1 were most common in the portacaval space and porta hepatis.

2 ion, the right portal vein only being at the porta hepatis.

3                  Image noise at the level of porta hepatis and acetabulum was evaluated with a five-p

4 ension of the primary tumor infiltrating the porta hepatis and body of the pancreas.

5 tation, and the size of the bile duct at the porta hepatis and in the pancreatic head.

6  dilation of duct-like structures within the porta hepatis, and dysplastic changes.

7 s our experience that warm dissection in the porta hepatis as well as extensive organ mobilization du

8 ic bile duct were measured proximally at the porta hepatis, at the middle above the head of the pancr

9 nsplant/death was increased in patients with porta hepatis atresia (Ohi type II and III vs type I; HR

10 l age, gender, race, ethnicity, or extent of porta hepatis dissection.

11 of the peritoneal cavity, liver, lesser sac, porta hepatis, duodenum, transverse mesocolon, and celia

12 n both IMT and other malignant tumors of the porta hepatis, histologic examination should be consider

13 nto the hepatic vein(s), inferior vena cava, porta hepatis, or gallbladder fossa.

14                 Intermittent clamping of the porta hepatis (PHC) is commonly performed during liver s

15 t) at five levels: upper liver at diaphragm, porta hepatis, right kidney hilum, iliac crest, and uppe