1 at causes traditional CFC methods to perform
poorly.
2 exacerbations, some patients respond to them
poorly.
3 Yet, D. lumholtzi performed
poorly across all experimental treatment(s) relative to
4 Nifedipine-resistant RRP exocytosis was
poorly affected by 5 mm intracellular EGTA, suggesting t
5 the flower-rich site (all colonies performed
poorly at the flower poor site).
6 Vertebrate ocular microbiomes are
poorly characterized and virtually unexplored in wildlif
7 The protein encoded by TMEM106B is
poorly characterized but is reported to have a role in r
8 Early industrial trace metal loadings are
poorly characterized but potentially substantial sources
9 of transgene susceptibility to silencing is
poorly characterized in plants; thus, we evaluated sever
10 rovides functional insights into hundreds of
poorly characterized proteins while enhancing network-ba
11 The deep dorsal horn is a
poorly characterized spinal cord region implicated in pr
12 Intriguingly, mammalian genomes encode many
poorly characterized SR-like proteins, including subunit
13 rom healthy human lung, these subsets remain
poorly characterized transcriptionally and phenotypicall
14 zation of organelles within the cell remains
poorly characterized, as fluorescence imaging approaches
15 (RBPs) that govern cancer stem cells remains
poorly characterized.
16 ntal aspects of AAV's capsid assembly remain
poorly characterized.
17 PCs, and their functions in the plant remain
poorly characterized.
18 ommunities in urban green space soils remain
poorly characterized.
19 iseases, while broadly acknowledged, remains
poorly characterized.
20 non-structural protein (NSs), which remains
poorly characterized.
21 tus and ensuing CD4+ T-cell polarization are
poorly characterized.
22 Although the sequence of RAM is
poorly conserved, the linear patterning of oppositely ch
23 fect chains leading to the extinction remain
poorly constrained as Late Devonian stratigraphy is poor
24 ns of NADW and AABW and their properties are
poorly constrained by marine records, including delta(18
25 However, such analyses are
poorly constrained by measured CO2 exchange in drylands.
26 particularly with stadial intervals, remains
poorly constrained by paleoclimate records.
27 oceanic and terrestrial carbon cycle remain
poorly constrained in space and time.
28 t has not been documented to occur, and (ii)
poorly constrained zircon growth rates combined with the
29 ls on V mobility within coke deposits remain
poorly constrained.
30 short timescales (10(3)-10(5) years) remains
poorly constrained.
31 orted outcomes were assessed, including very
poorly controlled asthma based on National Heart, Lung,
32 ces on the ascending aorta, typically due to
poorly controlled hypertension, and heritable genetic va
33 logy of epileptic disorders, seizures remain
poorly controlled in approximately one-third of patients
34 and management of asthma, many patients have
poorly controlled or refractory asthma (RA).
35 Inputs to care are
poorly correlated with provision of evidence-based care
36 s influencing the transformation rate of the
poorly crystalline iron (oxyhydr)oxide, ferrihydrite, to
37 ocytes, have high trabecular bone volume and
poorly defined metaphyseal cortices.
38 uggested that UL37 plays an essential albeit
poorly defined role in intracellular capsid trafficking.
39 The functions of many RBR E3s are
poorly defined, and their regulation is complex, involvi
40 inic acetylcholine receptors (nAChRs) remain
poorly defined.
41 litis requiring intensive care treatment are
poorly defined.
42 gens, but its impact on pathogenesis remains
poorly defined.
43 pancreatic ductal adenocarcinoma (PDAC) are
poorly defined.
44 and the associated immune mechanisms remain
poorly defined.
45 s that mediate this protective effect remain
poorly defined.
46 g the accuracy of preoperative 4D-CTs remain
poorly defined.
47 prognosis and outcome of these patients are
poorly defined.
48 ssion to the ICU and adverse outcomes remain
poorly defined.
49 stem cells from oncogenic transformation are
poorly defined.
50 e immunoproteasome in neuropathology remains
poorly defined.
51 ctions affected upon inclusion formation are
poorly defined.
52 al properties of this T cell response remain
poorly defined.
53 nical stimuli regulate mTOR signaling remain
poorly defined.
54 naling mechanisms that impact thrombosis are
poorly defined.
55 ain, the role of Sema7A in the adult remains
poorly defined.
56 r to be well understood, its regulon remains
poorly defined.
57 B cell lymphocytosis, premalignant condition
poorly described in KT recipients.
58 egories of care that drive this increase are
poorly described.
59 on revealed a higher mean vessel density and
poorly differentiated extracellular matrix (ECM) in MDA-
60 nizing hormone gene, Lhb We demonstrate that
poorly differentiated gonadotropes express a TET1 isofor
61 e histological features of human RAS-driven,
poorly differentiated thyroid cancers.
62 nd pertuzumab expressed EMT markers and were
poorly differentiated, whereas tumors exposed to the com
63 [moderately differentiated], and high-grade [
poorly differentiated], respectively), chromogranin A, a
64 SfCP is an easily measured but
poorly documented gauge of well integrity.
65 but underlying circuit mechanisms have been
poorly explored by genetic approaches.
66 ns; however, its motivational aspects remain
poorly explored in humans.
67 otein subcellular localization, they perform
poorly for effectors.
68 To test the method, we compared well and
poorly healing cardiac ventricles using a transgenic fis
69 tion cephalosporins, including cefepime, are
poorly hydrolyzed by specific ESBL enzymes, and certain
70 in pregnancies in women with diabetes remain
poorly identified.
71 parallel retinal input, encodes orientation
poorly,
if at all.
72 tact tracing has been widely recommended but
poorly implemented in resource-constrained settings.
73 T and photograph reading by clinicians agree
poorly in CDR assessment.
74 Fertilized eggs develop
poorly into blastocysts, which results in significantly
75 UPR achieves ER homeostatic readjustment is
poorly investigated, as in most studies the ER stress th
76 (CSC) leading to therapy resistance remains
poorly investigated.
77 One subfield whose function is
poorly known is the subiculum.
78 astric stump carcinoma is an exceptional and
poorly known long-term complication after pancreaticoduo
79 species richness are well understood, it is
poorly known which environmental factors promote diversi
80 ms that control cellular IGF2R abundance are
poorly known.
81 Humans can be exposed to pathogens from
poorly managed animal feces, particularly in communities
82 was concluded that the survival rate of HLA
poorly matched living donor transplants is not inferior
83 lyzed and survival rates of transplants from
poorly matched living donors were compared with those fr
84 the resource pulses, the cost of foraging on
poorly matched resources, and the strength of interspeci
85 However, it is unclear why dynein-1 moves
poorly on its own or how it is activated by dynactin.
86 from human mesenchymal stem cells (hMSCs) is
poorly organized and unstable in vivo.
87 Overall, however, BP
poorly predicted downstream ischemia.
88 However, several samples were
poorly predicted suggesting that chemical heterogeneitie
89 hat is not captured by sediment traps and is
poorly quantified by most models and in situ measurement
90 r, this potentially important carbon pool is
poorly quantified in Afrotropical forests and the region
91 nts in category 1 had a higher proportion of
poorly reactive pupils (P < 0.001) and abnormal ocular m
92 tic microangiopathy (TA-TMA) is a common and
poorly recognized complication of hematopoietic stem cel
93 hich ultimately leads to their critical, but
poorly recognized role in regional greenhouse gas emissi
94 reliable, reliable, moderately reliable, or
poorly reliable with respect to the corresponding volume
95 ParkinKO mice, however,
poorly represent human PD symptoms as they only exhibit
96 raphical theory and global vegetation models
poorly represent recent forest die-off patterns.
97 ypical EPEC infection is rare in animals and
poorly reproduced in experimental animal models.
98 constrained as Late Devonian stratigraphy is
poorly resolved, compared to younger cataclysmic interva
99 ases but differences between individuals are
poorly resolved.
100 t progresses after enzalutamide treatment is
poorly responsive to further antiandrogen therapy, and p
101 individuals with the dG allele, where it was
poorly secreted but highly functional, even at concentra
102 A bottleneck for their synthesis had been a
poorly selective cyclopropanation with ethyl diazoacetat
103 Although specific, current guidelines were
poorly sensitive, identifying only 34% to 60% of subject
104 Currently available analgesics
poorly serve individuals suffering from chronic pain, an
105 Tc was removed from solution in the form of
poorly soluble hydrous Tc(IV)-oxides or Tc(IV)-sulfides
106 ormation by destabilizing the prepore into a
poorly structured dodecamer as visualized by electron mi
107 sis using TC10-deficient mice, we define the
poorly studied Rho GTPase TC10 as an immunomodulatory mo
108 LV health, the IUGR right ventricle remains
poorly studied.
109 rhinosinusitis with nasal polyps (CRSwNP) is
poorly studied.
110 pproaches, sometimes reaching conflicting or
poorly supported conclusions.
111 Poorly supported management can have detrimental, costly
112 In CTL hearts, 1.4% of myocytes were
poorly synchronized with neighboring cells and exhibited
113 ging data demonstrate that TFIID alone binds
poorly to native p53 target promoters.
114 because of discomfort, suggesting that it is
poorly tolerated as an early routine treatment of HG.
115 Charged residues were
poorly tolerated, conferring extreme temperature-sensiti
116 ronic inflammation and underlie a variety of
poorly treatable pathologies.
117 Insomnia remains under-diagnosed and
poorly treated despite its high economic and social cost
118 idneys from increased risk donors (IRD), but
poorly understand them.
119 ection that develops into cervical cancer is
poorly understood and practically unobservable.
120 itized reactions in ambient aerosols remains
poorly understood and unaccounted for in atmospheric mod
121 Especially
poorly understood are the very early stages of protein f
122 tails of the intermolecular interface remain
poorly understood at atomic resolution.
123 c and pathogenic relationships, but is often
poorly understood at the molecular level.
124 However, its role in C4 photosynthesis is
poorly understood because it has been historically diffi
125 of diet on the metabolism-epigenome axis is
poorly understood but could alter gene expression and in
126 f developing organs requires coordinated but
poorly understood changes in epithelial cell-cell adhesi
127 quasi-particles arguably explain a range of
poorly understood experiments in gated graphene structur
128 The bacterial hibernating 100S ribosome is a
poorly understood form of the dimeric 70S particle that
129 However, it remains
poorly understood how long-lived PCs (LLPCs) are generat
130 However, it remains
poorly understood how the non-catalytic ISWI subunits BA
131 novel insight into the function of IFT56, a
poorly understood IFTB protein.
132 ntelligence and socio-cognitive abilities is
poorly understood in animals and humans.
133 nitely repeated games of cooperation remains
poorly understood in part because their dynamics play ou
134 studied in suspension cultures, but remains
poorly understood in substrate-dependent cells.
135 Mechanical forces are critical but
poorly understood inputs for organogenesis and wound hea
136 in development and cerebral injury through a
poorly understood mechanism known to involve neuroinflam
137 g one membranous subunit of V-ATPase or have
poorly understood mechanisms of action.
138 inked to different clinical diseases through
poorly understood mechanisms.
139 netic markers has provided insights into the
poorly understood phenomenon of AIV airborne transmissib
140 This
poorly understood population is referred to as minimal r
141 form multinucleated myotubes, a critical but
poorly understood process.
142 is class of enzymes is emphasized, given the
poorly understood roles performed by multiple, auxiliary
143 hat regulate membrane traffic, including the
poorly understood small transmembrane proteins neural-sp
144 aortic valve (BAV)-associated aortopathy is
poorly understood, and no prognostic biomarker is curren
145 The HTLV-1 particle structure is still
poorly understood, and previous studies analyzed viruses
146 cs of bacterial growth in such conditions is
poorly understood, and, unlike that for liquid culture,
147 hanisms triggered by these various agents is
poorly understood, but it might explain the differential
148 se freshwater shrimp genus Caridina is still
poorly understood, despite its vast distribution - from
149 tterns and drivers of shrub expansion remain
poorly understood, inhibiting accurate incorporation of
150 uantities in end-of-life vehicles (ELVs) are
poorly understood, mainly due to a limited understating
151 e introduction of biologically relevant, but
poorly understood, modifications to cytosine affect the
152 phosphorylation effects remains nonetheless
poorly understood, particularly for intrinsically disord
153 e the economics of water quality testing are
poorly understood, the extent to which cost may be a bar
154 The breeding habitats of this species are
poorly understood, which greatly hampers practical resea
155 ity ratios in children with tuberculosis are
poorly understood-particularly those among children with
156 ic factors of late-life depression are still
poorly understood.
157 The mechanism of action of GM-CSF in EAE is
poorly understood.
158 , the nature and timing of these changes are
poorly understood.
159 ) pathology and expression of AD symptoms is
poorly understood.
160 he factors that promote DCIS invasion remain
poorly understood.
161 ic determinants of body fat distribution are
poorly understood.
162 ral activity creates this motivation remains
poorly understood.
163 gene networks in the innate immune cells are
poorly understood.
164 gation of chromosomes during mitosis remains
poorly understood.
165 t the underlying molecular mechanisms remain
poorly understood.
166 ructure and dynamics in this process remains
poorly understood.
167 hanisms underlying these interactions remain
poorly understood.
168 spatiotemporal dynamics during behavior are
poorly understood.
169 ions in glioblastomas with wild-type IDH are
poorly understood.
170 the specificity of such interactions remain
poorly understood.
171 liminated to maintain protein homeostasis is
poorly understood.
172 nctions in hematological malignancies remain
poorly understood.
173 verexpression of this receptor in cancer are
poorly understood.
174 biological basis of these sex differences is
poorly understood.
175 s and organization in motile cells are still
poorly understood.
176 globally distributed, cryptic environment is
poorly understood.
177 arts; however, the underlying mechanisms are
poorly understood.
178 Basic mechanisms for this association are
poorly understood.
179 e cancer development and progression remains
poorly understood.
180 alian arcopallial and amygdala subregions is
poorly understood.
181 s underlying sex-specific development remain
poorly understood.
182 ontribute to MSC phenotype in cancer remains
poorly understood.
183 underlying lysosomal deficits in AD remains
poorly understood.
184 activity and breast cancer formation remain
poorly understood.
185 f such sequences are encoded in the brain is
poorly understood.
186 tein aggregates from more innocuous ones are
poorly understood.
187 erlying determinants of this variability are
poorly understood.
188 e expectancy, the underlying biology remains
poorly understood.
189 LA3 and its variant I148M in this process is
poorly understood.
190 echanisms favoring PMN-MDSC responses remain
poorly understood.
191 intervals of intraplate faults therefore are
poorly understood.
192 athobiology of early squamous lung cancer is
poorly understood.
193 such expansions cause neurodegeneration are
poorly understood.
194 systems interact to regulate bone growth is
poorly understood.
195 tical oscillations and neuronal responses is
poorly understood.
196 ation of this process leads to cancer remain
poorly understood.
197 influencing risk and disease progression are
poorly understood.
198 trol expression of RP genes in metazoans are
poorly understood.
199 erlying opposition to Polycomb silencing are
poorly understood.
200 However, its mechanisms of action remain
poorly understood.
201 iratory disease and lung cancer mortality is
poorly understood.
202 , and the spectrum of microbes affected, are
poorly understood.
203 ingle AD trial, and this variability remains
poorly understood.
204 uch as the incorporation of molecular O2 are
poorly understood.
205 erization impacts DNA-binding specificity is
poorly understood.
206 antipsychotic efficacy and side effects are
poorly understood.
207 nteractions with crop quality parameters, is
poorly understood.
208 alter the course of Leishmania infection are
poorly understood.
209 alter the course of AD neuropathology remain
poorly understood.
210 ons underlying this aberrant behavior remain
poorly understood.
211 (TF) occupancy and enhancer activity remains
poorly understood.
212 the underlying molecular mechanism is still
poorly understood.
213 he impact of SCNAs on tumour biology remains
poorly understood.
214 tion in idiopathic pulmonary fibrosis remain
poorly understood.
215 facilitate dynamic stomatal responses remain
poorly understood.
216 ing properties of its small RNA guide remain
poorly understood.
217 ch tau organizes microtubule networks remain
poorly understood.
218 ures in a solvent-free environment are still
poorly understood.
219 min-1 (syt1) in regulation of endocytosis is
poorly understood.
220 , but the mechanism of this activity remains
poorly understood.
221 ER shaping is linked to neuronal defects is
poorly understood.
222 ose resulting in return to theater (RTT) are
poorly understood.
223 alcifying and silicifying haptophytes remain
poorly understood.
224 how it communicates with neural elements is
poorly understood.
225 ogically distinct plasma membrane domains is
poorly understood.
226 w connections, a process whose regulation is
poorly understood.
227 s, communication between these cells remains
poorly understood.
228 proliferation potential of LRP cells remains
poorly understood.
229 ly disease in the context of prediabetes are
poorly understood.
230 -22 expression in the human intestine remain
poorly understood.
231 gulation of differentiation-related genes is
poorly understood.
232 f specific residues in the nervous system is
poorly understood.
233 main, the mechanisms for Tie2 activation are
poorly understood.
234 and maintain gut bacterial diversity remain
poorly understood.
235 e response to chemotherapeutic drugs remains
poorly understood.
236 ce sensory stimuli into action potentials is
poorly understood.
237 experience on interneuron plasticity remain
poorly understood.
238 er (MDD) patients with suicidal ideation are
poorly understood.
239 to cellular behavior, but their interplay is
poorly understood.
240 the mechanism underlying this association is
poorly understood.
241 d stands of varying species richness remains
poorly understood.
242 drivers of fine-root trait diversity remain
poorly understood.
243 ve black holes in the early universe remains
poorly understood.
244 the human pathology and NMDAR dysfunction is
poorly understood.
245 , transcription initiation by mtRNAP remains
poorly understood.
246 circuits underlying appetitive behaviors is
poorly understood.
247 velopment, but regulation of this process is
poorly understood.
248 tive value in predicting clinical events are
poorly understood.
249 and structure of these cytoskeletons remain
poorly understood.
250 thern peripheries of ancient Mesoamerica are
poorly understood.
251 s syndrome (ARDS), joblessness is common but
poorly understood.
252 re of R5 during silica precipitation remains
poorly understood.
253 processes, the role of the daughter remains
poorly understood.
254 among specific age and sex groups are still
poorly understood.
255 chanisms accounting for hypersensitivity are
poorly understood.
256 hanisms that orchestrate these events remain
poorly understood.
257 re determined during differentiation remains
poorly understood.
258 ons to circadian photoreception is currently
poorly understood.
259 hanisms mediating delamination are, however,
poorly understood.
260 pathogenesis of these complications remains
poorly understood.
261 enzymatic activity, but how they function is
poorly understood.
262 ogy is maintained in the adult brain remains
poorly understood.
263 s that control its proapoptotic function are
poorly understood.
264 tissue-specific, epigenetic pattern is still
poorly understood.
265 ergy and autoimmune disorders is complex and
poorly understood.
266 his evolutionary conserved diversity remains
poorly understood.
267 s in the loss of tolerance to gluten remains
poorly understood.
268 way, but sensing of physical stimuli remains
poorly understood.
269 cal diseases, its role at nerve terminals is
poorly understood.
270 ion to antisocial behavior and adversity are
poorly understood.
271 CD4 T cells and in HIV-infected children is
poorly understood.
272 f chondrocyte recognition by NK cells remain
poorly understood.
273 g the infection to cancer development remain
poorly understood.
274 control during mammalian development remain
poorly understood.
275 et the key events that drive aging are still
poorly understood.
276 lter tumour progression, their regulation is
poorly understood.
277 , protein dynamics within the 3D nucleus are
poorly understood.
278 ary gland morphogenesis and homoeostasis are
poorly understood.
279 al mechanisms underlying this process remain
poorly understood.
280 id innate and adaptive allergic responses is
poorly understood.
281 irmed independent cancer risk factor, remain
poorly understood.
282 uneven distribution within endemic areas is
poorly understood.
283 lar mechanisms regulating this transport are
poorly understood.
284 s, but the mechanisms involved are generally
poorly understood.
285 , the underlying molecular mechanisms remain
poorly understood.
286 enesis of severe asthma in childhood remains
poorly understood.
287 precise mechanism underlying gating remains
poorly understood.
288 onses are regulated by other stimuli remains
poorly understood.
289 physiological basis of this communication is
poorly understood.
290 ch Cdx members impact their transcription is
poorly understood.
291 the regulation of MAVS-mediated apoptosis is
poorly understood.
292 he roles of CDC42 in tumorigenesis are still
poorly understood.
293 e translocase; however, its function is only
poorly understood.
294 fills its controlling role, however, remains
poorly understood.
295 athophysiology of carcinoid heart disease is
poorly understood; however, chronic exposure to excessiv
296 humoral but not cellular immunity, and have
poorly-
understood mechanisms.
297 Formulating a
poorly water-soluble drug substance into nanocrystals of
298 ween the 2 approaches, MSI3-slices performed
poorly when MSI was <0.50.
299 While electrophilicities E correlate
poorly with the LUMO energies and with Parr's electrophi
300 NIK transgenic Tregs competed
poorly with WT Tregs in vivo and produced pro-inflammato