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1 means they influence the pathophysiology of pneumococcal meningitis.
2 e associated with morbidity and mortality of pneumococcal meningitis.
3 long-term hearing function in a rat model of pneumococcal meningitis.
4 IgR and PECAM-1 has the potential to prevent pneumococcal meningitis.
5 in CSF specimens from patients and mice with pneumococcal meningitis.
6 ammation and potential therapeutic target in pneumococcal meningitis.
7 ions for use of adjunctive dexamethasone for pneumococcal meningitis.
8 be associated with outcome in patients with pneumococcal meningitis.
9 t of CNS inflammation in an in vivo model of pneumococcal meningitis.
10 We identified 1379 cases of pneumococcal meningitis.
11 in play crucial roles in the pathogenesis of pneumococcal meningitis.
12 is plays in neuronal death in the context of pneumococcal meningitis.
13 rognosis for patients, especially those with pneumococcal meningitis.
14 e to the neuropathology commonly observed in pneumococcal meningitis.
15 en and adults with haemophilus meningitis or pneumococcal meningitis.
16 uggesting new neuroprotective strategies for pneumococcal meningitis.
17 ironment needed to establish nonhematogenous pneumococcal meningitis.
18 ebrospinal fluid compartment in experimental pneumococcal meningitis.
19 fatality was 8% (25/329) and was higher for pneumococcal meningitis (5/26 [19%]) than GBS meningitis
20 in the cerebrospinal fluid of patients with pneumococcal meningitis (6.8 to 3,900 ng of muramic acid
21 lysis was restricted to patients with proven pneumococcal meningitis (68 of 129 patients receiving co
22 genetic marker of morbidity and mortality of pneumococcal meningitis and also suggests a potential ro
24 tive, nationwide cohort of 405 patients with pneumococcal meningitis and in 329 controls matched for
25 multaneous in vivo biophotonic monitoring of pneumococcal meningitis and the accompanying neuronal in
28 association study in 469 community-acquired pneumococcal meningitis cases and 2072 population-based
31 reducing the incidence of hearing loss from pneumococcal meningitis, especially if therapy is instit
36 llin-susceptible and cephalosporin-resistant pneumococcal meningitis in rabbits, staphylococcal and e
37 investigated the progression and outcome of pneumococcal meningitis in Rag1(-/-) mice lacking functi
38 To better understand the high incidence of pneumococcal meningitis in the African meningitis belt,
39 ective antibody seroprevalence in preventing pneumococcal meningitis in the meningitis belt requires
40 Despite a substantially higher historical pneumococcal meningitis incidence in Burkina Faso, the g
47 y of being employed as a former patient with pneumococcal meningitis or herpes simplex encephalitis v
48 nt pneumococcal conjugate vaccine (PCV13) on pneumococcal meningitis (PM) in US children is unknown.
52 te recent advances in antimicrobial therapy, pneumococcal meningitis remains a life-threatening disea
55 tween multiple variants in a gene region and pneumococcal meningitis susceptibility yielded one signi
57 rabbit model of hearing loss in experimental pneumococcal meningitis to evaluate the therapeutic effe
58 pectively) in the progression and outcome of pneumococcal meningitis, using Kaplan-Meier survival cur
60 of brain biopsies from patients who died of pneumococcal meningitis, we observe that pneumococci col
63 st signals associated with susceptibility to pneumococcal meningitis were rs139064549 on chromosome 1
64 nctive steroid treatment is underutilized in pneumococcal meningitis, where it has shown to decrease
65 f all patients and in 39.3% of patients with pneumococcal meningitis, with an associated decrease in
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