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1 the aortitis of Cogan syndrome and relapsing polychondritis.
2 similar to those symptoms in human relapsing polychondritis.
3 important in susceptibility to experimental polychondritis.
4 maging assessment in patients with relapsing polychondritis.
6 is also a frequent target organ in relapsing polychondritis, and proptosis is a well-recognized manif
7 nd histologic manifestations of experimental polychondritis are similar to those symptoms in human re
8 anisms of pathogenesis involved in relapsing polychondritis as well as the influence of the expressio
9 of NOD.DQ8 mice develop severe experimental polychondritis, auricular chondritis, and polyarthritis,
11 habeta tg mice developed severe experimental polychondritis, exhibiting both polyarthritis and auricu
12 om malignant transformation of the relapsing polychondritis-induced inflammatory pseudotumor and emph
19 a man who was diagnosed as having relapsing polychondritis (RP) when he was 18 years of age and was
22 ' experience with 36 patients with relapsing polychondritis who were followed from 1980 to 1997, 30 p
23 t in the majority of patients with relapsing polychondritis who were referred for airway imaging, yet
24 nically diagnosed or biopsy-proved relapsing polychondritis who were referred for CT airway imaging d
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