ライフサイエンスコーパス: poor control

1 recognized as a risk for asthma severity and poor control.

2 ardiovascular events than those with fair or poor control.

3 duals diagnosed with the condition remain in poor control.

4 , whereas those with the worst score (4) had poor control (29.4%) and high hazard of hemorrhage (haza

5 L) within the past year; only 9% experienced poor control (a HbA1c level > 12% or mean blood glucose

6 Inefficient induction of ISGs contributes to poor control and persistence of hepatitis C virus infect

7 .e., control and clearance versus partial or poor control and persistent infection).

8 Patients with severe illness and poor control appear to bear the most burden in terms of

9 itoring especially in those with evidence of poor control before pregnancy.

10 Severe asthmatics often exhibit poor control despite high doses of inhaled corticosteroi

11 In patients who have persistently poor control despite optimal medication compliance, newl

12 GHb <7) soon after induction of diabetes, or poor control for 6 months with 6 months' good control.

13 ozotocin-induced diabetes were in a state of poor control (GHb >11%) for 12 months, good control (GHb

14 4 years of follow-up, subjects who were in "poor" control (glycosylated hemoglobin (GHb) > or = 11%)

15 their HbA(1c) was in the lowest quartile and poor control if it was in the highest quartile.

16 roid therapy (ICS) is a major contributor to poor control in difficult asthma, yet it is challenging

17 ivity observed in particular cases is due to poor control in ylide conformation or due to partial rev

18 ndings suggest that in diabetic adolescents, poor control is associated with a significant depletion

19 Poor control of a local activation process of this syste

20 res knowledge of the factors associated with poor control of asthma symptoms.

21 d CXCL1/keratinocyte-derived chemokine (KC), poor control of bacterial replication, and exacerbated i

22 f core outcome measures used by studies, and poor control of coexisting psychopathology.

23 Poor control of electrode properties can lead to subopti

24 alian cells by high background fluorescence, poor control of expression, and low GFP maturation effic

25 Poor control of eye movement may lead to unstable binocu

26 Poor control of gastric pH was not associated with feedi

27 Poor control of HIV replication and detectable EBV repli

28 tion, CD8(+) T-cell alveolitis, smoking, and poor control of human immunodeficiency virus (HIV) are f

29 ack race, were independently associated with poor control of hypertension among those who were aware

30 These data reveal the persistently poor control of pneumococcal colonization in HIV-infecte

31 has recently been challenged with reports of poor control of reflux and the inability to prevent prog

32 functions, and lack of either element led to poor control of responder cell proliferation and cytokin

33 functions, and lack of either element led to poor control of responder cell proliferation and cytokin

34 In case of poor control of symptoms despite guideline-directed phar

35 ector size and volume of injected fluid, and poor control of the amount of injected material.

36 cessing annealing step at 140 degrees C with poor control of the bonding geometry.

37 eneral nonpermissiveness of primate cells or poor control of the IFN response.

38 dysregulated immunity, which contributes to poor control of viral infection.

39 Poor control of viral replication as well as TNF-alpha a

40 ected individuals or humanized mice reported poor control of virus replication and the rapid emergenc

41 Negative immune regulation can result in the poor control of virus replication in chronic infections

42 database who began insulin (2000-2007) after poor control on oral glucose-lowering agents (OGLD) were

43 We report that, relative to GTs, STs have poor control over attentional performance, due in part t

44 entrations during the polymerization provide poor control over film thickness.

45 otein matrices that suffer from variability, poor control over matrix biochemistry, and inability to

46 n such systems is often difficult because of poor control over molecular morphology.

47 e reintubated within the same day because of poor control over secretions, airway spasm, or hypoventi

48 ibitory control deficits possibly underlying poor control over stereotyped and repetitive and compuls

49 o- and heterosolvated dimers but demonstrate poor control over structure.

50 olloidal assemblies, but also because of the poor control over the assembly of nanocrystals within a

51 +/- 1.0 kg/ m(2), HbA(1c) 5.9 +/- 0.2%) and poor control (PC) (n = 10, age 50.0 +/- 2.5 years, BMI 2

52 ld male patient with diabetes mellitus under poor control presented to our emergency room with fever,

53 In rats with poor control, retinal GAPDH activity and expressions wer

54 marked decrease in PEPCK content, suggesting poor control strength for this enzyme in gluconeogenic r

55 otein, supporting the concept that PEPCK has poor control strength over the gluconeogenic pathway in

56 /toluene mixture, while no polymerization or poor control was observed with Ru(bpy)3.

57 Subjects who were in poor control were somewhat more likely to develop overt

58 In contrast, patient level predictors of poor control were: short acting bronchodilator overuse [