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1 l brain regions (thalamus, hypothalamus, and pons).
2 ed all the way to the ventrolateral pons (vl-pons).
3  descended ventromedially through the rabbit pons.
4 d the lowest concentration in the cerebellum/pons.
5  ancestor of the three families of mammalian PONs.
6 ppocampus, striatum, thalamus, midbrain, and pons.
7 poral cortex, insula, pulvinar, caudate, and pons.
8 r week; 95% CI: -0.05, 0.28) was seen in the pons.
9 generator plus a portion of the dorsolateral pons.
10 tivity results in hypertrophy of the ventral pons.
11 ncluding thalamus, cerebellum, brainstem and pons.
12 ages were normalized to mean activity in the pons.
13 (PBel-inner) cell groups of the dorsolateral pons.
14 al plane, extending through the midbrain and pons.
15 ties of the cerebral cortex, cerebellum, and pons.
16 he midbrain; and parabrachial nucleus in the pons.
17 nual anterior cingulate cortex and brainstem pons.
18 igating host-pathogen interactions and lacks PONs.
19  projections to the spinal cord, tectum, and pons.
20           [18F]FDG images were normalized to pons.
21 ect to the parabrachial nucleus (PBN) of the pons.
22 nt in the white tracts of the cerebellum and pons.
23 um, midbrain and anterior part of cerebellum/pons.
24 m from the entry point of the nerve into the pons.
25 ardiorespiratory centers of the dorsolateral pons.
26 bellum, right fusiform, parahippocampus, and pons.
27  neostriatum, superior colliculus, and basal pons.
28 ry visceral sensory areas of the medulla and pons.
29 fetal lung maturation and development of the pons.
30 caudal pressor area (CPA) to the medulla and pons.
31 throughout the rostral- caudal extent of the pons.
32 ional anisotropy values, uniquely within the pons.
33 ersing from the opposite, intact side of the pons.
34 ly placed curved lamellae in mid- and caudal pons.
35 egrity of a number of regions in the rostral pons.
36 jections encircle the peduncle in the caudal pons.
37 lum, and characteristic flattened dystrophic pons.
38 iratory centers of the medulla oblongata and pons.
39 volume deficits in both the thalamus and the pons.
40 nsection of the brainstem through the caudal pons.
41 served at locus ceruleus and in anteromedial pons.
42 bnormal processes in the cerebral cortex and pons.
43 bus pallidus, dentate nucleus, thalamus, and pons.
44 men, thalamus, and caudate, and midbrain and pons.
45  were observed primarily in the rostromedial pons.
46  instead were restricted to the intermediate pons.
47 the distribution of labeled terminals in the pons.
48 morphometric analyses of the postnatal human pons (0-18 years; n = 6-14/timepoint).
49 tly higher (P < 0.05) in the medulla (176%), pons (146%), midbrain (101%), hippocampus (85%), thalamu
50 In MSA-P, atrophy rates were greatest in the pons: 4.5% (3.2%), over 20 times that in controls and th
51 ment, in 1 in the cerebellum and in 1 in the pons), abnormally dilated extraaxial fluid collections i
52 red molecular compositions, we estimate that pONs account for 3% and 8% of total submicrometer organi
53 teral (n = 7) and were located either in the pons alone (n = 4) or in the upper pons and the midbrain
54                  Compared with cerebellum or pons alone, reference regions that included subcortical
55 insoluble tangle-tau isolated from the basal pons also differed significantly.
56 lliker-Fuse nucleus (KF) in the dorsolateral pons, an important centre for control of respiratory rhy
57 ssion and TrkB activation in the medulla and pons and (2) breathing dysfunction, characterized by inc
58 d posterior inferior cerebellum) and middle (pons and anterior inferior cerebellum) territories were
59 s to distant subcortical targets such as the pons and as far caudal as the pyramidal decussation and
60  ratio 3D-SSP values were computed using the pons and cerebellar cortex as reference regions.
61 on between the white matter integrity of the pons and cerebellar gray matter volume associated with h
62  genome, in frontal cortex, temporal cortex, pons and cerebellum from 387 human donors between the ag
63             In addition to reduced volume of pons and cerebellum, affected individuals had microcepha
64 ders characterized by impaired growth of the pons and cerebellum, which frequently follows a degenera
65 riatal correlations with thalamus, midbrain, pons and cerebellum.
66 enatal onset, characterized by hypoplasia of pons and cerebellum.
67  ratio differences were calculated for DN-to-pons and DN-to-middle cerebellar peduncle (MCP) ratios b
68 ast MR examination were calculated for DN-to-pons and DN-to-middle cerebellar peduncle (MCP) ratios i
69 ilinear gadolinium enhancement peppering the pons and extending variably into the medulla, brachium p
70    The difference in mean SI ratios of DN to pons and GP to thalamus on unenhanced T1-weighted images
71 d no group differences were found when DN-to-pons and GP-to-pulvinar ratios were compared (DN-to-pons
72 (PBN) is located in the rostral dorsolateral pons and has been identified as a critical relay for car
73 maging sequences predominantly involving the pons and hippocampi.
74 y of labeled varicosities was highest in the pons and lowest in MI.
75  was most abundant in striatum, hippocampus, pons and medulla, and cortex.
76 pyramidal tracts and medial lemniscus of the pons and medulla.
77 g showed that the rostral projections to the pons and midbrain and caudal projections to the spinal c
78  was proposed that neural populations in the pons and midline cerebellum compute an independent, inte
79 uzole-treated patients with ALS (P < .05 for pons and motor cortex) and healthy controls (P < .05 for
80 or cortex) and healthy controls (P < .05 for pons and motor cortex).
81              The midline central gray of the pons and nucleus incertus receive input from the rostral
82 guingly, DIPGs are restricted to the ventral pons and occur during a narrow window of middle childhoo
83  the GABAergic neurons of the RVM and caudal pons and performed double labeling to evaluate the expre
84 ne selectively blocked activity in pulvinar, pons and posterior insula.
85  (GPi) (P < 0.001), as well as in the dorsal pons and primary motor cortex (P < 0.0001).
86 s of TrkB phosphorylation in the medulla and pons and restored wild-type breathing frequency.
87 rtex had the highest VC content, whereas the pons and spinal chord had the lowest.
88 c increased T2-weighted signal in the dorsal pons and spinal cord.
89 ons in MI project most strongly to the basal pons and superior colliculus.
90 s present in the parabrachial nucleus of the pons and that these receptors serve to modulate feeding
91 l projections and innervate the dorsolateral pons and the ipsilateral ventral respiratory column (VRC
92 er in the pons alone (n = 4) or in the upper pons and the midbrain (n = 5).
93 entially through two sites: the dorsolateral pons and the paraventricular thalamic nucleus.
94  tegmental area, periaqueductal grey, dorsal pons and various cortical areas including occipital, tem
95 al and trigeminal information in multiple dl-pons and vl-pons structures in the rat.
96 d fluffy brainstem lesions, often located in pons and/or adjacent to fourth ventricle.
97 nce of lesion involving posterior paramedian pons and/or medial thalamus.
98 I) in the DN was normalized to the SI of the pons, and a one-sample t test was used to test for diffe
99 the clustering of 33 of 34 cerebella, 7 of 7 pons, and all 3 livers.
100 d: whole cerebellum, cerebellar gray matter, pons, and centrum semiovale.
101 eposition (such as subcortical white matter, pons, and cerebellum).
102 al areas, including the cortex, hippocampus, pons, and cerebellum.
103 ctivity within amygdala, anterior insula and pons, and engendered different effects on blood pressure
104 bers were found, while more rostrally in the pons, and in the midbrain-pontine junction part of the n
105 pontine terminals were absent in the rostral pons, and instead were restricted to the intermediate po
106 emporal pattern beginning in the cerebellum, pons, and internal capsule; proceeding caudocranially fr
107 bundance was highest in the hypothalamus and pons, and lowest in the cerebellum and medulla.
108 dala, and neocortex, whereas in spinal cord, pons, and medulla GPR88 expression remains discrete.
109 expressed predominantly in the hypothalamus, pons, and medulla of posthatch chick brains, but not in
110 in the putamen, midbrain, caudate, thalamus, pons, and medulla.
111 r SERT-rich regions (hypothalamus, striatum, pons, and prefrontal cortex).
112 , the dorsal vagal complex, the dorsolateral pons, and selected hypothalamic nuclei (dorsomedial, lat
113 he ventral thalamus, hypothalamus, midbrain, pons, and spinal cord.
114  gray matter [CGM], whole cerebellum [WCER], pons, and subcortical white matter [SWM]) were studied.
115 oss perceptual conditions in the cerebellum, pons, and subthalamic nucleus.
116 ion to the middle cerebellar peduncle (MCP), pons, and thalamus after repeated administration of the
117 nterior horns of the spinal cord, the dorsal pons, and the medulla can be clearly seen on MRI.
118 eport a newly identified family of bacterial PONs, and the reconstruction of the ancestor of the thre
119 cerebellum (WC), WC with brainstem (WC + B), pons, and white matter (WM).
120 ion is medial and ventral in rostral and mid-pons; and arm function is represented ventral and latera
121 use studies have demonstrated that all three PONs are atheroprotective.
122  presented, in which the dorsal midbrain and pons are implicated.
123                                Paraoxonases (PONs) are a family of lactonases with promiscuous enzyme
124                                Paraoxonases (PONs) are a family of proteins that may play a significa
125                          Serum paraoxonases (PONs) are detoxifying lactonases that were first identif
126            CG, WC, and WC + B, but not WM or pons, are reliable reference regions for amyloid imaging
127 ion from 60 to 90 min were created using the pons as a reference region and nine regions of interest
128 g potential values can be assessed using the pons as a reference region, with a test-retest repeatabi
129 BPND images in NHP template space, using the pons as a reference region.
130 ited by extended EDTA treatment, implicating PONs as the major enzymes inactivating 3OC12-HSL.
131              Automated quantification (using pons as the reference region) demonstrated 91% sensitivi
132                                     With the pons as the reference region, the optimal z score thresh
133           Significant SUVR changes using the pons as the RR were detected only at 2 y (P = 0.46 [1 y]
134                                       In the pons, Barrington's nucleus and the norepinephrine (NE) n
135 end axons to the dorsal aspect of the SC and pons but avoid ventral SC and the pyramidal tract, where
136 no evidence of postnatal neurogenesis in the pons, but each progenitor compartment produces new astro
137 maller volumes in the thalamus, splenium and pons, but not in the caudate or putamen.
138 vely; maximally in the subiculum and ventral pons, but often present elsewhere.
139 s suggest that lesions confined to the upper pons can cause coma in humans even in the absence of dam
140              Additionally, whole cerebellum, pons, centrum semiovale, and a composite region were exa
141 ts and lower volume in the basilar (ventral) pons, cerebellar white matter and visual cortex.
142         nNOS was abundantly expressed in the pons cerebellum and hypothalamus and less so in the cort
143 nt in the frontal cortex, midbrain, putamen, pons, cerebellum, and corpus callosum.
144 t in thalamus, intermediate in the midbrain, pons, cerebellum, and cortex; and least in white matter.
145 white matter of the infused frontal lobe and pons compared to control.
146       NAD(P)H oxide was most abundant in the pons compared to other regions.
147 , putamen, pallidum, thalamus, midbrain with pons (comprising a region of interest that includes the
148 tive/oxidative stressors on brainstem (upper pons, containing pedunculopontine and lateraldorsal tegm
149                            The ventrolateral pons contains the A5 group of noradrenergic neurons whic
150                                          The pons controls crucial sensorimotor and autonomic functio
151 on whether they project subcortically to the pons [corticopontine (CPn)] or to the contralateral cort
152 membrane properties: those projecting to the pons (CPn) and those projecting across the commissure to
153 d medial parabrachial nuclei of dorsolateral pons (dl-pons) plays an important role in respiratory ph
154 y projections to neurons in the dorsolateral pons (DLP) that project to the spinal cord dorsal horn.
155 one of the parabrachial nucleus (PBN) in the pons eliminate the salt (sodium chloride; NaCl) appetite
156 s containing WM (as opposed to cerebellum or pons) enabled detection of cortical change that was more
157 ar gray matter, whole cerebellum, brain stem/pons, eroded subcortical white matter [WM], and 2 additi
158 rity of proliferative cells in the postnatal pons expressed the transcription factor Olig2, suggestin
159 o drive the excitatory burst neurones in the pons (feed-forward).
160 uptake, normalized to cerebellum for PiB and pons for (18)F-FDG, were compared.
161 ed the specific activities of purified human PONs for 3OC12-HSL hydrolysis, including the common PON1
162 , loss of Nfib results in defects in basilar pons formation and hippocampus development that are not
163                     In addition, the basilar pons forms normally in dystrophin-deficient mice.
164    Neuronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus of these 23 deceased
165    Neuronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus were harvested and a
166 were used to compare SI and SI ratios (DN to pons, GP to thalamus) between case patients and control
167                                DN/MCP, DN-to-pons, GP-to thalamus, and GP-to-cerebrospinal fluid rati
168  are localized to rostral and medial basilar pons; hand coordination is medial and ventral in rostral
169                                   The dorsal pons has long been implicated in the generation of rapid
170 p generation, and the neural circuits in the pons have since been studied extensively.
171 ion, including the medial prefrontal cortex, pons, hippocampus, and anterior insula.
172 s but catalase and GPX were more abundant in pons, hypothalamus and medulla and less so in the cortex
173 can replicate the observed pON assuming that pONs (i) are produced in the gas phase and rapidly estab
174 d (all 3 regions in 6 patients, midbrain and pons in 39, and medulla and pons in 8).
175 ts, midbrain and pons in 39, and medulla and pons in 8).
176 l was higher than controls in the vermis and pons in AOA2 and in the vermis in FRDA.
177 tified abnormalities in the thalamus and the pons in both schizophrenia and alcoholism.
178 on mutations in ACVR1 occur in tumors of the pons in conjunction with histone H3.1 p.Lys27Met substit
179 ctivity of forty four neurons in the rostral pons in hypocretin knock-out mice.
180 ic nuclei in (and a broader role for) the dl-pons in integrating respiratory and nonrespiratory infor
181 campus, septum, thalamus, mesencephalon, and pons in knock-out mice than wild-type mice.
182                            The appearance of PONs in metazoa is likely to relate to innate immunity r
183 g of unit respiratory rhythmic neurons in dl-pons in urethane-anesthetized, vagotomized, paralyzed, a
184 e superior cerebellar peduncle in the rostal pons, in the corticopontocerebellar fibers, and in Purki
185 (R2=0.29; P<.01 corrected) and midbrain with pons, including the locus coeruleus (R2=0.18; P<.01 corr
186                The BBB in the cerebellum and pons/interpeduncular nuclei was highly sensitive to decr
187  Large glycosyltransferase gene, the basilar pons is absent.
188                          A large fraction of pONs is highly functionalized, possessing between six an
189 The activation pattern in the dorsal rostral pons is highly suggestive of a role for this structure i
190 r the topography of prefrontal inputs to the pons is similar in rats and rabbits.
191            Located within this region of the pons is the sublaterodorsal nucleus (SLD), a structure t
192 otor corticopontine studies suggest that the pons is topographically organized, but details remain un
193 N) resides in the rostral medulla and caudal pons, is implicated in cardiovascular regulation and cra
194 were spread across the dorsal portion of the pons just below the fourth ventricle.
195         Catecholaminergic cell groups in the pons (LC) and medulla (VLM, NTS) were also activated by
196 nd increased connectivity in the cerebellum, pons, left amygdala, and orbitofrontal cortices (cluster
197 labeled terminals were most prevalent in the pons, less prevalent in neostriatum and superior collicu
198       The resulting implications for mPFC or pons manipulations for studies of trace eyeblink in each
199                           The dorsal rostral pons may be a locus of neuromodulation by suboccipital s
200 gest the possibility that mPFC inputs to the pons may be integrated with different sources of cortica
201                                The mammalian PONs may therefore relate to a newly identified family o
202 conveyed to cerebellum via a pathway through pons, may engage the cerebellum and allow for the expres
203 used by ischemia of the posterior paramedian pons, medial thalamus, or cerebellum.
204 ficits at different brain regions, including pons, medulla oblongata, and cerebellum.
205  in animals with MeCP2 removed from specific pons medullary respiratory circuits, we performed whole-
206 ird-order premotor neurons were found in the pons, midbrain, and cerebellum, including dorsolateral a
207 y expressed in large neurons in the medulla, pons, midbrain, and thalamus.
208 sites such as the cervicomedullary junction, pons, midbrain, or the tectum.
209                              The cerebellum, pons, middle insula, and amygdala responded to intensity
210                        The dorsolateral (DL) pons modulates the respiratory pattern.
211   After atrophy correction and adjusting for pons MRglc, PET MRglc reductions were found in all FAD s
212            Lesions were predominantly in the pons (n = 8) and cerebellum (n = 6).
213 rebral cortex (n=35), cerebellum (n=34), and pons (n=7), along with liver samples (n=3) from 43 indiv
214 gions using whole-brain-normalized (WBN) and pons-normalized (PN) activity.
215 e olfactory bulb, in the cerebral cortex, in pons nuclei, in the red nucleus, in all cranial nerve nu
216  Cre recombinase (AAV-Cre) into the midbrain/pons of mice carrying a loxP-conditional tryptophan hydr
217                                          The pons of rats and rabbits, however, shows divergence in g
218 reted protein normally expressed only in the pons of the brain and sweat glands.
219  versus an increase of centrality within the pons of the brainstem, highlighting the important role o
220 e examined in the rostral medulla and caudal pons of the cat after (i) sham, (ii) stimulation of the
221 ) in the cortex, hippocampus, cerebellum and pons of the Eker rats.
222 basal forebrain, diencephalon, midbrain, and pons of the minke whale, a mysticete cetacean.
223     The sublaterodorsal nucleus (SLD) in the pons of the rat is a locus supporting short-latency indu
224 basal forebrain, diencephalon, midbrain, and pons of the river hippopotamus, one of the closest extan
225 ed for the normal development of the basilar pons, one of several precerebellar nuclei of the hindbra
226 ptomeningeal disease; metastases in medulla, pons, or midbrain; or liver metastases.
227 diffuse intrinsic gliomas, most often in the pons, or the nondiffuse brainstem tumors originating at
228 for the production and phase partitioning of pONs, organic aerosol mass, and reactive nitrogen specia
229 01), subcortical white matter (P < .05), and pons (P < .01) and higher levels of myo-inositol in the
230 em activation was seen in the dorsal lateral pons (P < 0.05 after small volume correction) during the
231 GP), thalamus (T), dentate nucleus (DN), and pons (P) were measured on unenhanced T1-weighted images.
232 icant in frontal cortex, temporal cortex and pons: P ranging from 4.8 x 10(-12) to 3.4 x 10(-3).
233              After transection of the caudal pons, part of the remaining tone was removed by inhibiti
234 l remarkable postnatal growth in the ventral pons, particularly during infancy when cells are most pr
235                  These findings suggest that PONs, particularly PON2, could be an important mechanism
236 parabrachial nuclei of dorsolateral pons (dl-pons) plays an important role in respiratory phase switc
237 ergic agonist carbachol into the dorsomedial pons produces an REM sleep-like state with muscle atonia
238 e, the dichotomy of motor-predominant caudal pons projections to cerebellar anterior lobe, contrasted
239 trasted with associative-predominant rostral pons projections to cerebellar posterior lobe, is consis
240                    Our data suggest that the pons provides a necessary excitatory drive to an additio
241                      We show that the murine pons quadruples in volume postnatally; growth is fastest
242  GBCA applications: DN: r = -0.254, P = .31; pons: r = -0.097, P = .65; SN: r = -0.194, P = .38; GP:
243 f administered GBCA: DN: r = 0.091, P = .72; pons: r = 0.106, P = .62; SN: r = -0.165, P = .45; GP: r
244 caudal NTS, area postrema, VRC, dorsolateral pons, raphe nuclei, lateral hypothalamus, central amygda
245 an SI ratio increase (Bayes factor for DN-to-pons ratio = 0.09 and that for DN-to-MCP ratio = 0.12).
246 d GP-to-pulvinar ratios were compared (DN-to-pons ratio in case mean, 1.0083 +/- 0.0373 [standard dev
247 .0083 +/- 0.0373 [standard deviation]; DN-to-pons ratio in control mean, 1.0183 +/- 0.01917; P = .37;
248 s did not differ significantly from 0 (DN-to-pons ratio: -0.0012 +/- 0.0101, P = .436; DN-to-MCP rati
249 s did not differ significantly from 0 (DN-to-pons ratio: -0.0032 +/- 0.0154, P = .248; DN-to-MCP rati
250 against an SI ratio increase (0.10 for DN-to-pons ratio; 0.27 for DN-to-MCP ratio).
251                             DN/MCP and DN-to-pons ratios were significantly different between control
252              The lateral central gray of the pons receives bilateral input from the lateral IP, which
253 ogenous GABA localized to this region of the pons reduced the incidence of apnoea and the respiratory
254 ed better registration in the cerebellum and pons reference region area.
255 nterior cingulate cortex (ACC) and brainstem pons region.
256 f soma size and axonal pathology even in the pons region.
257 e (PON) family members, we hypothesized that PONs regulate P. aeruginosa virulence in vivo.
258 ite matter growth of the corpus callosum and pons relative to nondrinkers.
259 tion, the postnatal development of the human pons remains poorly understood.
260 lts DN/MCP (rho = 0.51, P < .0001) and DN-to-pons (rho = 0.41, P = .0001) ratios correlated positivel
261                                       WM and pons showed larger YC variances than other regions.
262 Globus pallidus-thalamus and dentate nucleus-pons SI ratios were calculated and compared between grou
263 ed, preferentially in the caudal half of the pons, situated in close proximity to traversing corticof
264 the putamen/globus pallidus, dorsal midbrain/pons, STN, and ventral thalamus.
265 minal information in multiple dl-pons and vl-pons structures in the rat.
266 ritic trees that spanned several of these dl-pons subnuclei, often with terminal dendrites ending in
267 f interest (ROIs): the dentate nucleus (DN), pons, substantia nigra (SN), pulvinar thalami, and globu
268 anges were not observed in the cerebellum or pons, suggesting that cortex is more susceptible to oxid
269 ifferent tau isoform deposition in the basal pons suggests that this may ultimately prove to be a dis
270 FDG uptake were also quantified as target-to-pons SUV ratios in 12 regions of interest (ROIs).
271 twork characterized by increased activity in pons, thalamus, medial frontal and sensorimotor areas, h
272     Higher MTRs were seen in the thalami and pons than in the BG (P < .05), indicating earlier matura
273 rus were significantly less prevalent in the pons than the medulla.
274 providing an input to the cerebellum via the pons that bridges the temporal gap between conditioning
275  large laterodorsal tegmental nucleus of the pons that has both parvocellular and magnocellular choli
276 lesions involving the neurons of the basilar pons that link the ipsilateral cerebral cortex with the
277 ive malignant glial neoplasms of the ventral pons that, due to their location within the brain, are u
278 re calculated in the brainstem (midbrain and pons), the cerebellum, the lateral and third ventricles
279 t assessing the neurochemical profile of the pons, the cerebellar hemisphere and the vermis in patien
280 n projects mainly to the ipsilateral basilar pons, the MI whisker region has significantly more conne
281 inergic cell groups in the mesencephalon and pons, the principal nucleus of the trigeminal nerve, and
282                             2) In the dorsal pons, the PVH projects heavily to the pre-locus coeruleu
283 ntal nuclei, the median raphe nucleus of the pons, the ventral part of the medullary reticular nucleu
284 rogressing rostrally from the medulla to the pons, then to midbrain and substantia nigra, limbic stru
285 onary origins and substrate specificities of PONs therefore remain poorly understood.
286  patients with focal infarcts in the basilar pons to determine whether pontine lacunar syndromes conf
287  overlapping specificities of some mammalian PONs versus the singular specificity of others.
288 were traced all the way to the ventrolateral pons (vl-pons).
289                                              Pons volume increased 6-fold from birth to 5 years, foll
290 bus pallidus, thalamus, dentate nucleus, and pons was measured at unenhanced T1-weighted MR imaging.
291                 In the medulla oblongata and pons, we detected Hoxa5 expression in many precerebellar
292 onderance of cerebellar afferents within the pons, we observed a significant positive correlation bet
293 sures of the left and right thalamus and the pons were derived from magnetic resonance imaging scans
294            The PV-ir cells of RVM and caudal pons were found medial to the facial nucleus and lateral
295       CBF responses to LD in the putamen and pons were relatively greater in patients exhibiting drug
296   Speciated particle-phase organic nitrates (pONs) were quantified using online chemical ionization M
297                        Compared with SWM and pons, which do not fulfil the RR requirements and have l
298 e most common location for anomalies was the pons, which was involved in 114 patients.
299 rfusion of the primate right frontal lobe or pons with infusate.
300 eg coordination is in the caudal half of the pons, with lateral predominance.

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