1 so exploited by bacteriocins to translocate into cells by
a poorly understood process.
2 ng how tissue specificity of their function is achieved
are poorly understood.
3 but links between specific dietary fats and cell fates
are poorly understood.
4 tory domain impair NHE6 activity and endosomal function
are poorly understood.
5 d States; however, the patterns for grants and grantees
are poorly understood.
6 s and whether it predicts the response to immunotherapy
are poorly understood.
7 ment and during cardiac neovascularization after injury
are poorly understood.
8 pers its treatment; the underlying molecular mechanisms
are poorly understood and warrant investigation given the inadequ
9 er, the function of GPR139 and its signaling mechanisms
are poorly understood.
10 impact and functional relevance of these modifications
are poorly understood.
11 matopoietic stem cells (HSCs) within the leukemic niche
are poorly understood, especially in the human context.
12 es that occur in the brain as this learning takes place
are poorly understood.
13 nces, but the neural mechanisms underlying this process
are poorly understood.
14 strate galactosylceramide, and the origin of psychosine
are poorly understood.
15 ond to target stimuli while ignoring distractor stimuli
are poorly understood.
16 papilla matrix to reach taste receptors, processes that
are poorly understood.
17 composed of millipedes and centipedes, is a fascinating
but poorly understood branch of life, including species with a hi
18 cognitive impairment (MCI) and Alzheimer's disease (AD)
is poorly understood, particularly at early stages preceding neu
19 s of actions, but how these are implemented in the brain
is poorly understood.
20 acetylase activities and site-selectivities in chromatin
is poorly understood.
21 injury induced under pathologically relevant conditions
is poorly understood.
22 oroquinolone treatment failure for Mycoplasma genitalium
is poorly understood.
23 egration is a rare event which significance for immunity
is poorly understood.
24 lements may have played in the evolution of bat immunity
is poorly understood.
25 How this resistance is mediated
is poorly understood, particularly during long-term exposure.
26 hat underlie disparities in cancer incidence and outcome
is poorly understood.
27 y recapitulate key disease features, and pathophysiology
is poorly understood.
28 However, its mechanism of transport into plants
is poorly understood.
29 angroves to high rates of relative sea level rise (RSLR)
is poorly understood.
30 nd characteristics of monsoonal precipitation and runoff
is poorly understood.
31 Neurobiological heterogeneity in schizophrenia
is poorly understood and confounds current analyses.
32 , the contribution of this function to tumor suppression
is poorly understood.
33 ion, some fundamental aspects of protein-DNA binding
remain poorly understood(1,2).
34 expression of Tim-3, but the molecular determinants
remain poorly understood.
35 equent as droughts, are overlooked and their impacts
remain poorly understood.
36 ity, and the mechanisms linking these two phenomena,
remain poorly understood.
37 g light and temperature signaling pathways in plants
remain poorly understood.
38 lipid mixing on the membrane biophysical properties
remain poorly understood.
39 g the relationship between cell number and body size
remain poorly understood.
40 that underpin mutational selection in normal tissues
remain poorly understood.
41 istal regulatory elements, yet how this is achieved
remains poorly understood.
42 ng this dysregulation, particularly in human cells,
remains poorly understood.
43 gation, but how they affect substrate conformations
remains poorly understood.
44 ates processing higher up in the cortical hierarchy
remains poorly understood.
45 nt immunity against hemibiotrophic fungal infection
remains poorly understood.
46 identified, precisely how gamma-TuRC nucleates a MT
remains poorly understood.
47 ensively investigated, the genetic etiology of NTDs
remains poorly understood.
48 on turnover to large-scale hydroclimate variability
remains poorly understood.
49 ls respond to the R loop-associated genomic stress is
still poorly understood.
50 e adaptor protein complex 4 (AP-4) lead to prototypical
yet poorly understood forms of childhood-onset and complex heredi