ライフサイエンスコーパス: pontine

1 iations between age and the mean cortical to pontine 18F-florbetapir standard uptake value ratios, pr

2 neurones, and increased c-fos expression in pontine A7 and other noradrenergic neurones.

3 rome is bilateral horizontal gaze palsy from pontine abducens nuclear defects, rather than abducens n

4 ate laterality, it was found that the dorsal pontine activation was ipsilateral in the right-sided an

5 espiratory drive; (b) even though changes in pontine activity are transient, they can persist after n

6 We hypothesized that DL pontine activity during delayed-I tests would be compara

7 (splenium and genu), two projection (cortico-pontine and anterior thalamic), and five bilateral assoc

8 formation and cell loss is prominent in the pontine and dentate nuclei, with variable cell loss in o

9 Central pontine and extrapontine myelinolysis (CPM/EPM) are seve

10 Central pontine and extrapontine myelinolysis patients experienc

11 orphic sets of small-field neurons including pontine and fb-eb neurons, and also isomorphic sets of l

12 cuitry governing REM sleep is located in the pontine and medullary brainstem and includes ascending a

13 and strong FLNa labeling was evident in the pontine and medullary raphe nuclei and in sensory and sp

14 ior colliculus, dorsal raphe, mesencephalic, pontine and medullary reticular formation, and the follo

15 The present study was undertaken to identify pontine and medullary structures that respond to noxious

16 logical examination revealed extreme loss of pontine and olivary nuclei and Purkinje cells, with pres

17 Its receptor, FGFR2, is expressed by pontine and vestibular neurons when their axons (mossy f

18 those areas included limbic, basal ganglia, pontine, and cerebellar sites.

19 pare the power of template-based cerebellar, pontine, and cerebral white matter reference regions to

20 nges in an arousal network including limbic, pontine, and cortical areas underlying the decreased per

21 eral, parvicellular, caudal pontine, ventral pontine, and oral pontine reticular nuclei; the dorsomed

22 Parkinson's disease tremor is localized to pontine- and mesencephalic-cerebellar-thalamic circuits,

23 a cerebrovascular accident caused by a right pontine arteriovenous malformation and destruction of th

24 that in controls and three times the rate of pontine atrophy in PSP.

25 oradic PAPT, the combination of brainstem or pontine atrophy, parkinsonism, autonomic dysfunction or

26 including 1 proband from the first reported pontine autosomal dominant microangiopathy with leukoenc

27 ange dorsal guidance of inferior olivary and pontine axons after crossing the midline.

28 ed defects of the medial deep cerebellar and pontine axons observed in Unc5c(-/-) embryos, demonstrat

29 eus, reticular nuclei, nucleus raphe magnus, pontine blink premotor area, and superior salivatory nuc

30 d reciprocal inhibitory interactions between pontine brainstem monoaminergic and cholinergic neurons.

31 havioral arousal via descending input to the pontine brainstem.

32 The ability of pontine carbachol to elicit any cortical, hippocampal or

33 Both pontine cell groups express the transcription factor Fox

34 Pontine cells that receive inputs from visual cortical a

35 , the median raphe, caudal dorsal raphe, and pontine central gray are major recipients of LHb efferen

36 VTA, median raphe, caudal dorsal raphe, and pontine central gray reside in characteristic, but somet

37 s, and via caudal pontine reticular nucleus, pontine central gray, and MS, reached EC.

38 ral division of the periaqueductal gray, and pontine central gray.

39 s in the LC; medial GABAergic neurons in the pontine central gray; ventromedial, small GABAergic neur

40 may be bridged by forebrain regions through pontine-cerebellar nuclear connections that can bypass c

41 omparison with other mammals, the numbers of pontine cholinergic (126,776) and noradrenergic (122,878

42 itative analysis reveals that the numbers of pontine cholinergic (259,578) and noradrenergic (127,752

43 tative analysis revealed that the numbers of pontine cholinergic (274,242) and noradrenergic (203,686

44 enlarged posterior commissure, supernumerary pontine cholinergic and noradrenergic cells, and an enla

45 arged posterior commissure and supernumerary pontine cholinergic and noradrenergic neurons indicate t

46 ipulations of limbic cortico-striato-pallido-pontine circuitry.

47 sions, and may result from disruption of the pontine component of associative corticopontocerebellar

48 isite recordings were made within a putative pontine-cortical micturition circuit from the pontine mi

49 ot dystrophin is required for normal basilar pontine development.

50 Pontine efferent axons terminate mainly in V and rostral

51 Cortico-pontine fibers arose from the PcG, the caudal/medial SFG

52 Cortico-pontine fibers travel through the cerebral peduncles and

53 the generation of active sleep is gated by a pontine GABAergic system that exerts its effects postsyn

54 Analysis of pontine gene expression revealed that distinct programs

55 aracteristics in pediatric diffuse intrinsic pontine glioma (DIPG) and to correlate these metrics wit

56 ch tumors in children with diffuse intrinsic pontine glioma (DIPG) by measuring the tumor uptake of (

57 Diffuse intrinsic pontine glioma (DIPG) comprise a subset of HGG that aris

58 Diffuse intrinsic pontine glioma (DIPG) is a childhood brainstem tumor wit

59 Diffuse intrinsic pontine glioma (DIPG) is a fatal brain cancer that arise

60 Diffuse intrinsic pontine glioma (DIPG) is a fatal childhood cancer.

61 Diffuse intrinsic pontine glioma (DIPG) is a fatal pediatric cancer with l

62 Diffuse intrinsic pontine glioma (DIPG) is a highly aggressive pediatric b

63 survival for children with diffuse intrinsic pontine glioma (DIPG) is less than 10%, and new therapeu

64 survival for children with diffuse intrinsic pontine glioma (DIPG) is less than one year.

65 R1 have been identified in diffuse intrinsic pontine glioma (DIPG) tumors.

66 tic mutations in pediatric diffuse intrinsic pontine glioma (DIPG), we performed whole-genome sequenc

67 tic oligodendroglioma, and diffuse intrinsic pontine glioma (DIPG).

68 tal pediatric brain tumor, diffuse intrinsic pontine glioma (DIPG).

69 tric high-grade glioma and diffuse intrinsic pontine glioma and 20 publicly available datasets in an

70 d vascular morphology in a diffuse intrinsic pontine glioma patient was performed.

71 childhood brainstem tumor diffuse intrinsic pontine glioma to harbor somatic mutations in ACVR1.

72 w- and high-grade gliomas, diffuse intrinsic pontine glioma, and ependymoma) and some selected rare t

73 explain lack of effect in diffuse intrinsic pontine glioma.

74 pediatric HGGs, including diffuse intrinsic pontine gliomas (DIPGs) and non-brainstem HGGs (NBS-HGGs

75 Diffuse Intrinsic Pontine Gliomas (DIPGs) are deadly paediatric brain tumo

76 Diffuse intrinsic pontine gliomas (DIPGs) are highly aggressive tumors of

77 Diffuse intrinsic pontine gliomas (DIPGs) are highly infiltrative malignan

78 We report that human diffuse intrinsic pontine gliomas (DIPGs) containing the K27M mutation dis

79 n childhood HGG, including diffuse intrinsic pontine gliomas, and gene expression analyses supported

80 target for pediatric HGG, including diffuse pontine gliomas.

81 easure the progress in management of diffuse pontine gliomas.

82 r and superior colliculi, periaqueductal and pontine gray matter, and the red nucleus.

83 of postnatal Sox2(+)Olig2(+) progenitors in pontine growth and oligodendrogenesis.

84 ediatric gliomas; however, the mechanisms of pontine growth remain poorly understood.

85 As pontine hypoplasia has been reported in some clinical ca

86 alities, in particular callosal agenesis and pontine hypoplasia, delayed myelination and, less freque

87 re characterized by the presence of multiple pontine infarcts in all symptomatic mutation carriers.

88 Medial rectus motoneurons receive two main pontine inputs: abducens internuclear neurons, whose axo

89 It has been recently shown that the pontine intertrigeminal region (ITR) plays an important

90 y described anatomical pathway involving the pontine intertrigeminal region (ITR), has a physiologica

91 nization at inferior levels and the midbrain-pontine isthmus suggests a vulnerable region of passage

92 on of fiber passage occurred at the midbrain-pontine isthmus where all of the fiber bundles overlappe

93 e rostrally in the pons, and in the midbrain-pontine junction part of the nucleus, orexin-A-immunopos

94 superior cerebellar peduncle at the midbrain pontine junction.

95 Since the pontine Kolliker-Fuse area (KF) drives post-inspiration,

96 The pontine Kolliker-Fuse nucleus (KF) controls upper airway

97 Neurons in the pontine Kolliker-Fuse nucleus (KF) were inhibited with b

98 t-inspiration, achieved by inhibition of the pontine Kolliker-Fuse nucleus, removed post-inspiratory

99 teral nucleus of the solitary tract, and the pontine Kolliker-Fuse, intertrigeminal region, and later

100 cts in the basilar pons to determine whether pontine lacunar syndromes conform to discrete clinical e

101 r nucleus of the hypothalamus (PVN), and the pontine lateral parabrachial nucleus (PBL; an important

102 ical laughter result from rostral and medial pontine lesions, and may result from disruption of the p

103 SX, transection of the brain stem at the mid-pontine level abolished PD in response to unilateral ESV

104 and transection of the brain stem at the mid-pontine level blocks access of vagus-induced activity th

105 Finally, the pontine locus coeruleus, and two medullary regions, the

106 control posit a key modulatory role for the pontine locus coeruleus-norepinephrine (LC-NE) system.

107 We used anaesthetized rats in which pontine microinjections of a cholinergic agonist, carbac

108 corticotropin-releasing hormone (Crh) in the pontine micturition center (PMC) is electrophysiological

109 ontine-cortical micturition circuit from the pontine micturition center (PMC), locus coeruleus (LC) a

110 strally, bladder neurons were located in the pontine micturition center and external urethral sphinct

111 hat depletion of CRF neurons in the putative pontine micturition center may contribute to the severe

112 erruption of the descending pathway from the pontine micturition center to the sacral spinal cord in

113 trigger voiding and thereby function as the "pontine micturition center." Lacking detailed informatio

114 been disconnected by spinal injury from the pontine micturition centre, vanilloid-sensitive fibres a

115 s specifically arrest the extension of their pontine mossy fiber afferents, leading us to propose tha

116 In the caudal pontine MTN, only scattered orexin-A-immunoreactive fibe

117 t (ICU) admission, and occurrence of central pontine myelinolysis (CPM).

118 coencephalitis, leucodystrophies and central pontine myelinolysis), infections [malaria, acquired imm

119 (no-inflation (no-I or delayed-I) tests), DL pontine neuronal activity increased the strength and con

120 We conclude that (a) DL pontine neurones receive both vagal-dependent excitatory

121 f the NTS) and the second-order dorsolateral pontine neurons (pre-LC and PB-el inner neurons).

122 Instead, ectopic clusters of pontine neurons are found lateral to their normal site,

123 ontrol animals, and that significantly fewer pontine neurons are produced.

124 enic expression of Unc5c in deep neurons and pontine neurons by the Atoh1 promoter rescued defects of

125 However, abnormal migration of facial and pontine neurons found in NMHC II-B mutant and ablated mi

126 naline-containing fibers that originate from pontine neurons in the A5, locus coeruleus (LC), and A7

127 Netrin-1 fails to attract pontine neurons lacking Robo3, and attraction can be res

128 the basilar pontine nuclei is delayed, with pontine neurons migrating 1-2 days later than in control

129 We also identified a novel set of pontine neurons that connect contralateral segments in t

130 by YO at 5.0 mg/kg BW included medullary and pontine neurons that project to the central nucleus of t

131 Pontine neurons, along with other precerebellar neurons

132 chain in migrating, compared with stationary pontine neurons, supports an active role for myosin II i

133 5c(-/-) mice, as were anterior migrations of pontine neurons.

134 myocytes and in migration of the facial and pontine neurons.

135 recerebellar migratory stream, including the pontine neurons.

136 nuclei, but similar values were obtained in pontine neurons.

137 ribrachial region, are the primary source of pontine noradrenergic afferents to the cochlear nucleus

138 ly, DNPI/VGLUT2 mRNA was undetectable in the pontine noradrenergic cell groups (A5 and A6/locus coeru

139 Thus, the pontine noradrenergic cell groups project in a roughly t

140 The pontine noradrenergic cell groups, A5, A6 (locus coerule

141 They also innervate pontine noradrenergic cell groups, including the locus c

142 t is, at least in part, due to activation of pontine noradrenergic neurones, but is not mediated by O

143 e re-examined the effect of C1 activation on pontine noradrenergic neurons (LC and A5) using a more s

144 requency stimulation of C1 neurons activates pontine noradrenergic neurons and sympathetic nerve disc

145 ess tau-pathology in motor cortex, striatum, pontine nuclei and cerebellum in PSP-PNLA.

146 formance and prevented neuronal cell loss in pontine nuclei and substantia nigra regions.

147 presses high levels of Nfi proteins, and the pontine nuclei are greatly reduced in mice lacking a fun

148 VGLUT1 and -2 were strongly expressed in the pontine nuclei but could not be detected in LC or seroto

149 Coupling discrete stimulation of pontine nuclei controlling vigilance state with analytic

150 Ultimately, we wanted to determine if the pontine nuclei could be a component of the descending au

151 ne inferior colliculus of 10 animals and the pontine nuclei examined under a light microscope to dete

152 eled fibers were observed in the ipsilateral pontine nuclei in 70% of the animals.

153 ojection from the inferior colliculus to the pontine nuclei in guinea pig.

154 ojection from the inferior colliculus to the pontine nuclei in guinea pig.

155 ndicates that the development of the basilar pontine nuclei is delayed, with pontine neurons migratin

156 lt intake, c-Fos activation increased within pontine nuclei that relay gustatory (caudal medial PB) a

157 Tracer overlap in the pontine nuclei was significantly higher than in the othe

158 ere observed in the dorsolateral area of the pontine nuclei, adjacent to the lateral lemniscus.

159 input to the thalamus, thalamic input to the pontine nuclei, and cerebellar feedback to the thalamus.

160 such population, the neurons of the basilar pontine nuclei, expresses high levels of Nfi proteins, a

161 ene including tooth buds, thymic epithelium, pontine nuclei, fastigial cerebellar nuclei, and cerebra

162 ent (striatum, substantia nigra, olivary and pontine nuclei, hippocampus, forebrain and thalamus, ant

163 nucleus, paralemniscal nucleus, red nucleus, pontine nuclei, inferior colliculus and the parvocellula

164 us, in the thalamus, hypothalamus, amygdala, pontine nuclei, spinal cord, and dorsal root ganglion.

165 s hypoglossi, the subtrigeminal nucleus, the pontine nuclei, the dorsal tegmental nucleus, the locus

166 eus, dorsal raphe nucleus, medial lemniscus, pontine nuclei, vagus nerve, inferior olive, abducens nu

167 put from the medial auditory thalamus to the pontine nuclei, which in turn affects input to the cereb

168 cephalic and pontine reticular formation and pontine nuclei.

169 tercalatus, reticularis tegmenti pontis, and pontine nuclei.

170 cerebellum by way of a synaptic link in the pontine nuclei.

171 ed axonal growth in the deafferented basilar pontine nuclei.

172 t of neuropil in the superior colliculus and pontine nuclei.

173 xternal granule layer and development of the pontine nuclei.

174 of RA, including the inferior olive and the pontine nuclei.

175 h were particularly numerous in the residual pontine nuclei.

176 biotinylated dextran amine into the lateral pontine nucleus (PL), and led to extensive retrograde la

177 nucleus (IPN) of the cerebellum and basilar pontine nucleus (PN) during different phases of training

178 nce in the frequency of NCIs in the putamen, pontine nucleus and inferior olivary nucleus between the

179 e tracing experiments identified the basilar pontine nucleus at the confluence of outputs from CeA th

180 -/-) mice and found that they have disrupted pontine nucleus development.

181 In mammals, the pontine nucleus locus ceruleus (LC) is the sole source o

182 oliation defects, motor impairments, partial pontine nucleus migration defects, cochlear hair cell de

183 c nuclei, superior colliculus, zona incerta, pontine nucleus, multiple other brainstem areas, and the

184 olfactory bulb, red nucleus, facial nucleus, pontine nucleus, oculomotor nucleus, substantia nigra, d

185 a (LHb), ventral tegmental area, and lateral pontine nucleus.

186 egion are most medial and include the median pontine nucleus.

187 om the medial central nucleus to the basilar pontine nucleus.

188 tegmentum and can be traced into the medial pontine nucleus.

189 oxb2 deletion resulted in a reduction of the pontine oligodendrogenic domain.

190 cortex, hippocampus, hypothalamus, midbrain, pontine olivary nuclei, trigeminal nuclei, cerebellum, a

191 system by removing the potent inhibition of pontine omnipause neurons (OPNs).

192 mine which, if any, of the various groups of pontine or medullary monoaminergic neurons express DNPI/

193 The pontine oral reticular nucleus, gigantocellular reticula

194 eruleus, median raphe, parabrachial complex, pontine oralis, pedunculopontine and ventral tegmental a

195 from the basolateral amygdala (BLA) and the pontine parabrachial (PB) area in brain slices from cont

196 electrical stimulation of afferents from the pontine parabrachial area (part of the spino-parabrachio

197 NTS) and their efferent target nuclei in the pontine parabrachial complex (PB) in rats during sodium

198 The pontine parabrachial nucleus (PBN) and medullary reticul

199 oral roles for cannabinoid mechanisms of the pontine parabrachial nucleus (PBN) in modulating intake

200 e CGRP innervation of BNST originates in the pontine parabrachial nucleus and targets its anterolater

201 provide evidence that the convergent basilar pontine pathways carry corollary discharges from upper b

202 sory (upper body proprioceptive) and basilar pontine pathways onto individual granule cells and mappe

203 Chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

204 Chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

205 rts on chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

206 named 'chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids'

207 idea that the NI is a distinct region of the pontine periventricular gray, and together with the supe

208 Nfia and Nfix null mice exhibit no apparent pontine phenotype, implying specificity in the action of

209 mplitude of the anticipatory decrease in the pontine portion of DBS was associated with greater activ

210 also appears to be active in normal ventral pontine precursor-like cells of the mouse, and unregulat

211 ith a clinically and radiologically distinct pontine-predominant encephalomyelitis we have named 'chr

212 cardiovascular nucleus tractus solitarii to pontine preganglionic neurons labeled retrogradely from

213 of arterial baroreceptor nerves, projects to pontine preganglionic neurons whose stimulation elicits

214 es differential innervation of medullary and pontine preganglionic parasympathetic neurons by differe

215 cardiovascular nucleus tractus solitarii to pontine preganglionic parasympathetic neurons that proje

216 cular nucleus tractus solitarii projected to pontine preganglionic parasympathetic neurons, but more

217 Although not projecting to pontine preganglionic parasympathetic neurons, regions l

218 ulus input to the cerebellum via the thalamo-pontine projection.

219 absent in Fezl(-/-) mice, corticotectal and pontine projections were severely reduced, and Fezl-expr

220 ls retrogradely, BDA3k was injected into the pontine pyramidal tract.

221 the RTN is innervated by both medullary and pontine raphe and receives inputs from thyrotropin-relea

222 ar stem, periaqueductal gray, area postrema, pontine raphe nucleus, gracile nucleus, spinal trigemina

223 cholinergic and noradrenergic nuclei of the pontine region revealed that in comparison with other ma

224 rther used retrograde tracing from that same pontine region to the cardiovascular nucleus tractus sol

225 ths, and activity in limbic, paralimbic, and pontine regions decreased.

226 activity in the right prefrontal cortex and pontine regions in the brainstem; no brain regions showe

227 rainstem respiratory centers via a brainstem pontine relay located in the parabrachial/Kolliker-Fuse

228 the stria terminalis) that in turn regulates pontine REM generator regions.

229 rahypoglossal and dorsal parafacial regions, pontine respiratory group, and ventromedial medulla.

230 (DLSC), periaqueductal grey (PAG), or caudal pontine reticular formation (cPRF), which are implicated

231 duce a rise in activity in the mesencephalic-pontine reticular formation (MPRF), an area of the DPMS

232 KA subunits were also measured in the medial pontine reticular formation (mPRF), medial prefrontal co

233 The oral pontine reticular formation (PnO) of rat is one region i

234 or of adenylyl cyclase into the caudal, oral pontine reticular formation (PnOc) of the rat induces a

235 superior colliculus (SC) and the paramedian pontine reticular formation (PPRF).

236 ic and glycinergic fibers ascending from the pontine reticular formation (PRF) of the brainstem evoke

237 Aergic transmission suggest that GABA in the pontine reticular formation (PRF) promotes wakefulness a

238 ons with characteristic locations within the pontine reticular formation (PRF).

239 nucleus of Darkschewitsch, mesencephalic and pontine reticular formation and pontine nuclei.

240 ity of the PCC to the ventral tegmental area/pontine reticular formation and thalamus, in addition to

241 ventral striatum, amygdala, hippocampus and pontine reticular formation are new observations that ar

242 In the same pontine reticular formation area of B6 mouse where in vi

243 sis studies revealed that ACh release in the pontine reticular formation is significantly altered by

244 ated by unique combinations of medullary and pontine reticular formation nuclei such as the subnucleu

245 The caudal pontine reticular formation nucleus (cPRF) is implicated

246 , zona incerta (ZI), anterior pretectum, and pontine reticular formation) provides temporally precise

247 ojections to the facial nucleus, surrounding pontine reticular formation, and spinal cord.

248 n the brainstem, including the mesencephalic pontine reticular formation, and the anterior thalami re

249 cleus of medial longitudinal fasciculus, the pontine reticular formation, and the lateral periaqueduc

250 ort to discrete regions of the medullary and pontine reticular formation, cerebellum, parabrachial nu

251 nucleus annularis, central superior nucleus, pontine reticular formation, lateral geniculate nucleus,

252 rinic receptor agonists into the caudal oral pontine reticular formation.

253 nt magnitudes of G protein activation in the pontine reticular formation.

254 gion, including the locus coeruleus (LC) and pontine reticular formation.

255 r, caudal pontine, ventral pontine, and oral pontine reticular nuclei; the dorsomedial tegmental, sub

256 specific binding of [(35)S]GTPgammaS in the pontine reticular nucleus, caudal part (79%); pontine re

257 ontine reticular nucleus, caudal part (79%); pontine reticular nucleus, oral part (131%); laterodorsa

258 the hypothesis that GABA(A) receptors in the pontine reticular nucleus, oral part (PnO) of mouse modu

259 ed from the cochlear nucleus, and via caudal pontine reticular nucleus, pontine central gray, and MS,

260 crest, facial mesenchyme, mesencephalon, and pontine reticular nucleus.

261 We recently characterized physiologically a pontine reticulospinal (pRS) projection in the neonatal

262 p and the connectivity of these neurons with pontine sites implicated in producing REM sleep: the lat

263 nergic neurons in the NTS, and from the same pontine sites that receive major inputs from the HSD2 ne

264 Cortical-to-pontine standard-uptake value ratios were used to charac

265 blink-conditioned responses established with pontine stimulation in 12-day-old rats were reversibly a

266 show associative eyeblink conditioning when pontine stimulation is used in place of an external (e.g

267 isolated CNS histoplasmosis presenting with pontine stroke and meningitis.

268 , and that ipsilateral dysmetria after large pontine stroke represents a disconnection syndrome.

269 s anatomical arrangement suggests that small pontine strokes spare sufficient decussating pontocerebe

270 agnetic resonance imaging revealed bilateral pontine strokes, and the working diagnosis was ischemic

271 greement with the general modulatory role of pontine structures in autonomic activities including res

272 are at risk for alcohol-related reduction of pontine structures that are not necessarily affected by

273 cate that medullary circuits, independent of pontine structures, are sufficient to produce 5-HT induc

274 tion of the state required activation of the pontine subceruleus (SubC) by cholinergic inputs.

275 Cerebral-to-white matter, cerebellar, and pontine SUVRs were characterized in terms of their longi

276 pure motor hemiplegia) to incomplete basilar pontine syndrome and restricted deficits after small foc

277 ve neurons in the laterodorsal and pedunculo-pontine tegmental nuclei (LDT and PPT), compared with ad

278 dorsal, pedunculopontine, and subpeduncular pontine tegmental nuclei.

279 riaqueductal grey matter (vlPAG) and lateral pontine tegmentum (LPT)) and vice versa.

280 the neuroaxis and is located in the rostral pontine tegmentum extending from the level of the inferi

281 ithin the nucleus pontis oralis (NPO) of the pontine tegmentum is critically involved in the generati

282 active neurons were identified in the dorsal pontine tegmentum just ventral to the locus ceruleus.

283 on of nerve growth factor (NGF) into the cat pontine tegmentum rapidly induces rapid eye movement (RE

284 distribution of noradrenergic neurons in the pontine tegmentum that project to the cochlear nucleus w

285 to NREM were found bilaterally in the dorsal pontine tegmentum, hypothalamus, basal forebrain, ventra

286 ; amygdala; hippocampus; and dorsal midbrain/pontine tegmentum, including the periaqueductal gray/nuc

287 e ventrolateral periaqueductal gray, lateral pontine tegmentum, locus ceruleus, and dorsal raphe.

288 iculus, the ventral central gray matter, the pontine tegmentum, the amygdala, the reticular formation

289 um lesion overlap was centred in the core of pontine tegmentum.

290 nalysis of the rat LC and neighboring dorsal pontine tegmentum.

291 are distributed within the brainstem dorsal pontine tegmentum.

292 ensive production of oligodendrocytes in the pontine territory (r4d) and delayed and reduced oligoden

293 on of forskolin-stimulated cAMP in dissected pontine tissue slices containing the PnOc incubated with

294 hypercapnia (7-10% CO(2)) was abolished with pontine transections or chemical suppression of retrotra

295 ts, Losartan, followed by pre-collicular and pontine transections, failed to reduce tSNA, whereas tra

296 lined the ventral and rostral aspects of the pontine trigeminal sensory nucleus.

297 In the primary pontine tumor, all samples showed similar vascular morph

298 antocellular, lateral, parvicellular, caudal pontine, ventral pontine, and oral pontine reticular nuc

299 hese animals exhibited an increase in phasic pontine-wave (P-wave) activity during post-training REM

300 nt rapid eye movement (REM) sleep and phasic pontine-wave (P-wave) activity, followed by improvement