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1  linked to a contributory virus (Merkel cell polyomavirus).
2 c acid, is the major attachment receptor for polyomavirus.
3 virus (MWPyV) is a recently identified human polyomavirus.
4 uce MCC in mice using oncoproteins from this polyomavirus.
5 Merkel cell carcinoma virus, which is also a polyomavirus.
6 ent of trichodysplasia spinulosa caused by a polyomavirus.
7 nty-one percent were seropositive for the JC polyomavirus.
8  sequencing of nucleic acid revealed a novel polyomavirus.
9 amily profile or the presence of Merkel cell polyomavirus.
10 r frequently associated with the Merkel cell polyomavirus.
11 cancers associated with a recently described polyomavirus.
12  CD8 T cell response to infection with mouse polyomavirus.
13  the rearrangements typical for reactivating polyomaviruses.
14 ion of Fe/S coordination in the sTs of other polyomaviruses.
15 ural history of the more recently discovered polyomaviruses.
16 uses, including two papillomaviruses and two polyomaviruses.
17 ds sialylated glycan receptors in many other polyomaviruses.
18 ommon ancestor of a large clade of mammalian polyomaviruses.
19 noviruses (AdV), papillomaviruses (HPV), and polyomaviruses.
20 elated overall to human and nonhuman primate polyomaviruses.
21 nt of the recently reported MWPyV and HPyV10 polyomaviruses.
22                                        Human polyomavirus 6 (HPyV6) and HPyV7 are commonly found on h
23                                        Human polyomavirus 6 (HPyV6) is most often detected at the ski
24 rus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9, and Trichodysplasi
25                                        Human polyomavirus 7 (HPyV7) is one of 11 HPyVs recently disco
26 re included herpesviruses, papillomaviruses, polyomaviruses, adenoviruses and anelloviruses.
27    In this Gem, we will discuss two viruses, polyomavirus and cytomegalovirus, in which cell culture
28      VP1 is the major coat protein of murine polyomavirus and forms virus-like particles (VLPs) in vi
29 thway for cytosolic entry varied between the polyomaviruses and between different cell types, namely,
30 ructures of closely related animal and human polyomaviruses and examine their strategies for engaging
31  patterns and determinants of acquisition of polyomaviruses and herpesviruses in childhood.
32 ersely associated with the seroprevalence of polyomaviruses and herpesviruses.
33                This pore is conserved across polyomaviruses and suggests either that these viruses ha
34 tical evidence in favor of an association of polyomaviruses and their hosts over millions of years.
35 V9, and Trichodysplasia spinulosa-associated polyomavirus) and examined factors associated with MCPyV
36 almonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine rotavirus A were present most f
37                                    Mammalian polyomaviruses are characterized by establishing persist
38                                              Polyomaviruses are nonenveloped viruses with capsids com
39                                              Polyomaviruses are opportunistic pathogens that are asso
40 viruses and explain the rarity of some novel polyomavirus-associated diseases.
41 nfiltrates in renal biopsy specimens with BK polyomavirus-associated nephropathy (BKPyVAN) and specim
42                                              Polyomavirus-associated nephropathy (PVAN) after BK viru
43 tomatic infections in most humans and causes polyomavirus-associated nephropathy (PVAN) and other pat
44                                              Polyomavirus-associated nephropathy (PVAN) occurs in a s
45 o a specific diagnosis), infections, such as polyomavirus-associated nephropathy, calcineurin inhibit
46 m a pediatric kidney transplant patient with polyomavirus-associated nephropathy.
47 .9, and 9.7 months for viruria, viremia, and polyomavirus-associated nephropathy.
48 sustained viremia, and 17 (7%) biopsy-proven polyomavirus-associated nephropathy.
49 factors that underlie the pathophysiology of polyomavirus-associated nephropathy.
50 imental data on unperturbed adenoviruses and polyomaviruses, at the same time providing insight into
51 ear if desensitization increases the risk of polyomavirus BK (BKV) viremia.
52 ear if desensitization increases the risk of polyomavirus BK (BKV) viremia.
53                          Reactivation of the polyomavirus BK has been associated with de novo antibod
54 onstrating "decoy" cells, and/or significant polyomavirus BK viremia.
55 ented: elucidating the transmission route of polyomaviruses BK and JC and estimating the basic reprod
56                                           BK polyomavirus (BKPyV) affects mostly kidney and bone marr
57                                           BK polyomavirus (BKPyV) frequently reactivates in kidney tr
58 t APOBEC3B is specifically upregulated by BK polyomavirus (BKPyV) infection in primary kidney cells a
59                                           BK polyomavirus (BKPyV) is a widespread human pathogen that
60                                           BK polyomavirus (BKPyV) is the most common viral pathogen a
61 is study, we investigated the role of the BK polyomavirus (BKPyV) miRNA in regulating virus replicati
62                                           BK polyomavirus (BKPyV) reactivation is associated with sev
63     Here, we demonstrate the detection of BK Polyomavirus (BKPyV), an emerging indicator of microbiol
64 d immunoglobulin G seroreactivity against 10 polyomaviruses (BKPyV, JCPyV, KIPyV, WUPyV, MCPyV, HPyV6
65 irus (CMV), Epstein-Barr virus (EBV), and BK polyomavirus (BKV) at transplant was a risk factor for p
66                                           BK polyomavirus (BKV) causes significant urinary tract path
67                                           BK polyomavirus (BKV) establishes persistent, low-level, an
68 uppression in kidney transplant patients, BK polyomavirus (BKV) has been shown to induce nephropathy
69  Recent case series describe detection of BK polyomavirus (BKV) in urinary tract cancers in kidney tr
70                       The pathogenesis of BK polyomavirus (BKV) infection and associated nephropathy
71                                           BK polyomavirus (BKV) infection remains a significant cause
72                                           BK polyomavirus (BKV) is an emerging pathogen in immunocomp
73                                           BK polyomavirus (BKV)-associated nephropathy is a threat to
74       Middle T antigen (MT), the oncogene of polyomavirus, can drive tumor formation in a variety of
75               Nephropathy associated with BK polyomavirus causes kidney allograft dysfunction and fai
76 affects the quantity and quality of the anti-polyomavirus CD8 T cell response and its differentiation
77 e 1; hepatitis E virus; bocavirus; KI and WU polyomaviruses; coronaviruses HKU1 and NL63; influenza,
78      No trichodysplasia spinulosa-associated polyomavirus could be isolated from the skin lesions; ho
79                                  Merkel cell polyomavirus could contribute to the origin of this derm
80 ly related to the middle T antigen of murine polyomavirus despite almost no sequence similarity.
81                                              Polyomaviruses encode a large T Ag (LT), a multifunction
82                                        Other polyomavirus-encoded large T antigens also increase the
83  these mutants will provide new insight into polyomavirus entry, pathogenesis, and evolution.
84 omaviruses, Merkel cell carcinoma-associated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-a
85 d 58.9% of eyebrow hair specimens) among all polyomaviruses examined.
86 y, and, ultimately, pathogenicity within the polyomavirus family and highlight the need for structure
87 hylogenetic relationships within the growing polyomavirus family and perhaps also for other viruses.
88  DNA tumor viruses, including members of the polyomavirus family, often result in tumor formation in
89               This was also hypothesized for polyomaviruses (family Polyomaviridae), a group comprisi
90                                The number of polyomaviruses found in the human body continues to grow
91 evidence that, consistent with this idea, BK polyomavirus genomes are depleted of APOBEC3B-preferred
92                                Whereas mouse polyomavirus has been studied in a fair amount of detail
93                                       Murine polyomavirus has repeatedly provided insights into tumor
94                                              Polyomavirus-Haufen counts showed excellent correlation
95                                              Polyomavirus-Haufen were counted in 40 urine samples fro
96                                  Several new polyomaviruses have been discovered in the last decade,
97                                              Polyomaviruses have repeating sequences at their origins
98            Both the papillomaviruses and the polyomaviruses have served as tools for understanding pa
99                                              Polyomaviruses have shown the importance of phosphotyros
100 titative PCR to test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus,
101 avirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9
102 ors and a novel mechanism of stability for a polyomavirus in cancer.
103  hybridization confirmed the presence of the polyomavirus in endothelial cells at sites of myositis a
104 rare skin cancer associated with Merkel cell polyomavirus in most cases.
105 zation of the noncoding control region of JC polyomavirus in vivo, mainly in CSF and blood.
106 n of large tumor T antigens (TAg) encoded by polyomaviruses in mammalian cells results in increased t
107                         The natural cycle of polyomaviruses in mammals is to persist in the host with
108 vides insight into how DDRs are activated by polyomaviruses in primary cells with intact cell cycle c
109 viously undefined protein encoded by several polyomaviruses including MCPyV, but also provides insigh
110                             Eleven new human polyomaviruses, including the skin viruses HPyV6 and HPy
111 ory of these viruses and the evolution of JC polyomavirus-induced progressive multifocal leukoencepha
112 BDCA-1(+) mDCs localized in proximity to the polyomavirus-infected tubular epithelial cells.
113                                Although both polyomavirus infection and T cell-mediated rejection (TC
114 P2.139-specific CD8 T cell response to mouse polyomavirus infection depends on CD4 T cell help and CD
115  is known about the initial host response to polyomavirus infection.
116  of herpes simplex, Epstein-Barr virus or BK polyomavirus infections.
117  Tumour oncogenesis is linked to Merkel cell polyomavirus integration and ultraviolet-radiation-induc
118                                  Merkel cell polyomavirus is a newly discovered human cancer virus th
119 e and rare event, the oncogenic potential of polyomavirus is primarily evaluated in laboratory animal
120                                           BK polyomavirus is ubiquitous, with a seropositivity rate o
121 tides found in the origins of replication of polyomaviruses is discussed.
122  acid-containing glycan receptors in related polyomaviruses is obstructed, and VP1 of HPyV6 and HPyV7
123 en, as well as large T antigens from related polyomaviruses, is alone capable of upregulating APOBEC3
124 the basis by which MCPyV, among all 12 human polyomaviruses, is the only one that causes cancer in hu
125 the presence of this pore is conserved among polyomaviruses, its functional role in infection or asse
126  document a unique DNA recombination between polyomavirus JC (JC virus [JCV]) and Epstein-Barr virus
127 ting disease caused by the human neurotropic polyomavirus JC (JCV) and is found almost exclusively in
128 elinating disease of the brain caused by the polyomavirus JC (JCV), has evolved tremendously.
129                        Isolates of the human polyomavirus JC virus from patients with the frequently
130  the white matter of the brain caused by the polyomavirus JC virus, which typically occurs only in im
131                                Classic human polyomaviruses (JC and BK viruses) become pathogenic whe
132 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human po
133                                 The human JC polyomavirus (JCPyV) causes the rapidly progressing demy
134                                 The human JC polyomavirus (JCPyV) establishes an asymptomatic, persis
135 a fatal disease caused by reactivation of JC polyomavirus (JCPyV) in immunosuppressed individuals and
136                                           JC polyomavirus (JCPyV) infection of immunocompromised indi
137                                           JC polyomavirus (JCPyV) is reactivated in approximately 20%
138 nally, endocytosis-dependent infection by JC polyomavirus (JCPyV) was reduced in human cells with dec
139 rebral manifestations are associated with JC polyomavirus (JCPyV) which are diagnosed by detection of
140  the receptor-binding properties of human JC polyomavirus (JCPyV), a virus that resides in the kidney
141 arget and neutralize its etiologic agent, JC polyomavirus (JCPyV).
142 from infection of oligodendrocytes by the JC polyomavirus (JCPyV).
143                                    The human polyomavirus, JCPyV, is the causative agent of progressi
144  life-threatening infections caused by human polyomaviruses JCV and BKV.
145 tients that treatment with interleukin 7, JC polyomavirus (JCV) capsid protein VP1, and a Toll-like r
146           The relationship between latent JC polyomavirus (JCV) infection and progressive multifocal
147  of the central nervous system (CNS) with JC polyomavirus (JCV) usually occur as a result of immunoco
148 s from lytic infection of the glia by the JC polyomavirus (JCV); JCV granule cell neuronopathy is cau
149 d persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyo
150     Combined, these results demonstrate that polyomaviruses lacking miRNAs can arise infrequently and
151 A-mediated viral gene autoregulation in some polyomavirus life cycles.
152 erkel cell carcinoma (MCC) is an aggressive, polyomavirus-linked skin cancer.
153 iated with clinical presentation: intrarenal polyomavirus load levels and Banff interstitial fibrosis
154 f DNA translocation by papillomavirus E1 and polyomavirus LTag hexameric helicases involves consecuti
155 ic infection of oligodendrocytes by human JC polyomavirus may result in the development of progressiv
156  CD8 T cell responses to infections by human polyomaviruses may be influenced by VP1 mutations involv
157 PyV-6 DNA load and VP1 protein suggests that polyomaviruses may contribute to the epithelial prolifer
158 ve to be the first case in which Merkel cell polyomavirus (MCPyV) and human papillomavirus subtype 17
159 rcinomas (MCCs) that contain the Merkel cell polyomavirus (MCPyV) and the clinical significance of tu
160                   Infection with Merkel cell polyomavirus (MCPyV) can lead to Merkel cell carcinoma (
161                                  Merkel cell polyomavirus (MCPyV) causes the majority of cases of Mer
162                                  Merkel cell polyomavirus (MCPyV) causes the majority of MCC cases du
163          MCCs frequently contain Merkel cell polyomavirus (MCPyV) DNA and express viral transforming
164      In at least 80% of all MCC, Merkel cell polyomavirus (MCPyV) DNA has undergone clonal integratio
165                                  Merkel cell polyomavirus (MCPyV) expressing viral T antigens is a co
166 ng evidence indicates a role for Merkel cell polyomavirus (MCPyV) in the development of Merkel cell c
167                                  Merkel cell polyomavirus (MCPyV) is frequently associated with Merke
168 yV infecting hominine hosts, the Merkel cell polyomavirus (MCPyV) lineage.
169                                  Merkel cell polyomavirus (MCPyV) may contribute to tumorigenesis in
170                                  Merkel cell polyomavirus (MCPyV) plays an important role in Merkel c
171 ns, copy number alterations, and Merkel cell polyomavirus (MCPyV) sequence were analyzed and compared
172 l integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC tumor cells express putati
173                              The Merkel cell polyomavirus (MCPyV), discovered in 2008, drives the dev
174                                  Merkel cell polyomavirus (MCPyV), identified in the majority of MCCs
175 me (ALTO) in the early region of Merkel cell polyomavirus (MCPyV), the causative agent of most Merkel
176 ure to ultraviolet light and the Merkel-cell polyomavirus (MCPyV).
177 ed in the last decade, including Merkel cell polyomavirus (MCPyV).
178  incidence, prevalence, and persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus,
179                                  Merkel cell polyomavirus (MCV) causes an aggressive human skin cance
180                                  Merkel cell polyomavirus (MCV) causes an aggressive skin cancer afte
181            Clonal integration of Merkel cell polyomavirus (MCV) DNA into the host genome has been obs
182                                  Merkel cell polyomavirus (MCV) infection and DNA integration into th
183                                  Merkel cell polyomavirus (MCV) is a newly discovered human cancer vi
184                                  Merkel cell polyomavirus (MCV) small T (sT) oncoprotein induces cent
185  skin cancer associated with the Merkel cell polyomavirus (MCV).
186 and tractable model for a novel mechanism of polyomavirus-mediated oncogenesis.
187 ntly associated with clonal integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC
188 aneous tumor system with mice expressing the polyomavirus middle T (PyMT) oncogene under control of t
189                                          The polyomavirus middle T antigen (PyMT) oncogene activates
190 In addition, mammary gland tumors induced by polyomavirus middle T antigen in JNK2(-/-) mice were mor
191 r cells derived from mice transgenic for the polyomavirus middle T oncogene to ionizing radiation res
192 RT-deficient mice displayed marked delays in polyomavirus middle T oncogene-induced (PyMT-induced) ma
193 tors in the mouse mammary tumor virus (MMTV)-polyomavirus middle T oncoprotein (PyMT) mouse model of
194 ased mammary tumorigenesis in ovariectomized polyomavirus middle T-antigen mice.
195 k Institute set out to determine whether the polyomavirus middle T-transforming protein had a similar
196  of breast cancer, including the widely used polyomavirus middle-T antigen (PyVmT) model, which provi
197 rovide evidence for a unique function of the polyomavirus miRNA that may have important implications
198                          The function of the polyomavirus miRNA was investigated in archetype BKPyV,
199                                  Using mouse polyomavirus (MPyV) infection, we previously showed that
200                                        Mouse polyomavirus (MPyV) is a ubiquitous persistent natural m
201                                Using a mouse polyomavirus (MPyV) kidney transplant model, we investig
202                                       Malawi polyomavirus (MWPyV) is a recently identified human poly
203 vergent polyomavirus, provisionally named MX polyomavirus (MXPyV), in stool samples from children.
204                                              Polyomavirus-negative matched patients (n = 943) were de
205                                              Polyomavirus nephropathy (PVAN) is a common cause of kid
206                                              Polyomavirus nephropathy (PVN) is a common viral infecti
207 alitative highly predictive urinary test for polyomavirus nephropathy (PVN) is the PV-Haufen test.
208 ated with hemorrhagic cystitis and also with polyomavirus nephropathy (PVN).
209 finitive PVN from the Banff Working Group on Polyomavirus Nephropathy, comprising nine transplant cen
210 novo or recurrent glomerular pathologies and polyomavirus nephropathy.
211                                   New Jersey polyomavirus (NJPyV-2013) is a novel polyomavirus that m
212 ding for middle T antigen (MT) is the murine polyomavirus oncogene most responsible for tumor formati
213         Middle T antigen (MT) is the primary polyomavirus oncogene responsible for tumor formation.
214 MCPyV), and MCC tumor cells express putative polyomavirus oncoprotein small T antigen (sTAg) and trun
215 mavirus sequence repeats in complex with the polyomavirus origin-binding domain reveals that only thr
216                                              Polyomavirus origins of replication contain multiple occ
217 viruses, herpesviruses, papillomaviruses and polyomaviruses) over time were observed in individuals w
218 ttempted to reveal the composition of the JC polyomavirus population (the quasispecies, i.e., the who
219                   For the first time, the JC polyomavirus population contained in different body comp
220                                              Polyomavirus-positive patients (n = 943) were defined as
221 ach, we identified a novel, highly divergent polyomavirus, provisionally named MX polyomavirus (MXPyV
222                                              Polyomaviruses (PV) have been historically associated wi
223 ) mice are susceptible to tumor induction by polyomavirus (Py), while C57BR/cdJ (BR) mice are resista
224                      The nonenveloped simian polyomavirus (PyV) simian virus 40 (SV40) hijacks the en
225                             The nonenveloped polyomavirus (PyV) simian virus 40 (SV40) traffics from
226                                         Nine polyomavirus (PyV) species are known to productively inf
227 rovide some simian virus 40 (SV40) or murine polyomavirus (PyV) ST functions.
228 ng these infections can be studied in murine polyomavirus (PyV)-infected mice as a model.
229                                              Polyomaviruses (PyV) are potentially tumorigenic in huma
230           It has long been hypothesized that polyomaviruses (PyV; family Polyomaviridae) codiverged w
231                                              Polyomavirus (PyVs) are small DNA pathogens that contain
232                                              Polyomaviruses (PyVs) are a group of emerging pathogens
233                                              Polyomaviruses (PyVs) are small DNA viruses, long known
234                                              Polyomaviruses (PyVs) cause devastating human diseases,
235                            Several different polyomaviruses (PyVs) encode microRNAs (miRNAs) that reg
236                       Other tumor-associated polyomaviruses (PyVs), including simian virus 40 (SV40),
237                                      Raccoon polyomavirus (RacPyV) is associated with 100% of neurogl
238                            Recently, raccoon polyomavirus (RacPyV) was identified in neuroglial tumor
239                       Unlike other oncogenic polyomaviruses, RacPyV was episomal, not integrated, in
240                            Seroprevalence of polyomaviruses ranged from 38.5% to 99.8% in cord blood
241                                The effect of polyomavirus reactivation (BK viremia or JC viruria) on
242 study (n=40) to explore associations between polyomavirus reactivation and immune responses to the se
243     Furthermore, these results indicate that polyomavirus reactivation associates with immune respons
244                                              Polyomavirus reactivation can cause significant morbidit
245 ore prevalent during year 1 in subjects with polyomavirus reactivation than in those without reactiva
246  conserved VP1 proteins from closely related polyomaviruses recognize different oligosaccharides.
247 titative histologic assessment of intrarenal polyomavirus replication/load levels.
248 crystal structure of the four central murine polyomavirus sequence repeats in complex with the polyom
249 how that miRNAs from related variants of the polyomavirus simian vacuolating virus 40 (SV40) have dif
250              In the case of the nonenveloped polyomavirus simian virus 40 (SV40), the virus penetrate
251 comparing the cytosolic entry of the related polyomavirus simian virus 40 (SV40), we found that depen
252 usage affect tropism, we studied the primate polyomavirus simian virus 40 (SV40), which uses the gang
253 revealed by a viral oncoprotein, Merkel cell polyomavirus small T (MCV sT).
254                                       Murine polyomavirus small t antigen (PyST) regulates cell cycle
255                We recently demonstrated that polyomavirus small T antigen (ST) binds YAP, a major eff
256                                              Polyomavirus small t antigen is a viral oncogene that co
257 t cell transformation induced by Merkel cell polyomavirus small T antigen viral oncoprotein.
258 xamined whether some of the functions of the polyomavirus small T antigens (ST) are shared by the E6
259 mavirus E7 has the same effect as the murine polyomavirus small T protein.
260  presence of a tenth apparently human-tropic polyomavirus species, which we name HPyV10.
261 yomavirus, we show that brain-resident mouse polyomavirus-specific CD8 T cells, unlike memory cells i
262                                       Animal polyomavirus sT oncoproteins have been found to cause ex
263 not based on PD-L1 expression or Merkel cell polyomavirus status.
264 ace morphology from those of all other known polyomavirus structures.
265  for the transforming properties of nonhuman polyomaviruses, such as simian virus 40 (SV40), but is n
266                                Here we probe polyomavirus SV40 endoplasmic reticulum (ER)-to-cytosol
267 model is described that predicts patterns of polyomavirus SV40 infections and associated cancers in h
268          Here we probe how the non-enveloped polyomavirus SV40 penetrates the endoplasmic reticulum (
269 tosol membrane transport of the nonenveloped polyomavirus SV40, a decisive infection step, a cytosoli
270 bserved for the beta-herpesvirus CMV and the polyomaviruses SV40 and SA12.
271          These studies uncover the action of polyomavirus T antigens on cellular CBP/p300 and suggest
272                                     Like all polyomavirus T antigens, PyST functions largely via its
273  Jersey polyomavirus (NJPyV-2013) is a novel polyomavirus that may have tropism for vascular endothel
274  question by focusing on a single lineage of polyomaviruses that infect both humans and their closest
275                                    In murine polyomavirus, the central region of the origin contains
276                In contrast to better-studied polyomaviruses, the routes of infection, cell tropism, a
277 cell carcinoma (MCC), making MCPyV the first polyomavirus to be clearly associated with human cancer.
278                           MCPyV is the first polyomavirus to be clearly associated with human cancer.
279 rt the complete genome sequence of the first polyomavirus to be isolated from a horse.
280 in complex, a tumor suppressor in some other polyomavirus transformation models, but was strictly dep
281 en (MT), the principal oncoprotein of murine polyomavirus, transforms by association with cellular pr
282 we show that manifestations of the causative polyomavirus (TSPyV) occur during primary infection of t
283  witnessed the discovery of eleven new human polyomaviruses, two of which cause unusual and rare canc
284                                          Any polyomavirus urinary shedding was more frequent in liver
285 arison with other structurally characterized polyomavirus VP1 proteins enhances our understanding of
286                                           In polyomaviruses, VP1 commonly determines antigenicity as
287 ere found with the expression of recombinant polyomavirus Vp1s and human papillomavirus L1s in COS-7
288                                  Merkel cell polyomavirus was detected in 32 of 38 specimens (84%).
289                                           JC polyomavirus was detected in the CSF at the time of pres
290                                           JC polyomavirus was detected within the graft's collecting
291             In that same year a second human polyomavirus was discovered in the urine of a kidney tra
292                     In 1971, the first human polyomavirus was isolated from the brain of a patient wh
293                                            A polyomavirus was isolated from the eyes of horses, and t
294                                           JC polyomavirus was not detected in pretransplant serum, ho
295                                      A human polyomavirus was recently discovered in Merkel cell carc
296                                  Using mouse polyomavirus, we demonstrate that a single amino acid di
297                                  Using mouse polyomavirus, we show that brain-resident mouse polyomav
298 lutionary divergence and diverse sampling of polyomaviruses, we propose evolutionary transitions that
299   We review what is currently known about JC polyomavirus, what is suspected, and what remains to be
300 (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPy

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