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   1 therapy for patients with advanced malignant pleural mesothelioma.                                   
     2  talc pleurodesis in patients with malignant pleural mesothelioma.                                   
     3 ed 68 with advanced ASS1-deficient malignant pleural mesothelioma.                                   
     4 os and silica seemed to increase the risk of pleural mesothelioma.                                   
     5 e treatment of recurrent and/or unresectable pleural mesothelioma.                                   
     6  best available serum biomarker of malignant pleural mesothelioma.                                   
     7 rol groups and 1,026 patients with malignant pleural mesothelioma.                                   
     8 e of action for asbestos in the induction of pleural mesothelioma.                                   
     9 modality therapy in stage I to III malignant pleural mesothelioma.                                   
    10 apeutic option in the treatment of malignant pleural mesothelioma.                                   
    11 estos and 76 patients with surgically staged pleural mesothelioma.                                   
    12 ty in patients with PD-L1-positive malignant pleural mesothelioma.                                   
    13 ng of the tumor cells in 36 of 38 samples of pleural mesothelioma.                                   
    14 rom those with exposure to asbestos who have pleural mesothelioma.                                   
    15 h cisplatin alone in patients with malignant pleural mesothelioma.                                   
    16 on in patients with ASS1-deficient malignant pleural mesothelioma.                                   
    17 f disease in patients with diffuse malignant pleural mesothelioma.                                   
    18 in the palliation of patients with malignant pleural mesothelioma.                                   
    19 d were significantly higher in patients with pleural mesothelioma (105+/-7 ng per milliliter in the D
    20 s were significantly higher in patients with pleural mesothelioma (694+/-37 ng per milliliter in the 
  
    22  investigated the presence of osteopontin in pleural mesothelioma and determined serum osteopontin le
    23  Patients with measurable advanced malignant pleural mesothelioma and disease progression after one o
    24 dality therapy in the treatment of malignant pleural mesothelioma and identify prognostic factors.   
    25 h cisplatin is approved for the treatment of pleural mesothelioma and is active in malignant peritone
  
    27 ins contribute to the malignant phenotype of pleural mesotheliomas and indicate that interventions de
    28  has been reported in varying proportions of pleural mesotheliomas and other tumours, but data are co
    29 ients with pleural effusion due to malignant pleural mesothelioma, and talc pleurodesis might be pref
  
    31     The successful treatment of unresectable pleural mesothelioma awaits the discovery of active drug
    32 type I IGF receptor in a subset of malignant pleural mesothelioma cell lines and determined the corre
  
    34 results are in favor of an increased risk of pleural mesothelioma for subjects exposed to both asbest
  
    36 d quality of life in patients with malignant pleural mesothelioma have, to our knowledge, not been as
  
    38 d Affymetrix U133A microarray analysis on 99 pleural mesotheliomas, in which multivariate analysis sh
  
    40  literature of clinical studies in malignant pleural mesothelioma, including phase II trials of the n
  
  
  
  
  
    46 sed immunotherapy in patients with malignant pleural mesothelioma is feasible, well-tolerated, and ca
  
  
    49 eadily detected in genomic DNA isolated from pleural mesothelioma lines; however, levels of SV40 T/t 
  
  
    52  malignant mesothelial tissues and malignant pleural mesothelioma (MPM) cell lines as compared to ben
  
  
  
  
  
  
  
  
  
  
  
  
  
  
    67 ed in patients with ASS1-deficient malignant pleural mesothelioma (MPM) or non-small-cell lung cancer
  
  
    70 tor-1 (PAR1, F2R) on the growth of malignant pleural mesothelioma (MPM), using human MPM cells that l
  
  
  
  
  
    76 rgoing cancer-directed surgery for malignant pleural mesothelioma (MPM); however, it is unclear if th
    77 e pathological distinction between malignant pleural mesothelioma (MPM)and adenocarcinoma (ADCA) of t
    78 en shown to be highly expressed in malignant pleural mesotheliomas (MPM), was detected in serum using
  
    80 ns and expression levels unique to malignant pleural mesotheliomas (MPMs) and not present in control 
  
    82 oncoproteins have been detected in malignant pleural mesotheliomas (MPMs), their role in the pathogen
  
  
    85 ria In Solid Tumors (RECIST) version 1.0 for pleural mesothelioma or RECIST version 1.1 for peritonea
  
    87 ocedure-tract metastases (PTMs) in malignant pleural mesothelioma remains controversial, and clinical
  
    89 ur previous microarray analysis of malignant pleural mesothelioma revealed alterations in components 
  
    91  were significantly higher in the group with pleural mesothelioma than in the group with exposure to 
    92  system components represent novel malignant pleural mesothelioma therapeutic targets for investigati
    93 port on their use of a murine model of human pleural mesothelioma to explore potential factors that l
    94  benefit reported in patients with malignant pleural mesothelioma treated in a phase 1 study of vorin
  
    96  (18)F-FDG-CI in the assessment of malignant pleural mesothelioma using histopathology as the gold st
  
    98 atients with histologically proven malignant pleural mesothelioma were enrolled (26 male patients and
    99 eated patients with PD-L1-positive malignant pleural mesothelioma were enrolled from 13 centres in si
  
   101 ears; 10 women and 29 men) with unresectable pleural mesothelioma were treated with repetitive transa
  
   103 spite several attempts at treating malignant pleural mesothelioma with various modalities, mortality 
  
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