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1 orbital inflammation, or granulomatosis with polyangiitis.
2 f the PR3 autoantigen in granulomatosis with polyangiitis.
3 toplasmic antibodies) in granulomatosis with polyangiitis.
4 ith diagnosed Wegener disease or microscopic polyangiitis.
5 ce injury in MPO-ANCA-associated microscopic polyangiitis.
6 reatment of eosinophilic granulomatosis with polyangiitis.
7 as Wegener's granulomatosis) and microscopic polyangiitis.
8 ther Wegener's granulomatosis or microscopic polyangiitis.
9 condition a separate entity from microscopic polyangiitis.
10 is as Wegener granulomatosis and microscopic polyangiitis.
11 egener's granulomatosis [WG], 12 microscopic polyangiitis, 16 Churg-Strauss syndrome [CSS]), and anti
12 rolled patients (87 with granulomatosis with polyangiitis, 23 with microscopic polyangiitis, and 5 wi
14 c autoantibody (ANCA)-associated microscopic polyangiitis and glomerulonephritis (excluding Wegener's
15 tic distinctions between granulomatosis with polyangiitis and microscopic polyangiitis that are assoc
16 vasculitis and also that granulomatosis with polyangiitis and microscopic polyangiitis were genetical
17 resistance in 107 patients with microscopic polyangiitis and necrotizing and crescentic glomerulonep
19 d AAV (Wegener granulomatosis or microscopic polyangiitis) and were aged 18 to 75 years at diagnosis.
20 tosis with polyangiitis, 23 with microscopic polyangiitis, and 5 with renal-limited ANCA-associated v
23 with Wegener granulomatosis and microscopic polyangiitis are derived by extrapolating from data abou
25 esponses in eosinophilic granulomatosis with polyangiitis (Churg-Strauss syndrome); and matrix metall
28 (MPA), and eosinophilic granulomatosis with polyangiitis (Churg-Strauss; EGPA), also collectively re
30 (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, formerly Churg-Strauss), are a group
31 phils from patients with granulomatosis with polyangiitis expressing high membrane PR3 levels showed
32 ing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis
34 a 15-year-old girl with granulomatosis with polyangiitis, formerly known as Wegener's granulomatosis
35 tion with categories for granulomatosis with polyangiitis (GPA) (Wegener's), microscopic polyangiitis
36 mal gland involvement in granulomatosis with polyangiitis (GPA) commonly accompanies orbital disease,
40 omatous lesions found in granulomatosis with polyangiitis (GPA, formerly Wegener's) or the developmen
41 associated vasculitides, granulomatosis with polyangiitis (GPA, formerly Wegener's), microscopic poly
44 al features of kidney disease in microscopic polyangiitis have been associated with clinical presenta
45 monstrates greater similarity to microscopic polyangiitis in relapse rate than previously reported.
50 nagement of renal involvement in microscopic polyangiitis is discussed, with emphasis on prognosis an
51 PR3), the autoantigen in granulomatosis with polyangiitis, is expressed at the plasma membrane of res
52 sis, Churg-Strauss syndrome, and microscopic polyangiitis, is impeded by the small number of pediatri
53 ipants with eosinophilic granulomatosis with polyangiitis, mepolizumab resulted in significantly more
54 cludes Wegener's granulomatosis, microscopic polyangiitis (microscopic polyarteritis), the Churg-Stra
55 y diagnosed or relapsing granulomatosis with polyangiitis, microscopic polyangiitis, or renal-limited
57 egener's granulomatosis (WG) and microscopic polyangiitis (MPA) have been reported to be pathological
59 iitis (GPA, formerly Wegener's), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with
60 h polyangiitis (Wegener's; GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with
61 polyangiitis (GPA) (Wegener's), microscopic polyangiitis (MPA), and kidney-limited disease; 2) the E
62 hy include polyarteritis nodosa, microscopic polyangiitis (MPA), rheumatoid vasculitis, Churg-Strauss
64 ve patients (64 with WG, 14 with microscopic polyangiitis [MPA], and 526 others) and 45 healthy volun
65 creased in patients with granulomatosis with polyangiitis, MVs generated from neutrophils expressing
66 ived a KTX for ESRD secondary to microscopic polyangiitis (n=43) or Wegener's granulomatosis (n=42).
67 oncluded that most patients with microscopic polyangiitis or necrotizing and crescentic glomeruloneph
68 matosis with polyangiitis versus microscopic polyangiitis), or new diagnosis (versus relapsing diseas
69 ranulomatosis with polyangiitis, microscopic polyangiitis, or renal-limited ANCA-associated vasculiti
70 rythematosus (P = 2.7 x 10(-8)), microscopic polyangiitis (P = 2.9 x 10(-4)) and Wegener's granulomat
71 il cytoplasmic antibody-positive microscopic polyangiitis, resulting in pulmonary hemorrhage and acut
73 ulomatosis with polyangiitis and microscopic polyangiitis that are associated with ANCA specificity,
74 ntrolling for age, ANCA pattern, microscopic polyangiitis versus glomerulonephritis alone, and pulmon
75 NCA type, AAV diagnosis (granulomatosis with polyangiitis versus microscopic polyangiitis), or new di
81 b (RTX) in patients with granulomatosis with polyangiitis (Wegener's; GPA), microscopic polyangiitis
84 itivity and eosinophilic granulomatosis with polyangiitis, which accounted for 34.1% and 12.8% of cas
85 refractory eosinophilic granulomatosis with polyangiitis who had received treatment for at least 4 w
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