ライフサイエンスコーパス: polymorphonuclear

1 The infiltration of polymorphonuclear and macrophage cells was associated wi

2 m of ICOS (ICOS-Fc) inhibits the adhesion of polymorphonuclear and tumor cell lines to HUVECs; thus,

3 ere immunostained for IL-6R, gp130, CD15(+) (polymorphonuclear), and CD3(+) (T cell) inflammatory cel

4 D11b+CD33+) phenotype and a subpopulation of polymorphonuclear CD11b+CD33+CD15+ cells.

5 nstrated that Kupffer cell depletion reduces polymorphonuclear cell (neutrophil) (PMN) and matrix met

6 human leukocytes to purified Pic resulted in polymorphonuclear cell activation, but impaired chemotax

7 coli, an abnormal ultrasonographic finding, polymorphonuclear cell count of greater than 60%, C-reac

8 Polymorphonuclear cell Ecto expressed MHC class I and in

9 els of proinflammatory mediators (decreasing polymorphonuclear cell infiltration), increasing anti-in

10 Enriched NPs dramatically reduced polymorphonuclear cell influx in murine peritonitis, sho

11 es in vivo and platelet/P-selectin-dependent polymorphonuclear cell migration in vitro were exclusive

12 reased IL-1beta and MIP-2 proteins, reducing polymorphonuclear cell number in the infected cornea.

13 yanidin also decreased KC concentrations and polymorphonuclear cell recruitment in bronchoalveolar la

14 Platelet, polymorphonuclear cell, mononuclear cell, and erythrocyt

15 acrine manner in the suppressive activity of polymorphonuclear cell-derived Ecto.

16 n comparison with ADCC by mononuclear cells, polymorphonuclear cell-mediated ADCC and complement-depe

17 Polymorphonuclear cells (neutrophils) are the first cell

18 eptible to phagocytosis and killing by human polymorphonuclear cells (P = 0.01 and P = 0.006, respect

19 In 22/29 cases of active cryptitis, polymorphonuclear cells (PMN) clearly expressed HSP70i,

20 The engulfment of apoptotic polymorphonuclear cells (PMN) during the resolution of i

21 or cells such as mononuclear cells (MNC) and polymorphonuclear cells (PMN) to exert Ab-dependent cell

22 CD97 was upregulated on polymorphonuclear cells (PMNC) of patients with psoriasi

23 loroquine (CQ) on the antifungal capacity of polymorphonuclear cells (PMNs) and on the inflammatory r

24 formed to determine the total levels of oral polymorphonuclear cells (PMNs) before and 3 months after

25 BLP activated human polymorphonuclear cells (PMNs) ex vivo to adhere to dena

26 In inflammation, they activate and recruit polymorphonuclear cells (PMNs) through binding of the ch

27 ers in the lung, a robust alveolar influx of polymorphonuclear cells (PMNs), and a risk of systemic s

28 addition, in the presence of complement and polymorphonuclear cells (PMNs), antibodies to Ata were h

29 We have previously reported that neutrophil (polymorphonuclear cells [PMNs]) accumulation in culprit

30 robust alveolar infiltration of neutrophils (polymorphonuclear cells [PMNs]) that can promote systemi

31 te flow, clearance of apoptotic neutrophils (polymorphonuclear cells [PMNs]), production of specializ

32 nfiltration, consisting of mainly neutrophil polymorphonuclear cells and monocytes/macrophages, cente

33 ages and dendritic cells, and recruitment of polymorphonuclear cells are likely to contribute to this

34 totoxicity (ADCC) by NK cells, monocytes, or polymorphonuclear cells as well as complement-dependent

35 2, 3, and 8, and E-NPP1, 2, and 3, in their polymorphonuclear cells by immunofluorescence and qPCR.

36 NA) in THP-1 (human monocytic cell line) and polymorphonuclear cells from patients with sepsis enhanc

37 nflammatory genes, decreased infiltration of polymorphonuclear cells into the T-cell zone of lymphoid

38 gocytosis of platelets using FcgammaRIIIb(+) polymorphonuclear cells or FcgammaRIIIa(+) monocytes as

39 e >40 years, African American race, and >/=5 polymorphonuclear cells per high-power field on urethral

40 ondition where macrophages, eosinophils, and polymorphonuclear cells play an important role in its pa

41 Properdin released from human polymorphonuclear cells stimulated with PMA did not bind

42 the migration of IL-17(+)CD8(+) T cells and polymorphonuclear cells to infected tissues.

43 tion of c-Jun, leading to the recruitment of polymorphonuclear cells to the cochlea.

44 Furthermore, secondary tethering of polymorphonuclear cells was blocked by DREG-200 but sign

45 The generation of reactive oxygen species by polymorphonuclear cells was significantly lower when ora

46 city (ADCC) by isolated monocytes, activated polymorphonuclear cells, and human whole blood.

47 In the presence of complement and polymorphonuclear cells, antisera to 9Glc-NH(2)-TT media

48 lates in phagocytes, monocytes, fibroblasts, polymorphonuclear cells, macrophages, and lymphocytes.

49 Also, the islets of Langerhans attracted polymorphonuclear cells, possibly via release of IL-6, I

50 At day 3, the grafts were surrounded by polymorphonuclear cells, which were replaced by a notabl

51 such as natural killer cells, monocytes, and polymorphonuclear cells.

52 influenzae-induced cochlear infiltration of polymorphonuclear cells.

53 as, sclerosis of the surrounding tissue, and polymorphonuclear cells.

54 uction of TRAIL, and reduced infiltration of polymorphonuclear cells.

55 parent differences between splenic and tumor polymorphonuclear cells/granulocytic myeloid-derived sup

56 ion would enable gonococci to survive within polymorphonuclear cells; however, an active LgtD in a fe

57 to monocytic (mononuclear) and granulocytic (polymorphonuclear) cells using the Ly6C and Ly6G markers

58 Using resting human polymorphonuclear granulocytes (PMN) from peripheral blo

59 alpha, Il-1beta, and Il-6 and attenuation of polymorphonuclear inflammatory cells into the placental

60 Recently, we demonstrated that blood polymorphonuclear leucocytes (PMNs) in ARDS are basally

61 accompanied by impaired defense functions of polymorphonuclear leucocytes (PMNs), increased patient s

62 pheral insulin resistance and alterations in polymorphonuclear leukocyte (PML) function.

63 ids participate in the suppression of murine polymorphonuclear leukocyte (PMN) bactericidal activitie

64 In vitro assays with human Polymorphonuclear leukocyte (PMN) demonstrated that CFTR

65 (2) formation was analyzed by lipidomics and polymorphonuclear leukocyte (PMN) infiltration quantifie

66 Polymorphonuclear leukocyte (PMN) is the predominant inn

67 Polymorphonuclear leukocyte (PMN) migration across the i

68 eltaybcL) failed to suppress transepithelial polymorphonuclear leukocyte (PMN) migration in vitro, a

69 ation but had no effect on bacterial load or polymorphonuclear leukocyte (PMN) numbers in the lung.

70 es (CORMs) suppress inflammation by reducing polymorphonuclear leukocyte (PMN) recruitment to the aff

71 lular adhesion molecule 1 (ICAM-1)-dependent polymorphonuclear leukocyte (PMN) recruitment, functiona

72 n species (ROS) are critical for neutrophil (polymorphonuclear leukocyte (PMN)) microbicidal function

73 rate that epinephrine alters the neutrophil (polymorphonuclear leukocyte (PMN))-dependent inflammator

74 ant throughout the observation period, while polymorphonuclear leukocyte (PMN), S100A8, and interleuk

75 entify novel virulence factors in evasion of polymorphonuclear leukocyte (PMN)-mediated innate immuni

76 Neutrophil [polymorphonuclear leukocyte (PMN)] transepithelial migra

77 y with IL-1beta to promote early neutrophil (polymorphonuclear leukocyte [PMN]) recruitment.

78 In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were

79 but reduced cytokines/chemokines as well as polymorphonuclear leukocyte and macrophage numbers.

80 r early source of chemokines associated with polymorphonuclear leukocyte and monocyte/macrophage infi

81 blood counts were performed and examined for polymorphonuclear leukocyte content.

82 The most polymorphonuclear leukocyte infiltration in periodontal

83 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infiltration in response to

84 ion, increased cell apoptosis, and decreased polymorphonuclear leukocyte infiltration in the healing

85 phagocytosis of Escherichia coli, decreased polymorphonuclear leukocyte infiltration, and counter-re

86 attenuated pulmonary membrane thickening and polymorphonuclear leukocyte infiltration, reduced NF-kap

87 ls of erythrocyte lysis, resistance to human polymorphonuclear leukocyte killing, and pathogenesis in

88 We show that the specific immunodepletion of polymorphonuclear leukocyte neutrophils using anti-Ly6G

89 ng inflammation, characterized by increasing polymorphonuclear leukocyte recruitment, is a major caus

90 s, but PG formation was not even detected in polymorphonuclear leukocyte supernatants from control mi

91 ntercellular adhesion molecule-1 expression, polymorphonuclear leukocyte transendothelial migration,

92 MRSA exotoxins also caused neutrophil (polymorphonuclear leukocyte) activation, as measured by

93 volving either platelet-monocyte or platelet-polymorphonuclear leukocyte.

94 Neutrophil (polymorphonuclear leukocyte; PMN) inflammatory functions

95 cytosis of G. bethesdensis by normal and CGD polymorphonuclear leukocytes (CGD PMN) required heat-lab

96 ce expressing human alpha-defensins in their polymorphonuclear leukocytes (Def(+/+)).

97 Polymorphonuclear leukocytes (neutrophils) are the prima

98 trates the epidermis and reaches the dermis, polymorphonuclear leukocytes (PMLs) accumulate and an ab

99 gue associated with extensive recruitment of polymorphonuclear leukocytes (PMN or neutrophils) to the

100 Polymorphonuclear leukocytes (PMN) achieve an intermedia

101 Polymorphonuclear leukocytes (PMN) are the first respond

102 Polymorphonuclear leukocytes (PMN) from patients with ch

103 tussis is able to avoid bacterial killing by polymorphonuclear leukocytes (PMN) if specific opsonic a

104 expression in human uroepithelial cells and polymorphonuclear leukocytes (PMN) in vitro and in bladd

105 flammation is traditionally characterized by polymorphonuclear leukocytes (PMN) influx followed by ph

106 Transendothelial migration (TEM) of polymorphonuclear leukocytes (PMN) involves a carefully

107 ng upon this model, we investigated the role polymorphonuclear leukocytes (PMN) play in the control o

108 xperiments, conditioned media collected from polymorphonuclear leukocytes (PMN) selectively increased

109 . bethesdensis resists killing by serum, CGD polymorphonuclear leukocytes (PMN), and antimicrobial pe

110 dal polypeptides secreted by macrophages and polymorphonuclear leukocytes (PMN).

111 , sustained recruitment to the lymph node of polymorphonuclear leukocytes (PMN, or neutrophils), the

112 st immune responses, including the influx of polymorphonuclear leukocytes (PMN; neutrophils).

113 by the provider, who recorded the number of polymorphonuclear leukocytes (PMNLs) per epithelial cell

114 The migration of polymorphonuclear leukocytes (PMNs) across the intestina

115 We hypothesized that polymorphonuclear leukocytes (PMNs) and less mature myel

116 nstrate its ability in separating/recovering polymorphonuclear leukocytes (PMNs) and mononuclear leuk

117 s a host foreign body response, during which polymorphonuclear leukocytes (PMNs) and then monocytes (

118 Polymorphonuclear leukocytes (PMNs) are innate immune ce

119 e hypersensitivity and autoimmunity in which polymorphonuclear leukocytes (PMNs) are involved.

120 Polymorphonuclear leukocytes (PMNs) are key in innate im

121 e, we demonstrate that RvD1 actions on human polymorphonuclear leukocytes (PMNs) are pertussis toxin

122 Y. pestis and that CD11b(+) cells other than polymorphonuclear leukocytes (PMNs) are selectively lost

123 oeae recruits and interacts extensively with polymorphonuclear leukocytes (PMNs) during infection.

124 We tested cytotoxicity of LukGH toward polymorphonuclear leukocytes (PMNs) from mice, rabbits,

125 progenitor cells (HSPCs) differentiate into polymorphonuclear leukocytes (PMNs) in the bone marrow.

126 ntratracheal administration of LPS increased polymorphonuclear leukocytes (PMNs) in the bronchoalveol

127 f pathogenesis of pneumonia is the influx of polymorphonuclear leukocytes (PMNs) into the lungs.

128 Massive infiltration of polymorphonuclear leukocytes (PMNs) to the corneal endot

129 nococcus), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

130 A few polymorphonuclear leukocytes (PMNs) were already present

131 On necropsy, a large number of necrotic polymorphonuclear leukocytes (PMNs) were observed in the

132 ite the fundamental function of neutrophils (polymorphonuclear leukocytes (PMNs)) in innate immunity,

133 ing in septic plasma required C5a receptors, polymorphonuclear leukocytes (PMNs), and the Nacht-, LRR

134 Neutrophil granulocytes, also called polymorphonuclear leukocytes (PMNs), extrude molecular l

135 Our method reproducibly recovers 94.0% of polymorphonuclear leukocytes (PMNs), with up to 10(5) PM

136 , as they prevented an influx of Siglec-F(+) polymorphonuclear leukocytes (PMNs).

137 din (PVL), a pore-forming toxin that targets polymorphonuclear leukocytes (PMNs).

138 infections depends on bacterial clearance by polymorphonuclear leukocytes (PMNs); however, excessive

139 hemorrhage, and accumulation of neutrophils [polymorphonuclear leukocytes (PMNs)] in the alveolar com

140 Neutrophil (polymorphonuclear leukocytes [PMN]) infiltration plays a

141 ed to increased infiltration of neutrophils (polymorphonuclear leukocytes [PMN]), macrophages (MPhi),

142 Human neutrophils (polymorphonuclear leukocytes [PMNs]) generate inflammato

143 n to avoid destruction by human neutrophils (polymorphonuclear leukocytes [PMNs]), which are crucial

144 n by human epithelial cells and neutrophils (polymorphonuclear leukocytes [PMNs]).

145 ous system (CNS) include infiltrating cells (polymorphonuclear leukocytes [PMNs], macrophages, and na

146 Human polymorphonuclear leukocytes adhesion to endothelial cel

147 associated with decreases in infiltration of polymorphonuclear leukocytes and activation of microglia

148 re susceptible to oxidative killing by human polymorphonuclear leukocytes and displays decreased tiss

149 At 4 h, more polymorphonuclear leukocytes and fewer CD11c(+) cells we

150 Munro's microabscesses contain polymorphonuclear leukocytes and form specifically in th

151 Primary polymorphonuclear leukocytes and keratinocytes were used

152 ment and reduced intrahepatic recruitment of polymorphonuclear leukocytes and NKT cells after islet i

153 Measurement of sputum polymorphonuclear leukocytes and other analytes, cortiso

154 it both apoptosis and the oxidative burst in polymorphonuclear leukocytes but were unable to identify

155 labeled bacteria was tested in primary mouse polymorphonuclear leukocytes by flow cytometry.

156 blood and during phagocytic interaction with polymorphonuclear leukocytes ex vivo We conclude that Fa

157 .)-deficient monocytes, dendritic cells, and polymorphonuclear leukocytes from gp91(PHOX)- or p47(PHO

158 f the bacteria compared with macrophages and polymorphonuclear leukocytes from wild-type mice in whic

159 , and decreases interstitial infiltration of polymorphonuclear leukocytes in a mouse model of TNF-alp

160 The transepithelial migration of polymorphonuclear leukocytes in response to chemokine ex

161 Large numbers of polymorphonuclear leukocytes in the amnion and chorion d

162 uired to counter toxic effectors secreted by polymorphonuclear leukocytes in the tissues.

163 ice had increased efferocytosis of apoptotic polymorphonuclear leukocytes instilled into the peritone

164 act of fruit reduced the edema, migration of polymorphonuclear leukocytes into the peritoneal cavity,

165 Peritoneal macrophages and polymorphonuclear leukocytes isolated from iNOS-/- mice

166 timulated COX-2 expression was suppressed in polymorphonuclear leukocytes isolated from MacKOs, but P

167 Neutrophils are polymorphonuclear leukocytes of the phagocytic system th

168 with NGU (visible urethral discharge or >/=5 polymorphonuclear leukocytes per high-power field [PMNs/

169 The total number of bronchoalveolar lavage polymorphonuclear leukocytes recovered from the mice exp

170 Bacterial invasion of host tissues triggers polymorphonuclear leukocytes to release DNA [neutrophil

171 iae-infected Mmp2/9(-/-) mice recruited more polymorphonuclear leukocytes to the lung but had higher

172 ies of AnxA1 become operative to finely tune polymorphonuclear leukocytes transmigration to the site

173 ly, specific immunodepletion of neutrophils (polymorphonuclear leukocytes) blocked hCNS-SCns astrogli

174 ption factor FOXO1, and increased numbers of polymorphonuclear leukocytes, all of which were signific

175 ular phagocytosis with mouse NK cells, mouse polymorphonuclear leukocytes, and mouse macrophages.

176 reus abscesses, including the involvement of polymorphonuclear leukocytes, and provide a brief overvi

177 n ligand uptake or C5aR endocytosis in human polymorphonuclear leukocytes, distinguishing its role fr

178 gion increase resistance to killing by human polymorphonuclear leukocytes, increase bacterial prolife

179 In polymorphonuclear leukocytes, SipA or other Salmonella p

180 the assembly of the LT synthetic complex in polymorphonuclear leukocytes.

181 ed potent ADCC with mouse NK cells and mouse polymorphonuclear leukocytes.

182 the alpha-subunit after G-CSF expression on polymorphonuclear leukocytes.

183 P12-associating lectin-1 (MDL-1) on immature polymorphonuclear leukocytes.

184 strong activity for stimulating migration of polymorphonuclear leukocytes.

185 ive inflammation with granulation tissue and polymorphonuclear leukocytes.

186 enuated for survival upon challenge by human polymorphonuclear leukocytes.

187 ls (PMVECs), alveolar macrophages (AMs), and polymorphonuclear leukocytes.

188 nuated in ability to resist killing by human polymorphonuclear leukocytes.

189 n, epithelial exfoliation, and the influx of polymorphonuclear leukocytes.

190 NO is produced by polymorphonuclear lymphocytes (PMNs) and cervical endoth

191 or-bearing mice with reduced accumulation of polymorphonuclear MDSC but not monocytic MDSC (M-MDSC),

192 t not monocytic MDSC (M-MDSC), and decreased polymorphonuclear MDSC suppression in vitro through the

193 cells (MDSCs), monocytic MDSCs (M-MDSCs) and polymorphonuclear MDSCs (PMN-MDSCs) regulate immune resp

194 t also an accumulation of tumor-infiltrating polymorphonuclear mononuclear cells caused by Cxcl-1 rel

195 adenosine triphosphate (ATP) contributes to polymorphonuclear neutrophil (PMN) activation leading to

196 iphosphate (ATP) and adenosine in regulating polymorphonuclear neutrophil (PMN) activation, fibrin fo

197 Myeloperoxidase (MPO) is released during polymorphonuclear neutrophil (PMN) degranulation, and me

198 Polymorphonuclear neutrophil (PMN) extravasation require

199 To determine whether platelet and polymorphonuclear neutrophil (PMN) functions require spe

200 metabolite, adenosine, are key regulators of polymorphonuclear neutrophil (PMN) functions.

201 cin treatment also increased clinical score, polymorphonuclear neutrophil (PMN) infiltration (determi

202 heral organs in the two groups, WNV-infected polymorphonuclear neutrophil (PMN) infiltration and vira

203 onfirmed with synthetic MaR1, i.e., limiting polymorphonuclear neutrophil (PMN) infiltration in murin

204 r to clinical observations, a robust vaginal polymorphonuclear neutrophil (PMN) migration occurs in a

205 observations revealed that a robust vaginal polymorphonuclear neutrophil (PMN) migration occurs in s

206 in response to C. albicans, which stimulate polymorphonuclear neutrophil (PMN) migration to the vagi

207 of the junctional epithelium (P < 0.01) and polymorphonuclear neutrophil (PMN) recruitment (P < 0.00

208 -surface protein, plays an important role in polymorphonuclear neutrophil (PMN) transmigration across

209 tly expressed on pulmonary epithelium and on polymorphonuclear neutrophil (PMNs) after transepithelia

210 tic ketoacidosis is associated with systemic polymorphonuclear neutrophil activation and degranulatio

211 2-triggered neutrophilia and monocytosis and polymorphonuclear neutrophil and monocyte renal infiltra

212 Of all the polymorphonuclear neutrophil azurophilic enzymes examine

213 , possibly due to the release of destructive polymorphonuclear neutrophil azurophilic enzymes.

214 Polymorphonuclear neutrophil granulocytes (neutrophils)

215 translates into increased transmigration of polymorphonuclear neutrophil granulocytes across endothe

216 demonstrated an attenuated extravasation of polymorphonuclear neutrophil granulocytes in Sema7a-defi

217 of Blood, Sapey et al. report that the human polymorphonuclear neutrophil leukocyte (or neutrophil) u

218 endothelial cells that appear essential for polymorphonuclear neutrophil recruitment in IC disease.

219 To test individual mosaic subpopulations of polymorphonuclear neutrophil responses, we also develope

220 Interestingly, C5a-stimulated endothelial polymorphonuclear neutrophil transmigration, but not che

221 Polymorphonuclear neutrophilic granulocytes (PMN) as cel

222 h reactive oxygen species production by oral polymorphonuclear neutrophils (oPMNs) in chronic periodo

223 /80(-)I-A(-) intraglomerular macrophages and polymorphonuclear neutrophils (PMN) are important in ind

224 Polymorphonuclear neutrophils (PMN) are potent inflammat

225 Polymorphonuclear neutrophils (PMN) have distinct phenot

226 Because polymorphonuclear neutrophils (PMN) interaction with ECs

227 Polymorphonuclear neutrophils (PMN) play a central role

228 e of alphaMbeta2 affected the recruitment of polymorphonuclear neutrophils (PMN) to bacterial and fun

229 t-mediated phagocytosis and firm adhesion of polymorphonuclear neutrophils (PMN) under physiological

230 is expressed on human leukocytes, including polymorphonuclear neutrophils (PMN), monocytes, and macr

231 iated with significant tumor infiltration by polymorphonuclear neutrophils (PMN).

232 how that stimulation of alpha7nAChR on human polymorphonuclear neutrophils (PMNs) and blood mononucle

233 its ligand, CXCL12, mediate the retention of polymorphonuclear neutrophils (PMNs) and hematopoietic s

234 vitro model of transepithelial migration of polymorphonuclear neutrophils (PMNs) and in human intest

235 mice with ALI had greater increases in lung polymorphonuclear neutrophils (PMNs) and macrophage coun

236 Polymorphonuclear neutrophils (PMNs) and macrophages are

237 Polymorphonuclear neutrophils (PMNs) and NK cells are bo

238 Polymorphonuclear neutrophils (PMNs) are critical for th

239 Polymorphonuclear neutrophils (PMNs) are innate effector

240 Polymorphonuclear neutrophils (PMNs) are innate immune s

241 Polymorphonuclear neutrophils (PMNs) are largely conside

242 During pneumonia, polymorphonuclear neutrophils (PMNs) are recruited by ch

243 this infection METHODS: To determine whether polymorphonuclear neutrophils (PMNs) are required for di

244 Notably, polymorphonuclear neutrophils (PMNs) can capture circula

245 Human polymorphonuclear neutrophils (PMNs) counteracted the pr

246 its functional relevance for recruitment of polymorphonuclear neutrophils (PMNs) during acute inflam

247 Trafficking of polymorphonuclear neutrophils (PMNs) during inflammation

248 Polymorphonuclear neutrophils (PMNs) express a number of

249 Polymorphonuclear neutrophils (PMNs) form the first line

250 nfection in skin involves the recruitment of polymorphonuclear neutrophils (PMNs) from the bone marro

251 Polymorphonuclear neutrophils (PMNs) have previously bee

252 uces pro-inflammatory cytokine production by polymorphonuclear neutrophils (PMNs) of diabetic individ

253 DGKalpha function is altered in polymorphonuclear neutrophils (PMNs) of patients with lo

254 unexpected activity of a CD49d(+) subset of polymorphonuclear neutrophils (PMNs) that are found in t

255 Bone marrow (BM) holds a large reserve of polymorphonuclear neutrophils (PMNs) that are rapidly mo

256 These activate human polymorphonuclear neutrophils (PMNs) through formyl pept

257 Recruitment of polymorphonuclear neutrophils (PMNs) to sites of acute i

258 ammatory cytokines followed by attraction of polymorphonuclear neutrophils (PMNs) to the site of infl

259 During inflammation polymorphonuclear neutrophils (PMNs) traverse venular wa

260 Eighty-six genes in WG PBMCs and 40 in WG polymorphonuclear neutrophils (PMNs) were significantly

261 count for this absence, we hypothesized that polymorphonuclear neutrophils (PMNs), recruited massivel

262 ADAM9 is also a product of human and murine polymorphonuclear neutrophils (PMNs).

263 ogous to CD16A and a major FcgammaR on human polymorphonuclear neutrophils (PMNs).

264 Neutrophils (polymorphonuclear neutrophils [PMNs]) play an elaborate

265 cluded albumin leak into lung and buildup of polymorphonuclear neutrophils and cytokines/chemokines i

266 With human polymorphonuclear neutrophils and macrophages, RvD1, RvD

267 icates the inflammatory response mediated by polymorphonuclear neutrophils and monocytes as the key e

268 hlamydia-specific immunoglobulin G (IgG) and polymorphonuclear neutrophils and show the importance of

269 Polymorphonuclear neutrophils constitute the first line

270 Infiltration of polymorphonuclear neutrophils into the lung is an import

271 ed as visible urethral discharge and/or >/=5 polymorphonuclear neutrophils per high-powered field; n

272 Blood polymorphonuclear neutrophils provide immune protection

273 whereas adoptive transfer of MD-2-competent polymorphonuclear neutrophils restored it.

274 te inflammation by blocking the migration of polymorphonuclear neutrophils to initiate resolution.

275 Elevated counts of polymorphonuclear neutrophils were detectable at 15 minu

276 Myeloperoxidase, a heme enzyme released by polymorphonuclear neutrophils, accumulates within ischem

277 ing the migration of monocytes, neutrophils, polymorphonuclear neutrophils, and dendritic cells into

278 d Y. pestis' ability to counter lysozyme and polymorphonuclear neutrophils, but it did not affect the

279 and possibly inflammatory monocytes, but not polymorphonuclear neutrophils, in clearing HSV-1 from th

280 igration process of host inflammatory cells (polymorphonuclear neutrophils, macrophages) into the inf

281 the adhesion of a type of white blood cell--polymorphonuclear neutrophils--to the lining of the circ

282 profile and impaired antifungal activity of polymorphonuclear neutrophils.

283 rs (degree of fibrosis) and concentration of polymorphonuclear neutrophils.

284 ollagenase thought to be expressed mainly by polymorphonuclear neutrophils.

285 is and reactive oxygen species generation in polymorphonuclear neutrophils.

286 sis in children was associated with elevated polymorphonuclear neutrophils.

287 that this oxysterol increases the number of polymorphonuclear-neutrophils and gammadelta-T cells at

288 tions of 27-hydroxycholesterol requires both polymorphonuclear-neutrophils and gammadelta-T cells, an

289 hrough its actions on gammadelta-T cells and polymorphonuclear-neutrophils, 27-hydroxycholesterol fun

290 CD11b(+) Ly6C(int) Ly6G(+) cells exhibited a polymorphonuclear or band-shaped nuclear morphology.

291 decreased accumulation and/or activation of polymorphonuclear (PMN) and CD4(+) and CD8(+) T cells, a

292 epithelial cells (SECs) and high numbers of polymorphonuclear (PMN) cells are regarded as indicative

293 neal epithelial wound closure and attenuated polymorphonuclear (PMN) leukocyte responses, a phenotype

294 antitative and qualitative reconstitution of polymorphonuclear (PMN), CD4, CD8, and natural killer (N

295 we demonstrate that monocytic (M)-MDSCs and polymorphonuclear (PMN)-MDSCs can be detected using seve

296 consist of two major subsets, monocytic and polymorphonuclear (PMN).

297 receptor I (FcgammaRI) CD64 on neutrophils (polymorphonuclear [PMN] CD64 index) in flow cytometry wa

298 ver disease (ALD) with intense neutrophilic (polymorphonuclear; PMN) inflammation and high mortality.

299 eratinocyte-derived cytokine measurement and polymorphonuclear recruitment in bronchoalveolar lavage

300 indicate that properdin in serum as well as polymorphonuclear-released properdin is unable to bind a