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1 y, or had received previous treatment with a poly(ADP-ribose) polymerase inhibitor.
2 reaks (DSBs), and increased sensitivity to a poly(ADP-ribose) polymerase inhibitor.
3 eficient cells were sensitive to olaparib, a poly(ADP-ribose) polymerase inhibitor.
4 Ialpha inhibitor, L67, in combination with a poly (ADP-ribose) polymerase inhibitor.
5 ation) activity and is affected by canonical poly(ADP-ribose) polymerase inhibitors.
6 h in neurons, all of which were prevented by poly(ADP-ribose) polymerase inhibitors.
7 vic radiotherapy, or previous treatment with poly (ADP-ribose) polymerase inhibitors.
8 ndicated by the cytoprotective effect of the poly(ADP-ribose) polymerase inhibitor 3-aminobenzamide.
10 2 deficient and sensitive to cisplatin and a poly(ADP-ribose) polymerase inhibitor, AG14361, whereas
13 mor-derived DNA were resistant to platin- or poly ADP ribose polymerase inhibitor-based chemotherapy.
16 lethality anticancer therapeutics, including poly ADP-ribose polymerase inhibitors for BRCA1- and BRC
22 Promising novel therapeutic agents such as poly (ADP-ribose) polymerase inhibitors have increased a
24 IEL3 provides further evidence that use of a poly(ADP-ribose) polymerase inhibitor in the maintenance
25 gagement of the chemotherapeutic Olaparib, a poly(ADP-ribose) polymerase inhibitor, in live cells and
26 ion with doxorubicin, bortezomib, as well as poly(ADP-ribose) polymerase inhibitor NU1025 and Fas-act
27 d treatments such as antiangiogenic drugs or poly (ADP-ribose) polymerase inhibitors offer potential
28 We investigated in vitro sensitivity to the poly (ADP-ribose) polymerase inhibitor olaparib (AZD2281
29 rpose Durable and long-term responses to the poly (ADP-ribose) polymerase inhibitor olaparib are obse
31 tions initially respond well to platinum and poly(ADP-ribose) polymerase inhibitor (PARPi) therapy; h
32 uely responsible for cellular sensitivity to poly(ADP-ribose) polymerase inhibitors (PARPi) in BRCA1-
34 Combination studies of PCI-24781 with the poly(ADP-ribose) polymerase inhibitor PJ34, an agent tho
35 rate alpha-ketoglutarate or treatment with a poly(ADP ribose) polymerase inhibitor protects reductive
39 cells with mutant p53 were resistant to the poly(ADP-ribose) polymerase inhibitor, veliparib (2-[(2R
42 receptor tyrosine kinase inhibitor XL184 and poly (ADP-ribose) polymerase inhibitors which are in ear
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