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1 (e.g., multidrug-resistant tuberculosis and pneumococcus).
2 s species, such as Streptococcus pneumoniae (pneumococcus).
3 spiratory pathogen Streptococcus pneumoniae (pneumococcus).
4 Influenza affects host susceptibility to pneumococcus.
5 swabs yielded an alternative serotype or no pneumococcus.
6 y recombinational loss, which is frequent in pneumococcus.
7 ed on RelA after instillation of LPS but not pneumococcus.
8 cific CDS when compared to the pan-genome of pneumococcus.
9 natural resistance and cellular immunity to pneumococcus.
10 arynx, the principal ecological niche of the pneumococcus.
11 ns than control subjects who did not acquire pneumococcus.
12 ed 5157 specimens, of which 3525 (68.4%) had pneumococcus.
13 against pulmonary challenge with serotype 3 pneumococcus.
14 nges seen after infection with the wild-type pneumococcus.
15 ly involved in the innate immune response to pneumococcus.
16 fatal infections was taking prophylaxis for Pneumococcus.
17 alleled in the incidence of OM due to non-VT pneumococcus.
18 relative change in incidence of OM due to VT pneumococcus.
19 t immunity, which may ultimately benefit the pneumococcus.
20 esponse to nasopharyngeal acquisition of the pneumococcus.
21 0) of those with positive blood cultures for pneumococcus.
22 before FtsZ and guides septum positioning in pneumococcus.
23 e effectiveness of within host selection for pneumococcus.
24 a highly virulent, but immunogenic, form of pneumococcus.
25 an and infer its role in the division of the pneumococcus.
26 cholesterol-dependent cytolysin produced by pneumococcus.
27 6.11 [IQR, 20.41-76.28] FDA U/mL; P < .001), pneumococcus (17.24 [IQR, 11.33-40.25] mg/L vs 31.97 [IQ
28 vs 1.34 [IQR, 0.15-4.82] mg/L; P = .009) and pneumococcus (33.47 [IQR, 4.03-69.43] mg/L vs 50.84 [IQR
29 .7 [IQR, 27.9-168.4] FDA U/mL; P = .006) and pneumococcus (47.32 [IQR, 32.56-77.80] mg/L vs 14.77 [IQ
34 antibody levels could not be determined for pneumococcus, almost half of the patients (47%) develope
40 w insights into the synergistic link between pneumococcus and influenza virus in the context of otiti
44 and selection in the population genomics of pneumococcus and provide proof of principle of the consi
45 thus discovered a novel interaction between pneumococcus and rBPI21, a potent antimicrobial peptide
46 e elapsed between the first isolation of the pneumococcus and the coming millennium, much of fundamen
47 -independent CCR mechanism identified in the pneumococcus and the first example of lethality from los
48 ation are likely to affect the spread of the pneumococcus and the rate of pneumococcal disease in the
49 olunteers that were naturally colonized with pneumococcus and, after clearance of their natural carri
50 of all strains of Streptococcus pneumoniae (pneumococcus) and induces antibodies which protect again
51 tween the pathogen Streptococcus pneumoniae (pneumococcus) and its human host is the ability of this
53 RK (VicRK) TCSs of Streptococcus pneumoniae (pneumococcus) and other Streptococcus species show numer
54 reservoir for Streptococcus pneumoniae (the pneumococcus) and the source for both horizontal spread
55 y all serotypes of Streptococcus pneumoniae (pneumococcus), and all have been studied separately for
56 -shaped, ovococcal Streptococcus pneumoniae (pneumococcus), and depletion of GpsB leads to formation
58 colonized adults were given a type 23F or 6B pneumococcus, and a portion of these subjects became col
61 r a nonserotypeable isolate was or was not a pneumococcus, and the sequence of the ply gene fragment
64 Infections with Streptococcus pneumoniae (pneumococcus) are a cause of significant child mortality
66 er, the method by which neutrophils kill the pneumococcus as well as other Gram-positive bacteria, is
67 The success of Streptococcus pneumoniae (the pneumococcus) as a pulmonary pathogen is related to its
68 R mechanism and an essential activity in the pneumococcus, as an HPr point mutation abolishing HPrK/P
70 n(19F)-14 (ST236) to 19A ST320 has made this pneumococcus better able to colonize of the nasopharynx.
71 thal intratracheal challenge with serotype 8 pneumococcus, but it does not promote polymorphonuclear
72 ction against the homologous serotype of the pneumococcus, but the efficacies of type-specific IgM an
73 but previous attempts to study this gene in pneumococcus by generating a dpr mutant were unsuccessfu
76 The prevalence of Streptococcus pneumoniae (pneumococcus) carriage is higher in adults who are infec
82 e immunized with heat-killed type 3 serotype pneumococcus cells are impaired in generating pneumococc
85 ficant impairment of IgM humoral response to pneumococcus compared with controls (IgM titer 79.0 vs 2
86 quenced strains of Streptococcus pneumoniae (pneumococcus) contain a version of the blp locus which i
87 Streptococcus pneumoniae, also known as the pneumococcus, contains several surface proteins that alo
88 We hypothesized that the surface charge of pneumococcus contributes to its success in nasopharyngea
89 e CSP6.1, formerly reported as an "atypical" pneumococcus, CSPps1 to reflect its occurrence in S. pse
90 3 blinded nasopharyngeal specimens that were pneumococcus culture positive, the TAC pan-pneumococcus
91 th broad protection against meningococci and pneumococcus, develop an effective vaccine against group
93 sed to HIV become carriers of nasopharyngeal pneumococcus earlier and more frequently than infants wh
94 or hepoxilin-A3, an eicosanoid required for pneumococcus-elicited neutrophil trans-epithelial migrat
95 We conclude that type 1 pilus expression in pneumococcus exhibits a bistable phenotype, which is dep
101 c antibody titers and subsequent carriage of pneumococcus expressing a particular antigen variant.
102 TIGR4, a serotype 4 isolate, caused discrete pneumococcus-filled microscopic lesions (microlesions),
103 how genomics has transformed the use of the pneumococcus for the pursuit of new antibiotics, and mad
107 Invasive disease, defined as isolation of pneumococcus from a sterile site, was identified in chil
108 ing received immunizations for influenza and pneumococcus (from any source) (in 2000, 77% of MMC vs 6
114 quired immunity to Streptococcus pneumoniae (pneumococcus) has long been assumed to depend on the pre
116 emic patients lacked protective responses to Pneumococcus in 14/47 (30%), diphtheria in 15%, and teta
117 showed Cryptococcus in 64 (19.5%) patients, pneumococcus in 8 (2.4%), and meningococcus in 2 (0.6%).
118 investigated human neutrophil killing of the pneumococcus in a complement-dependent opsonophagocytic
119 variance effective population size (N(e)) of pneumococcus in a mouse colonization model by monitoring
120 eumonia (MCPP) was confirmed by detection of pneumococcus in a relevant normally sterile body fluid.
121 Ne was also evident for the colonization of pneumococcus in BALB/c mice exposed to cholera toxin 4 w
124 -control study tested whole blood by PCR for pneumococcus in children aged 1-59 months hospitalized w
127 to implement vaccines against rotavirus and pneumococcus in LMICs, and the roll out of the MenAfriVa
129 s essential for the host defense response to pneumococcus in the lungs and that RelA in airway epithe
130 es on immunizations, including varicella and pneumococcus in the post-vaccine era, use of a polyvalen
132 serum to rPstS did not inhibit growth of the pneumococcus in vitro, suggesting that antibodies do not
133 re shown to be present on the surface of the pneumococcus in vivo during pneumococcal pneumonia.
135 complex, kills Streptococcus pneumoniae (the pneumococcus) in a manner that shares features with acti
136 The proportion of CAP cases attributable to pneumococcus increased from 27.1% to 52.5% using that cu
138 ccumb to challenge with live type 3 serotype pneumococcus, indicating that TACI is required for T cel
142 though both pathways have been implicated in pneumococcus-induced neuronal cell death, their relative
144 s (leukotrienes), but the mechanism by which pneumococcus induces production of leukotrienes in the m
146 al, lower transcript levels were detected in pneumococcus-infected than in NTHi-infected animals.
147 utinin in the combined influenza A virus and pneumococcus infection cohort suggested that there were
148 have a crucial role in immunity to systemic pneumococcus infection, because both vaccinated wild-typ
149 ster of multiresistant invasive serogroup 19 pneumococcus infections, including two fatalities, was r
150 For prevention of Streptococcus pneumoniae (pneumococcus) infections in infancy, protein-conjugated
151 ith influenza virus and then challenged with pneumococcus, influenza viruses of any subtype increased
177 petence regulon of Streptococcus pneumoniae (pneumococcus) is crucial for genetic transformation.
178 s of the bacterium Streptococcus pneumoniae (pneumococcus) is its transmission from host to host, the
179 pulmonary pathogen Streptococcus pneumoniae (pneumococcus) is required for both genetic transformatio
184 a-associated hospitalizations and IPP cases (pneumococcus isolated from normally sterile sites with d
185 e opaque phenotype was predominant among the pneumococcus isolates from the middle-ear fluid in the c
187 man mAbs to the major surface antigen of the pneumococcus, its capsular polysaccharide, and tested th
188 atory tract of mice with influenza virus and pneumococcus leads to synergistic stimulation of type I
189 ecimens but also by the misidentification of pneumococcus-like viridans group streptococci (P-LVS) as
190 e pneumococcus culture positive, the TAC pan-pneumococcus lytA assay was positive in 21 (91% sensitiv
192 rate that morphological heterogeneity in the pneumococcus may promote colonization of the upper respi
194 ewborns represent a consistent population of pneumococcus-naive individuals in which to estimate the
195 data, which underscore the plasticity of the pneumococcus, need to be confirmed with in vivo analyses
196 of infants against Streptococcus pneumoniae (pneumococcus) needs substantial investment by government
198 gen test, or whole-blood lytA rtPCR revealed pneumococcus or if lytA rtPCR from NP swabs gave a resul
199 nations (OR, 4.25; 95% CI, 2.10-8.60), adult pneumococcus or tetanus vaccinations (OR, 5.14; 95% CI,
203 e important in predicting how changes in the pneumococcus (Pnc)-specific B-cell repertoire will influ
207 regation, and division site selection in the pneumococcus, providing a simple way to ensure equally s
211 e vaccine era, Streptococcus pneumoniae (the pneumococcus) remains a leading cause of otitis media, a
214 between a respiratory virus protein and the pneumococcus resulting in increased bacterial virulence
215 mes caused by Haemophilus influenzae type b, pneumococcus, rotavirus, and early infant influenza.
216 transporter is likely unique and integral to pneumococcus's strategy of carbon catabolite repression
218 ch as the pathogen Streptococcus pneumoniae (pneumococcus), side-wall (peripheral) peptidoglycan (PG)
219 ruption in IgA expression, it was found that pneumococcus-specific IgA played a significant role in t
222 of pneumonic lesions, using a bioluminescent pneumococcus, suggested that the effect of NA inhibition
223 ant from degranulated neutrophils killed the pneumococcus, suggesting a role for granular products.
225 spiratory pathogen Streptococcus pneumoniae (pneumococcus) synthesizes AcP by the conventional pathwa
226 er in children who were culture positive for pneumococcus than in those who were culture negative for
228 As discussed here in the context of the pneumococcus, the study of PavB highlights the central r
230 ty by tyrosine phosphorylation may allow the pneumococcus to adapt to the requirements of both coloni
231 mplex regulation of pneumocins may allow the pneumococcus to reserve the secretion of active peptides
233 covalently to the surface polysaccharides of pneumococcus type 14, Shigella flexneri type 2a, and Esc
234 re intranasally inoculated with 10(3) cfu of pneumococcus type 3 or type 19F and placed in a cage con
235 mutation in either comC or luxS rendered the pneumococcus unable to produce early biofilms on HREC.
237 es tested were naive responses to rabies and pneumococcus vaccines, delayed-type hypersensitivity ski
240 of proteins from a pspA-deficient strain of pneumococcus was eluted in a choline-dependent fashion.
247 ved from choline-binding protein A (CbpA) of pneumococcus were identified and then genetically fused
248 imals, and animals infected with listeria or pneumococcus, were 2.3, 2.5, and 2.6, respectively.
249 much greater C3 deposition onto the PspA(-) pneumococcus when exposed to normal mouse serum from wil
250 e supernatant (CCS) from a type 14 strain of pneumococcus which contained secreted pneumococcal prote
251 ation of the multiple adhesive properties of pneumococcus which, in turn, may correlate to diminished
252 synergism exists between influenza virus and pneumococcus, which likely accounts for excess mortality
254 (PG) synthesis in Streptococcus pneumoniae (pneumococcus); yet, mechanisms of this switching remain
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