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1 linked to a contributory virus (Merkel cell polyomavirus).
2 c acid, is the major attachment receptor for polyomavirus.
3 virus (MWPyV) is a recently identified human polyomavirus.
4 uce MCC in mice using oncoproteins from this polyomavirus.
5 Merkel cell carcinoma virus, which is also a polyomavirus.
6 ent of trichodysplasia spinulosa caused by a polyomavirus.
7 nty-one percent were seropositive for the JC polyomavirus.
8 sequencing of nucleic acid revealed a novel polyomavirus.
9 amily profile or the presence of Merkel cell polyomavirus.
10 r frequently associated with the Merkel cell polyomavirus.
11 cancers associated with a recently described polyomavirus.
12 CD8 T cell response to infection with mouse polyomavirus.
13 the rearrangements typical for reactivating polyomaviruses.
14 ion of Fe/S coordination in the sTs of other polyomaviruses.
15 ural history of the more recently discovered polyomaviruses.
16 uses, including two papillomaviruses and two polyomaviruses.
17 ds sialylated glycan receptors in many other polyomaviruses.
18 ommon ancestor of a large clade of mammalian polyomaviruses.
19 noviruses (AdV), papillomaviruses (HPV), and polyomaviruses.
20 elated overall to human and nonhuman primate polyomaviruses.
21 nt of the recently reported MWPyV and HPyV10 polyomaviruses.
24 rus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9, and Trichodysplasi
27 In this Gem, we will discuss two viruses, polyomavirus and cytomegalovirus, in which cell culture
29 thway for cytosolic entry varied between the polyomaviruses and between different cell types, namely,
30 ructures of closely related animal and human polyomaviruses and examine their strategies for engaging
34 tical evidence in favor of an association of polyomaviruses and their hosts over millions of years.
35 V9, and Trichodysplasia spinulosa-associated polyomavirus) and examined factors associated with MCPyV
36 almonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine rotavirus A were present most f
41 nfiltrates in renal biopsy specimens with BK polyomavirus-associated nephropathy (BKPyVAN) and specim
43 tomatic infections in most humans and causes polyomavirus-associated nephropathy (PVAN) and other pat
45 o a specific diagnosis), infections, such as polyomavirus-associated nephropathy, calcineurin inhibit
50 imental data on unperturbed adenoviruses and polyomaviruses, at the same time providing insight into
55 ented: elucidating the transmission route of polyomaviruses BK and JC and estimating the basic reprod
58 t APOBEC3B is specifically upregulated by BK polyomavirus (BKPyV) infection in primary kidney cells a
61 is study, we investigated the role of the BK polyomavirus (BKPyV) miRNA in regulating virus replicati
63 Here, we demonstrate the detection of BK Polyomavirus (BKPyV), an emerging indicator of microbiol
64 d immunoglobulin G seroreactivity against 10 polyomaviruses (BKPyV, JCPyV, KIPyV, WUPyV, MCPyV, HPyV6
65 irus (CMV), Epstein-Barr virus (EBV), and BK polyomavirus (BKV) at transplant was a risk factor for p
68 uppression in kidney transplant patients, BK polyomavirus (BKV) has been shown to induce nephropathy
69 Recent case series describe detection of BK polyomavirus (BKV) in urinary tract cancers in kidney tr
76 affects the quantity and quality of the anti-polyomavirus CD8 T cell response and its differentiation
77 e 1; hepatitis E virus; bocavirus; KI and WU polyomaviruses; coronaviruses HKU1 and NL63; influenza,
84 omaviruses, Merkel cell carcinoma-associated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-a
86 y, and, ultimately, pathogenicity within the polyomavirus family and highlight the need for structure
87 hylogenetic relationships within the growing polyomavirus family and perhaps also for other viruses.
88 DNA tumor viruses, including members of the polyomavirus family, often result in tumor formation in
91 evidence that, consistent with this idea, BK polyomavirus genomes are depleted of APOBEC3B-preferred
100 titative PCR to test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus,
101 avirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9
103 hybridization confirmed the presence of the polyomavirus in endothelial cells at sites of myositis a
106 n of large tumor T antigens (TAg) encoded by polyomaviruses in mammalian cells results in increased t
108 vides insight into how DDRs are activated by polyomaviruses in primary cells with intact cell cycle c
109 viously undefined protein encoded by several polyomaviruses including MCPyV, but also provides insigh
111 ory of these viruses and the evolution of JC polyomavirus-induced progressive multifocal leukoencepha
114 P2.139-specific CD8 T cell response to mouse polyomavirus infection depends on CD4 T cell help and CD
117 Tumour oncogenesis is linked to Merkel cell polyomavirus integration and ultraviolet-radiation-induc
119 e and rare event, the oncogenic potential of polyomavirus is primarily evaluated in laboratory animal
122 acid-containing glycan receptors in related polyomaviruses is obstructed, and VP1 of HPyV6 and HPyV7
123 en, as well as large T antigens from related polyomaviruses, is alone capable of upregulating APOBEC3
124 the basis by which MCPyV, among all 12 human polyomaviruses, is the only one that causes cancer in hu
125 the presence of this pore is conserved among polyomaviruses, its functional role in infection or asse
126 document a unique DNA recombination between polyomavirus JC (JC virus [JCV]) and Epstein-Barr virus
127 ting disease caused by the human neurotropic polyomavirus JC (JCV) and is found almost exclusively in
130 the white matter of the brain caused by the polyomavirus JC virus, which typically occurs only in im
132 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human po
135 a fatal disease caused by reactivation of JC polyomavirus (JCPyV) in immunosuppressed individuals and
138 nally, endocytosis-dependent infection by JC polyomavirus (JCPyV) was reduced in human cells with dec
139 rebral manifestations are associated with JC polyomavirus (JCPyV) which are diagnosed by detection of
140 the receptor-binding properties of human JC polyomavirus (JCPyV), a virus that resides in the kidney
145 tients that treatment with interleukin 7, JC polyomavirus (JCV) capsid protein VP1, and a Toll-like r
147 of the central nervous system (CNS) with JC polyomavirus (JCV) usually occur as a result of immunoco
148 s from lytic infection of the glia by the JC polyomavirus (JCV); JCV granule cell neuronopathy is cau
149 d persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyo
150 Combined, these results demonstrate that polyomaviruses lacking miRNAs can arise infrequently and
153 iated with clinical presentation: intrarenal polyomavirus load levels and Banff interstitial fibrosis
154 f DNA translocation by papillomavirus E1 and polyomavirus LTag hexameric helicases involves consecuti
155 ic infection of oligodendrocytes by human JC polyomavirus may result in the development of progressiv
156 CD8 T cell responses to infections by human polyomaviruses may be influenced by VP1 mutations involv
157 PyV-6 DNA load and VP1 protein suggests that polyomaviruses may contribute to the epithelial prolifer
158 ve to be the first case in which Merkel cell polyomavirus (MCPyV) and human papillomavirus subtype 17
159 rcinomas (MCCs) that contain the Merkel cell polyomavirus (MCPyV) and the clinical significance of tu
164 In at least 80% of all MCC, Merkel cell polyomavirus (MCPyV) DNA has undergone clonal integratio
166 ng evidence indicates a role for Merkel cell polyomavirus (MCPyV) in the development of Merkel cell c
171 ns, copy number alterations, and Merkel cell polyomavirus (MCPyV) sequence were analyzed and compared
172 l integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC tumor cells express putati
175 me (ALTO) in the early region of Merkel cell polyomavirus (MCPyV), the causative agent of most Merkel
178 incidence, prevalence, and persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus,
187 ntly associated with clonal integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC
188 aneous tumor system with mice expressing the polyomavirus middle T (PyMT) oncogene under control of t
190 In addition, mammary gland tumors induced by polyomavirus middle T antigen in JNK2(-/-) mice were mor
191 r cells derived from mice transgenic for the polyomavirus middle T oncogene to ionizing radiation res
192 RT-deficient mice displayed marked delays in polyomavirus middle T oncogene-induced (PyMT-induced) ma
193 tors in the mouse mammary tumor virus (MMTV)-polyomavirus middle T oncoprotein (PyMT) mouse model of
195 k Institute set out to determine whether the polyomavirus middle T-transforming protein had a similar
196 of breast cancer, including the widely used polyomavirus middle-T antigen (PyVmT) model, which provi
197 rovide evidence for a unique function of the polyomavirus miRNA that may have important implications
203 vergent polyomavirus, provisionally named MX polyomavirus (MXPyV), in stool samples from children.
207 alitative highly predictive urinary test for polyomavirus nephropathy (PVN) is the PV-Haufen test.
209 finitive PVN from the Banff Working Group on Polyomavirus Nephropathy, comprising nine transplant cen
212 ding for middle T antigen (MT) is the murine polyomavirus oncogene most responsible for tumor formati
214 MCPyV), and MCC tumor cells express putative polyomavirus oncoprotein small T antigen (sTAg) and trun
215 mavirus sequence repeats in complex with the polyomavirus origin-binding domain reveals that only thr
217 viruses, herpesviruses, papillomaviruses and polyomaviruses) over time were observed in individuals w
218 ttempted to reveal the composition of the JC polyomavirus population (the quasispecies, i.e., the who
221 ach, we identified a novel, highly divergent polyomavirus, provisionally named MX polyomavirus (MXPyV
223 ) mice are susceptible to tumor induction by polyomavirus (Py), while C57BR/cdJ (BR) mice are resista
242 study (n=40) to explore associations between polyomavirus reactivation and immune responses to the se
243 Furthermore, these results indicate that polyomavirus reactivation associates with immune respons
245 ore prevalent during year 1 in subjects with polyomavirus reactivation than in those without reactiva
246 conserved VP1 proteins from closely related polyomaviruses recognize different oligosaccharides.
248 crystal structure of the four central murine polyomavirus sequence repeats in complex with the polyom
249 how that miRNAs from related variants of the polyomavirus simian vacuolating virus 40 (SV40) have dif
251 comparing the cytosolic entry of the related polyomavirus simian virus 40 (SV40), we found that depen
252 usage affect tropism, we studied the primate polyomavirus simian virus 40 (SV40), which uses the gang
258 xamined whether some of the functions of the polyomavirus small T antigens (ST) are shared by the E6
261 yomavirus, we show that brain-resident mouse polyomavirus-specific CD8 T cells, unlike memory cells i
265 for the transforming properties of nonhuman polyomaviruses, such as simian virus 40 (SV40), but is n
267 model is described that predicts patterns of polyomavirus SV40 infections and associated cancers in h
269 tosol membrane transport of the nonenveloped polyomavirus SV40, a decisive infection step, a cytosoli
273 Jersey polyomavirus (NJPyV-2013) is a novel polyomavirus that may have tropism for vascular endothel
274 question by focusing on a single lineage of polyomaviruses that infect both humans and their closest
277 cell carcinoma (MCC), making MCPyV the first polyomavirus to be clearly associated with human cancer.
280 in complex, a tumor suppressor in some other polyomavirus transformation models, but was strictly dep
281 en (MT), the principal oncoprotein of murine polyomavirus, transforms by association with cellular pr
282 we show that manifestations of the causative polyomavirus (TSPyV) occur during primary infection of t
283 witnessed the discovery of eleven new human polyomaviruses, two of which cause unusual and rare canc
285 arison with other structurally characterized polyomavirus VP1 proteins enhances our understanding of
287 ere found with the expression of recombinant polyomavirus Vp1s and human papillomavirus L1s in COS-7
298 lutionary divergence and diverse sampling of polyomaviruses, we propose evolutionary transitions that
299 We review what is currently known about JC polyomavirus, what is suspected, and what remains to be
300 (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPy
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