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1 s may have predisposed to the development of polyarthritis.
2 thritidis is a rat pathogen causing a severe polyarthritis.
3 animal model of T cell-mediated inflammatory polyarthritis.
4 a natural pathogen of rats, causing an acute polyarthritis.
5 ancer survival in patients with inflammatory polyarthritis.
6 stent disease or disease that evolved into a polyarthritis.
7 patients following the onset of inflammatory polyarthritis.
8  in the cohort of patients with inflammatory polyarthritis.
9  compared with patients without inflammatory polyarthritis.
10 are-based register of new-onset inflammatory polyarthritis.
11 s ranging from severe meningoencephalitis to polyarthritis.
12 ) were at an increased risk for inflammatory polyarthritis.
13 5 years after presentation with inflammatory polyarthritis.
14 reatment, both control dogs developed severe polyarthritis.
15 o present with undifferentiated inflammatory polyarthritis.
16 a variety of autoimmune disorders, including polyarthritis.
17  monitored the impact on SCW-induced erosive polyarthritis.
18 tly reduced the severity of adjuvant-induced polyarthritis.
19 r manifestations in a patient with carcinoma polyarthritis.
20 rder which can be associated with a JRA-like polyarthritis.
21 elationship of their immunodeficiency to the polyarthritis.
22  the rat hindpaw as well as adjuvant-induced polyarthritis (21 d).
23 m tuberculosis H37Ra leads to development of polyarthritis (adjuvant-induced arthritis; AA) that shar
24                     Two of the patients with polyarthritis also had IgA deficiency.
25 om fetal human cartilage induces progressive polyarthritis, an experimental disease similar to human
26              B10.DQ8 transgenic mice develop polyarthritis and Ab's to CII only.
27 experimental polychondritis, exhibiting both polyarthritis and auricular chondritis.
28 ype, mortality in patients with inflammatory polyarthritis and cancer was significantly increased (ha
29  to create a new model of chronic autoimmune polyarthritis and characterize the associated CII-specif
30  this is the first animal model showing both polyarthritis and heart disease as a direct result of TN
31 n a model of Streptococcal cell wall-induced polyarthritis and liver inflammation in rats.
32                             The induction of polyarthritis and polyarthralgia is a hallmark of arthri
33 OR 1.8, 95% CI 1.4-2.4) between inflammatory polyarthritis and the presence of any shared epitope (SE
34 henotypically characterized by granulomatous polyarthritis and uveitis.
35 ll lung carcinoma, the signs and symptoms of polyarthritis and vasculitis had disappeared completely.
36 tis, 45 with rheumatoid factor [RF]-negative polyarthritis, and 21 with systemic disease) were isolat
37 for the systemic, rheumatoid factor-positive polyarthritis, and enthesitis-related arthritis variants
38  ERA, persistent oligoarthritis, RF-negative polyarthritis, and systemic JIA patients, respectively.
39  with persistent oligoarthritis, RF-negative polyarthritis, and systemic JIA subtypes, up-regulation
40 y care-based inception study of inflammatory polyarthritis, and were matched for age and sex to 2 con
41 ch as systemic lupus erythematosus (SLE) and polyarthritis are characterized by chronic cytokine over
42 s that cause weeks to months of debilitating polyarthritis/arthralgia, which is often poorly managed
43                                   Often, the polyarthritis at presentation of SLE cannot be distingui
44 ing 1996, the clinical spectrum of the acute polyarthritis caused by parvovirus B19 was further delin
45 al was reduced in patients with inflammatory polyarthritis compared with patients without inflammator
46 y was rescued by loss of STING function, and polyarthritis completely prevented because cytosolic DNA
47 de thyroid disease, neutrophilic dermatoses, polyarthritis, connective tissue diseases, vasculitis, a
48                            Finally, a severe polyarthritis developed in a majority of HLA-DQ8+,H-2Ab0
49  placebo-controlled trial, SLE patients with polyarthritis, discoid lesions, or pleuritis and/or peri
50  with persistent or extended oligoarthritis, polyarthritis (either rheumatoid factor [RF] positive or
51  (SCW)-induced model of inflammatory erosive polyarthritis, endogenous SLPI was unexpectedly upregula
52 phenotypes of JIA, including oligoarthritis, polyarthritis, enthesitis-related arthritis and psoriati
53 ligoarthritis and rheumatoid factor-negative polyarthritis, erythrocyte sedimentation rate (ESR), C-r
54 matic disease, such as severe polyarthralgia/polyarthritis, for weeks to years.
55 causes a chronic debilitating polyarthralgia/polyarthritis, for which current treatments are often in
56  We recruited 532 patients with inflammatory polyarthritis from the Norfolk Arthritis Register and ty
57 ; treatment differences were greatest in the polyarthritis group.
58                          All 3 patients with polyarthritis had evidence of impaired T cell function.
59                          All 3 patients with polyarthritis had evidence of more extensive immunoregul
60 -dose oral methotrexate (MTX) for persistent polyarthritis has been shown to be effective by the USA/
61 ause mortality in patients with inflammatory polyarthritis (hazard ratio, 1.16 [95% CI, 1.03-1.31], P
62 abetes, synovitis and tenosynovitis (ICD10), polyarthritis (ICD10), neck or shoulder pain, and substa
63 e adjuvant, resulting in a chronic relapsing polyarthritis in >80% of the mice 4 weeks after immuniza
64 s the development of seronegative, symmetric polyarthritis in a patient with relapsing-remitting MS,
65                          We report a case of polyarthritis in a young man with PRP in which magnetic
66  new spontaneous mutation that causes severe polyarthritis in bone phenotype spontaneous mutation 1 (
67 s (CHIKV), an alphavirus that causes chronic polyarthritis in humans and is associated with reservoir
68 avirus that causes epidemics of debilitating polyarthritis in humans.
69   The spontaneous development of destructive polyarthritis in mice transgenic for an autoreactive T-c
70                            The prevalence of polyarthritis in patients with chromosome 22q11.2 deleti
71          Mycoplasma arthritidis causes acute polyarthritis in rats and chronic proliferative arthriti
72                             Adjuvant-induced polyarthritis in rats was used as a model of chronic inf
73       We describe 2 patients presenting with polyarthritis in whom the synovial fluid (1 patient) or
74 elected patients with new-onset inflammatory polyarthritis (IP) has not been examined.
75 f RA in subjects with new-onset inflammatory polyarthritis (IP) over the first 5 years of observation
76 utcome and OC use in women with inflammatory polyarthritis (IP).
77 sability in patients with early inflammatory polyarthritis (IP).
78  patients newly presenting with inflammatory polyarthritis (IP).
79 n together, proteoglycan-induced progressive polyarthritis is dictated by three major components: gen
80 ture regarding IFNalpha- and IFNbeta-induced polyarthritis is reviewed, and possible mechanisms for I
81     When present with inflammatory symmetric polyarthritis, it is pathognomonic of rheumatoid arthrit
82 ne K/BxN arthritis model, a distal symmetric polyarthritis mediated by IgG immune complexes.
83 t patients, ages 18-70, with an inflammatory polyarthritis of <12 months' duration who were recruited
84 f 680 consecutive patients with inflammatory polyarthritis, of whom 404 satisfied the American Colleg
85 iated with an increased risk of inflammatory polyarthritis or rheumatoid arthritis.
86 actor-positive or rheumatoid factor-negative polyarthritis, or systemic JIA without active systemic f
87 ountered pathology in three rodent models of polyarthritis: rat and mouse collagen-induced arthritis,
88 f all patients who had onset of inflammatory polyarthritis (swelling of > or =2 joints) during 1990.
89  joint involvement, small joint involvement, polyarthritis, symmetric arthritis, spinal pain, fever,
90 attle, responsible for chronic pneumonia and polyarthritis syndrome (CPPS).
91 ibes a patient who presented with an unusual polyarthritis syndrome.
92 eumatoid factor (RF)-positive or RF-negative polyarthritis, systemic JIA, or extended oligoarthritis
93                 Furthermore, the progressive polyarthritis that is characteristic in susceptible C3H/
94  twice daily) was a symptomatic inflammatory polyarthritis that persisted for up to 8 weeks after dis
95 yel-KO) mice) triggers a spontaneous erosive polyarthritis that resembles rheumatoid arthritis in pat
96 hritis (RA) causes a symmetric, inflammatory polyarthritis that results in joint destruction and sign
97  long-term inflammatory pain models (CFA and polyarthritis) the same pattern of SP release and SPR ac
98 ith chromosome 22q11.2 deletion syndrome and polyarthritis underwent laboratory evaluations of immuno
99 ures on outcome in patients with unexplained polyarthritis (UPA) (from the early undifferentiated CTD
100 terations in nociceptive pathways induced by polyarthritis using the collagen antibody-induced arthri
101     The risk for development of inflammatory polyarthritis was compared between subjects in the highe
102                           Chronic autoimmune polyarthritis was induced and was dependent on the antig
103                                              Polyarthritis was induced in rats by injection of Freund
104 .7%] of 2400) and rheumatoid factor-negative polyarthritis was more frequent in North America (165 [3
105 ng this cohort of patients with inflammatory polyarthritis was not increased compared with that in th
106 cts who sought primary care for inflammatory polyarthritis were analyzed.
107  and 2002, 88 new patients with inflammatory polyarthritis were identified and matched with 176 contr
108      Patients with new cases of inflammatory polyarthritis were identified by linkage with the Norfol
109 iting toxicity (DLT) was severe inflammatory polyarthritis, which seemed to be a cumulative toxicity.
110 neally with pristane oil to induce a chronic polyarthritis, which was monitored by visual scoring.
111 eumatoid arthritis is a chronic inflammatory polyarthritis whose etiology remains uncertain.
112  allows differentiation of presentation of a polyarthritis with a good prognosis (spondyloarthropathy
113 l lesions, whereas all control dogs had mild polyarthritis with periarteritis.
114 IA) represents an animal model of autoimmune polyarthritis with significant similarities to human rhe
115 IA) represents an animal model of autoimmune polyarthritis with similarities to human rheumatoid arth
116 al polychondritis, auricular chondritis, and polyarthritis, with clinical and histological similariti

 
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