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1 at causes traditional CFC methods to perform poorly.
2 exacerbations, some patients respond to them poorly.
3                  Yet, D. lumholtzi performed poorly across all experimental treatment(s) relative to
4      Nifedipine-resistant RRP exocytosis was poorly affected by 5 mm intracellular EGTA, suggesting t
5 the flower-rich site (all colonies performed poorly at the flower poor site).
6            Vertebrate ocular microbiomes are poorly characterized and virtually unexplored in wildlif
7           The protein encoded by TMEM106B is poorly characterized but is reported to have a role in r
8    Early industrial trace metal loadings are poorly characterized but potentially substantial sources
9  of transgene susceptibility to silencing is poorly characterized in plants; thus, we evaluated sever
10 rovides functional insights into hundreds of poorly characterized proteins while enhancing network-ba
11                    The deep dorsal horn is a poorly characterized spinal cord region implicated in pr
12  Intriguingly, mammalian genomes encode many poorly characterized SR-like proteins, including subunit
13 rom healthy human lung, these subsets remain poorly characterized transcriptionally and phenotypicall
14 zation of organelles within the cell remains poorly characterized, as fluorescence imaging approaches
15 (RBPs) that govern cancer stem cells remains poorly characterized.
16 ntal aspects of AAV's capsid assembly remain poorly characterized.
17 PCs, and their functions in the plant remain poorly characterized.
18 ommunities in urban green space soils remain poorly characterized.
19 iseases, while broadly acknowledged, remains poorly characterized.
20  non-structural protein (NSs), which remains poorly characterized.
21 tus and ensuing CD4+ T-cell polarization are poorly characterized.
22              Although the sequence of RAM is poorly conserved, the linear patterning of oppositely ch
23 fect chains leading to the extinction remain poorly constrained as Late Devonian stratigraphy is poor
24 ns of NADW and AABW and their properties are poorly constrained by marine records, including delta(18
25                   However, such analyses are poorly constrained by measured CO2 exchange in drylands.
26 particularly with stadial intervals, remains poorly constrained by paleoclimate records.
27  oceanic and terrestrial carbon cycle remain poorly constrained in space and time.
28 t has not been documented to occur, and (ii) poorly constrained zircon growth rates combined with the
29 ls on V mobility within coke deposits remain poorly constrained.
30 short timescales (10(3)-10(5) years) remains poorly constrained.
31 orted outcomes were assessed, including very poorly controlled asthma based on National Heart, Lung,
32 ces on the ascending aorta, typically due to poorly controlled hypertension, and heritable genetic va
33 logy of epileptic disorders, seizures remain poorly controlled in approximately one-third of patients
34 and management of asthma, many patients have poorly controlled or refractory asthma (RA).
35                           Inputs to care are poorly correlated with provision of evidence-based care
36 s influencing the transformation rate of the poorly crystalline iron (oxyhydr)oxide, ferrihydrite, to
37 ocytes, have high trabecular bone volume and poorly defined metaphyseal cortices.
38 uggested that UL37 plays an essential albeit poorly defined role in intracellular capsid trafficking.
39            The functions of many RBR E3s are poorly defined, and their regulation is complex, involvi
40 inic acetylcholine receptors (nAChRs) remain poorly defined.
41 litis requiring intensive care treatment are poorly defined.
42 gens, but its impact on pathogenesis remains poorly defined.
43  pancreatic ductal adenocarcinoma (PDAC) are poorly defined.
44  and the associated immune mechanisms remain poorly defined.
45 s that mediate this protective effect remain poorly defined.
46 g the accuracy of preoperative 4D-CTs remain poorly defined.
47  prognosis and outcome of these patients are poorly defined.
48 ssion to the ICU and adverse outcomes remain poorly defined.
49 stem cells from oncogenic transformation are poorly defined.
50 e immunoproteasome in neuropathology remains poorly defined.
51 ctions affected upon inclusion formation are poorly defined.
52 al properties of this T cell response remain poorly defined.
53 nical stimuli regulate mTOR signaling remain poorly defined.
54 naling mechanisms that impact thrombosis are poorly defined.
55 ain, the role of Sema7A in the adult remains poorly defined.
56 r to be well understood, its regulon remains poorly defined.
57 B cell lymphocytosis, premalignant condition poorly described in KT recipients.
58 egories of care that drive this increase are poorly described.
59 on revealed a higher mean vessel density and poorly differentiated extracellular matrix (ECM) in MDA-
60 nizing hormone gene, Lhb We demonstrate that poorly differentiated gonadotropes express a TET1 isofor
61 e histological features of human RAS-driven, poorly differentiated thyroid cancers.
62 nd pertuzumab expressed EMT markers and were poorly differentiated, whereas tumors exposed to the com
63 [moderately differentiated], and high-grade [poorly differentiated], respectively), chromogranin A, a
64               SfCP is an easily measured but poorly documented gauge of well integrity.
65  but underlying circuit mechanisms have been poorly explored by genetic approaches.
66 ns; however, its motivational aspects remain poorly explored in humans.
67 otein subcellular localization, they perform poorly for effectors.
68     To test the method, we compared well and poorly healing cardiac ventricles using a transgenic fis
69 tion cephalosporins, including cefepime, are poorly hydrolyzed by specific ESBL enzymes, and certain
70 in pregnancies in women with diabetes remain poorly identified.
71  parallel retinal input, encodes orientation poorly, if at all.
72 tact tracing has been widely recommended but poorly implemented in resource-constrained settings.
73 T and photograph reading by clinicians agree poorly in CDR assessment.
74                      Fertilized eggs develop poorly into blastocysts, which results in significantly
75  UPR achieves ER homeostatic readjustment is poorly investigated, as in most studies the ER stress th
76  (CSC) leading to therapy resistance remains poorly investigated.
77               One subfield whose function is poorly known is the subiculum.
78 astric stump carcinoma is an exceptional and poorly known long-term complication after pancreaticoduo
79  species richness are well understood, it is poorly known which environmental factors promote diversi
80 ms that control cellular IGF2R abundance are poorly known.
81      Humans can be exposed to pathogens from poorly managed animal feces, particularly in communities
82  was concluded that the survival rate of HLA poorly matched living donor transplants is not inferior
83 lyzed and survival rates of transplants from poorly matched living donors were compared with those fr
84 the resource pulses, the cost of foraging on poorly matched resources, and the strength of interspeci
85    However, it is unclear why dynein-1 moves poorly on its own or how it is activated by dynactin.
86 from human mesenchymal stem cells (hMSCs) is poorly organized and unstable in vivo.
87                         Overall, however, BP poorly predicted downstream ischemia.
88                However, several samples were poorly predicted suggesting that chemical heterogeneitie
89 hat is not captured by sediment traps and is poorly quantified by most models and in situ measurement
90 r, this potentially important carbon pool is poorly quantified in Afrotropical forests and the region
91 nts in category 1 had a higher proportion of poorly reactive pupils (P < 0.001) and abnormal ocular m
92 tic microangiopathy (TA-TMA) is a common and poorly recognized complication of hematopoietic stem cel
93 hich ultimately leads to their critical, but poorly recognized role in regional greenhouse gas emissi
94  reliable, reliable, moderately reliable, or poorly reliable with respect to the corresponding volume
95                      ParkinKO mice, however, poorly represent human PD symptoms as they only exhibit
96 raphical theory and global vegetation models poorly represent recent forest die-off patterns.
97 ypical EPEC infection is rare in animals and poorly reproduced in experimental animal models.
98 constrained as Late Devonian stratigraphy is poorly resolved, compared to younger cataclysmic interva
99 ases but differences between individuals are poorly resolved.
100 t progresses after enzalutamide treatment is poorly responsive to further antiandrogen therapy, and p
101 individuals with the dG allele, where it was poorly secreted but highly functional, even at concentra
102  A bottleneck for their synthesis had been a poorly selective cyclopropanation with ethyl diazoacetat
103   Although specific, current guidelines were poorly sensitive, identifying only 34% to 60% of subject
104               Currently available analgesics poorly serve individuals suffering from chronic pain, an
105  Tc was removed from solution in the form of poorly soluble hydrous Tc(IV)-oxides or Tc(IV)-sulfides
106 ormation by destabilizing the prepore into a poorly structured dodecamer as visualized by electron mi
107 sis using TC10-deficient mice, we define the poorly studied Rho GTPase TC10 as an immunomodulatory mo
108  LV health, the IUGR right ventricle remains poorly studied.
109 rhinosinusitis with nasal polyps (CRSwNP) is poorly studied.
110 pproaches, sometimes reaching conflicting or poorly supported conclusions.
111                                              Poorly supported management can have detrimental, costly
112         In CTL hearts, 1.4% of myocytes were poorly synchronized with neighboring cells and exhibited
113 ging data demonstrate that TFIID alone binds poorly to native p53 target promoters.
114 because of discomfort, suggesting that it is poorly tolerated as an early routine treatment of HG.
115                        Charged residues were poorly tolerated, conferring extreme temperature-sensiti
116 ronic inflammation and underlie a variety of poorly treatable pathologies.
117         Insomnia remains under-diagnosed and poorly treated despite its high economic and social cost
118 idneys from increased risk donors (IRD), but poorly understand them.
119 ection that develops into cervical cancer is poorly understood and practically unobservable.
120 itized reactions in ambient aerosols remains poorly understood and unaccounted for in atmospheric mod
121                                   Especially poorly understood are the very early stages of protein f
122 tails of the intermolecular interface remain poorly understood at atomic resolution.
123 c and pathogenic relationships, but is often poorly understood at the molecular level.
124    However, its role in C4 photosynthesis is poorly understood because it has been historically diffi
125  of diet on the metabolism-epigenome axis is poorly understood but could alter gene expression and in
126 f developing organs requires coordinated but poorly understood changes in epithelial cell-cell adhesi
127  quasi-particles arguably explain a range of poorly understood experiments in gated graphene structur
128 The bacterial hibernating 100S ribosome is a poorly understood form of the dimeric 70S particle that
129                          However, it remains poorly understood how long-lived PCs (LLPCs) are generat
130                          However, it remains poorly understood how the non-catalytic ISWI subunits BA
131  novel insight into the function of IFT56, a poorly understood IFTB protein.
132 ntelligence and socio-cognitive abilities is poorly understood in animals and humans.
133 nitely repeated games of cooperation remains poorly understood in part because their dynamics play ou
134  studied in suspension cultures, but remains poorly understood in substrate-dependent cells.
135           Mechanical forces are critical but poorly understood inputs for organogenesis and wound hea
136 in development and cerebral injury through a poorly understood mechanism known to involve neuroinflam
137 g one membranous subunit of V-ATPase or have poorly understood mechanisms of action.
138 inked to different clinical diseases through poorly understood mechanisms.
139 netic markers has provided insights into the poorly understood phenomenon of AIV airborne transmissib
140                                         This poorly understood population is referred to as minimal r
141 form multinucleated myotubes, a critical but poorly understood process.
142 is class of enzymes is emphasized, given the poorly understood roles performed by multiple, auxiliary
143 hat regulate membrane traffic, including the poorly understood small transmembrane proteins neural-sp
144  aortic valve (BAV)-associated aortopathy is poorly understood, and no prognostic biomarker is curren
145       The HTLV-1 particle structure is still poorly understood, and previous studies analyzed viruses
146 cs of bacterial growth in such conditions is poorly understood, and, unlike that for liquid culture,
147 hanisms triggered by these various agents is poorly understood, but it might explain the differential
148 se freshwater shrimp genus Caridina is still poorly understood, despite its vast distribution - from
149 tterns and drivers of shrub expansion remain poorly understood, inhibiting accurate incorporation of
150 uantities in end-of-life vehicles (ELVs) are poorly understood, mainly due to a limited understating
151 e introduction of biologically relevant, but poorly understood, modifications to cytosine affect the
152  phosphorylation effects remains nonetheless poorly understood, particularly for intrinsically disord
153 e the economics of water quality testing are poorly understood, the extent to which cost may be a bar
154    The breeding habitats of this species are poorly understood, which greatly hampers practical resea
155 ity ratios in children with tuberculosis are poorly understood-particularly those among children with
156 ic factors of late-life depression are still poorly understood.
157  The mechanism of action of GM-CSF in EAE is poorly understood.
158 , the nature and timing of these changes are poorly understood.
159 ) pathology and expression of AD symptoms is poorly understood.
160 he factors that promote DCIS invasion remain poorly understood.
161 ic determinants of body fat distribution are poorly understood.
162 ral activity creates this motivation remains poorly understood.
163 gene networks in the innate immune cells are poorly understood.
164 gation of chromosomes during mitosis remains poorly understood.
165 t the underlying molecular mechanisms remain poorly understood.
166 ructure and dynamics in this process remains poorly understood.
167 hanisms underlying these interactions remain poorly understood.
168  spatiotemporal dynamics during behavior are poorly understood.
169 ions in glioblastomas with wild-type IDH are poorly understood.
170  the specificity of such interactions remain poorly understood.
171 liminated to maintain protein homeostasis is poorly understood.
172 nctions in hematological malignancies remain poorly understood.
173 verexpression of this receptor in cancer are poorly understood.
174 biological basis of these sex differences is poorly understood.
175 s and organization in motile cells are still poorly understood.
176 globally distributed, cryptic environment is poorly understood.
177 arts; however, the underlying mechanisms are poorly understood.
178    Basic mechanisms for this association are poorly understood.
179 e cancer development and progression remains poorly understood.
180 alian arcopallial and amygdala subregions is poorly understood.
181 s underlying sex-specific development remain poorly understood.
182 ontribute to MSC phenotype in cancer remains poorly understood.
183  underlying lysosomal deficits in AD remains poorly understood.
184  activity and breast cancer formation remain poorly understood.
185 f such sequences are encoded in the brain is poorly understood.
186 tein aggregates from more innocuous ones are poorly understood.
187 erlying determinants of this variability are poorly understood.
188 e expectancy, the underlying biology remains poorly understood.
189 LA3 and its variant I148M in this process is poorly understood.
190 echanisms favoring PMN-MDSC responses remain poorly understood.
191 intervals of intraplate faults therefore are poorly understood.
192 athobiology of early squamous lung cancer is poorly understood.
193  such expansions cause neurodegeneration are poorly understood.
194  systems interact to regulate bone growth is poorly understood.
195 tical oscillations and neuronal responses is poorly understood.
196 ation of this process leads to cancer remain poorly understood.
197 influencing risk and disease progression are poorly understood.
198 trol expression of RP genes in metazoans are poorly understood.
199 erlying opposition to Polycomb silencing are poorly understood.
200     However, its mechanisms of action remain poorly understood.
201 iratory disease and lung cancer mortality is poorly understood.
202 , and the spectrum of microbes affected, are poorly understood.
203 ingle AD trial, and this variability remains poorly understood.
204 uch as the incorporation of molecular O2 are poorly understood.
205 erization impacts DNA-binding specificity is poorly understood.
206  antipsychotic efficacy and side effects are poorly understood.
207 nteractions with crop quality parameters, is poorly understood.
208 alter the course of Leishmania infection are poorly understood.
209 alter the course of AD neuropathology remain poorly understood.
210 ons underlying this aberrant behavior remain poorly understood.
211 (TF) occupancy and enhancer activity remains poorly understood.
212  the underlying molecular mechanism is still poorly understood.
213 he impact of SCNAs on tumour biology remains poorly understood.
214 tion in idiopathic pulmonary fibrosis remain poorly understood.
215 facilitate dynamic stomatal responses remain poorly understood.
216 ing properties of its small RNA guide remain poorly understood.
217 ch tau organizes microtubule networks remain poorly understood.
218 ures in a solvent-free environment are still poorly understood.
219 min-1 (syt1) in regulation of endocytosis is poorly understood.
220 , but the mechanism of this activity remains poorly understood.
221  ER shaping is linked to neuronal defects is poorly understood.
222 ose resulting in return to theater (RTT) are poorly understood.
223 alcifying and silicifying haptophytes remain poorly understood.
224  how it communicates with neural elements is poorly understood.
225 ogically distinct plasma membrane domains is poorly understood.
226 w connections, a process whose regulation is poorly understood.
227 s, communication between these cells remains poorly understood.
228 proliferation potential of LRP cells remains poorly understood.
229 ly disease in the context of prediabetes are poorly understood.
230 -22 expression in the human intestine remain poorly understood.
231 gulation of differentiation-related genes is poorly understood.
232 f specific residues in the nervous system is poorly understood.
233 main, the mechanisms for Tie2 activation are poorly understood.
234  and maintain gut bacterial diversity remain poorly understood.
235 e response to chemotherapeutic drugs remains poorly understood.
236 ce sensory stimuli into action potentials is poorly understood.
237  experience on interneuron plasticity remain poorly understood.
238 er (MDD) patients with suicidal ideation are poorly understood.
239 to cellular behavior, but their interplay is poorly understood.
240 the mechanism underlying this association is poorly understood.
241 d stands of varying species richness remains poorly understood.
242  drivers of fine-root trait diversity remain poorly understood.
243 ve black holes in the early universe remains poorly understood.
244 the human pathology and NMDAR dysfunction is poorly understood.
245 , transcription initiation by mtRNAP remains poorly understood.
246  circuits underlying appetitive behaviors is poorly understood.
247 velopment, but regulation of this process is poorly understood.
248 tive value in predicting clinical events are poorly understood.
249  and structure of these cytoskeletons remain poorly understood.
250 thern peripheries of ancient Mesoamerica are poorly understood.
251 s syndrome (ARDS), joblessness is common but poorly understood.
252 re of R5 during silica precipitation remains poorly understood.
253  processes, the role of the daughter remains poorly understood.
254  among specific age and sex groups are still poorly understood.
255 chanisms accounting for hypersensitivity are poorly understood.
256 hanisms that orchestrate these events remain poorly understood.
257 re determined during differentiation remains poorly understood.
258 ons to circadian photoreception is currently poorly understood.
259 hanisms mediating delamination are, however, poorly understood.
260  pathogenesis of these complications remains poorly understood.
261 enzymatic activity, but how they function is poorly understood.
262 ogy is maintained in the adult brain remains poorly understood.
263 s that control its proapoptotic function are poorly understood.
264 tissue-specific, epigenetic pattern is still poorly understood.
265 ergy and autoimmune disorders is complex and poorly understood.
266 his evolutionary conserved diversity remains poorly understood.
267 s in the loss of tolerance to gluten remains poorly understood.
268 way, but sensing of physical stimuli remains poorly understood.
269 cal diseases, its role at nerve terminals is poorly understood.
270 ion to antisocial behavior and adversity are poorly understood.
271  CD4 T cells and in HIV-infected children is poorly understood.
272 f chondrocyte recognition by NK cells remain poorly understood.
273 g the infection to cancer development remain poorly understood.
274  control during mammalian development remain poorly understood.
275 et the key events that drive aging are still poorly understood.
276 lter tumour progression, their regulation is poorly understood.
277 , protein dynamics within the 3D nucleus are poorly understood.
278 ary gland morphogenesis and homoeostasis are poorly understood.
279 al mechanisms underlying this process remain poorly understood.
280 id innate and adaptive allergic responses is poorly understood.
281 irmed independent cancer risk factor, remain poorly understood.
282  uneven distribution within endemic areas is poorly understood.
283 lar mechanisms regulating this transport are poorly understood.
284 s, but the mechanisms involved are generally poorly understood.
285 , the underlying molecular mechanisms remain poorly understood.
286 enesis of severe asthma in childhood remains poorly understood.
287  precise mechanism underlying gating remains poorly understood.
288 onses are regulated by other stimuli remains poorly understood.
289 physiological basis of this communication is poorly understood.
290 ch Cdx members impact their transcription is poorly understood.
291 the regulation of MAVS-mediated apoptosis is poorly understood.
292 he roles of CDC42 in tumorigenesis are still poorly understood.
293 e translocase; however, its function is only poorly understood.
294 fills its controlling role, however, remains poorly understood.
295 athophysiology of carcinoid heart disease is poorly understood; however, chronic exposure to excessiv
296  humoral but not cellular immunity, and have poorly-understood mechanisms.
297                                Formulating a poorly water-soluble drug substance into nanocrystals of
298 ween the 2 approaches, MSI3-slices performed poorly when MSI was <0.50.
299         While electrophilicities E correlate poorly with the LUMO energies and with Parr's electrophi
300                NIK transgenic Tregs competed poorly with WT Tregs in vivo and produced pro-inflammato

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