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1 expression of GAD67 and PV by neurons at the pontomedullary border implies that PV may not be a valid
2 D67-ir cells were found at the ventrolateral pontomedullary border in areas adjacent to the A5 noradr
3 al tone depends upon at least 3 sites of the pontomedullary brainstem and that a significant proporti
4 Previous studies suggested that unilateral pontomedullary brainstem lesions cause ipsiversive roll-
5 ds on neurons in at least three sites of the pontomedullary brainstem, and much of it arises independ
8 tary tract and in the locus caeruleus at the pontomedullary junction as well as in the fastigial nucl
11 from the mesencephalic locomotor region and pontomedullary medial reticular formation responsible fo
13 the ESOc and ESOt activate different sets of pontomedullary nuclei and different physiological respon
14 mulation are mediated through a relay in the pontomedullary raphe magnus (RM) and adjacent nucleus re
18 situ preparation in young rats that retained pontomedullary respiratory circuits and spinal pathways
19 on, and (4) use a computational model of the pontomedullary respiratory network to suggest neuronal m
20 nvestigated how single unit discharge in the pontomedullary reticular formation (PMRF) modulated duri
22 ribed previously, suggesting that individual pontomedullary reticular formation neurons may coordinat
24 d effects are SEROTONERGIC, the responses of pontomedullary SEROTONERGIC-LIKE cells to PAG stimulatio
26 REM sleep and motor control circuitry in the pontomedullary structures cause REM sleep behaviour diso
27 as a result of degeneration of olfactory and pontomedullary structures could theoretically begin many
28 s of the ventral medulla, ventral and dorsal pontomedullary structures, basal forebrain, and cerebell
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