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1 PEL, which arises in a hypoxic environment (pleural effusions).
2 nical characters, such as refractoriness and pleural effusion.
3 peutic strategy for the control of malignant pleural effusion.
4 e II (n = 88) studies for the development of pleural effusion.
5 schedule may result in a higher incidence of pleural effusion.
6 patient irritability, small pericardial +/- pleural effusion.
7 o be secondary to a decrease or absence of a pleural effusion.
8 they showed any mismatched V/Q defect or any pleural effusion.
9 l or reticulonodular pattern with or without pleural effusion.
10 graphy for PE with radiographic evidence for pleural effusion.
11 t upper lobe mass along with a moderate-size pleural effusion.
12 diffuse interstitial pattern with or without pleural effusion.
13 owed mild pulmonary edema with a small right pleural effusion.
14 rentially diagnose tuberculous and malignant pleural effusion.
15 aphy showed an enlarged heart with bilateral pleural effusion.
16 ostoperative AF or postoperative pericardial/pleural effusion.
17 nts were postoperative AF and pericardial or pleural effusion.
18 esis is performed to identify the cause of a pleural effusion.
19 ractory MPP children or in MPP children with pleural effusion.
20 s. 19+/-6 bpm, p<0.01) than those with lower pleural effusion.
21 anulomas as well as ascites and a left-sided pleural effusion.
22 tein, release of inflammatory cytokines, and pleural effusion.
23 xudative pleural effusions from transudative pleural effusions.
24 cy and the presence, size, and laterality of pleural effusions.
25 e may reduce the incidence of pneumonia with pleural effusions.
26 ed as factors associated with development of pleural effusions.
27 s Fontan morbidity, particularly duration of pleural effusions.
28 ain the prominent and persistent hemorrhagic pleural effusions.
29 ne, it induced transient production of large pleural effusions.
30 ty, thickened bronchial walls, and bilateral pleural effusions.
31 loped low cardiac output, and 3 patients had pleural effusions.
32 rodesis was successful in treating malignant pleural effusions.
33 evaluated; (c) unsuspected pneumonia; or (d) pleural effusions.
34 lycans mimicked the inhibition observed with pleural effusions.
35 hymal opacities, cavitation, adenopathy, and pleural effusions.
36 onventional inpatient treatment of malignant pleural effusions.
37 all thickening, nodules, Kerley B lines, and pleural effusions.
38 eft pulmonary artery thrombosis, and chronic pleural effusions.
39 ntional T2-weighted sequences revealed small pleural effusions.
40 airment were more likely to have significant pleural effusions.
41 PCs (13.2% vs 40.5%, P < 0.001), symptomatic pleural effusion (11.6% vs 26.4%, P = 0.003), pleural ef
42 -up data were available for 31 of 39 treated pleural effusions: 11 (35%) had CR, 18 (58%) had PR, and
43 ), thoracic duct dilatation (4% vs 0, P=.3), pleural effusion (12% vs 6%, P=.2), or ascites (10% vs 6
44 (20%), intrathoracic lymphadenopathy (16%), pleural effusion (12%), reticular infiltration (4%), and
46 ophic scar formation (35), atelectasis (12), pleural effusion (13), recurrent sternal depression (5),
47 %), oral ulcers (24.2%), joint pain (23.0%), pleural effusion (16.5%) and increase in abdominal girth
48 rapy was most effective in the resolution of pleural effusions (2 of 2), ascites (6 of 8), and hydrop
52 ily resulted in significantly lower rates of pleural effusion (all grades, 7% v 16%; P = .024) and gr
53 y-proven PE, 77 (47%) had at least one large pleural effusion and 86 (53%) had a small effusion; 33 (
54 ome features was strong (eg, alpha = .78 for pleural effusion and ascites) but was lower for others (
55 ded the use of ultrasonography for ruling-in pleural effusion and assisting its drainage, ascites dra
56 ersistent fever caused by pneumonia (n = 2), pleural effusion and atelectasis (n = 1), or liver absce
57 adiologically as interstitial pneumonia with pleural effusion and clinically as hypoxemic respiratory
59 th dasatinib compared with imatinib, whereas pleural effusion and grade 3/4 thrombocytopenia were mor
60 ion in lung injury, as assessed by volume of pleural effusion and histological analyses (significant
63 unrelated to alectinib: acute renal failure; pleural effusion and pericardial effusion; and brain met
65 urs in mouse models of malignant lung cancer pleural effusion and spontaneous colon cancer metastasis
66 lations of leukocytes and malignant cells in pleural effusions and accurately predict disease state i
69 n of peritoneal spread, presence and size of pleural effusions and ascites, lymphadenopathy, and dist
71 nt has altered the traditional management of pleural effusions and empyema in children, resulting in
72 tered to improve the drainage of complicated pleural effusions and empyemas and it is the most effect
76 gnificantly post antibiotic treatment in the pleural effusions and pleural macrophages up-regulated m
77 by progression of respiratory insufficiency, pleural effusions and pulmonary edema, and, ultimately,
78 s observed, in the other one, pneumonia with pleural effusion, and as a consequence of inflammatory i
79 primary tumor, pleural metastases, malignant pleural effusion, and ascites obtained during disease pr
81 ata suggest a strong impact of PCV13 on CAP, pleural effusion, and documented pneumococcal pneumonia,
84 sults suggest that drainage of the malignant pleural effusion, and perhaps enzymatic pretreatment of
85 pic support, thoracostomy tube drainage of a pleural effusion, and prolonged antimicrobial therapy.
87 required no further treatment), two cases of pleural effusion, and two cases of moderate pain (one ca
89 y, and most animals also developed leukemia, pleural effusion, and, in some cases, ascites associated
92 samples (tumor and normal pleura), malignant pleural effusions, and in established mesothelioma cell
93 of thiourea, W/D increased to 5.5 and marked pleural effusions appeared, but there were no difference
97 heir presence in urine, chylous ascites, and pleural effusions are consistent with their ability to m
99 ts corresponding to radiographically-evident pleural effusions are of intermediate probability for PE
100 nvolvement-thoracic duct dilatation, chylous pleural effusion, ascites, and LALM-is more common in LA
101 that increased gall bladder wall thickness, pleural effusion, ascites, hepatomegaly, and splenomegal
102 ngs included enlarged abdominal lymph nodes, pleural effusions, ascites, and dilatation of the thorac
103 estigated the risk factors and management of pleural effusion associated with dasatinib therapy for c
104 sion may be instrumental to the treatment of pleural effusion-associated lung restriction and cyclica
105 was designed to determine the prevalence of pleural effusion at approximately 28 days after cardiac
106 ion, but 1 patient developed pericardial and pleural effusion attributed to pericardial instrumentati
110 rom 41.3% to 29.7% (P < .001), the number of pleural effusion cases decreased by 53% (167 to 79; P <
111 e believe that this is the first report of a pleural effusion caused by HSV II, and suggest that this
115 ntestinal and/or pulmonary lymphangiectasia, pleural effusions, chylothoraces and/or pericardial effu
118 -2.2% to 17.6%) or postoperative pericardial/pleural effusion (colchicine, 103 patients [57.2%]; plac
119 phosphorylation in MT cells, derived from a pleural effusion, compared with cells from the primary t
120 adiograph revealed hilar adenopathy and left pleural effusion; computed tomography scan showed a 2-cm
121 s predictive of CT outcome, although lack of pleural effusion correlated with clinical stability.
122 investigated whether fibulin-3 in plasma and pleural effusions could meet sensitivity and specificity
123 In cytology-negative suspected malignant pleural effusions, CT-guided pleural biopsy is a better
126 ever, higher morbidity of tachypnea/dyspnea, pleural effusion, diarrhea, hepatosplenomegaly, consciou
127 )CD16(-) NK cells isolated from inflammatory pleural effusions display a potent regulatory activity.
128 to improve overall survival in patients with pleural effusion due to malignant pleural mesothelioma,
130 ion defect(s), and (g) perfusion defect from pleural effusion equal to one third or more of the pleur
131 ential diagnosis of an unexplained exudative pleural effusion, especially in an immunocompromised hos
134 s immunosuppressive therapy, who had a large pleural effusion, found on a chest radiograph during the
136 Organ-specific pleural complications include pleural effusion from hepatic venoocclusive disease, spo
137 after bone marrow transplantation, and early pleural effusion from urinothorax and late effusion from
138 d we also detected CXCL12-gamma in malignant pleural effusions from patients with breast cancer.
139 values can help in differentiating exudative pleural effusions from transudative pleural effusions.
140 -172 mL vs. 80+/-87 mL, p<0.0001, for higher pleural effusion group vs. lower pleural effusion group)
141 /-124 mL vs. 23+/-29 mL, p<0.0001 for higher pleural effusion group vs. lower pleural effusion group,
142 for higher pleural effusion group vs. lower pleural effusion group) was greater than the estimated l
144 Patients were divided into higher and lower pleural effusion groups according to the median value (2
145 s following induction of an antigen-specific pleural effusion, guinea pigs were injected intrapleural
148 ommonly seen pulmonary complications include pleural effusion, hemothorax, atelectasis, pulmonary ede
149 n 480 (39.5%) patients including symptomatic pleural effusion in 366 (30.1%) patients, respiratory in
150 63 patients, CT demonstrated an increase in pleural effusion in 46 (73%), periaortic changes in 21 (
151 d JIMT-1 breast cancer cells, derived from a pleural effusion in a patient who displayed clinical res
154 ions in 13 group A versus 4 group B fetuses, pleural effusions in 2 group A versus 0 group B, ascites
156 Small-bore catheter drainage of persistent pleural effusions in lung transplant recipients is usual
158 sion defects and elimination of lungs with a pleural effusion indicate that triple matches with PE (r
161 s a primary target organ with serosanguinous pleural effusions, intra-alveolar edema, and hemorrhagic
163 analysis revealed that histology, malignant pleural effusion, intraparenchymal liver metastasis, and
166 areas of consolidation, areas of cavitation, pleural effusions, linear opacities, septal thickening,
172 rantly accumulated in latently KSHV-infected pleural effusion lymphoma cells and results in increased
175 ecreased intracellular bacterial survival in pleural-effusion macrophages of the guinea pigs infected
180 us pleural effusion (TPE, n = 50), malignant pleural effusion (MPE, n = 41), other cases including pn
181 quantified MCs in human and murine malignant pleural effusions (MPEs) and evaluated the fate and func
182 revealed a globular dilated heart, bilateral pleural effusions, myocyte apoptosis, and transmural myo
183 areas of hazy increased attenuation (n = 1), pleural effusion (n = 1), and areas of cavitation (n = 1
184 e included patients with pneumonia (n = 28), pleural effusion (n = 13), pleural empyema (n = 4), lung
185 l thickening (n = 7), consolidation (n = 5), pleural effusion (n = 4), and solitary endobronchial les
186 onal lymph node invasion (n = 8), ascites or pleural effusion (n = 7), and metastases to liver (n = 5
188 Adverse events included procedure-related pleural effusion (n=2), cholecystitis (n=1), and additio
190 Perfusion defects associated with small pleural effusions (obliteration of the costophrenic angl
196 mouse xenograft (Sparky-X) from a malignant pleural effusion of a 12-year-old Persian male with auto
197 a population established in culture from the pleural effusion of a breast cancer patient showed disti
199 cancer cell lines in vitro, or isolated from pleural effusions of mesothelioma patients triggered dow
203 re (HR, 2.55; 95% CI, 1.15 to 5.63; P=0.02), pleural effusion on chest radiograph (HR, 2.56; 95% CI,
204 lking status, and ascites, moderate-to-large pleural effusion on CT images was significantly associat
205 reasing periaortic hematoma, and hemorrhagic pleural effusion on imaging identify patients with compl
207 hematocrit <30% (OR, 2.0; 95% CI, 1.3-3.2), pleural effusion on presenting chest x-ray (OR, 1.6; 95%
208 L/One hundred and twenty eight patients with pleural effusions on thoracic CT who underwent thoracent
209 hospital admissions for bronchitis (one) and pleural effusion (one), grade 3 increase in aminotransfe
210 Methods Patients with NSCLC (stage IIIB with pleural effusion or stage IV according to American Joint
212 l of patients with advanced (stage IIIB with pleural effusion or stage IV) non-small-cell lung cancer
215 uce a pleurodesis in patients with recurrent pleural effusions or pneumothorax should be investigated
216 chemotherapy-naive patients with stage IIIB (pleural effusion) or IV NSCLC, without restrictions by h
217 chemotherapy-naive patients with stage IIIB (pleural effusion) or stage IV NSCLC of any histology or
218 her findings (mediastinal edema, adenopathy, pleural effusion, or a sternal or lung abnormality).
219 Nodular opacities, reticular opacities, pleural effusion, or lymphadenopathy were not observed i
221 ned or recurrent pain (P<0.0001), increasing pleural effusion (P=0.0003), and both the maximum diamet
222 0.009), pulmonary hypertension (P < 0.001), pleural effusions (P = 0.01), myositis (P = 0.02), and a
223 that regulate the pathogenesis of malignant pleural effusion (PE) associated with advanced stage of
225 riables studied were the occurrence of PPCs, pleural effusion, pneumonia, and pulmonary embolism.
226 s (mediastinal bleeding, pneumothoraces, and pleural effusions), postdischarge complications, reopera
228 There were 4 independent risk factors for pleural effusion: prolonged surgery (OR = 1), surgery on
231 of confirmed or suspected mesothelioma with pleural effusion, recruited from 12 hospitals in the UK.
232 leural effusion (11.6% vs 26.4%, P = 0.003), pleural effusion requiring drainage (1.7% vs 9.9%, P = 0
233 = 49, 12%), incisional hernia (n = 22, 6%), pleural effusion requiring intervention (n = 21, 5%), ne
234 cantly (P<0.0001) associated with pneumonia, pleural effusions requiring drainage, and maximum postop
235 l gene transfer to cancer cells in malignant pleural effusions revealed that transduction was markedl
240 cancer were thrombocytopenia (eight [11%]), pleural effusion (six [8%]), and increased lipase (five
241 This study was conducted to determine if pleural effusion size affects ventilation/perfusion (V/Q
245 ine (SUM-52PE), originating from a malignant pleural effusion specimen, that can be cultured under se
246 assays and biochemical analyses of malignant pleural effusion specimens revealed CS in relevant conce
248 ociations among themselves and with ascites, pleural effusions, subcutaneous edema, and low mean seru
249 ion of the heart, increased lung weight, and pleural effusion, suggesting that they died of congestiv
252 sed included noncalcified pulmonary nodules, pleural effusion, thoracic duct dilatation, hepatic and
253 the selected conditions ranged from 0.22 for pleural effusion to 0.0004 for tension pneumothorax.
255 We enrolled 91 cases, including tuberculous pleural effusion (TPE, n = 50), malignant pleural effusi
256 ned in 31 lung transplant recipients who had pleural effusions treated with catheter drainage were re
257 , and information on pleural fluid findings, pleural effusion treatment, and cardiac surgery was obta
258 imary malignancy and a symptomatic malignant pleural effusion underwent small-bore-catheter thoracost
263 rsus 30%) were more prevalent with imatinib; pleural effusion was more common with dasatinib (17% ver
264 functional residual capacity by 368 mL when pleural effusion was present and by 184 mL when intra-ab
267 ents identified in either treatment arm, and pleural effusion was the only drug-related, nonhematolog
269 loss and septal thickening, with or without pleural effusion, was never seen in the absence of acute
270 us pneumoniae responsible for pneumonia with pleural effusion, we determined the capsular polysacchar
278 tocytologically proven symptomatic malignant pleural effusions were included in this phase I toxicity
285 and asthenia (40 [5%] vs 23 [3%]); grade 3-4 pleural effusions were uncommon (ten [1%] vs three [<1%]
286 er radiologic findings (multiple nodules and pleural effusion) were less frequent, but appeared later
287 nia in children is frequently complicated by pleural effusions, which rarely progress to empyema.
288 (1:1) for which 106 patients with malignant pleural effusion who had not previously undergone pleuro
290 o of right to left lung mass, association of pleural effusion with other conditions, and frequency of
291 Larger doses of TGF-beta(2) induced larger pleural effusions with relatively low pleural fluid WBC
292 tomography (CT) findings for characterizing pleural effusions with the use of attenuation values.
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