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1 ndular (25%) forms of tularemia, followed by pneumonic (12%), typhoidal (10%), oculoglandular (3%), a
5 opositive animals detected in 9/9 versus 0/9 pneumonic and nonpneumonic populations, respectively [P
12 MyD88-dependent pathways may be important in pneumonic B. thailandensis infection but that MyD88-inde
15 presence of M. ovipneumoniae in the lungs of pneumonic bighorn sheep in this study, and M. ovipneumon
16 high-quality diagnostic specimens from nine pneumonic bighorn sheep in three populations and analyze
21 here was moderate to severe inflammation and pneumonic consolidation in isolated areas at 5 and 7 day
22 patients from both outbreaks presented with pneumonic disease and although aerosol transmission has
23 Francisella tularensis causes acute, lethal pneumonic disease following infection with only 10 CFU.
25 infection but only slight attenuation by the pneumonic-disease model, closely mimicking the virulence
26 es and rabbits against challenge with lethal pneumonic doses of fully virulent Ames strain spores.
27 ve agent of tularemia, is most deadly in the pneumonic form; therefore, mucosal immunity is an import
29 immunity and IFN-gamma production following pneumonic infection with B. mallei and therefore may als
30 with the pathogen Salmonella Typhimurium or pneumonic infection with Burkholderia thailandensis, the
31 ng bubonic infection and in the lungs during pneumonic infection, suggesting a role for the Yaps duri
32 e of MGL1 in controlling neutrophilia during pneumonic infection, thus playing an important role in r
37 pha is required for the prompt resolution of pneumonic legionellosis and point to a direct role for T
40 tory disease and macroscopic and microscopic pneumonic lesions were more severe and persistent in M.
41 examination of the lungs and live imaging of pneumonic lesions, using a bioluminescent pneumococcus,
42 quency CD8(+) tetramer(+) populations in the pneumonic lung and mediastinal lymph nodes fell rapidly
43 c sequences of two bovine isolates, one from pneumonic lung and the other from healthy prepuce, have
44 was detected as a predominant member of the pneumonic lung flora in lambs with early lesions of bron
45 onse profile in both lymphoid tissue and the pneumonic lung has no obvious deleterious consequences.
46 g lesions of these cattle with a majority of pneumonic lung lobes exhibiting fibronecrotic and exudat
47 magnitude of the inflammatory process in the pneumonic lung, though replication of this influenza vir
50 d virus-specific CD4+ T cell response in the pneumonic lung; 2) enhanced primary antiviral Ab-forming
52 nfluenza epitope recovered directly from the pneumonic lungs of mice, this technique determined that
53 heimia haemolytica serotype A2 isolated from pneumonic lungs of two different ruminant species, one f
54 ating cytokine expressed in the airspaces of pneumonic lungs, but its physiological significance in t
57 eumonia but was ineffective against severely pneumonic mice, despite effective bacterial killing.
59 roximately half of RSV-infected persons, and pneumonic opacities were typically small and unilateral.
60 nfected with a closely related gram-negative pneumonic organism (Klebsiella pneumoniae) suggesting th
61 echnology to render them less susceptible to pneumonic pasteurellosis and concomitant economic losses
62 One of the pathological hallmarks of bovine pneumonic pasteurellosis is an influx of neutrophils int
63 imia haemolytica is the etiological agent of pneumonic pasteurellosis of cattle and sheep; two differ
64 bronecrotic and exudative changes typical of pneumonic pasteurellosis, but other lung lobules had his
65 hospholipase products in the pathogenesis of pneumonic pasteurellosis, development and use of anti-in
72 rat as an alternative small animal model for pneumonic plague and characterized both the efficacy and
73 studying T-cell-mediated protection against pneumonic plague and demonstrates the capacity for live,
74 n CO92 and screened them in a mouse model of pneumonic plague at a dose equivalent to 5 50% lethal do
75 dicate that Y. pestis was capable of causing pneumonic plague before it evolved to optimally cause in
76 lly virulent in animal models of bubonic and pneumonic plague but also break through immune responses
77 is essential for Y. pestis to cause primary pneumonic plague but is less important for dissemination
78 d Yersinia pestis confers protection against pneumonic plague but is not considered safe for general
79 ation protected mice from lethal bubonic and pneumonic plague caused by CO92, a wild-type F1+ strain,
80 ation resulted in partial protection against pneumonic plague challenge with 250 MLD Y. pestis CO92,
81 ague have highlighted a significant role for pneumonic plague during outbreaks of Y. pestis infection
82 lence testing in mouse models of bubonic and pneumonic plague found only a modest increase in surviva
83 course, severity, and difficulty of treating pneumonic plague highlight how differences in the route
84 severely attenuated Y. pestis CO92 to evoke pneumonic plague in a mouse model while retaining the re
88 ented Y. pestis were reported to cause fatal pneumonic plague in mice, suggesting a useful model for
91 ting type III secretion in the prevention of pneumonic plague in rats and reveal critical contributio
100 double mutant was still fully virulent in a pneumonic plague model but had an approximately 90-fold
101 he Deltalpp or DeltamsbB single mutant, in a pneumonic plague model were significantly protected agai
102 Surprisingly, via intranasal instillation (pneumonic plague model), we saw a difference in the viru
108 caf mutant was as virulent as WT CO92 in the pneumonic plague mouse model; however, it was attenuated
112 We hypothesized that the pathophysiology of pneumonic plague resulting from expression of proteins e
115 ulence factors required for the induction of pneumonic plague that are independent of iron scavenging
116 utant and comparing its ability in mediating pneumonic plague to that of the wild type in two animal
117 ary Y. pestis challenge, and we suggest that pneumonic plague vaccines should aim to induce mixed typ
118 vations strongly suggest that development of pneumonic plague vaccines should strive to prime both CD
119 To aid the development of safe and effective pneumonic plague vaccines, we are deciphering mechanisms
120 y host response during the course of primary pneumonic plague was investigated in two mouse strains,
122 ulmonary infection by Yersinia pestis causes pneumonic plague, a necrotic bronchopneumonia that is ra
125 ulmonary infection by Yersinia pestis causes pneumonic plague, a rapidly progressing and often fatal
126 pestis is the causative agent of bubonic and pneumonic plague, an acute and often fatal disease in hu
127 am-negative bacterium Yersinia pestis causes pneumonic plague, an acutely lethal septic pneumonia.
128 he potential virulence properties of Psa for pneumonic plague, an Escherichia coli strain expressing
130 on with the bacterium Yersinia pestis causes pneumonic plague, an often-fatal disease for which no va
131 ficantly affected by the Pla protease during pneumonic plague, and although A2AP participates in immu
132 ynergistically in protecting animals against pneumonic plague, and we have demonstrated an immunologi
133 onsequences of neutrophil recruitment during pneumonic plague, and we studied the susceptibility of C
134 rsinia pestis-laden aerosols that results in pneumonic plague, arming both the mucosal and systemic i
137 is review we describe the characteristics of pneumonic plague, focusing on its disease progression an
138 Yersinia pestis, which causes bubonic and pneumonic plague, forms pigmented red colonies on Congo
140 deeply rooted strains of Y. pestis to cause pneumonic plague, indicating that Y. pestis was primed t
141 m-negative bacterium that causes bubonic and pneumonic plague, is able to rapidly disseminate to othe
144 that protect mice against bubonic plague and pneumonic plague, suggesting that rV10 may serve as an i
145 ared to the WT bacterium in a mouse model of pneumonic plague, the Deltalpp Deltaail double mutant an
146 le this modification is unnecessary to cause pneumonic plague, the substitution is instead needed to
147 tis virulence in mouse models of bubonic and pneumonic plague, we characterized an msbB in-frame dele
148 sing the C57BL/6 mouse models of bubonic and pneumonic plague, we determined that all of these genes
149 produces a severe primary pneumonia known as pneumonic plague, which is contagious and highly lethal
150 esidues 271-300, elicited protection against pneumonic plague, which seemed to be based on conformati
192 protease is essential for the development of pneumonic plague; however, the complete repertoire of su
193 a pestis, the causative agent of bubonic and pneumonic plagues, has undergone detailed study at the m
197 se-type lectin-1 (MGL1), a mammalian CLR, in pneumonic sepsis, a deadly immune disorder frequently as
198 il sequestration and edema formation at that pneumonic site with or without pretreatment with endotox
199 onducted a case-control study of adults with pneumonic tularemia and investigated the environment to
200 on of Francisella tularensis biovar A causes pneumonic tularemia associated with high morbidity and m
207 The only previously reported outbreak of pneumonic tularemia in the United States also occurred o
210 n the summer of 2000, an outbreak of primary pneumonic tularemia occurred on Martha's Vineyard, Massa
211 ar results were obtained in a mouse model of pneumonic tularemia using the highly virulent F. tularen
212 mutant because this strain was attenuated in pneumonic tularemia yet induced a protective immune resp
214 eyard who had symptoms suggestive of primary pneumonic tularemia, were ill between May 15 and October
225 n of this agent with bronchopneumonia (16/34 pneumonic versus 0/17 nonpneumonic sheep were PCR positi
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