ライフサイエンスコーパス: poly G

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1 r (VEGF) proximal promoter region contains a poly G/C-rich element that is essential for basal and in

2 Phase variation of PorA is mediated by a poly-G repeat tract present within the promoter, leading

3 mation at presumed endogenous RFBs formed by poly G/C tracts in the absence of DOG-1.

4 s showed that lgtA, lgtC, and lgtD contained poly-G tracts within the coding frames, leading to the h

5 In a cohort of 22 patients, we detect poly-G variants in 91% of tumors.

6 These findings establish poly-G ODN as a novel type of cancer immunotherapy.

7 We generate poly-G mutation profiles of spatially separated samples

8 A homopolymeric tract of Gs (poly-G) is present in the pgtA gene of many GC strains,

9 tream 3' splice site and downstream intronic poly-G runs functioned redundantly to protect an exon fr

10 Mechanistically, poly-G ODN directly induced the phosphorylation of Lck (

11 The antitumor activity of poly-G ODN was mediated through CD8 T cells in a TLR9-in

12 RNA polymerase will synthesize a 'ladder' of poly-G RNA products.

13 elation was observed between the presence of poly-G in pgtA and the dissemination of GC infection.

14 matic variation in hypermutable polyguanine (poly-G) repeats can provide a rapid and reliable assessm

15 the intratumoral injection of polyguanosine (poly-G) oligonucleotides (ODN) has such an effect, boost

16 e, we show that synthetic polyriboguanosine (poly G) and oligo-deoxyriboguanosine (oligo G) reduce en

17 tic mutation frequency, suggesting that some poly-G variants accumulate before the onset of carcinoge

18 Finally, we show that poly-G mutations can be found in other cancers than colo

19 ase pair insertions and deletions within the poly G:C run there, suggesting that a high affinity PNA

20 s in the number of guanosine residues in the poly-G tracts might be responsible for the high frequenc

21 ted gonorrhea (UG) patients, pgtA lacked the poly-G.

22 However, in many other GC, pgtA lacks the poly-G and is expressed constitutively without Pv.

23 Strikingly, we found that the poly-G run, a known intronic splicing enhancer, was the

24 We use poly-G repeat genotyping to study the evolution of colon

25 gene of many GC strains, and this pgtA with poly-G can undergo phase variation (Pv).

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