1 he Nalp3 inflammasome in the pathogenesis of pneumoconiosis.

2 e-response relation for simple coal workers' pneumoconiosis.

3  were found to have radiographic evidence of pneumoconiosis.

4  had classic lesions of simple coal workers' pneumoconiosis.

5                      To evaluate the risk of pneumoconiosis among workers in a Midwestern automotive

6 stantially more radiographs with evidence of pneumoconiosis and classified higher small opacity profu

7 rs in the US National Study of Coal Workers' Pneumoconiosis and its implications on the exposure-resp

8 nce of pleural abnormalities consistent with pneumoconiosis, as determined by consensus of two Nation

9 's pneumoconiosis, silicosis, and mixed dust pneumoconiosis), coal miners are at risk for dust-relate

10 hat lead to the development of coal workers' pneumoconiosis (CWP).

11 s for the following lesions of coal workers' pneumoconiosis (CWP): macules, micro- and macronodules (

12 ur findings suggest that rapidly progressive pneumoconiosis in these miners was associated with expos

13    Recent recognition of rapidly progressive pneumoconiosis in younger miners, mainly in the eastern

14 ers from the National Study of Coal Workers' Pneumoconiosis (NSCWP), researchers examined changes in

15 gistic model fitting of simple coal workers' pneumoconiosis prevalence to the cumulative coal dust ex

16 .38; 95% CI: 1.35, 1.41) and identified more pneumoconiosis (prevalence ratio, 1.22; 95% CI: 1.20, 1.

17 al interstitial lung diseases (coal worker's pneumoconiosis, silicosis, and mixed dust pneumoconiosis

18 etermine if criteria for rapidly progressive pneumoconiosis were confirmed.

19 or chronic obstructive pulmonary disease and pneumoconiosis; widely dispersed throughout the Southwes