ライフサイエンスコーパス: plexiform

1 ial hypertension (PAH) that recapitulate the plexiform and obliterative arteriopathy seen in PAH pati

2 aration of neurosensory retina between outer plexiform and outer nuclear layers.

3 dian manner, in interneurons in the external plexiform and periglomerular layers, whereas VPAC2R is e

4 hereas STIM2 is mainly confined to the outer plexiform and RGC layers.

5 l layers (nerve fiber, ganglion cells, inner plexiform, and inner nuclear layers) of eyes with previo

6 ipheral displacement of ganglion cell, inner plexiform, and inner nuclear layers.

7 he retinal nerve fiber, ganglion cell, inner plexiform, and outer plexiform layers and increased thic

8 ciently reaching the inner nuclear and outer plexiform, and to a lesser extent the outer nuclear laye

9 oliferation and selection of a proliferative/plexiform EC phenotype.

10 inner plexiform layer (GCL+IPL), RNFL, outer plexiform/inner nuclear layers (OPL+INL), and outer nucl

11 0.25 mum/y) and the ganglion cell (GC)/inner plexiform layer (0.29 mum/y) on optical coherence tomogr

12 lls that terminate in stratum 3 of the inner plexiform layer (DB4) express more Ret-PCP2 than those t

13 including both glomerular layer and external plexiform layer (EPL) computations and incorporating bot

14 We found that, whereas external plexiform layer (EPL) interneurons show broadly distribu

15 , including granule cells (GCs) and external plexiform layer (EPL) interneurons.

16 odendritic synaptic circuits in the external plexiform layer (EPL).

17 f the perifoveal retinal ganglion cell-inner plexiform layer (GC-IPL) and the peripapillary retinal n

18 e fiber layer (RNFL) and ganglion cell-inner plexiform layer (GC-IPL) of patients with DOA were evalu

19 Rates of ganglion cell + inner plexiform layer (GCIP) and whole-brain (r = 0.45; p < 0.

20 AAs (p = 0.047), whereas ganglion cell/inner plexiform layer (GCIP) thickness did not differ by race.

21 nd that of the ganglion cell layer and inner plexiform layer (GCIP, -11.3 mum), whereas the thickness

22 The common ganglion cell and inner plexiform layer (GCIPL) and inner nuclear layer (INL) vo

23 NFL) and macular retinal ganglion cell-inner plexiform layer (GCIPL) change over time in healthy and

24 of the macular ganglion cell layer and inner plexiform layer (GCIPL) was -16.42 mum (-19.23 to -13.60

25 n cell complex (GCC) and ganglion cell inner plexiform layer (GCIPL), with the accuracy of RNFL param

26 of the retinal ganglion cell layer and inner plexiform layer (GCL + IPL).

27 Thicknesses of the ganglion cell layer/inner plexiform layer (GCL+IPL), RNFL, outer plexiform/inner n

28 layer (GCL) (nasally and temporally), inner plexiform layer (IPL) (nasally), outer nuclear layer (ON

29 with ONHD had a significantly thinner inner plexiform layer (IPL) (P = 0.02), nerve fiber layer (P =

30 es stratify at different levels in the inner plexiform layer (IPL) and can interact with costratifyin

31 apses in the innermost ON layer of the inner plexiform layer (IPL) and from dopaminergic amacrine cel

32 rve fiber layer (NFL), and also in the inner plexiform layer (IPL) and inner nuclear layer (INL).

33 o a discrete synaptic layer called the inner plexiform layer (IPL) and only rarely extend processes i

34 The location of the inner plexiform layer (IPL) and outer plexiform layer (OPL) wa

35 from bipolar and amacrine cells in the inner plexiform layer (IPL) and send information to the brain

36 urite targeting defects in the retinal inner plexiform layer (IPL) and tectal neuropil.

37 nal amacrine cells migrate towards the inner plexiform layer (IPL) and then retract their trailing pr

38 branching in the outermost part of the inner plexiform layer (IPL) and weakly melanopsin-positive M2

39 t dACs send processes into the forming inner plexiform layer (IPL) before migrating through it and in

40 he division between the ON and the OFF inner plexiform layer (IPL) is not structurally absolute.

41 In the inner plexiform layer (IPL) of the mouse retina, ~70 neuronal

42 te arbors and form synapses within the inner plexiform layer (IPL) of the vertebrate retina.

43 ls ramifying between 0% and 30% of the inner plexiform layer (IPL) receive mixed inputs from rods and

44 upregulated in regrowing RGC axons and inner plexiform layer (IPL) synapses, respectively.

45 cific arbor specializations within the inner plexiform layer (IPL) that occur consistently at defined

46 dor columns through synapses in the internal plexiform layer (IPL) to produce an intrabulbar map.

47 scleral half or "Off" sublamina of the inner plexiform layer (IPL) undergo the greatest changes, wher

48 e combined nerve fiber layer (NFL) and inner plexiform layer (IPL) were manually segmented and thickn

49 form functional neural circuits in the inner plexiform layer (IPL), a laminar region that is conventi

50 In the distal 80% of the inner plexiform layer (IPL), dense GC dendrites coexisted with

51 SAC, found at the outer border of the inner plexiform layer (IPL), forms a synaptic subband "a" with

52 cally branched into sublamina a of the inner plexiform layer (IPL), i.e., the OFF inner plexiform sub

53 on the dendritic stratification in the inner plexiform layer (IPL), those monostratified in the Off s

54 l axon terminals in sublamina-b of the inner plexiform layer (IPL), we investigated the possibility t

55 regation of ON and OFF pathways in the inner plexiform layer (IPL), where glutamate is released from

56 nals in the innermost sublamina of the inner plexiform layer (IPL), which is typical for mammals.

57 ; (4) restricted lamination within the inner plexiform layer (IPL), which renders J-RGCs responsive t

58 odied as separate strata that span the inner plexiform layer (IPL).

59 ple retinal neuron subtypes within the inner plexiform layer (IPL).

60 mified in strata 1, 3, 4, and 5 of the inner plexiform layer (IPL).

61 the inner or the outer portion of the inner plexiform layer (IPL).

62 ayering, or lamination, of the retinal inner plexiform layer (IPL).

63 to the outer plexiform layer (OPL) and inner plexiform layer (IPL); the beta(3) subunit was localized

64 parameters and the ganglion cell layer-inner plexiform layer (mGCL-IPL) was determined by combining t

65 ar ganglion cell layer (mGCL), macular inner plexiform layer (mIPL), macular inner nuclear layer (mIN

66 ar inner nuclear layer (mINL), macular outer plexiform layer (mOPL), macular outer nuclear layer (mON

67 ng at the ganglion cell layer (n = 1), outer plexiform layer (n = 4), outer nuclear layer (n = 12), o

68 lion cell layer (NFL/GCL), NFL/GCL and inner plexiform layer (NFL/GCL + IPL), and total retina thickn

69 ment (OS) and outer nuclear layer plus outer plexiform layer (ONL+) thicknesses fell below the 95% co

70 nesses of the outer nuclear layer plus outer plexiform layer (ONL+), outer segment (OS), and retinal

71 S) layer, the outer nuclear layer plus outer plexiform layer (ONL+), the retinal pigment epithelium p

72 er ganglion cell layer (P = 0.003) and outer plexiform layer (OPL) (P < 0.001) compared with controls

73 pointing toward the inner limit of the outer plexiform layer (OPL) adjacent to the margin between the

74 hese areas included: subsidence of the outer plexiform layer (OPL) and inner nuclear layer (INL), and

75 ; the beta(2) subunit localized to the outer plexiform layer (OPL) and inner plexiform layer (IPL); t

76 es of HC axons fail to stratify in the outer plexiform layer (OPL) and invade the outer nuclear layer

77 l and optical property features of the outer plexiform layer (OPL) and the complex formed by the gang

78 n above (type 1) or below (type 2) the outer plexiform layer (OPL) at 6 tertiary referral centers.

79 correlating confirmed expansion of the outer plexiform layer (OPL) by optical coherence tomography (O

80 or terminals are ensheathed within the outer plexiform layer (OPL) by the processes of one type of gl

81 tinal layers limited externally by the outer plexiform layer (OPL) in 15 eyes (93.7%).

82 receptors mediating this action in the outer plexiform layer (OPL) is not clear.

83 is localized primarily throughout the outer plexiform layer (OPL) of the distal retina, a synaptic l

84 l confined to the inner portion of the outer plexiform layer (OPL) on PD-OCT.

85 drites (ORDs) either ramify within the outer plexiform layer (OPL) or the inner nuclear layer, and wh

86 of the inner plexiform layer (IPL) and outer plexiform layer (OPL) was identified at each age, and it

87 ribbon synapses established within the outer plexiform layer (OPL), initiating retinal visual process

88 body, near the distal boundary of the outer plexiform layer (OPL), suggesting that apical synapses a

89 tina expresses several laminins in the outer plexiform layer (OPL), where they may provide an extrace

90 n the organization and assembly of the outer plexiform layer (OPL).

91 (ELM), outer nuclear layer (ONL), and outer plexiform layer (OPL).

92 orm a third independent network in the outer plexiform layer (OPL).

93 the inner nuclear layer (INL), and the outer plexiform layer (OPL).

94 ral macular retinal ganglion cell plus inner plexiform layer (RGC+IPL) loss identified by spectral-do

95 nglion cell layer (I3 and N6 sectors), inner plexiform layer (S6 and N6 sectors), inner nuclear layer

96 nuclear layer (T6 and N6 sectors), and outer plexiform layer (S6 sector), as well as the overall reti

97 ed by OB neurons in the superficial external plexiform layer (sEPL) and glomerular layer (GL).

98 rom the retinal ganglion cell layer to outer plexiform layer (standardized beta = 0.657 to 0.777, all

99 us inner plexiform layer, the INL plus outer plexiform layer (the combined thickness of these layers

100 cular (including the ganglion cell and inner plexiform layer [GCIPL], inner retina [IR], outer retina

101 l of their axon terminal system in the inner plexiform layer and in immunoreactivity for recoverin an

102 while the abnormal hyperreflectance of outer plexiform layer and inner nuclear layer on spectral-doma

103 estored; however, the thickness of the inner plexiform layer and one measure of axon branching were s

104 parameters, such as the ganglion cell inner plexiform layer and optic nerve head parameters, also ar

105 lls arborized at various levels of the inner plexiform layer and over fields of different diameters,

106 g: rods retracted their axons from the outer plexiform layer and partially degenerated, whereas cones

107 Cone pedicles remained in the outer plexiform layer and preserved synaptic contacts with OFF

108 ition of an RGC's dendrites within the inner plexiform layer and that of its axon within the retinore

109 stance between the outer border of the outer plexiform layer and the inner border of the ellipsoid zo

110 er and immunoreactive processes in the inner plexiform layer and the outer plexiform layer.

111 es depolarize TH cell dendrites in the inner plexiform layer and these depolarizations propagate to t

112 Although the inner plexiform layer appears earlier than the outer plexiform

113 ition, the synaptic connections in the outer plexiform layer are defective in Oc1-null mice, and phot

114 er, the synaptic mechanisms within the inner plexiform layer are not well characterized within specif

115 ression and synaptic structures in the outer plexiform layer are preserved, and visual responses are

116 e attributed to the disorganization of inner plexiform layer cells that occurs in the Dscam mutant re

117 drant, and a thinner ganglion cell and inner plexiform layer complex (GCL-IPL).

118 photoreceptor axons, which changed the outer plexiform layer from a thin sheet of synaptic pedicles i

119 cted to specific sublaminae within the inner plexiform layer in adulthood, but acquire their restrict

120 of significantly thicker GCL, IPL, and outer plexiform layer in the central retinal area (i.e., fovea

121 zed to the outer nuclear layer and the outer plexiform layer in the CNGB3(-/-) retina.

122 ed thickness of the ON sublayer of the inner plexiform layer in the microbat retina, more ON than OFF

123 irregularity (18%), outer nuclear and outer plexiform layer irregularity (8%), and inner nuclear lay

124 that Gbeta5S expression in the retinal outer plexiform layer is eliminated, as is the ERG b-wave.

125 suggests that the organization of the outer plexiform layer is more complex than classically thought

126 ing from the inner nuclear layer (INL)/outer plexiform layer junction to involve the full-thickness I

127 Average and quadrant ganglion cell-inner plexiform layer measures demonstrated CVs </=4.5% with e

128 plexiform layer neurites, and varicose outer plexiform layer neurites all bear spines, that some of t

129 TH cell somata, tapering and varicose inner plexiform layer neurites, and varicose outer plexiform l

130 was localized to the inner segment and outer plexiform layer of rod photoreceptors.

131 select neurons concentrated in the internal plexiform layer of the main olfactory bulb.

132 tina and were found to interact in the outer plexiform layer of the retina containing the photorecept

133 he organization of cells making up the outer plexiform layer of the retina in the absence of Dscam.

134 Synapses in the inner plexiform layer of the retina undergo short-term plastic

135 dependent cellular interactions in the outer plexiform layer overcome this variability to ensure the

136 of idiopathic ERM, deformation of the outer plexiform layer progresses and is associated with decrea

137 ent types of amacrine cells across the inner plexiform layer prompts that they should be also involve

138 specific types of RGCs and of specific inner plexiform layer sublaminae, opening new avenues for iden

139 naptic layers beginning in stereotyped inner plexiform layer sublaminae.

140 The inner plexiform layer surround inhibition comprised GABAergic

141 Minimum rim width (MRW), ganglion cell-inner plexiform layer thickness (GC-IPLT), and circumpapillary

142 (ETDRS </=35) had normal ganglion cell-inner plexiform layer thickness and normal mfERG findings.

143 between CS at 6 cpd and ganglion cell/inner plexiform layer thickness at inferotemporal and inferona

144 al retinal thickness and ganglion cell-inner plexiform layer thickness were measured using custom-des

145 reproducible measures of ganglion cell-inner plexiform layer thickness.

146 polar cell markers and preservation of outer plexiform layer thickness.

147 The ganglion cell layer and inner plexiform layer thicknesses could predict axonal damage

148 well as composite ganglion cell layer+inner plexiform layer thicknesses in the eyes of patients with

149 is and increased nerve fibre layer and inner plexiform layer thicknesses.

150 be obtained by measuring the areas of outer plexiform layer thinning (adjusted R(2) = 0.93), externa

151 lls and the ON-OFF organization in the inner plexiform layer was largely preserved.

152 and stratification of terminals in the outer plexiform layer were comparable among coneless, conefull

153 l complement, and the extension of the outer plexiform layer were diminished.

154 The synapses in the outer plexiform layer were extensively degenerated and replace

155 copically, the inner nuclear layer and outer plexiform layer were the most affected retinal structure

156 f the combined outer nuclear layer and outer plexiform layer when we compared MSNON or MSON eyes with

157 dritic reduction to sublamina b of the inner plexiform layer without retinal ganglion cell loss, show

158 ing exclusively in sublamina S5 of the inner plexiform layer, (2) bistratified cells with dendrites i

159 ayers, whereas PKG II was found in the outer plexiform layer, amacrine cells, and somata in the gangl

160 present within the nerve fiber layer, inner plexiform layer, and inner and outer nuclear layers and

161 GCs co-stratify their dendrites in the inner plexiform layer, and that Tenm3(+) ACs require Tenm3 to

162 cells occupy strata 2, 3, and 4 of the inner plexiform layer, between the two bands formed by choline

163 major targets of histamine are in the outer plexiform layer, but the retinopetal axons containing hi

164 ter degree in the OFF sublamina of the inner plexiform layer, corroborating the hypothesis that RGCs

165 t 50%) of the GlyRalpha4 puncta in the inner plexiform layer, however, was found to lack GlyRbeta and

166 s localized primarily in puncta in the inner plexiform layer, in amacrine cells, and in somata in the

167 splicing in the retinal ganglia cells, outer plexiform layer, inner nuclear layer, and outer nuclear

168 ation of photoreceptor synapses in the outer plexiform layer, leading to a progressive functional det

169 ses extending into the ON-layer of the inner plexiform layer, similar to A8 amacrine cells described

170 exiform layer appears earlier than the outer plexiform layer, synaptic proteins, and ribbons are firs

171 re layer, the ganglion cell layer plus inner plexiform layer, the INL plus outer plexiform layer (the

172 nglion cell layer (GCL) as well as the inner plexiform layer, the inner nuclear layer (INL), and the

173 Imaging throughout the inner plexiform layer, we found transient, rectified release a

174 his functional diversity arises in the inner plexiform layer, where inhibitory amacrine cells modulat

175 GL-dSACs are located in the internal plexiform layer, where they integrate centrifugal cholin

176 s in the inner plexiform layer and the outer plexiform layer.

177 e functional ON/OFF subdivision of the inner plexiform layer.

178 centralmost (on and off) bands of the inner plexiform layer.

179 ing to new, more distal regions of the inner plexiform layer.

180 milar to those found in the vertebrate inner plexiform layer.

181 mifying in the different layers of the inner plexiform layer.

182 mify within the outermost layer of the inner plexiform layer.

183 d to both ON and OFF strata within the inner plexiform layer.

184 on their dendrites and throughout the inner plexiform layer.

185 the dendrites in the sublaminae of the inner plexiform layer.

186 s, sparing the outer retina except the outer plexiform layer.

187 r the outer or the inner border of the inner plexiform layer.

188 , redirecting their dendrites into the inner plexiform layer.

189 r layer and in synaptic boutons in the inner plexiform layer.

190 e consistent with synaptic loss in the inner plexiform layer.

191 glion cells and their processes in the inner plexiform layer.

192 neuronal connexin, is expressed in the outer plexiform layer.

193 atification in the ON sublamina of the inner plexiform layer.

194 containing histamine terminate in the inner plexiform layer.

195 al M1 positive) in diffusely condensed outer plexiform layer.

196 od vessels abnormally localized in the outer plexiform layer.

197 colocalizations of GluR1 and TH in the inner plexiform layer.

198 interactions, respectively, within the inner plexiform layer.

199 wo to three specific sublaminae in the inner plexiform layer.

200 macrine cells located laterally in the inner plexiform layer.

201 y complex, and horizontal cells in the outer plexiform layer.

202 ynapse of the retina as well as in the inner plexiform layer.

203 tion of dendrites and axons within the inner plexiform layer.

204 as to their processes and tips in the outer plexiform layer.

205 athway within the Off sublamina of the inner plexiform layer.

206 es as well as glutamate release in the outer plexiform layer.

207 ter degree in the OFF sublamina of the inner plexiform layer.

208 retina and dendrites growing into the inner plexiform layer.

209 dendrites in the OFF sublamina of the inner plexiform layer.

210 omplex formed by the ganglion cell and inner plexiform layers (GCL + IPL) provided the highest probab

211 the combined retinal ganglion cell and inner plexiform layers (RGCL+), and the inner nuclear layer (I

212 cose axons arborizing in the inner and outer plexiform layers after glutamatergic synapses depolarize

213 r, ganglion cell, inner plexiform, and outer plexiform layers and increased thickness in the inner nu

214 The SNAP25 immunoreactivity in the plexiform layers and outer nuclear layer fell into at le

215 tes are segregated within the inner or outer plexiform layers are not known.

216 duction-related machinery is present in both plexiform layers by fetal week 13.

217 and their terminals in the outer nuclear and plexiform layers in a developmentally regulated manner.

218 While the developmental sequence of plexiform layers in human retina has been characterized,

219 ll structures in the inner nuclear and outer plexiform layers in paraneoplastic vitelliform retinopat

220 Reduction of the ganglion cell and inner plexiform layers predicted greater axonal damage in pati

221 uronal cell types are constrained within the plexiform layers, allowing for establishment of retinal

222 ndria of the ganglion cells, outer and inner plexiform layers, and photoreceptor inner segments.

223 iated with progression of deformation of the plexiform layers, as central retinal thickness (CRT) did

224 the visibility of the SCS: disarrangement of plexiform layers, CRT, and multiple adhesion points betw

225 at the margin of the inner nuclear and inner plexiform layers, rather than the ganglion cell layer.

226 mple, changes in the ganglion cell and inner plexiform layers, the sites of the retinal ganglion cell

227 d in bipolar cells, ganglion cells, and both plexiform layers, whereas PKG II was found in the outer

228 re generated in both the outer and the inner plexiform layers.

229 e inner and outer nuclear layers and in both plexiform layers.

230 protein predominantly in the inner and outer plexiform layers.

231 cavities in the outer and inner nuclear and plexiform layers.

232 port the robust expression of SNAP25 in both plexiform layers.

233 The plexiform lesion is the hallmark of severe pulmonary art

234 Plexiform lesions (PLs), the hallmark of plexogenic pulm

235 Vascular occlusion and complex plexiform lesions are hallmarks of the pathology of seve

236 ng, which had similar numbers of profiles of plexiform lesions as those in lungs with more pronounced

237 ion of pulmonary arterioles and formation of plexiform lesions composed of hyperproliferative endothe

238 dothelium of remodeled pulmonary vessels and plexiform lesions of patients with pulmonary arterial hy

239 The number of profiles of plexiform lesions was significantly lower in lungs of ma

240 , media hypertrophy, adventitial thickening, plexiform lesions, vascular pruning) in this disease.

241 molecular features closely resembling human plexiform lesions.

242 the presence of early and advanced complex (plexiform) lesions, when compared with either the SIV-in

243 ation, and various forms of obliterative and plexiform-like lesions in pulmonary arteries, similar to

244 remodeling, including vascular occlusion and plexiform-like lesions, resembling the hallmarks of the

245 irtually pathognomonic finding of NF1 is the plexiform neurofibroma (PN), a benign, likely congenital

246 , and facial structures (orbital-periorbital plexiform neurofibroma [OPPN]) can result in significant

247 ues define the cell of origin for murine Nf1 plexiform neurofibroma and leverage this finding to deve

248 Previous studies found that plexiform neurofibroma formation in a mouse model requir

249 urine models that closely recapitulate human plexiform neurofibroma formation indicate that tumorigen

250 In mouse models of plexiform neurofibroma formation, Nf1 haploinsufficient

251 dies implicating the hematopoietic system in plexiform neurofibroma genesis, delineate the physiology

252 cursors (SCP), which have been implicated in plexiform neurofibroma initiation.

253 Extent of orbitotemporal plexiform neurofibroma involvement was assessed clinical

254 Orbitotemporal plexiform neurofibroma location was classified as isolat

255 Thus, the plexiform neurofibroma microenvironment involves a tumor

256 that mast cells underpin inflammation in the plexiform neurofibroma microenvironment of neurofibromat

257 for sensitive measurement of orbitotemporal plexiform neurofibroma size, and larger volumes were ass

258 nt coincides with enhanced susceptibility to plexiform neurofibroma tumorigenesis.

259 l subjects with amblyopia had orbitotemporal plexiform neurofibroma volumes greater than 10 mL.

260 Amblyopia secondary to the orbitotemporal plexiform neurofibroma was present in 13 subjects (62%)

261 ngineered mouse model that accurately models plexiform neurofibroma-MPNST progression in humans would

262 include myeloid leukemia, optic glioma, and plexiform neurofibroma.

263 and to have NF1 and a clinically significant plexiform neurofibroma.

264 ically treated a patient with a debilitating plexiform neurofibroma.

265 nalysis to measure the change in size of the plexiform neurofibroma.

266 Plexiform neurofibromas (PNs) involving the eyelid, orbi

267 life, whereas loss in adulthood caused large plexiform neurofibromas and morbidity beginning 4 months

268 Major superficial plexiform neurofibromas and symptomatic spinal neurofibr

269 Plexiform neurofibromas are common NF1 tumors carrying a

270 Plexiform neurofibromas are one of the most common tumor

271 Plexiform neurofibromas are peripheral nerve sheath tumo

272 Plexiform neurofibromas are slow-growing chemoradiothera

273 ptic pathway gliomas (OPGs) and orbitofacial plexiform neurofibromas are two of the more common ophth

274 with neurofibromatosis type 1 and inoperable plexiform neurofibromas benefited from long-term dose-ad

275 (NF-1), malignant transformation of internal plexiform neurofibromas carries a poor prognosis, in par

276 Imatinib mesylate could be used to treat plexiform neurofibromas in patients with NF1.

277 the volume burden of clinically significant plexiform neurofibromas in patients with NF1.

278 of Nf1 resulted in the development of small plexiform neurofibromas late in life, whereas loss in ad

279 pe with neither externally visible cutaneous/plexiform neurofibromas nor other tumors.

280 viable therapeutic options for patients with plexiform neurofibromas that cannot be surgically remove

281 eurofibromatosis type 1 (NF1) develop benign plexiform neurofibromas that frequently progress to beco

282 had neurofibromatosis type 1 and inoperable plexiform neurofibromas to determine the maximum tolerat

283 e or decrease from baseline in the volume of plexiform neurofibromas) was monitored by using volumetr

284 Patients commonly present with plexiform neurofibromas, benign but debilitating growths

285 han half of NF1 children with orbitotemporal plexiform neurofibromas, most commonly because of ptosis

286 reatment of neurofibromatosis type 1-related plexiform neurofibromas, which are characterized by elev

287 l databases for children with orbitotemporal plexiform neurofibromas.

288 firmed RUNX1 protein overexpression in human plexiform neurofibromas.

289 This unique murine model of PAH-like plexiform/obliterative arteriopathy induced via a two-hi

290 dels and human patients, induce formation of plexiform/obliterative lesions and defined the molecular

291 INL) than in the complex formed by the outer plexiform (OPL) and the Henle fiber layers (HFL): 5.0 x

292 s to ameliorate and, perhaps, reverse the EC plexiform phenotype in severe human PAH.

293 correlated with the cellular features of her plexiform schwannoma.

294 mary endpoint was a 20% or more reduction in plexiform size by sequential volumetric MRI imaging.

295 Dense and plexiform structure of AVM nidus as well as a low number

296 r plexiform layer (IPL), i.e., the OFF inner plexiform sublamina.

297 0% or more decrease in volume of one or more plexiform tumours.

298 mples in the odontoblast cluster were of the plexiform type (p < 0.05).

299 localized to the abluminal side of the outer plexiform vascular endothelial cells, Muller glia cells,

300 nantly to endothelial cells in occlusive and plexiform vascular lesions.