ライフサイエンスコーパス: polysomnography

1 apnea-hypopnea index >/=15 events per hour (polysomnography).

2 SDB was assessed at baseline with full polysomnography.

3 ltaNC) and leg fluid volume before and after polysomnography.

4 s assessed on separate nights using standard polysomnography.

5 ging from normal to obese underwent attended polysomnography.

6 raphy that followed 1 night of accommodation polysomnography.

7 iary care medical center underwent overnight polysomnography.

8 red questions instead of the 'gold standard' polysomnography.

9 presence of OSA was determined by overnight polysomnography.

10 concurrent UES and esophageal manometry and polysomnography.

11 lance Test, diaries of patients, and daytime polysomnography.

12 per hour of sleep, measured by in-laboratory polysomnography.

13 nd without underlying lung disease underwent polysomnography.

14 nnaire, and underwent oximetry and two-night polysomnography.

15 BP) in 4,409 subjects referred for overnight polysomnography.

16 5 to February 1998 and characterized by home polysomnography.

17 Thirty-five patients underwent polysomnography.

18 r hour of sleep, was measured during in-home polysomnography.

19 on) [corrected], obtained by unattended home polysomnography.

20 of spontaneous awakenings was determined by polysomnography.

21 ness characterized by insomnia, confirmed by polysomnography.

22 somnia with demonstration of the disorder by polysomnography.

23 ian time (phase), and sleep was monitored by polysomnography.

24 s per hour of sleep, measured during in-home polysomnography.

25 breathing was excluded by complete overnight polysomnography.

26 ning by specialists in patients referred for polysomnography.

27 ilateral cortical windows and electrodes for polysomnography.

28 had undiagnosed sleep apnea, as detected by polysomnography.

29 f rapid eye movement sleep without atonia on polysomnography.

30 th functional magnetic resonance imaging and polysomnography.

31 All of the subjects underwent polysomnography.

32 h as electroencephalographic recordings from polysomnography.

33 inical-biochemical assessment and a standard polysomnography.

34 nd obstructive sleep apnoea, as confirmed by polysomnography.

35 sleep obtained from overnight in-laboratory polysomnography.

36 me on actigraphy and apnea-hypopnea index on polysomnography.

37 of SV by digital photoplethysmography during polysomnography.

38 ilated subjects were monitored by continuous polysomnography.

39 n tests, transthoracic echocardiography, and polysomnography 3 months after ICU discharge.

40 In 89 patients with NAION who underwent polysomnography, 67 (75%) had OSAS.

41 Measurements included polysomnography, a multiple sleep latency test, an oral

42 smography (RIP) with simultaneously recorded polysomnography-acquired nasal end-tidal CO(2) (PET(CO(2

43 thods to measure sleep in the ICU, including polysomnography, actigraphy, and questionnaires.

44 All patients received follow-up polysomnography after 6 months.

45 roblem severity and depression severity, and polysomnography after at least 2 weeks of abstinence.

46 rice-weekly hemodialysis underwent overnight polysomnography along with measurement of total body ext

47 d patients and 13 healthy subjects underwent polysomnography and [(18)F]fluorodeoxyglucose positron e

48 Polysomnography and activity monitoring revealed a profo

49 ndex (mean score=49; range=15-111)-underwent polysomnography and completed the Center for Epidemiolog

50 who were diagnosed with OSA after overnight polysomnography and control children matched on the basi

51 Full montage home-polysomnography and fasting glucose were available on al

52 dren (ages 4.8-12 years) underwent overnight polysomnography and fasting homeostatic model (HOMA) of

53 a fasting morning blood sample at diagnostic polysomnography and follow-up 1.3 +/- 0.6 yr later.

54 o completed baseline and follow-up overnight polysomnography and had complete questionnaire-based dat

55 of 112 Minnesota residents who had undergone polysomnography and had died suddenly from cardiac cause

56 n=10) subjects were assessed with concurrent polysomnography and LFP recordings from the DBS electrod

57 Polysomnography and lumbar puncture were performed in OS

58 with a wide range of SDB severity underwent polysomnography and measurement of high-sensitivity CRP.

59 eep Heart Health Studies underwent overnight polysomnography and measurement of high-sensitivity trop

60 nts (mean age, 62.5 +/- 5.5 years) underwent polysomnography and measurement of hs-TnT.

61 ence analysis on 127 patients with nocturnal polysomnography and MSLT, including 25 with narcolepsy w

62 f 2 weeks of actigraphy at home, 2 nights of polysomnography and multiple sleep latency tests in the

63 recruited from public schools and underwent polysomnography and neurocognitive assessments of intell

64 ects (9 women, age 22 to 45 years) underwent polysomnography and simultaneous recording of ECG, blood

65 Polysomnography and spirometry results from 5,954 partic

66 The patients underwent polysomnography and studies of their ventilatory respons

67 5.71) healthy subjects underwent a nocturnal polysomnography and T1 MRI.

68 rior history of hypothyroidism who underwent polysomnography and thyroid function testing, four new c

69 ) underwent 3 consecutive nights of standard polysomnography and weight and height assessments as par

70 Participants underwent baseline polysomnography and were followed for a mean 7.3 years f

71 ltisite community-based study that conducted polysomnography and wrist actigraphy.

72 a Index (AHI) greater than 15 as assessed by polysomnography, and in 14 older adults (age +/- SD: 62.

73 diffusion tensor imaging and structural MRI, polysomnography, and neuropsychological assessments.

74 affeine versus placebo underwent actigraphy, polysomnography, and parental sleep questionnaires.

75 of SDB was based on the results of overnight polysomnography, and severe SDB was defined as an apnea-

76 ions by magnetoencephalography together with polysomnography, and source-localized the origins of osc

77 cohort study, consecutive patients underwent polysomnography, and subsequent events (strokes and deat

78 The subjects underwent clinical assessment, polysomnography, and wrist actigraphy.

79 ter sleep onset, derived from sleep diaries; polysomnography; and symptoms of fatigue, sleepiness, an

80 In a clinical research facility, overnight polysomnography, anthropometry, and 9 blood pressure mea

81 Findings on nocturnal polysomnography are described.

82 Using polysomnography as the criterion standard, we prospectiv

83 seline to 6 months, measured by a full-night polysomnography assessed by masked investigators in a co

84 ransplant recipients who underwent one-night polysomnography at baseline and were followed for a medi

85 ividuals who underwent overnight, unattended polysomnography at home.

86 ta, personal and treatment history, and full polysomnography at home.

87 ires, 24-hour blood pressure monitoring, and polysomnography at the end of follow-up.

88 ) was determined by unattended, single-night polysomnography at the participant's home.

89 a group of patients with OSA diagnosed with polysomnography between 1992 and 2004 (apnea-hypopnea in

90 eep center in Zaragoza, Spain, for nocturnal polysomnography between January 1, 1994, and December 31

91 ocardiography, range-of-motion measurements, polysomnography, clinical laboratory evaluations, measur

92 Polysomnography, cognitive performance, and sleepiness w

93 PVI between 2007 and 2010, 62 patients had a polysomnography-confirmed diagnosis of OSA.

94 Eighty-seven patients with polysomnography-confirmed IRBD underwent (123) I-FP-CIT

95 Polysomnography data from 2121 people were included in t

96 Blood pressure and overnight polysomnography did not change after fat gain or loss.

97 ng sound recordings during in-lab full-night polysomnography, drug-induced sleep endoscopy (DISE), an

98 Eleven OSA subjects underwent a night of polysomnography during which the physiological traits we

99 as superior to placebo on all subjective and polysomnography end points at night 1/week 1, month 1, a

100 patients at night 1, month 1, and month 3 by polysomnography end points of wakefulness after persiste

101 ts were tested with 1 night of in-laboratory polysomnography followed by a cognitive evaluation the n

102 , the apnea-hypopnea index was determined by polysomnography followed by determination of anatomic (p

103 to investigate BP homeostasis, we conducted polysomnography followed by tilt-table testing on 15 sub

104 istory alone, but some may require nocturnal polysomnography for accurate diagnosis and determining a

105 RECOMMENDATION 2: ACP recommends polysomnography for diagnostic testing in patients suspe

106 rty patients undergoing nocturnal diagnostic polysomnography for sleep apnea underwent transcranial D

107 ipants were assessed overnight by 18-channel polysomnography for sleep-disordered breathing, as defin

108 es in each of 38 adult patients evaluated by polysomnography for sleep-disordered breathing.

109 336 consecutive adult patients who underwent polysomnography for suspected OSA.

110 ay be a simpler alternative to in-laboratory polysomnography for the management of more symptomatic p

111 and respiratory parameters were recorded by polysomnography from 4 PM to 9 AM on the second, third,

112 ants were individuals referred for overnight polysomnography from March 1 to August 30, 2015.

113 nd slow sleep spindle duration in full-night polysomnography has only been reported in females but no

114 Polysomnography has specific preoperative indications.

115 We monitored Pes during polysomnography in 155 patients and compared their sleep

116 challenge (nasal occlusion) during overnight polysomnography in 31 women (45 to 55 yr).

117 herefore measured blood pressure (BP) during polysomnography in 41 children with OSAS, compared to 26

118 We also assessed its accuracy compared with polysomnography in a sample of the study population.

119 e (OSAS) is usually diagnosed with overnight polysomnography in a sleep laboratory.

120 mated with oesophageal pressure, Pes) during polysomnography in four adult volunteers and applied the

121 sual Analogue Scale measures) and sleep (via polysomnography), including increased REM and NREM sleep

122 The cost-utility of polysomnography instead of home study or no testing in t

123 Polysomnography is invaluable for the evaluation of slee

124 increased neck circumference), but overnight polysomnography is needed to confirm presence of the dis

125 es as an alternative to polysomnography when polysomnography is not available for diagnostic testing.

126 Therefore, polysomnography is not necessary for most patients with

127 Polysomnography is the diagnostic standard, but is often

128 ss (6 mo) of home respiratory polygraphy and polysomnography management protocols in patients with in

129 SD] age: 82.3 [3.2] years) who had overnight polysomnography measured between January 2002 and April

130 Confirmatory tests such as polysomnography, multiple sleep latency test, and actigr

131 = 4,307-10,332), actigraphy (n = 1,513), or polysomnography (n = 3,021).

132 and dermatitis, sleep variables measured by polysomnography, nocturnal urinary levels of 6-sulfatoxy

133 t were adequately specific (77%) to rule out polysomnography noninsomnia.

134 MSLTs were conducted following nocturnal polysomnography (NPSG) and daily sleep diaries in 289 ma

135 ur asymptomatic subjects underwent nocturnal polysomnography (NPSG) with monitoring of flow (nasal ca

136 leep Latency Test (MSLT) following nocturnal polysomnography (NPSG).

137 mode of mechanical ventilation, we performed polysomnography on 11 critically ill patients.

138 sleep apnea, have improvements documented by polysomnography on the night of surgery following adenot

139 patients and 17 healthy volunteers underwent polysomnography on two consecutive nights.

140 n a population of 10,701 adults referred for polysomnography, OSA predicted incident SCD, and the mag

141 The incremental charges for polysomnography over home study or no testing were about

142 the mean oxygen desaturation index among the polysomnography parameters (P = 0.023).

143 ng a night of sleep, which we monitored with polysomnography, participants encoded a second set of fa

144 Polysomnography plays an important role in addressing pi

145 y polygraphy protocol was noninferior to the polysomnography protocol based on the Epworth scale.

146 were randomized to respiratory polygraphy or polysomnography protocols.

147 orientation was assessed in 21 patients with polysomnography-proven idiopathic REM sleep behaviour di

148 Under almost all modeled conditions, polysomnography provided maximal quality-adjusted life-y

149 ive care unit (ICU) patients with continuous polysomnography (PSG) and environmental noise measuremen

150 n patients with advanced cancer, but 24-hour polysomnography (PSG) examinations of these patterns hav

151 asingly conducted at home) versus laboratory polysomnography (PSG) for diagnosing obstructive sleep a

152 l patients and controls underwent over-night polysomnography (PSG) for the diagnosis of OSAS and calc

153 esults of the complementary tests, including polysomnography (PSG), brain imaging and genetic analysi

154 ho underwent overnight unattended 12-channel polysomnography (PSG).

155 and tolerances in volunteers after overnight polysomnography (PSG).

156 Measurements: Polysomnography, pulmonary function tests, arterial bloo

157 Among individuals referred for overnight polysomnography, quantitative markers of eyelid laxity w

158 ekly episodes (72 to 96 hours) of continuous polysomnography (r=0.94) and work logs that were validat

159 nasal CPAP, recalibration of nasal CPAP with polysomnography, regular downloading of home data, and a

160 The proper interpretation of infant polysomnography requires an understanding of normative d

161 tain information on thyroid function status, polysomnography results, levothyroxine use, and clinical

162 nce and severity of OSA were determined from polysomnography results.

163 Despite careful screening, polysomnography revealed that 1 of 25 normal-weight subj

164 seline to 2-year change in AHI on diagnostic polysomnography scored by staff blinded to randomization

165 AS is common in patients with NAION and that polysomnography should be considered in these patients.

166 In five of five patients, video polysomnography showed features of obstructive sleep apn

167 health-related quality-of-life assessments, polysomnography, spirometry, 6-minute-walk distance, dro

168 isconsin state employees to attend overnight polysomnography studies at 4-year intervals.

169 Adult patients undergoing diagnostic polysomnography studies at a private Australian universi

170 d covariate information were assessed during polysomnography studies through March 2013.

171 nts who were 30-70 years of age had baseline polysomnography studies to assess the presence of sleep-

172 ep diagnoses and sleep architecture based on polysomnography studies, actigraphy assessment, and 24-h

173 f <5 events/h and not treated) by 2 baseline polysomnography studies.

174 Of 1,001 polysomnography subjects, 90 with SDB defined as a respi

175 ctive sleep measures (e.g. wrist actigraphy, polysomnography) support links between disturbed sleep a

176 nts underwent a single night of experimental polysomnography that followed 1 night of accommodation p

177 video-electroencephalographic telemetry and polysomnography, the differential diagnostic challenges

178 Using polysomnography, the gold standard for sleep assessment,

179 Prior to overnight polysomnography, the participants were given a battery o

180 en ages 30 to 60 yr was studied by overnight polysomnography to assess the frequency of apneas and hy

181 Patients underwent polysomnography to detect OSAS and were prospectively fo

182 Nocturnal polysomnography to evaluate apnea-hypopnea index.

183 All participants underwent polysomnography to exclude obstructive sleep apnea or ot

184 All control subjects underwent overnight polysomnography to exclude the existence of occult OSA.

185 ervational study, we used clinical and video polysomnography to identify a novel sleep disorder in th

186 plus nocturnal pulse oximetry against using polysomnography to identify patients without apnea (Obje

187 (working memory measure) tests and overnight polysomnography to investigate the specific sleep-depend

188 HI) of at least 20 events per h, tested by a polysomnography, underwent device implantation and were

189 Polysomnography was assumed as the gold standard.

190 prevalence of objective insomnia assessed by polysomnography was higher than the prevalence of subjec

191 Home unattended polysomnography was performed and scored using similar p

192 Overnight polysomnography was performed during the 2 sessions.

193 Full night polysomnography was performed during two consecutive nig

194 Polysomnography was performed throughout the study.

195 teral local injection of orexin-saporin, and polysomnography was performed to measure baseline sleep

196 months after the LT; in each phase, standard polysomnography was performed, and anthropometric, patho

197 Full polysomnography was used to characterize SDB.

198 Polysomnography was used to characterize sleep architect

199 ed neuroimaging techniques [7, 8] as well as polysomnography, we found that the temporary sleep distu

200 Using simultaneous fiber photometry and polysomnography, we observed time-delineated dorsal raph

201 2omSOREMPs on an MSLT that follows nocturnal polysomnography, we reviewed data from 1,145 consecutive

202 ide ventriculography, Holter monitoring, and polysomnography were done.

203 se and heart failure at the time of baseline polysomnography were followed up for a median of 8.7 yea

204 ation averaged over 5 minutes, and overnight polysomnography were obtained in participants, each with

205 onnaires from patients and bed partners, and polysomnography were obtained on all subjects in compari

206 Quality of life, blood pressure, and polysomnography were similar between protocols.

207 of sleep apnea, derived from 12-channel home polysomnography, were the apnea-hypopnea index (average

208 t serious comorbidities as an alternative to polysomnography when polysomnography is not available fo

209 These five patients had been assessed with polysomnography, which was done in our sleep unit in one

210 ure (Pes) monitoring can be performed during polysomnography with a thin, water-filled catheter conne

211 in 146 participants who underwent overnight polysomnography with an epiglottic catheter to measure t

212 Some patients referred for polysomnography with complaints of excessive daytime sle

213 removed by UF, followed by repeat overnight polysomnography with fluid measurements.

214 (MrOS) Sleep Study cohort underwent in-home polysomnography with PLMS measurement and were followed

215 ygraphy management is similarly effective to polysomnography, with a substantially lower cost.

216 and related symptoms (all P < .01), but not polysomnography, with similar improvements in both group

217 clinical evaluation, subjective scales, four polysomnographies without nasal CPAP, recalibration of n