ライフサイエンスコーパス: poisoning

1 eam inflammatory and oxidative effects of CO poisoning.

2 a reduction in blood oxygen level and acute poisoning.

3 pal neurogenesis after acute carbon monoxide poisoning.

4 , and no specific therapy exists for sulfide poisoning.

5 marine food webs and leads to ciguatera fish poisoning.

6 uring the first 7 days after carbon monoxide poisoning.

7 initiated urgently in the context of severe poisoning.

8 formation on both nanocatalysis and catalyst poisoning.

9 e in the treatment of patients with methanol poisoning.

10 icant advance for treating and preventing CO poisoning.

11 drug therapy against acute organophosphorus poisoning.

12 real removal in the case of severe metformin poisoning.

13 alone kills at 20 mM, CN must potentiate HP poisoning.

14 gnostic marker of death after acute paraquat poisoning.

15 ns for extracorporeal treatment in metformin poisoning.

16 e for bacterial infectious diseases and food poisoning.

17 ons for extracorporeal treatment in methanol poisoning.

18 sk of suicide following discharge after self-poisoning.

19 containers for prevention of pesticide self-poisoning.

20 old pesticide storage reduces pesticide self-poisoning.

21 enteritis necroticans, gas gangrene and food poisoning.

22 c condition referred to as amnesic shellfish poisoning.

23 ay become valuable for early diagnosis of Mn poisoning.

24 d by the number of carcasses recovered after poisoning.

25 en implicated or suggested in scombroid fish poisoning.

26 his compound that could result in human food poisoning.

27 2 patients in a cluster outbreak of methanol poisoning.

28 breastfed infants are also at risk for lead poisoning.

29 ter the visual outcome, especially in severe poisoning.

30 osis, management, and prevention of acute CO poisoning.

31 need of emergency chelation therapy for lead poisoning.

32 ropriate flushing procedures to prevent lead poisoning.

33 ributing to the lack of heterocycle catalyst poisoning.

34 or storage and prevention of accidental self-poisoning.

35 resenting an established plasma biomarker of poisoning.

36 d musculoskeletal diseases, and injuries and poisoning.

37 , driven by mental illness and injury and/or poisoning.

38 ate to treat or prevent organophosphate (OP) poisoning.

39 se as a first specific treatment for sulfide poisoning.

40 emission, raising the possibility of (210)Po poisoning.

41 oach for adults with calcium channel blocker poisoning.

42 pylobacter jejuni is the major cause of food poisoning.

43 to 19 years who were hospitalized for opioid poisonings.

44 treatment is recommended in severe metformin poisoning (1D).

45 e-treated control, (2) acute carbon monoxide poisoning, (3) acute carbon monoxide poisoning followed

46 on died during the period studied, with lead poisoning a primary factor in six deaths.

47 In patients with paracetamol poisoning, a 12 h modified acetylcysteine regimen result

48 ent neurological and affective effects of CO poisoning, a portion of survivors still have substantial

49 etal alloyed catalysts with superior anti-CO poisoning ability and low usage of Pt and Ru for fuel ce

50 Pesticide self-poisoning accounts for 14-20% of suicides worldwide.

51 ited enhanced resistance to sintering and CO poisoning, achieving an order of magnitude increase in s

52 quifers are the source of widespread arsenic poisoning across the region.

53 position for topoisomerase IB (Top1) enzyme poisoning activity and cytotoxicity to human cancer cell

54 the 3-nitro group with minimal loss of Top1 poisoning activity.

55 Carbon monoxide (CO) poisoning affects 50,000 people a year in the United Sta

56 ed poisoning/STEM/EDX experiment whereby the poisoning agent is shown to be bound to the Fe NPs.

57 s using substoichiometric amounts of various poisoning agents.

58 : self-injury alone pound753 (SD 2061), self-poisoning alone pound806 (SD 1568), self-poisoning and s

59 1150 (70%) presentations were for self-poisoning alone, 367 (22%) for self-injury alone, and 13

60 Mercury poisoning and "hot filtration" experiments ruled out lea

61 rfringens enterotoxin (CPE) cause human food poisoning and antibiotic-associated diarrhea.

62 um perfringens enterotoxin (CPE) causes food poisoning and antibiotic-associated diarrhea.

63 (GI) symptoms of C. perfringens type A food poisoning and CPE-associated non-food-borne human GI dis

64 tion between biochemical markers of methanol poisoning and final VA.

65 ny human and animal diseases, including food poisoning and gas gangrene.

66 V) in people who have recovered from arsenic poisoning and in patients with acute promyelocytic leuke

67 Childhood lead poisoning and lead contamination were widespread in surv

68 Bacillus cereus a pathogen that causes food poisoning and life-threatening wound infections, secrete

69 Addition of detergent in milk can cause food poisoning and other complications.

70 ylactic therapeutics against organophosphate poisoning and other important targets.

71 nal bicyclopentadione) mediate topoisomerase poisoning and possibly many other activities of curcumin

72 llisions with man-made structures, vehicles, poisoning and predation by domestic pets, combine to kil

73 favour the adsorption of O2, alleviating CO poisoning and promoting the catalysis.

74 (upper gastrointestinal haemorrhage, alcohol poisoning and related head injury, and a completed suici

75 neurotoxins that cause neurotoxic shellfish poisoning and respiratory illness in humans, as well as

76 Self-poisoning and self-injury are associated with a high ris

77 elf-poisoning alone pound806 (SD 1568), self-poisoning and self-injury pound987 (SD 1823; p<0.0001).

78 aquatic pathogen responsible for severe food poisoning and septicemia in humans, secretes a PFT calle

79 10)Po) gained widespread notoriety after the poisoning and subsequent death of Mr Alexander Litvinenk

80 was an important modifiable risk factor for poisonings and thermal injuries (tests for trend p </= 0

81 of heterogeneous DNA damage that causes Top2 poisoning, and indicate that evaluation of Tdp2 status m

82 rink of extinction, in part, because of lead poisoning, and lead poisoning remains a significant thre

83 oints were highest for deaths due to injury, poisoning, and other external causes, in both men (7.89;

84 , recent history of intentional use for mass poisoning, and the absence of a known antidote raise pub

85 dministration as an antidote for (134/137)Cs poisoning, and use for (131/137)Cs radiotherapy (brachyt

86 saw increases in mortality from suicide and poisonings, and an overall increase in external cause mo

87 Early symptoms of (210)Po poisoning are indistinguishable from those of a wide ran

88 ratio=15.86, 95% CI=10.7-23.4; computed with poisoning as reference), and to a lesser extent after ev

89 edicinal use in the treatment of heavy metal poisonings as well as having potency for reducing inflam

90 lfish poisoning (DSP), and amnesic shellfish poisoning (ASP) toxins in seafood is a severe and growin

91 In adolescents aged 15 to 19 years, poisonings attributed to heroin were also identified.

92 Main Outcomes and Measures: Poisonings attributed to prescription opioids were ident

93 e theoretical basis behind carbon and sulfur poisoning, before examining the strategies toward carbon

94 In the absence of severe poisoning but if the methanol concentration is elevated

95 difiable risk factors for thermal injury and poisoning but not fractures in preschool children.

96 cyanide antidote in animal models of cyanide poisoning, but it is unstable in solution and poorly abs

97 thousands of accidental or intentional human poisonings, but it is of continued concern as a chemical

98 lgesics, apart from an increase in oxycodone poisonings, but numbers were small.

99 riggered by Top2 engagement of DNA damage or poisoning by anticancer drugs.

100 s O2 , and Ag/Pt surface proximity disfavors poisoning by CO or oxidized species.

101 bed on atop position, which suggests surface poisoning by CO or surface reconstruction.

102 eaction will be inhibited because of surface poisoning by CO.

103 HQNO induces specific self-poisoning by disrupting the flow of electrons through th

104 , moderate and severe acute organophosphorus poisoning by exposing zebrafish larvae to different conc

105 euRS), while the TM84-producer prevents self-poisoning by expressing a resistant LeuRS AgnB2.

106 antagonists is most commonly associated with poisoning by or surreptitious ingestion of warfarin, war

107 emory impairment after acute carbon monoxide poisoning by preserving adult neurogenesis via an increa

108 ever no efficient and approved treatment for poisoning by ricin inhalation, although there have been

109 These materials are resistant to poisoning by small reactive gases (CO and H2S) that may

110 Pt is notorious for its sensitivity to poisoning by strong CO adsorption.

111 Methanol poisoning can induce death and disability.

112 Organophosphate poisoning can occur from exposure to agricultural pestic

113 pressants, 4) street drugs, 5) sedatives, 6) poisoning (carbon monoxide, arsenic, or cyanide), 7) oth

114 injury cases, 4,050 fracture cases and 2,193 poisoning cases, matched on general practice to 94,620 c

115 phimurium (ST), a major source of human food poisoning, caused inflammation of murine intestinal tiss

116 toxicity in animal models and acute fluoride poisoning causes neurotoxicity in adults, very little is

117 ively related to incidence of ciguatera fish poisoning (CFP).

118 butable to accidental deaths (primarily drug poisonings), chronic liver disease and cirrhosis, and su

119 especially for young people and against self-poisoning, could potentially lower future costs at a tim

120 uicides and mischaracterization of most drug poisoning deaths as "accidents" on death certificates.

121 ol in the UK, there was a major reduction in poisoning deaths involving this drug, without apparent s

122 e SuperMICAR text entries to study the 7,817 poisoning deaths that occurred among Washington State re

123 g to 600 fewer deaths (-62%) when accidental poisoning deaths were included.

124 are often used to study the epidemiology of poisoning deaths, but the International Classification o

125 e control group; incidence of pesticide self-poisoning did not differ between groups (293.3 per 100 0

126 nal mining may combine to cause similar lead poisoning disasters elsewhere globally.

127 ISPR/Cas9-mediated chromosome cutting, or by poisoning DNA decatenation resulted in mistiming of MapZ

128 ellfish poisoning (PSP), diarrheic shellfish poisoning (DSP), and amnesic shellfish poisoning (ASP) t

129 estern Black Sea region of Turkey and causes poisoning due to consumption of grayanatoxins (GTX I and

130 eriod (AD 500-1450) show patterns of chronic poisoning due to exposure to geogenic arsenic.

131 posed to hospital admissions for injuries or poisonings due to self-harm, interpersonal violence, or

132 plication of palladacycle catalysts prevents poisoning during catalyst formation, thereby allowing fo

133 e admitted to hospital following injuries or poisonings during their childhood.

134 ed directing group capable of overriding the poisoning effect of a wide range of heterocycle substrat

135 ymer layer as a transducer did not show this poisoning effect.

136 tter tolerance for methanol crossover and CO poisoning effects, long-term durability than commercial

137 lity and free from any methanol-crossover/CO-poisoning effects.

138 Most suicides occur long after the index poisoning, emphasizing the importance of longitudinal, s

139 mental remediation to address childhood lead poisoning epidemic due to artisanal gold mining in Zamfa

140 identified all individuals with a first self-poisoning episode in Ontario, Canada, from April 1, 2002

141 A first self-poisoning episode is a strong predictor of subsequent su

142 Patients with a self-poisoning episode were also more likely to die because o

143 For each individual with a deliberate self-poisoning episode, we randomly selected 1 control from t

144 ears) who were discharged after a first self-poisoning episode.

145 der age, male sex, multiple intervening self-poisoning episodes, higher socioeconomic status, depress

146 ion in the liver, posing a risk of secondary poisoning especially in liver-failure patients.

147 illed medication prescriptions, and nonfatal poisoning events during the 30 days and 12 months before

148 ecular weight neurotoxin, is responsible for poisoning events that traditionally occur from consumpti

149 not limited to toxic waste sites and massive poisoning events.

150 induction period during catalysis as well as poisoning experiments using substoichiometric amounts of

151 luding kinetic studies, filtration tests, Hg poisoning experiments, transmission electron microscopy,

152 tically significant as determined by kinetic poisoning experiments.

153 w-income and middle-income countries (burns, poisoning, falling, or drowning) as suicide.

154 ns, especially in the setting of intentional poisoning, fire-related toxic gas exposures, and inhalat

155 onoxide poisoning, (3) acute carbon monoxide poisoning followed by 7-day hyperbaric oxygen treatment,

156 B-Fc) chimera, and (5) acute carbon monoxide poisoning followed by intracerebroventricular infusion o

157 nsor was 100% accurate in diagnosing cyanide poisoning for acutely exposed rabbits.

158 anI gene was absent from typical type A food poisoning (FP) strains carrying a chromosomal enterotoxi

159 ive OD were identified using ICD-9 codes for poisoning from opioids or adverse effects from opioids.

160 In this age group, poisonings from heroin increased from 0.96 (95% CI, 0.75

161 risk, and examine potential for similar lead poisoning globally.

162 We investigated the extent of childhood lead poisoning [>/= 1 child with a blood lead level (BLL) >/=

163 years or older, 611 cases of pesticide self-poisoning had occurred by 3 years in the intervention gr

164 The treatment of OP poisoning has not changed over the last 50 years.

165 herichia coli strains, and outbreaks of food poisoning have caused significant mortality rates as, fo

166 ders, including food allergy, scombroid food poisoning, histamine intolerance, irritable bowel syndro

167 s Frontieres (MSF) discovered extensive lead poisoning impacting several thousand children in rural n

168 t for the treatment of severe childhood lead poisoning in a resource-limited setting.

169 We developed a model of CO poisoning in anesthetized and mechanically ventilated mi

170 hot pellets constitutes the main cause of Pb poisoning in avifauna.

171 dren (R(2) = 0.68) and the incidence of lead poisoning in children under age 1.3 years (R(2) = 0.64).

172 d ammunition is the principle source of lead poisoning in condors.

173 ant as an early warning tool to prevent food poisoning in consumers.

174 letal muscle cytochrome c oxidase to sulfide poisoning in ethylmalonic encephalopathy, resulting from

175 aphylococcal enterotoxin B (SEB) causes food poisoning in humans.

176 ance against both carbon build-up and sulfur poisoning in hydrocarbon fuels.

177 t superior catalytic ability to withstand CO poisoning in methanol oxidation reaction (MOR) than do b

178 mary outcome was incidence of pesticide self-poisoning in people aged 14 years or older during 3 year

179 sk factors for thermal injury, fractures and poisoning in pre-school children in order to inform the

180 be-PVC-based membrane, suffered from sulfide poisoning in the deeper, sulfidic regions of the lake.

181 in reducing the incidence of childhood lead poisoning in the United States in the past three decades

182 major cause of mortality resulting from food poisoning in the United States.

183 ses addressed an unprecedented epidemic lead poisoning in Zamfara State, northern Nigeria.

184 ng practices and hospitalizations for opioid poisonings in adults.

185 ics of hospitalizations attributed to opioid poisonings in children and adolescents.

186 exist describing hospitalizations for opioid poisonings in children and adolescents.

187 those age 1-2 (OR = 2.43, 95%CI 2.23-2.65), poisonings in those age 2-3 (OR = 7.32, 95%CI 6.26-8.58)

188 ssels from Ireland in 1995 following a human poisoning incident.

189 ath in the previous year from suspected lead poisoning included the age of the child, the mother's wo

190 or extracorporeal treatment: Severe methanol poisoning including any of the following being attribute

191 spiratory complications after OP insecticide poisoning, including onset of delayed neuromuscular junc

192 DTA or ion exchange resin caused no catalyst poisoning, indicating that cobalt ions were not released

193 Extracorporeal Treatments in Poisoning inhibitors and folic/folinic acid should be co

194 ), an increase of 161% (P for trend, <.001); poisonings involving methadone increased from 0.10 (95%

195 d deaths (suicide, open verdicts, accidental poisonings) involving single analgesics.

196 Acetaminophen (APAP, paracetamol) poisoning is a leading cause of acute liver failure (ALF

197 Organophosphorus (OP) compound poisoning is a major global public health problem.

198 Agricultural pesticide self-poisoning is a major public health problem in rural Asia

199 Selenium poisoning is a significant health problem in parts of Pu

200 Staphylococcal food poisoning is caused by enterotoxins excreted into foods

201 Carbon monoxide (CO) poisoning is common in modern society, resulting in sign

202 Paracetamol poisoning is common worldwide.

203 t universally fatal, survival following self-poisoning is common, providing an opportunity for second

204 Food poisoning is one of the leading causes of morbidity and

205 Deliberate self-poisoning is the leading method of attempted suicide.

206 of suicide following a first episode of self-poisoning is unknown.

207 untermeasures against acute organophosphorus poisoning is urgently needed.

208 ociated with oceanic red tides and shellfish poisoning, is a potent inhibitor of electrical conductio

209 to organophosphate (OP) pesticides, without poisoning, is associated with adverse peripheral nervous

210 fishery products, responsible for scombroid poisoning, is essential to guarantee the safety of food.

211 soluble protein 4 (Ssp4) made by type A food poisoning isolates with a chromosomal cpe gene that also

212 Acute OP insecticide self-poisoning kills over 200,000 people every year, the majo

213 Phototherapy administered during severe CO poisoning limited the blood COHb increase and improved t

214 ases, we suggest that the very nature of CS2 poisoning may be related to interrupting or overwhelming

215 Here we show that metal poisoning may have caused these aberrant morphologies du

216 This "self-poisoning" mechanism explains the need to mix CO into th

217 al activity of EDP on food spoilage and food poisoning microorganisms was evaluated through the measu

218 ts can be obtained from others, stabbing and poisoning neighbouring cells, or colonising spaces while

219 inically described as neurological shellfish poisoning (NSP).

220 from a home marine aquarium involved in the poisoning of a whole family.

221 rent length scales for the analysis of metal poisoning of an individual catalyst particle.

222 ersibly store hydrogen prevents the hydrogen poisoning of Ru surfaces.

223 The poisoning of rural populations in South and Southeast As

224 ct experimental support to the well-accepted poisoning of the TPRE and IIT mechanisms, but also provi

225 re-entrant edge (TPRE) mechanism as well as poisoning of the TPRE mechanism are valid under certain

226 However, IIT, TPRE or poisoning of the TPRE mechanism cannot be used to interp

227 re-entrant edge, inducing TPRE mechanism or poisoning of the TPRE mechanism, and (iii) the segregati

228 mits formation of EPFRs due to inhibition or poisoning of the transition metal active sites necessary

229 reversal protects forks from breakage after poisoning of Topoisomerase 1.

230 On the other hand, phosphorus poisoning of zeolite-based catalysts, which are used for

231 ic deactivation due to hydrocarbon coking or poisoning of zeolites at an unprecedented spatial resolu

232 the multiple respiratory complications of OP poisoning offers additional therapeutic opportunities.

233 Accidental BoNT poisoning often occurs through ingestion of Clostridium

234 his coordination, which can lead to catalyst poisoning or C-H functionalization at an undesired posit

235 needed or evidence is present of a treatable poisoning or infection, seizures including non-convulsiv

236 erbivorous consumers, as opposed to directly poisoning or repelling them.

237 xford, Manchester, and Derby, UK, after self-poisoning or self-injury between Jan 1, 2000, and Dec 31

238 elf-harm (intentional acts of non-fatal self-poisoning or self-injury) is common, particularly in you

239 onspecific ocular chemical burn and chemical poisoning or toxic effects were eligible for inclusion.

240 , Medecins Sans Frontieres discovered a lead poisoning outbreak linked to artisanal gold processing i

241 During May-June 2010, a childhood lead poisoning outbreak related to gold ore processing was co

242 to determine the cause of the childhood lead poisoning outbreak, investigate risk factors for child m

243 ual incidence of hospitalizations for opioid poisonings per 100000 children aged 1 to 19 years rose f

244 he published, peer-reviewed literature on CO poisoning, placing emphasis on selecting studies that mo

245 ster experience may inform future local lead poisoning prevention policies in other communities.

246 Paralytic shellfish poisoning (PSP) is a serious human illness caused by the

247 The presence of paralytic shellfish poisoning (PSP), diarrheic shellfish poisoning (DSP), an

248 in part, because of lead poisoning, and lead poisoning remains a significant threat today.

249 Treatment of CO poisoning requires rapid removal of CO and restoration o

250 precious Pt and at the same time to enhance poisoning resistance.

251 Furthermore, acute carbon monoxide poisoning resulted in a delayed impairment of cognitive

252 Suicides by carbon monoxide poisoning resulting from burning barbecue charcoal reach

253 activity due to genetic aberrations or lead poisoning results in a mild to moderate nonspherocytic h

254 years, hospitalizations attributed to opioid poisonings rose nearly 2-fold in the pediatric populatio

255 which are commonly ingested in acts of self-poisoning, seem to be effective in reducing pesticide-sp

256 Palythoa- and Ostreopsis-related inhalatory poisonings showed many similarities including fever, res

257 Acute carbon monoxide poisoning significantly suppressed adult hippocampal neu

258 responding to their in vitro potency and the poisoning signs or toxicity they produced in intrathorac

259 n is critical for C. perfringens type A food poisoning since spores contribute to transmission and re

260 g area of research in which many traditional poisoning species have been used to promote catalytic ac

261 inal support is given in terms of a combined poisoning/STEM/EDX experiment whereby the poisoning agen

262 , a derivative of C. perfringens type A food-poisoning strain NCTC8798.

263 CO binding, EPR spectroscopy, and poisoning studies indicate that two different types of C

264 Four examined acute exposure: Three neonic poisoning studies reported two fatalities (n = 1,280 cas

265 Four examined acute Three neonic poisoning studies reported two fatalities (n = 1,280 cas

266 ignificant number of patients who survive CO poisoning suffer from long-term neurological and affecti

267 of potentially avoidable causes such as drug poisonings, suicide, and chronic liver disease and cirrh

268 increasing death rates from drug and alcohol poisonings, suicide, and chronic liver diseases and cirr

269 underestimated owing to misclassification of poisoning suicides and mischaracterization of most drug

270 tes a tightly regulated QS self-defense anti-poisoning system.

271 sis by nanoclusters was confirmed by mercury poisoning, temperature-dependent sigmoidal kinetic curve

272 vity may prevent iron hydride formation from poisoning the P3(B)Fe system.

273 In the absence of severe poisoning, the decision to use extracorporeal treatment

274 ransiently reducing paralysis following BoNT poisoning, the precise mechanisms by which aminopyridine

275 e determinant for C. perfringens type-A food poisoning, the second most common bacterial food-borne i

276 Notably, direct Top1 poisoning to elevate Top1cc levels phenocopied the neuro

277 no evidence of switching from pesticide self-poisoning to other forms of self-harm, with no significa

278 veral observations: Pt octahedra show higher poisoning tolerance in the electrooxidation of formic ac

279 in Nigeria to assess lead contamination and poisoning, treat victims, mitigate exposures, and remedi

280 Lead (Pb) poisoning via ingestion of shot pellets is a frequent ca

281 Acute carbon monoxide poisoning was achieved by exposing the rats to carbon mo

282 of lung phototherapy administered during CO poisoning was also studied.

283 of adult neurogenesis by the carbon monoxide poisoning was appreciably alleviated by early treatment

284 The risk of suicide following self-poisoning was markedly increased relative to controls (h

285 Thallium poisoning was suspected and investigated but ultimately

286 n method of suicide (125 [73%] of 171), self-poisoning was the main reason for presenting to hospital

287 Azaspiracid (AZA) poisoning was unknown until 1995 when shellfish harveste

288 Patients with acute paraquat poisoning were recruited.

289 Self inflicted injuries by poisoning were the most frequently reported sources acco

290 d models of acetaminophen and alpha-amanitin poisoning were used.

291 04) hospitalizations for prescription opioid poisonings were identified.

292 SEA is the main cause of food poisoning, while SEB is a common toxin manufactured as a

293 iated with major reductions in deaths due to poisoning with co-proxamol receiving verdicts of suicide

294 Metformin poisoning with lactic acidosis appears to be amenable to

295 ts a potential use for DFO in cases of acute poisoning with phosphorus pesticides.

296 ical course prompted active consideration of poisoning with radioactive material, with the diagnosis

297 The Extracorporeal Treatments in Poisoning workgroup aimed to develop evidence-based cons

298 Here, the Extracorporeal Treatments In Poisoning workgroup, comprising international experts re

299 ellosis is one of the leading causes of food poisoning worldwide.

300 (S. typhimurium) is a leading cause of food poisoning worldwide.