1 hip between allergy and autoimmune disorders is complex
and poorly understood.
2 and their relation to antisocial behavior and adversity
are poorly understood.
3 t the mechanisms that control its proapoptotic function
are poorly understood.
4 ns driving mammary gland morphogenesis and homoeostasis
are poorly understood.
5 ors, but the mechanisms accounting for hypersensitivity
are poorly understood.
6 However, protein dynamics within the 3D nucleus
are poorly understood.
7 Molecular mechanisms regulating this transport
are poorly understood.
8 iratory distress syndrome (ARDS), joblessness is common
but poorly understood.
9 morphogenesis of developing organs requires coordinated
but poorly understood changes in epithelial cell-cell adhesion an
10 direct projections to circadian photoreception is
currently poorly understood.
11 Especially poorly understood are the very early stages of protein foldin
12 ong nerve tracts, but the mechanisms involved are
generally poorly understood.
13 ted to targeting one membranous subunit of V-ATPase or
have poorly understood mechanisms of action.
14 -biological mechanisms mediating delamination are,
however,
poorly understood.
15 d molecular mechanisms triggered by these various agents
is poorly understood, but it might explain the differential resp
16 determines its uneven distribution within endemic areas
is poorly understood.
17 but its role on CD4 T cells and in HIV-infected children
is poorly understood.
18 tein (MOG)35-55 The mechanism of action of GM-CSF in EAE
is poorly understood.
19 thrive in this globally distributed, cryptic environment
is poorly understood.
20 (2+) flux into enzymatic activity, but how they function
is poorly understood.
21 ese processes alter tumour progression, their regulation
is poorly understood.
22 ely mediates said innate and adaptive allergic responses
is poorly understood.
23 erous neurological diseases, its role at nerve terminals
is poorly understood.
24 These novel quasi-particles arguably explain a range
of poorly understood experiments in gated graphene structures at
25 r both symbiotic and pathogenic relationships, but is
often poorly understood at the molecular level.
26 he mechanisms of chondrocyte recognition by NK cells
remain poorly understood.
27 chanisms linking the infection to cancer development
remain poorly understood.
28 f translational control during mammalian development
remain poorly understood.
29 Mechanisms that orchestrate these events
remain poorly understood.
30 trition, a confirmed independent cancer risk factor,
remain poorly understood.
31 tem cell (MaSC) activity and breast cancer formation
remain poorly understood.
32 tic dysfunction, the underlying molecular mechanisms
remain poorly understood.
33 he influence of experience on interneuron plasticity
remain poorly understood.
34 though the neural mechanisms underlying this process
remain poorly understood.
35 euronal morphology is maintained in the adult brain
remains poorly understood.
36 is extensively studied in suspension cultures, but
remains poorly understood in substrate-dependent cells.
37 , the molecular pathogenesis of these complications
remains poorly understood.
38 osome-dependent processes, the role of the daughter
remains poorly understood.
39 ne expression are determined during differentiation
remains poorly understood.
40 gnificance of this evolutionary conserved diversity
remains poorly understood.
41 y T (Treg) cells in the loss of tolerance to gluten
remains poorly understood.
42 rfacial structure of R5 during silica precipitation
remains poorly understood.
43 e cellular pathway, but sensing of physical stimuli
remains poorly understood.
44 The etiologic factors of late-life depression are
still poorly understood.
45 heir importance among specific age and sex groups are
still poorly understood.
46 al event, and yet the key events that drive aging are
still poorly understood.
47 The HTLV-1 particle structure is
still poorly understood, and previous studies analyzed viruses prod
48 the old, diverse freshwater shrimp genus Caridina is
still poorly understood, despite its vast distribution - from Afric
49 maintenance of tissue-specific, epigenetic pattern is
still poorly understood.
50 This poorly understood population is referred to as minimal residu