ライフサイエンスコーパス: polymeric IgA

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1 uch as agalactosylated glycoforms of IgG and polymeric IgA.

2 lumen, causing a dramatic increase in serum polymeric IgA.

3 nonmucosal VRP delivery, J chain-containing, polymeric IgA Abs were detected in the peripheral draini

4 nal anti-hemagglutinin IgG1, IgG2a, IgM, and polymeric IgA Abs were equally effective in preventing i

5 ice, which lack the receptor that transports polymeric IgA across the mucosal epithelium where it is

6 he polymeric receptor (pIgR) is to transport polymeric IgA across various mucosal epithelial layers.

7 ic immunoglobulin receptor (pIgR) transports polymeric IgA and IgM across epithelia to mucosal secret

8 cosal immunity that mediates the delivery of polymeric IgA and IgM to the apical surface of epithelia

9 produced upon association of plasma-derived polymeric IgA and IgM with a recombinant secretory compo

10 s a small polypeptide covalently attached to polymeric IgA and IgM.

11 ed accumulation of basolaterally endocytosed polymeric IgA and the polymeric IgA receptor in the peri

12 IgA is represented by secretory IgA, polymeric IgA, and monomeric IgA.

13 Sal4 is a monoclonal polymeric IgA antibody directed against the O antigen (O

14 al cells is inhibited by Sal4, a monoclonal, polymeric IgA antibody that binds an immunodominant epit

15 In contrast, polymeric IgA exists mostly as dimers with J chain.

16 Here we show that the polymeric IgA form of anti-HIV antibody inhibits HIV muc

17 main criteria are used to show that labeled polymeric IgA-ligand-receptor complexes are organized in

18 The persistence of polymeric IgA may facilitate binding and clearance of pn

19 distinct in the production of predominantly polymeric IgA over time.

20 e mucosal surface occurs via transcytosis of polymeric IgA (pIgA) across the epithelium, a process me

21 Polymeric IgA (pIgA) is transcytosed by the pIgA recepto

22 enza virus IgG and i.v. anti-influenza virus polymeric IgA (pIgA) mAb administered in amounts designe

23 this work, we sought to investigate whether polymeric IgA (pIgA) recovered from human plasma is able

24 capsule-specific IgA (monomeric IgA [mIgA], polymeric IgA [pIgA], and secretory IgA [SIgA]) on OPC a

25 using markers such as transferrin (TfR) and polymeric IgA (pIgR) receptors.

26 ase release of secretory component (SC), the polymeric IgA (plgA)-binding segment of the plgA recepto

27 ccumulation of transcytotic carriers for the polymeric IgA receptor (pIgA-R) and redistribution of se

28 ere enriched in the transcytosed form of the polymeric IgA receptor (pIgA-R), but lacked not only the

29 GPI-anchored and single TMD apical proteins; polymeric IgA receptor (pIgA-R), polytopic apical, and b

30 ssential for the cell-free reconstitution of polymeric IgA receptor (pIgA-R)-containing exocytic tran

31 ned not only ER markers (105-kDa form of the polymeric IgA receptor (pIgAR)) but also transcytotic ma

32 n polarized MDCK cells stably expressing the polymeric IgA receptor caused accumulation of basolatera

33 olaterally endocytosed polymeric IgA and the polymeric IgA receptor in the pericentrosomal region.

34 ns aminopeptidase N, 5'nucleotidase, and the polymeric IgA receptor were efficiently transcytosed.

35 sly, FRET confocal microscopy has shown that polymeric IgA-receptor (pIgA-R) is distributed in a clus

36 Polymeric IgA RV antibodies mediated an inhibitory effec

37 mponent, the cleaved epithelial receptor for polymeric IgA, was secreted in a pattern very similar to

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