ライフサイエンスコーパス: polymorphonuclear leukocyte

1 volving either platelet-monocyte or platelet-polymorphonuclear leukocyte.

2 enuated for survival upon challenge by human polymorphonuclear leukocytes.

3 ive inflammation with granulation tissue and polymorphonuclear leukocytes.

4 F1 decreases the antimicrobial activities of polymorphonuclear leukocytes.

5 nuated in ability to resist killing by human polymorphonuclear leukocytes.

6 ansfected HEK293T cells and peripheral blood polymorphonuclear leukocytes.

7 ct peribronchial infiltrate of predominantly polymorphonuclear leukocytes.

8 ls (PMVECs), alveolar macrophages (AMs), and polymorphonuclear leukocytes.

9 tage to the bacteria in evading infiltrating polymorphonuclear leukocytes.

10 n characterized by inflammation dominated by polymorphonuclear leukocytes.

11 n, epithelial exfoliation, and the influx of polymorphonuclear leukocytes.

12 the assembly of the LT synthetic complex in polymorphonuclear leukocytes.

13 ed potent ADCC with mouse NK cells and mouse polymorphonuclear leukocytes.

14 the alpha-subunit after G-CSF expression on polymorphonuclear leukocytes.

15 P12-associating lectin-1 (MDL-1) on immature polymorphonuclear leukocytes.

16 strong activity for stimulating migration of polymorphonuclear leukocytes.

17 MRSA exotoxins also caused neutrophil (polymorphonuclear leukocyte) activation, as measured by

18 olymerase chain reaction on peripheral blood polymorphonuclear leukocytes, acute rejection, and cardi

19 In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were

20 Human polymorphonuclear leukocytes adhesion to endothelial cel

21 ption factor FOXO1, and increased numbers of polymorphonuclear leukocytes, all of which were signific

22 The observed dynamics in polymorphonuclear leukocyte alterations in peripheral bl

23 but reduced cytokines/chemokines as well as polymorphonuclear leukocyte and macrophage numbers.

24 H. ducreyi evades uptake by polymorphonuclear leukocyte and macrophage-like cell lin

25 r early source of chemokines associated with polymorphonuclear leukocyte and monocyte/macrophage infi

26 y increased and prolonged the formation of a polymorphonuclear leukocyte and mononuclear cell infiltr

27 associated with decreases in infiltration of polymorphonuclear leukocytes and activation of microglia

28 subsequent impairment of lung recruitment of polymorphonuclear leukocytes and clearance of Streptococ

29 re susceptible to oxidative killing by human polymorphonuclear leukocytes and displays decreased tiss

30 At 4 h, more polymorphonuclear leukocytes and fewer CD11c(+) cells we

31 Munro's microabscesses contain polymorphonuclear leukocytes and form specifically in th

32 he SBA assay, OMV ELISA, and OPA using human polymorphonuclear leukocytes and human complement but no

33 Primary polymorphonuclear leukocytes and keratinocytes were used

34 Haemophilus ducreyi colocalizes with polymorphonuclear leukocytes and macrophages and evades

35 onstrated that the expression of FcRgamma on polymorphonuclear leukocytes and monocytes was not requi

36 ment and reduced intrahepatic recruitment of polymorphonuclear leukocytes and NKT cells after islet i

37 Measurement of sputum polymorphonuclear leukocytes and other analytes, cortiso

38 ROS generation by mononuclear cells and polymorphonuclear leukocytes and p47(phox) expression in

39 nsins are abundant antimicrobial peptides in polymorphonuclear leukocytes and play an important role

40 f CD11b adhesion molecules on the surface of polymorphonuclear leukocytes and status of iron-transfer

41 ROS generation by mononuclear cells and polymorphonuclear leukocytes and the expression of p47(p

42 anied by a pronounced expression of CD11b in polymorphonuclear leukocytes and tissue sequestration of

43 converted to several novel products by human polymorphonuclear leukocytes and whole blood as well as

44 the gamma subunit on the surface of porcine polymorphonuclear leukocytes and with several other uniq

45 evaluated for total white blood cell (WBC), polymorphonuclear leukocyte, and monocyte counts.

46 ular phagocytosis with mouse NK cells, mouse polymorphonuclear leukocytes, and mouse macrophages.

47 reus abscesses, including the involvement of polymorphonuclear leukocytes, and provide a brief overvi

48 ant, suggesting that macrophages (MP) and/or polymorphonuclear leukocytes are the key target cells no

49 7-fold increase in the number of circulatory polymorphonuclear leukocytes as early as 1 hr postexposu

50 ly, specific immunodepletion of neutrophils (polymorphonuclear leukocytes) blocked hCNS-SCns astrogli

51 it both apoptosis and the oxidative burst in polymorphonuclear leukocytes but were unable to identify

52 labeled bacteria was tested in primary mouse polymorphonuclear leukocytes by flow cytometry.

53 data further suggest that the expression of polymorphonuclear leukocyte CD11b and the response of ir

54 cytosis of G. bethesdensis by normal and CGD polymorphonuclear leukocytes (CGD PMN) required heat-lab

55 nd placenta components included and 2) using polymorphonuclear leukocyte characteristics to assign lo

56 Defective FPR1 expression and impaired polymorphonuclear leukocyte chemotaxis toward bacterial

57 Neutrophils, or polymorphonuclear leukocytes, comprise a crucial compone

58 Neutrophilic polymorphonuclear leukocytes contain glycosphingolipid-

59 blood counts were performed and examined for polymorphonuclear leukocyte content.

60 measures of cervical inflammation (elevated polymorphonuclear leukocyte count).

61 assessment of the dynamics of alteration in polymorphonuclear leukocyte counts and expression of CD1

62 Polymorphonuclear leukocyte counts increased through the

63 ce expressing human alpha-defensins in their polymorphonuclear leukocytes (Def(+/+)).

64 n ligand uptake or C5aR endocytosis in human polymorphonuclear leukocytes, distinguishing its role fr

65 etics of enhanced green fluorescence protein-polymorphonuclear leukocyte (EGFP-PMN) influx within a w

66 blood and during phagocytic interaction with polymorphonuclear leukocytes ex vivo We conclude that Fa

67 .)-deficient monocytes, dendritic cells, and polymorphonuclear leukocytes from gp91(PHOX)- or p47(PHO

68 f the bacteria compared with macrophages and polymorphonuclear leukocytes from wild-type mice in whic

69 eral strategy used by this pathogen to alter polymorphonuclear leukocyte function.

70 Hyperoxia also significantly increased polymorphonuclear leukocytes, growth-related oncogene-al

71 MP-inhibitor treatment significantly reduced polymorphonuclear leukocytes, growth-related oncogene/CI

72 The effect size was modest for polymorphonuclear leukocytes/high-powered field threshol

73 A definitions were constructed by 1) varying polymorphonuclear leukocytes/high-powered field threshol

74 , and decreases interstitial infiltration of polymorphonuclear leukocytes in a mouse model of TNF-alp

75 igration of naive murine bone marrow-derived polymorphonuclear leukocytes in an in vitro cell migrati

76 The transepithelial migration of polymorphonuclear leukocytes in response to chemokine ex

77 flammatory signaling to recruit and activate polymorphonuclear leukocytes in the airway.

78 Large numbers of polymorphonuclear leukocytes in the amnion and chorion d

79 since we observed increased accumulation of polymorphonuclear leukocytes in the colonic mucosa of ra

80 Early influx of polymorphonuclear leukocytes in the lungs of uninfected

81 uired to counter toxic effectors secreted by polymorphonuclear leukocytes in the tissues.

82 Depletion of polymorphonuclear leukocytes in vivo failed to prevent g

83 gion increase resistance to killing by human polymorphonuclear leukocytes, increase bacterial prolife

84 normal epithelial architecture but increased polymorphonuclear leukocyte infiltration and large lymph

85 llin pretreatment resulted in suppression of polymorphonuclear leukocyte infiltration at a late stage

86 d equipotent (at nanogram dosages), limiting polymorphonuclear leukocyte infiltration in a dose-depen

87 The most polymorphonuclear leukocyte infiltration in periodontal

88 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infiltration in response to

89 ion, increased cell apoptosis, and decreased polymorphonuclear leukocyte infiltration in the healing

90 phagocytosis of Escherichia coli, decreased polymorphonuclear leukocyte infiltration, and counter-re

91 attenuated pulmonary membrane thickening and polymorphonuclear leukocyte infiltration, reduced NF-kap

92 ontrol of immune-mediated cardiac injury and polymorphonuclear leukocyte inflammation.

93 Systemic treatment with PD1 reduced kidney polymorphonuclear leukocyte influx and, more importantly

94 ulted in improved oxygenation and diminished polymorphonuclear leukocyte influx into the isograft.

95 ice had increased efferocytosis of apoptotic polymorphonuclear leukocytes instilled into the peritone

96 ve of mobilization of the pool of marginated polymorphonuclear leukocytes into the free circulation.

97 However, the influx of polymorphonuclear leukocytes into the lung and the numbe

98 act of fruit reduced the edema, migration of polymorphonuclear leukocytes into the peritoneal cavity,

99 Peritoneal macrophages and polymorphonuclear leukocytes isolated from iNOS-/- mice

100 timulated COX-2 expression was suppressed in polymorphonuclear leukocytes isolated from MacKOs, but P

101 Upon stimulation of the human polymorphonuclear leukocyte, it was found that the abund

102 ls of erythrocyte lysis, resistance to human polymorphonuclear leukocyte killing, and pathogenesis in

103 aemophilus ducreyi is orchestrated by serum, polymorphonuclear leukocytes, macrophages, T cells, and

104 to invade epithelial cells and/or to induce polymorphonuclear leukocyte migration in a tissue cultur

105 culating blood elements including platelets, polymorphonuclear leukocytes, monocytes, and lymphocytes

106 (Hp) infection triggers a chronic influx of polymorphonuclear leukocyte neutrophils (PMNs) into the

107 We show that the specific immunodepletion of polymorphonuclear leukocyte neutrophils using anti-Ly6G

108 Polymorphonuclear leukocytes (neutrophils) are the prima

109 n of peri-implant inflammation: neutrophilic polymorphonuclear leukocytes (neutrophils) maximally acc

110 9 siRNA showed decreases in corneal opacity, polymorphonuclear leukocyte number, IL-12 and IFN-gamma

111 significantly elevated bacterial counts and polymorphonuclear leukocyte numbers at 1 and 3 days post

112 d latex bead-induced ROS production in human polymorphonuclear leukocytes occurred by the NADPH oxida

113 Neutrophils are polymorphonuclear leukocytes of the phagocytic system th

114 d for virulence and resistance to killing by polymorphonuclear leukocytes, one epa mutant (TX5179) wa

115 atment on the volume of nasal secretions, on polymorphonuclear leukocyte or interleukin-8 concentrati

116 ant for in vitro killing of P. gingivalis by polymorphonuclear leukocytes or AM and, moreover, the AM

117 with NGU (visible urethral discharge or >/=5 polymorphonuclear leukocytes per high-power field [PMNs/

118 ouse peritonitis and was more susceptible to polymorphonuclear leukocyte phagocytic killing.

119 pheral insulin resistance and alterations in polymorphonuclear leukocyte (PML) function.

120 ase of cytokines and chemokines that attract polymorphonuclear leukocytes (PML) and macrophages to th

121 trates the epidermis and reaches the dermis, polymorphonuclear leukocytes (PMLs) accumulate and an ab

122 inflammatory stimulus and results in tissue polymorphonuclear leukocyte (PMN) accumulation.

123 JAM-C mAb alone had no inhibitory effect on polymorphonuclear leukocyte (PMN) adhesion or transmigra

124 pothesis that these antibodies might augment polymorphonuclear leukocyte (PMN) adhesion to endotheliu

125 here that the saliva of ixodid ticks reduces polymorphonuclear leukocyte (PMN) adhesion via downregul

126 7 from Akt, prior to Akt activation, induced polymorphonuclear leukocyte (PMN) apoptosis.

127 ids participate in the suppression of murine polymorphonuclear leukocyte (PMN) bactericidal activitie

128 iodontitis (AgP) is associated with impaired polymorphonuclear leukocyte (PMN) chemotaxis toward bact

129 e, is susceptible to killing by a variety of polymorphonuclear leukocyte (PMN) components.

130 In vitro assays with human Polymorphonuclear leukocyte (PMN) demonstrated that CFTR

131 PD1 was a potent regulator of polymorphonuclear leukocyte (PMN) infiltration (approxim

132 logic sections showed more corneal edema and polymorphonuclear leukocyte (PMN) infiltration in contro

133 Histologic analysis suggested less polymorphonuclear leukocyte (PMN) infiltration into the

134 (2) formation was analyzed by lipidomics and polymorphonuclear leukocyte (PMN) infiltration quantifie

135 Polymorphonuclear leukocyte (PMN) infiltration was first

136 rsus the PBS-treated group were examined for polymorphonuclear leukocyte (PMN) infiltration, chemokin

137 Polymorphonuclear leukocyte (PMN) is the predominant inn

138 nslocation, biofilm formation, resistance to polymorphonuclear leukocyte (PMN) killing, and virulence

139 Extension of the polymorphonuclear leukocyte (PMN) leading edge toward a

140 Polymorphonuclear leukocyte (PMN) migration across the i

141 lts, larvae, and embryos), a transepithelial polymorphonuclear leukocyte (PMN) migration assay, and t

142 eltaybcL) failed to suppress transepithelial polymorphonuclear leukocyte (PMN) migration in vitro, a

143 ation but had no effect on bacterial load or polymorphonuclear leukocyte (PMN) numbers in the lung.

144 Using priming of the polymorphonuclear leukocyte (PMN) oxidase as a measure o

145 charide causes translocation of Rac from the polymorphonuclear leukocyte (PMN) rafts and does not ind

146 To model polymorphonuclear leukocyte (PMN) recognition of fungi u

147 a concentration where RvE1 potently reduced polymorphonuclear leukocyte (PMN) recruitment in zymosan

148 es (CORMs) suppress inflammation by reducing polymorphonuclear leukocyte (PMN) recruitment to the aff

149 ection, which coincided with the increase in polymorphonuclear leukocyte (PMN) recruitment to the lun

150 Polymorphonuclear leukocyte (PMN) recruitment to vascula

151 lular adhesion molecule 1 (ICAM-1)-dependent polymorphonuclear leukocyte (PMN) recruitment, functiona

152 ICAM-1) expression regulates the location of polymorphonuclear leukocyte (PMN) TEM.

153 tal staphylococcal infections by controlling polymorphonuclear leukocyte (PMN) trafficking.

154 nd counterregulatory actions that stop human polymorphonuclear leukocyte (PMN) transendothelial migra

155 sduction pathways that are engaged to induce polymorphonuclear leukocyte (PMN) transepithelial migrat

156 tactin phosphorylation in endothelium during polymorphonuclear leukocyte (PMN) transmigration through

157 n species (ROS) are critical for neutrophil (polymorphonuclear leukocyte (PMN)) microbicidal function

158 rate that epinephrine alters the neutrophil (polymorphonuclear leukocyte (PMN))-dependent inflammator

159 ant throughout the observation period, while polymorphonuclear leukocyte (PMN), S100A8, and interleuk

160 The polymorphonuclear leukocyte (PMN)-derived serine proteas

161 ction of cytoskeletal changes, inhibition of polymorphonuclear leukocyte (PMN)-endothelial cell inter

162 hydrogenase gene expression in macrophages, polymorphonuclear leukocyte (PMN)-like cells, and a mous

163 entify novel virulence factors in evasion of polymorphonuclear leukocyte (PMN)-mediated innate immuni

164 type 8 pneumococcus, but it does not promote polymorphonuclear leukocyte (PMN)-mediated pneumococcal

165 Polymorphonuclear leukocyte (PMN)-mediated tissue damage

166 t defense against microbial infection is the polymorphonuclear leukocyte (PMN).

167 Neutrophil [polymorphonuclear leukocyte (PMN)] transepithelial migra

168 gue associated with extensive recruitment of polymorphonuclear leukocytes (PMN or neutrophils) to the

169 Polymorphonuclear leukocytes (PMN) achieve an intermedia

170 taphylococcus aureus include mobilization of polymorphonuclear leukocytes (PMN) and extracellular gro

171 (4) (LTB(4)) is a potent chemoattractant for polymorphonuclear leukocytes (PMN) and other cells.

172 presentation of phagocytes in these lesions: polymorphonuclear leukocytes (PMN) are more prevalent in

173 Polymorphonuclear leukocytes (PMN) are the first respond

174 Polymorphonuclear leukocytes (PMN) from patients with ch

175 Depletion of circulating polymorphonuclear leukocytes (PMN) had a similar therape

176 tussis is able to avoid bacterial killing by polymorphonuclear leukocytes (PMN) if specific opsonic a

177 expression in human uroepithelial cells and polymorphonuclear leukocytes (PMN) in vitro and in bladd

178 duced chronic injury in the cornea triggered polymorphonuclear leukocytes (PMN) infiltration, patholo

179 flammation is traditionally characterized by polymorphonuclear leukocytes (PMN) influx followed by ph

180 Transendothelial migration (TEM) of polymorphonuclear leukocytes (PMN) involves a carefully

181 ng upon this model, we investigated the role polymorphonuclear leukocytes (PMN) play in the control o

182 Subcellular fractionation studies in polymorphonuclear leukocytes (PMN) revealed similar patt

183 sly localized the anion transporter ClC-3 to polymorphonuclear leukocytes (PMN) secretory vesicles an

184 xperiments, conditioned media collected from polymorphonuclear leukocytes (PMN) selectively increased

185 LFA-1 bonds help anchor polymorphonuclear leukocytes (PMN) to inflamed endotheli

186 We have shown that polymorphonuclear leukocytes (PMN) treated with the sali

187 trate by flow cytometric analysis that mouse polymorphonuclear leukocytes (PMN) up-regulate surface e

188 nfected individuals contain large numbers of polymorphonuclear leukocytes (PMN) with ingested gonococ

189 ays, we found that RAGE mediates neutrophil (polymorphonuclear leukocytes (PMN)) adhesion to, and sub

190 reported previously that human neutrophils (polymorphonuclear leukocytes (PMN)) express multiple SIR

191 . bethesdensis resists killing by serum, CGD polymorphonuclear leukocytes (PMN), and antimicrobial pe

192 J774 cells, murine polymorphonuclear leukocytes (PMN), human monocytes, and

193 depends on a prompt response by circulating polymorphonuclear leukocytes (PMN).

194 atterns of JNK/SAPK in wild-type and JNK2-/- polymorphonuclear leukocytes (PMN).

195 dal polypeptides secreted by macrophages and polymorphonuclear leukocytes (PMN).

196 spergillus are killed by normal, but not CGD polymorphonuclear leukocytes (PMN); however, the few stu

197 , sustained recruitment to the lymph node of polymorphonuclear leukocytes (PMN, or neutrophils), the

198 st immune responses, including the influx of polymorphonuclear leukocytes (PMN; neutrophils).

199 y with IL-1beta to promote early neutrophil (polymorphonuclear leukocyte [PMN]) recruitment.

200 Neutrophil (polymorphonuclear leukocytes [PMN]) infiltration plays a

201 Neutrophil (polymorphonuclear leukocytes [PMN]) transepithelial migr

202 ed to increased infiltration of neutrophils (polymorphonuclear leukocytes [PMN]), macrophages (MPhi),

203 Neutrophil (polymorphonuclear leukocyte; PMN) inflammatory functions

204 by the provider, who recorded the number of polymorphonuclear leukocytes (PMNLs) per epithelial cell

205 Polymorphonuclear leukocytes (PMNs or neutrophils) are e

206 Transmigration of polymorphonuclear leukocytes (PMNs) across CRECs monolay

207 The migration of polymorphonuclear leukocytes (PMNs) across the intestina

208 We hypothesized that polymorphonuclear leukocytes (PMNs) and less mature myel

209 nstrate its ability in separating/recovering polymorphonuclear leukocytes (PMNs) and mononuclear leuk

210 s a host foreign body response, during which polymorphonuclear leukocytes (PMNs) and then monocytes (

211 Human neutrophilic polymorphonuclear leukocytes (PMNs) are central to innat

212 Polymorphonuclear leukocytes (PMNs) are innate immune ce

213 e hypersensitivity and autoimmunity in which polymorphonuclear leukocytes (PMNs) are involved.

214 Polymorphonuclear leukocytes (PMNs) are key in innate im

215 e, we demonstrate that RvD1 actions on human polymorphonuclear leukocytes (PMNs) are pertussis toxin

216 Y. pestis and that CD11b(+) cells other than polymorphonuclear leukocytes (PMNs) are selectively lost

217 oeae recruits and interacts extensively with polymorphonuclear leukocytes (PMNs) during infection.

218 of CA-MRSA, we evaluated the lysis of human polymorphonuclear leukocytes (PMNs) during phagocytic in

219 A flow-cytometric analysis of polymorphonuclear leukocytes (PMNs) exposed to supernata

220 We tested cytotoxicity of LukGH toward polymorphonuclear leukocytes (PMNs) from mice, rabbits,

221 Polymorphonuclear leukocytes (PMNs) from subjects with l

222 into host cells, and both pathogens recruit polymorphonuclear leukocytes (PMNs) from the submucosa t

223 nd cell migration are essential functions of polymorphonuclear leukocytes (PMNs) in host defense.

224 orresponding decrease in the accumulation of polymorphonuclear leukocytes (PMNs) in lungs.

225 progenitor cells (HSPCs) differentiate into polymorphonuclear leukocytes (PMNs) in the bone marrow.

226 ntratracheal administration of LPS increased polymorphonuclear leukocytes (PMNs) in the bronchoalveol

227 n the percentage of dendritic cells (DCs) or polymorphonuclear leukocytes (PMNs) in the early stage o

228 del of LPS-induced lung injury, migration of polymorphonuclear leukocytes (PMNs) into the different c

229 lografts is initiated by the infiltration of polymorphonuclear leukocytes (PMNs) into the graft.

230 ine receptor 2 (CXCR2) mediates migration of polymorphonuclear leukocytes (PMNs) into the lung.

231 f pathogenesis of pneumonia is the influx of polymorphonuclear leukocytes (PMNs) into the lungs.

232 elial cells (HPMEC) prior to the addition of polymorphonuclear leukocytes (PMNs) or dendritic cells (

233 Bacteria and polymorphonuclear leukocytes (PMNs) per cornea were quan

234 on to releasing preformed granular proteins, polymorphonuclear leukocytes (PMNs) synthesize chemokine

235 Massive infiltration of polymorphonuclear leukocytes (PMNs) to the corneal endot

236 oeae (Gc), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

237 nococcus), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

238 opsonophagocytosis of P. gingivalis by human polymorphonuclear leukocytes (PMNs) using a fluorescent

239 A few polymorphonuclear leukocytes (PMNs) were already present

240 On necropsy, a large number of necrotic polymorphonuclear leukocytes (PMNs) were observed in the

241 ized by a mucopurulent exudate that contains polymorphonuclear leukocytes (PMNs) with intracellular g

242 flammatory response that is characterized by polymorphonuclear leukocytes (PMNs) with intracellular g

243 ite the fundamental function of neutrophils (polymorphonuclear leukocytes (PMNs)) in innate immunity,

244 Neutrophils (or polymorphonuclear leukocytes (PMNs)) readily clear blast

245 ing in septic plasma required C5a receptors, polymorphonuclear leukocytes (PMNs), and the Nacht-, LRR

246 e HUVEC monolayer permeability, and activate polymorphonuclear leukocytes (PMNs), as reflected in CD1

247 aggregated with human platelets and bound to polymorphonuclear leukocytes (PMNs), as shown by the sti

248 Neutrophil granulocytes, also called polymorphonuclear leukocytes (PMNs), extrude molecular l

249 by an inflammatory infiltrate that includes polymorphonuclear leukocytes (PMNs), monocytes, lymphocy

250 -derived alveolar macrophages (AMs), but not polymorphonuclear leukocytes (PMNs), to phagocytose bact

251 Our method reproducibly recovers 94.0% of polymorphonuclear leukocytes (PMNs), with up to 10(5) PM

252 din (PVL), a pore-forming toxin that targets polymorphonuclear leukocytes (PMNs).

253 ession in epithelial cells, fibroblasts, and polymorphonuclear leukocytes (PMNs).

254 pact interactions between N. gonorrhoeae and polymorphonuclear leukocytes (PMNs).

255 ion conferring protection against killing by polymorphonuclear leukocytes (PMNs).

256 eported, decrease membrane fluidity on mouse polymorphonuclear leukocytes (PMNs).

257 ositive strains is an elevation in levels of polymorphonuclear leukocytes (PMNs).

258 revisiae, release chemoattractants for human polymorphonuclear leukocytes (PMNs).

259 , as they prevented an influx of Siglec-F(+) polymorphonuclear leukocytes (PMNs).

260 infections depends on bacterial clearance by polymorphonuclear leukocytes (PMNs); however, excessive

261 hemorrhage, and accumulation of neutrophils [polymorphonuclear leukocytes (PMNs)] in the alveolar com

262 Human polymorphonuclear leukocytes (PMNs, or neutrophils) are

263 Human polymorphonuclear leukocytes (PMNs, or neutrophils) are

264 Polymorphonuclear leukocytes (PMNs, or neutrophils) are

265 The transmigration of polymorphonuclear leukocytes (PMNs; neutrophils) into th

266 Neutrophils (polymorphonuclear leukocytes [PMNs]) are critical to the

267 Human neutrophils (polymorphonuclear leukocytes [PMNs]) generate inflammato

268 ytes, the interactions of human neutrophils (polymorphonuclear leukocytes [PMNs]) with both TSP-4 var

269 n to avoid destruction by human neutrophils (polymorphonuclear leukocytes [PMNs]), which are crucial

270 n by human epithelial cells and neutrophils (polymorphonuclear leukocytes [PMNs]).

271 ous system (CNS) include infiltrating cells (polymorphonuclear leukocytes [PMNs], macrophages, and na

272 Neutrophils (polymorphonuclear leukocytes, PMNs) are vital to innate

273 O+/+) mice exhibited comparable increases in polymorphonuclear leukocytes, predominately neutrophils,

274 The total number of bronchoalveolar lavage polymorphonuclear leukocytes recovered from the mice exp

275 on chromosome 2, and a region affecting both polymorphonuclear leukocyte recruitment and TNF-alpha le

276 the omega-3 group correlated with decreased polymorphonuclear leukocyte recruitment, chemokine and c

277 ng inflammation, characterized by increasing polymorphonuclear leukocyte recruitment, is a major caus

278 Finally, depletion of polymorphonuclear leukocytes reversed the enhanced susce

279 thal disease, in association with widespread polymorphonuclear leukocyte-rich microabscesses but with

280 sion of NF-kappaB activity and restored lung polymorphonuclear leukocyte sequestration in response to

281 In polymorphonuclear leukocytes, SipA or other Salmonella p

282 s, but PG formation was not even detected in polymorphonuclear leukocyte supernatants from control mi

283 ated expression of MMP-26 in macrophages and polymorphonuclear leukocytes supports the functional rol

284 rom the biofilm were more resistant to human polymorphonuclear leukocytes than were in vitro-grown Y.

285 distribution in leukocytes (110 copies/10(5) polymorphonuclear leukocytes) than the R-/D+ subgroup.

286 mine (GPEtn) species after exposure of human polymorphonuclear leukocytes to A23187 and granulocyte m

287 e attributed to a defect in the migration of polymorphonuclear leukocytes to infected sites during ea

288 Bacterial invasion of host tissues triggers polymorphonuclear leukocytes to release DNA [neutrophil

289 iae-infected Mmp2/9(-/-) mice recruited more polymorphonuclear leukocytes to the lung but had higher

290 RvD1 regulates human polymorphonuclear leukocyte transendothelial migration a

291 ntercellular adhesion molecule-1 expression, polymorphonuclear leukocyte transendothelial migration,

292 AC753S mutant had reduced Salmonella-induced polymorphonuclear leukocytes transepithelial migration.

293 ies of AnxA1 become operative to finely tune polymorphonuclear leukocytes transmigration to the site

294 Polymorphonuclear leukocytes undergo directed movement t

295 multisubunit complex from Brij 96 lysates of polymorphonuclear leukocytes using the G7 mAb, which bin

296 The increase in circulatory polymorphonuclear leukocytes was accompanied by a decrea

297 acute inflammatory infiltrate consisting of polymorphonuclear leukocytes was detected in the urethra

298 sing the RosetteSep antibody cocktail, while polymorphonuclear leukocytes were separated with density

299 By 6 h after infection, polymorphonuclear leukocytes with intracellular spores w

300 y detection of viral DNA in peripheral blood polymorphonuclear leukocytes within the first few months