ライフサイエンスコーパス: popliteal

1 s were 199 superficial femoral arteries, 110 popliteal, 218 tibials, and 52 multilevel.

2 Accumulation of the nonspecific agent by the popliteal and axillary nodes at 6-hr postinjection was a

3 Popliteal and axillary nodes were then assayed for perce

4 p < 0.01) were directly associated with both popliteal and carotid intimal-medial thicknesses.

5 e present analyses are based on the baseline popliteal and carotid ultrasonography examination in 10,

6 ial (TRP) channel TRPC3 were present in both popliteal and first order intramuscular arterioles.

7 ode (LN) excision, consisting of ipsilateral popliteal and inguinal LN excision and to evaluate the i

8 ons in lymph drainage through tumor-draining popliteal and inguinal LNs versus contralateral uninvolv

9 The popliteal and inguinal lymph nodes were excised ipsilate

10 also related to early thickening of both the popliteal and the carotid artery walls.

11 imes to the common femoral artery (CFA), the popliteal and tibial arteries, and the corresponding vei

12 angioplasty, and atherectomy of the femoral, popliteal, and tibial vessels.

13 cond and first order arterioles vs. feed and popliteal arteries (58% and 16% vs. 5% and 3%; N = 10 im

14 surement of oxygen saturation in the femoral/popliteal arteries and veins during cuff-induced reactiv

15 based therapy in the superficial femoral and popliteal arteries in patients with peripheral artery di

16 or arterial remodelling, in the brachial and popliteal arteries of 13 healthy male subjects (21.6 +/-

17 flow (mean vessel sharpness: 44% vs 30% for popliteal arteries, 45% vs 28% for saphenous arteries; P

18 Popliteal arteries, subsequent gastrocnemius feed arteri

19 rtery disease of the superficial femoral and popliteal arteries.

20 ent placement for obstructive lesions of the popliteal artery achieves superior acute technical succe

21 U in the midabdominal aorta to 357 HU in the popliteal artery and 253 HU in the dorsalis pedis or pos

22 with additional 5 seconds +/- 2 to reach the popliteal artery and 7 seconds +/- 4 to reach the ankle

23 The relationship between PI values of the popliteal artery and the number of thrombosed calf veins

24 tion, the superficial femoral artery and the popliteal artery are subject to various forces e.g. thos

25 tentially associated with the development of popliteal artery atherosclerosis in a population-based s

26 tment of symptomatic superficial femoral and popliteal artery disease.

27 stress reaction, periostitis, claudication, popliteal artery entrapment, and peripheral nerve entrap

28 andomization, included the ICD and ACD, ABI, popliteal artery flow with duplex and QOL* at baseline*,

29 udication distances, pressure indices [ABI], popliteal artery flow, and QOL with the short-form 36 He

30 Brachial and popliteal artery FMD and DC did not change in control su

31 Similarly, popliteal artery FMD increased from baseline (6.2 +/- 0.

32 Patients with popliteal artery injuries over the 10-year period ending

33 e limb salvage in association with repair of popliteal artery injuries.

34 accepted factors impacting amputation after popliteal artery injury include blunt trauma, prolonged

35 ed to evaluate those factors associated with popliteal artery injury that influence amputation, with

36 ated spectral doppler characteristics of the popliteal artery on the same side as the isolated calf v

37 To investigate the usefulness of popliteal artery spectral doppler findings as a complime

38 The WSS in the narrow popliteal artery was more sensitive to a reduction in ra

39 n 5 cm in the superficial femoral artery and popliteal artery, and six patients had stenoses or occlu

40 y words were: "superficial femoral artery," "popliteal artery," "angioplasty," "drug-eluting balloon,

41 atients had stenoses or occlusions below the popliteal artery.

42 t to the pulsatility index (PI) value of the popliteal artery.

43 n, plasmid DNA was transferred to the distal popliteal artery.

44 class 2 to 5 who had a de novo lesion in the popliteal artery.

45 lar treatment for obstructive lesions of the popliteal artery.

46 luding superficial femoral, deep femoral and popliteal) artery models that were reconstructed from ma

47 Patients with symptomatic femoro-popliteal atherosclerotic disease undergoing percutaneou

48 Though popliteal blood flow appeared to be modulated by respira

49 ign also suggested a correlation between the popliteal-brachial gradient and aortic regurgitation sev

50 olume increased, in-hospital mortality after popliteal bypass decreased from 6.5% to 4.9% (P = 0.0045

51 thmia, aortic valve replacement, and femoral popliteal bypass graft) in isradipine (n=40; 9.05%) vs h

52 ased again by week 8 (6.5 +/- 0.6%), whereas popliteal DC progressively increased from baseline (8.9

53 Popliteal DCs transduced with an empty recombinant Adv u

54 rability for aortoiliac disease than femoral popliteal disease.

55 ude femorodistal bypass to ankle or foot and popliteal distal bypass using autogenous vein usually in

56 Iliofemoral DVT (n = 221 [71%]) and femoral-popliteal DVT (n = 79 [25%]) were treated with urokinase

57 y one patient required surgery for a delayed popliteal embolus.

58 h proximal deep-vein thrombosis of the legs (popliteal, femoral, or iliac vein thrombosis).

59 sions of the superficial femoral or proximal popliteal (femoropopliteal) artery.

60 5% CI, -0.2 to 2.1; P=0.116), peak hyperemic popliteal flow (0.0+/-0.4 mL/s; 95% CI, -0.8 to 0.8; P=0

61 Inter- and intragroup popliteal flow differences at 5/12 were small (P > 0.1).

62 time (PWT), collateral count, peak hyperemic popliteal flow, and capillary perfusion measured by magn

63 Skin grafts were placed distal to the popliteal fossa and mice were euthanized at day 10.

64 xcised collecting lymphatic vessels from the popliteal fossa of mice and removed their muscle cells t

65 nerve block was produced at the level of the popliteal fossa, and behavior was assessed using evoked

66 hy was performed from the iliac crest to the popliteal fossa.

67 ned contiguous images from the pelvis to the popliteal fossa.

68 om mouse collecting lymphatic vessels of the popliteal fossa.

69 eral erythematous halo was noted in the left popliteal fossa; the ulcer had begun as an asymptomatic

70 CT venograms from the iliac crests to the popliteal fossae were reviewed for presence and location

71 in sites relevant to AD: the antecubital and popliteal fossae, nasal tip, and cheek.

72 adjustment for covariates, both carotid and popliteal intimal-medial thicknesses were strongly assoc

73 of Obstructive Superficial Femoral Artery or Popliteal Lesions With A Novel Paclitaxel-Coated Percuta

74 and biomechanical properties of the oblique popliteal ligament (OPL).

75 ells were observed in maximal numbers in the popliteal LN at day 1 and in marginal zones and T-depend

76 and knee joint synovial volumes and draining popliteal LN volumes before and after 8 weeks of treatme

77 erations in contrast agent drainage into the popliteal LN, while lower molecular weight or albumin-bi

78 pliteal LNs, lymphatic drainage from paws to popliteal LNs, and the number of VEGF-C-expressing CD11b

79 he number of lymphatic vessels in joints and popliteal LNs, lymphatic drainage from paws to popliteal

80 reatment significantly decreased the size of popliteal LNs, the number of lymphatic vessels in joints

81 und the cortex and medulla of tumor-draining popliteal LNs, while they were restricted to the cortex

82 ine and chemokine production in the draining popliteal LNs.

83 y in lymphatic vessels afferent to collapsed popliteal LNs.

84 ion on lymph transport from paws to draining popliteal LNs.

85 l formation and the morphology of joints and popliteal LNs.

86 of VEGF-C-expressing CD11b+ myeloid cells in popliteal LNs.

87 model of inflammatory-erosive arthritis, the popliteal lymph node (PLN) enlarges during the pre-arthr

88 ural and functional changes of the adjoining popliteal lymph node (PLN), detectable by contrast-enhan

89 high CD25+) CD8+ T cells within the draining popliteal lymph node (PLN).

90 d by highly expanded B cells in the draining popliteal lymph node (PLN).

91 otyping, and gene expression profiles in the popliteal lymph node and inflamed joints, two pathogenic

92 the inflammatory infiltrate in the draining popliteal lymph node and the site of the infection using

93 The popliteal lymph node assay was used to study the role of

94 We analyzed clonally related VDJ genes from popliteal lymph node B cells responding to primary, seco

95 Ab blockade of ICOS ligand, expressed by popliteal lymph node B cells, but not dendritic cells, a

96 Flow cytometric analysis of popliteal lymph node cells demonstrated similar profiles

97 Spleen and popliteal lymph node cells from OVA-primed mice 3 or 7 d

98 vitro proliferative response of OVA-specific popliteal lymph node cells was assessed.

99 d vessel growth; however, the tumor-draining popliteal lymph node featured greatly increased lymphati

100 , amides show inhibitory activity in the rat popliteal lymph node hyperplasia assay.

101 CCR2(+) monocytic dendritic cells within the popliteal lymph node in comparison with B6.WT mice.

102 dary challenge (the increase in the draining popliteal lymph node mass, cell number, and lymphocyte t

103 C cell lines (P < 0.01) in vitro, as well as popliteal lymph node metastases of ESCC cells in nude mi

104 ype littermates to quantify the synovial and popliteal lymph node volumes and the patella and talus b

105 t respond to the host antigens measured by a popliteal lymph node weight gain assay.

106 of myeloid and lymphoid DCs in the draining popliteal lymph node, but not in other lymphoid organs.

107 l as from Ag-induced germinal centers of the popliteal lymph node.

108 -positive cells were readily detected in the popliteal lymph nodes (pLN) of VLP-inoculated mice.

109 arance at the site of infection and draining popliteal lymph nodes (PLNs), and impaired functions of

110 ed B cell accumulation within tumor-draining popliteal lymph nodes (TDLN).

111 Tissues (draining popliteal lymph nodes [LN], spleens, and thymi) were rem

112 Furthermore, cLTalpha(-/-) popliteal lymph nodes contained a higher proportion of n

113 estimulation, and secondary responses in the popliteal lymph nodes following in vivo challenge and in

114 erived myeloid dendritic cells trafficked to popliteal lymph nodes from paw pads, the expression of C

115 ces potent IL-4 expression by T cells in the popliteal lymph nodes of mice following footpad immuniza

116 B lymphocytes from popliteal lymph nodes or Peyer's patch activated in vitr

117 ompared with the paraaortic lymph nodes, the popliteal lymph nodes retain greater than 95% of the rad

118 nt and subsequent MRI of rabbit axillary and popliteal lymph nodes revealed significant contrast enha

119 e for a T cell residing 24 hours in a murine popliteal lymph nodes to interact with a DC was 8%, 58%,

120 In selected mice, popliteal lymph nodes underwent Cerenkov luminescence im

121 Fluorescence within all popliteal lymph nodes was easily detected by the FireFly

122 sis of Ly6C(+) macrophages in the ankles and popliteal lymph nodes, decreased migration of monocytes

123 One and 36 h after injection, popliteal lymph nodes, representing the SLNs, were disse

124 antly to early induced IL-4 responses in the popliteal lymph nodes.

125 local (mesenteric) and distant (inguinal and popliteal) lymph nodes of mice with induced polymicrobia

126 after a single fraction of 20 Gy radiation, popliteal lymphadenectomy, and lymphatic vessel ablation

127 e inguinal and popliteal nodes with draining popliteal lymphatic vessel significantly decreased the p

128 IFN-gamma secretion by cultured popliteal lymphocytes decreased in TG animals by 83% aft

129 minal centers in the spleen, plasma cells in popliteal lymphoid nodes, bone marrow cells and granuloc

130 gament in 48%, and the fibular origin of the popliteal muscle in 53% of the patients, whereas standar

131 Upon stimulation of popliteal node cells, in vitro induction of regulatory c

132 Necropsy at 24 h revealed that the popliteal node on the experimental leg receiving the avi

133 nd enhanced NK cell activity in the draining popliteal node.

134 At both doses (1.7 and 8.4 nmol), the popliteal nodes had higher (P < 0.050) optical fluoresce

135 Excision of the inguinal and popliteal nodes with draining popliteal lymphatic vessel

136 des) and partial systemic (inguinal, but not popliteal nodes) loss of DCs from lymph nodes in septic

137 ial occlusions and 24 (83%) of 29 SFA and/or popliteal occlusions were longer than 10 cm.

138 tients with tibial occlusions and SFA and/or popliteal occlusions, respectively, as scored with modif

139 ce of symptomatic or asymptomatic DVT in the popliteal or femoral veins.

140 mies, iliac or femoral arteries in 25 (18%), popliteal or tibial arteries in 25 (18%), carotid arteri

141 d with LTbetaR-Ig had no axillary, inguinal, popliteal, or peripancreatic lymph nodes.

142 as absent during mild calf contraction where popliteal outflow was phasic with the concentric phase o

143 onspecific agents were observed for both the popliteal (p < 0.006) and axillary (p < 0.012) nodes.

144 ciated with thickened carotid (p < 0.01) and popliteal (p < 0.05) intimal-medial thicknesses, hormone

145 nd with plasma triglycerides (women only for popliteal) (p < 0.01).

146 Popliteal percent injected dose at 24 hr was 3.00 +/- 0.

147 t 6 months, and reinterventions after femoro-popliteal percutaneous transluminal angioplasty up to 1

148 oated balloons in patients undergoing femoro-popliteal percutaneous transluminal angioplasty.

149 pain attributable to superficial femoral and popliteal peripheral artery disease were randomly assign

150 th CEAP clinical class (P < .01) in femoral, popliteal, posterior tibial, peroneal, gastrocnemial, an

151 Van der Woude syndrome (VWS) and popliteal pterygium syndrome (PPS) are autosomal dominan

152 Popliteal pterygium syndrome (PPS; OMIM 119500) is a dis

153 sufficiency of IRF6 causes Van der Woude and popliteal pterygium syndrome, 2 syndromic forms of cleft

154 ries of devastating birth defects, including popliteal pterygium syndrome, cocoon syndrome, and Barts

155 genital disorders Van der Woude syndrome and popliteal pterygium syndrome, have a hyperproliferative

156 ermal disorders Bartsocas-Papas syndrome and popliteal pterygium syndrome, respectively.

157 6 (IRF6) underlie Van der Woude syndrome and popliteal pterygium syndrome.

158 otypes in individuals with Van der Woude and popliteal pterygium syndromes, suggesting that the TGFbe

159 fore and after light pulses presented to the popliteal region (behind the knee).

160 R imaging sequence performed parallel to the popliteal tendon proximally was added to our routine stu

161 ral collateral ligament of the knee, and the popliteal tendon.

162 femoral vein (CFV), mid-SFV, distal SFV, and popliteal vein (PV).

163 ases (99%), either the common femoral or the popliteal vein was involved.

164 T) isolated to the calf veins (distal to the popliteal vein) is frequently detected with duplex ultra

165 ed US examinations of the common femoral and popliteal veins only, followed by traditional US of the

166 The common femoral and popliteal veins were assessed for compressibility.

167 ws thrombosis of the superficial femoral and popliteal veins.Onchest computed tomography (CT) angiogr

168 thrombus in the left superficial femoral and popliteal veins; follow-up chest CT angiogram shows no e

169 ive blood oxygenation time-course of femoral/popliteal vessels in: 1) young healthy subjects (YH) (n

170 ial and superior femoral artery [SFA] and/or popliteal vessels, n = 22).

171 otein cholesterol and carotid (p < 0.01) and popliteal (women only) (p < 0.05) intimal-medial thickne