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1 c TAT, different TAT variants, Antp, MTS and polyarginine.
2 as mimicked by the CaR agonists spermine and polyarginine.
3 ) molecular clone was effectively blocked by polyarginine.
4 ion reactions were faster in the presence of polyarginine.
5 cence polarization signal in the presence of polyarginine.
6 igher in the presence than in the absence of polyarginine.
7 ze the transfer of ADP-ribose from NAD(+) to polyarginine.
8 is some 50% larger with polylysine than with polyarginine.
9 possess inhibitory potency almost equal to L-polyarginines.
10 to 140% of the control in the presence of l-polyarginines.
12 evidence indicate that HvnA and HvnB mediate polyarginine ADP-ribosylation not by ARTase activity, bu
13 performing the reactions in the presence of polyarginine and continuously measuring the fluorescence
15 ptides with cationic polyamino acids such as polyarginine and polylysine by fluorescence polarization
16 A previously proposed model for binding of polyarginine and protamine to DNA provides a convenient
19 inhibited by CK2 effectors such as heparin, polyarginine, and histone H1, but was not affected by th
20 tial reading frames, generating polyproline, polyarginine, and polyalanine proteins, respectively.
21 ed cationic sequence derived from HIV Tat or polyarginine Arg8, and equals that of hydrocarbon-staple
23 and HvnB catalyzed ribosylation of not only polyarginine but also polylysine and polyhistidine, and
25 esent study was to investigate the effect of polyarginine chain length on topical delivery of surface
26 artificial cerebrospinal fluid (aCSF) using polyarginine-coated nanodiamonds (PA-coated NDs) as affi
28 yarginine peptides showed that inhibition by polyarginine-containing peptides appeared to depend on t
31 caused by mutations in KRT1, showing that a polyarginine frameshift in the keratin-1 tail can also c
32 -Arg-Arg-Arg-Arg-Arg or longer iterations of polyarginine have been shown to be competitive inhibitor
38 everal CPPs in prostate cancer cell lines, a polyarginine peptide (R11) seemed to be the best deliver
40 dentity of the cationic amino acid, with the polyarginine peptide giving the most favourable Delta G
42 ort that the cytosolic penetration of linear polyarginine peptides is dependent on the oxidation stat
44 sidechain binding enthalpies (polyhistidine, polyarginine, polylysine), weighted by numbers of such c
45 ng a fluorogenic assay, we demonstrated that polyarginine potently inhibited substrate-specific prote
46 ) values of five macromolecules: polylysine, polyarginine, protamine, low-density lipoprotein (LDL),
47 ting human immunodeficiency virus-1 and that polyarginine represents a lead example of such inhibitor
50 kable feature of these materials is that the polyarginine segments both direct structure for vesicle
52 cation studies showed that the most potent D-polyarginine tested was nona-D-arginine (D9R) amide with
54 d to determine the important elements within polyarginines that contribute to effective inhibition.
55 ific, as indicated by lack of effects by the polyarginine vehicle alone or a scrambled sequence of th
58 zymes, the reaction rates in the presence of polyarginine were found to be sensitive to the presence
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