pleural catheter was placed in the pleural effusion to drain the fluid fully.
255  We enrolled 91 cases, including tuberculous pleural effusion (TPE, n = 50), malignant pleural effusi
256 ned in 31 lung transplant recipients who had pleural effusions treated with catheter drainage were re
257 , and information on pleural fluid findings, pleural effusion treatment, and cardiac surgery was obta
258 imary malignancy and a symptomatic malignant pleural effusion underwent small-bore-catheter thoracost
259                                              Pleural effusion volume was determined on each CT scan s
260 d causing medium lobe atelectasis; bilateral pleural effusion was also present.
261                                              Pleural effusion was an expected and problematic toxicit
262                  Pneumococcal pneumonia with pleural effusion was associated with a limited number of
263 rsus 30%) were more prevalent with imatinib; pleural effusion was more common with dasatinib (17% ver
264  functional residual capacity by 368 mL when pleural effusion was present and by 184 mL when intra-ab
265                                              Pleural effusion was present at some point during the co
266                                              Pleural effusion was present on radiographs in seven pat
267 ents identified in either treatment arm, and pleural effusion was the only drug-related, nonhematolog
268  or absence of parenchymal abnormalities and pleural effusions was noted.
269  loss and septal thickening, with or without pleural effusion, was never seen in the absence of acute
270 us pneumoniae responsible for pneumonia with pleural effusion, we determined the capsular polysacchar
271             Among hospitalized patients with pleural effusions, we identified subgroup of patients in
272                         Patients with higher pleural effusion were older (62+/-16 yr vs. 54+/-17 yr,
273                         First occurrences of pleural effusion were reported with dasatinib, with the
274                                              Pleural effusions were classified as exudates or transud
275                    Mediastinal adenopathy or pleural effusions were classified as typical of recent i
276                        Orthotopic tumors and pleural effusions were clearly visualized at MR imaging
277 secutive patients with symptomatic malignant pleural effusions were enrolled.
278 tocytologically proven symptomatic malignant pleural effusions were included in this phase I toxicity
279                             The incidence of pleural effusions were not significantly different betwe
280                                              Pleural effusions were present in one-half of the patien
281 h cytologically negative suspected malignant pleural effusions were recruited.
282                                              Pleural effusions were seen in 16 (57%) patients with PE
283          However, mediastinal adenopathy and pleural effusions were significantly more common in HIV-
284                Thirty three (26%) of the 128 pleural effusions were transudates and 95 (74%) were exu
285 and asthenia (40 [5%] vs 23 [3%]); grade 3-4 pleural effusions were uncommon (ten [1%] vs three [<1%]
286 er radiologic findings (multiple nodules and pleural effusion) were less frequent, but appeared later
287 nia in children is frequently complicated by pleural effusions, which rarely progress to empyema.
288  (1:1) for which 106 patients with malignant pleural effusion who had not previously undergone pleuro
289                           The association of pleural effusion with other conditions mirrored that in
290 o of right to left lung mass, association of pleural effusion with other conditions, and frequency of
291   Larger doses of TGF-beta(2) induced larger pleural effusions with relatively low pleural fluid WBC
292  tomography (CT) findings for characterizing pleural effusions with the use of attenuation values.

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