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1 interaction between the azide group and the portal.
2 after the assembly of adaptor protein to the portal.
3 the web-based BankIt or the NCBI Submission Portal.
4 The survey was conducted through an online portal.
5 g a common tool and entered into a Web-based portal.
6 and visualization features through a single portal.
7 sociation, and the City of Toronto Open Data Portal.
8 indicating an induced fit on binding to the portal.
9 orm and through a controlled-access internet portal.
10 lopedia and the Cancer Therapeutics Response Portal.
11 ons at the second ureidyl C horizontal lineO portal.
12 based support, social platforms, and patient portals.
13 ...O horizontal lineC close contacts at both portals.
14 omponents are at slightly lower resolutions: portal (9.2 A), hub (8.5 A), tailspike (10.9 A), and nee
15 called the 'portal vertex' and consists of a portal, a DNA packaging ATPase and other components.
23 ct data is presented through the HipSci data portal and is downloadable from the associated FTP site
24 xpression omnibus (GEO), such as the GEO web portal and related applications, are optimized to reanal
25 ated through echographic measurements, while portal and systemic pressures were measured through poly
27 nal Clinical Trials Registry Platform Search Portal, and clinicaltrials.gov The date of the last sear
28 nal Clinical Trials Registry Platform Search Portal, and clinicaltrials.gov The date of the last sear
29 ens utilise this natural opening as an entry portal, and thus have an increasingly complex relationsh
30 Portosystemic shunting was demonstrated by portal angiography, which disclosed virtually complete p
31 RT: We present imaging findings in a case of portal annular pancreas in a 45-year-old male patient.
35 Biliary ductal cells proliferate from the portal areas of chronically damaged livers, but their si
36 iopsy, which was moderate to severe (<50% of portal areas with bile ducts) in 14 and mild (50%-75%) i
37 vascular structures, wall thickening of some portal arteries, and periductal fibrosis in Fut2(-/-)(hi
38 ular involvement of all hepatic veins (V) or portal bifurcation (P), contiguous extrahepatic tumour (
39 nstantly exposed to gut-derived antigens via portal blood and, as a consequence, they express a uniqu
43 organized and presented in a searchable web-portal called gene-drug Interaction for survival in canc
49 of gut-derived antigens that arrive through portal circulation at homeostasis and protect these orga
51 the TMA produced is passively absorbed into portal circulation, and hepatic flavin-dependent monooxy
52 l permeability, release of microbes into the portal circulation, and increased serum levels and liver
54 nction with the Cancer Therapeutics Response Portal (CTRP), a dataset with drug-response measurements
61 t and is associated with early activation of portal fibroblasts (PFs) that express Thy-1, fibulin 2,
62 letion results in a significant reduction of portal fibrosis and portal hypertension as well as of li
64 tter understanding of reciprocal dynamics of portal flow and regeneration between the graft and nativ
65 as to study hepatic blood flow and effect of portal flow modulation on graft outcomes in the setting
66 st the need for a study using a prespecified portal flow modulation protocol with defined indications
68 terial fraction, and arterial flow but lower portal flow, distribution volume, and mean transit time
70 mall-molecules profiled, and developed a web portal for browsing and searching predictive small-molec
71 The EMDataBank project provides a unified portal for deposition, retrieval and analysis of 3DEM de
72 and visualization, concise gene summaries, a portal for external resources, video tutorials and the F
73 d reinterpretation of sieve effects and as a portal for organizing and sharing the viral sequence dat
75 ant data for every strain, and a GWA mapping portal for studying natural variation in C. elegans Addi
76 ve contraction along the anterior intestinal portal generates tension to elongate the foregut and hea
78 l helix-loop observed in the Sf6-gp7 and P22 portal:gp4 complex represents the pre- and postassembly
79 GKRP, and it is activated postprandially by portal hyperglycemia and fructose through dissociation f
80 CLD), the presence of clinically significant portal hypertension (CSPH) and varices needing treatment
81 ssociated cirrhosis and clinical significant portal hypertension (CSPH, hepatic venous pressure gradi
82 rices (EVs) or having clinically significant portal hypertension (for presurgical risk stratification
83 nrolled patients with compensated cirrhosis, portal hypertension (hepatic venous pressure gradient [H
84 ss vary differently with respect to cause of portal hypertension (ie, congestion- or cirrhosis-induce
85 ed with progressively developed fibrosis and portal hypertension (mean stiffness at 80 Hz and 48-week
94 er cirrhosis is complicated by bleeding from portal hypertension but also by portal vein thrombosis (
95 ality Improvement Program (NSQIP) formed the portal hypertension cohort, and were case matched to pat
96 l hemorrhage is a concerning complication of portal hypertension in children, the first bleed appears
98 t leads to better control of RA secondary to portal hypertension in patients with cirrhosis, compared
99 lications related to liver insufficiency and portal hypertension in patients with heavy alcohol intak
103 ght subjects from six kindreds with onset of portal hypertension of indeterminate etiology during inf
104 rts of patients with idiopathic noncirrhotic portal hypertension undergoing TIPS in seven centers bet
106 s induced in rats by bile duct ligation, and portal hypertension was induced by partial portal vein l
107 In patients with idiopathic noncirrhotic portal hypertension who have normal kidney function or d
108 cutive patients with severe complications of portal hypertension who received placement of TIPS from
109 ephalopathy, esophageal varices, ascites, or portal hypertension) or liver transplant were estimated
110 scarring from any cause leads to cirrhosis, portal hypertension, and a progressive decline in renal
111 with hepatic "recompensation," reduction of portal hypertension, and eventually avoidance of liver t
113 , FAH(-/-) pigs developed liver fibrosis and portal hypertension, and thus may serve as a large-anima
114 varices, which are most commonly a result of portal hypertension, downhill esophageal varices result
115 early-onset familial idiopathic noncirrhotic portal hypertension, in which Mendelian mutations may ac
118 is and management of idiopathic noncirrhotic portal hypertension, its pathogenesis remains elusive.
119 with preoperative hierarchic interaction of portal hypertension, planned extension of hepatectomy, a
120 oportions of patients in the LVP+A group had portal hypertension-related bleeding (18% vs 0%; P = .01
136 esources have been added to the RCSB PDB web portal in support of a 'Structural View of Biology.' Rec
139 s (taurocholate), lobular (glycocholate) and portal inflammation (taurolithocholate), and hepatocyte
140 NASH), histologically defined by lobular and portal inflammation, and accompanied by marked oxidative
141 tal fibrosis, bridging, parenchymal nodules, portal inflammation, hepatocellular swelling, steatosis,
144 , insulin sensitivity and clearance, and the portal insulin:glucagon ratio.The addition of lactisole
147 delling studies indicate the structure of PC-portal is incompatible with DNA coaxially spooled around
150 While the important feature of this public portal is the ability for the users to build maps from t
152 ponsive element-binding protein and featured portal/lobular inflammation along with total, whole-body
153 ocellular swelling, steatosis, dilatation of portal lymphatics, and periductal fibrosis did not show
154 ere investigated in Genomic Data Common data portal miRNA-Seq dataset and The Cancer Genome Atlas (TC
155 kaging machine consists of three components: portal, motor (large terminase; TerL) and regulator (sma
160 serving as a likely permissive reservoir and portal of fetal transmission with risk of latent microce
161 attribute to the larger size of the carbonyl portals of CB[8]; this suggests routes to develop CB[8]
162 ontribution of fibroblastic stromal cells as portals of entry into the CNS was only recently uncovere
165 P=0.04), access their CHD risk via a patient portal (OR, 2.99 [CI, 1.35-7.04]; P=0.01), and discuss t
167 Remarkably, while the distance between the portal plane and most atoms at the guest end groups incr
168 atoms maintain a constant distance from the portal plane in all homologues, pointing at a strong att
175 derate exercise were safe and reduced BW and portal pressure in overweight/obese patients with cirrho
184 f the major head protein, 12 subunits of the portal protein and 120 subunits of the decoration protei
185 C-terminal arm, which may interact with the portal protein during motor assembly, as predicted for s
186 ere, we provide structural evidence that the portal protein of the bacteriophage P22 exists in two di
187 ly through binding to the -domain, while the portal protein regulates assembly into the correct T=13
189 on of the genome DNA within the channel, the portal protein would become a Brownian motor, which adop
190 in turn, affect the channel activity of the portal protein, GP10, embedded in the semipermeable caps
194 h as the alpha-helical barrel domains of P22 portal proteins and T7 proteins that form tail tube exte
195 nd compared them with the clip region of the portal proteins of bacteriophages phi29, SPP1 and T4.
202 omeCentral remains as the common data access portal, providing the ability to search for data sets in
203 tutional review board protocol 2 hours after portal reperfusion, followed by Western blot analyses.
204 The site of EHD affected survival, with portal, retroperitoneal nodes and multiple sites associa
205 The site of EHD affected survival, with portal, retroperitoneal nodes and multiple sites associa
210 nd a symmetric ring in the mature virion (MV-portal) that has negligible affinity for the packaging m
211 an asymmetric assembly in the procapsid (PC-portal) that is competent for high affinity binding to t
212 ximately 0.32 A above the C horizontal lineO portal; the observed 0.80 A spacing observed for CB[7].D
213 promotes a breach of BM vascular sinusoidal portals, thereby augmenting HSPC trafficking to the circ
214 hat enables its integration into third-party portals, thus providing 'Search as a Service' capabiliti
216 exploits the HER3 cell surface protein as a portal to sneak therapeutics into tumor cells by mimicki
218 ce for biomedical applications and open up a portal to the next generation of multi-functional electr
219 Patient and care partner access to online portal to view health information, participate in the ca
220 R in PBC livers (particularly in the hepatic portal tracks) support a disease mechanism in which the
221 uding liver-specific inflammation focused on portal tract areas, increased number and activation stat
224 rosis (>6 weeks) after DR migration from the portal tracts to the centrilobular site of injury, in as
226 c lobule or histologic nodular growth in the portal triad that effaced adjacent hepatic parenchyma.
227 double-heterozygous mice show well-developed portal triads around most portal veins, with no elevatio
229 r results were found across types of patient portal use (communicating by email, viewing laboratory t
230 ortal shunting vessels, increased numbers of portal vascular structures, wall thickening of some port
232 nderwent 2D phase-contrast MR imaging of the portal vein (PV) and infrahepatic and suprahepatic infer
236 ernative porto-caval shunt between the right portal vein and inferior vena cava detected on postnatal
238 model perfused with autologous blood via the portal vein at three flow rates (60, 80, 100 mL/min per
239 sion Unlike monopolar RF ablation, change in portal vein flow rates does not have a statistically sig
240 rpose To investigate the effect of change in portal vein flow rates on the size and shape of ablation
241 pectrometry with (ii) direct sampling of the portal vein following an intravenous glucose/arginine ch
243 the setting of hepatocellular carcinoma with portal vein invasion, and for radiation segmentectomy.
244 ation after extended partial hepatectomy and portal vein ligation for multiple bilobar CRLM were appl
245 ation after extended partial hepatectomy and portal vein ligation for multiple bilobar CRLM were appl
246 djustment in Associating Liver Partition and Portal Vein Ligation for Staged Hepatectomy (ALPPS) occu
248 r regeneration after partial hepatectomy and portal vein ligation, and increased the expression of ce
249 r regeneration after partial hepatectomy and portal vein ligation, and increased the expression of ce
252 vasion into the extra-hepatic portion of the portal vein or the development of distant metastases ren
253 creased clot formation rate, associated with portal vein platelet aggregates and reductions in protei
254 were mainly rejected for comorbidity (19%), portal vein thrombosis (16%), previous surgery (9%), obe
255 s of the liver in children with extrahepatic portal vein thrombosis (EHPVT), with surgical outcome af
257 ed-stage hepatocellular carcinoma (HCC) with portal vein thrombosis (PVT) treated with (90)Y radioemb
265 , 17 (17.3%) had cavernous transformation of portal vein, and 3 (3.1%) had post-transplant thrombosis
267 from the gastrointestinal tract through the portal vein, and thereby is exposed continuously to diet
268 x 2 cm x 2.1 cm in size with abutment of the portal vein-superior mesenteric vein confluence for less
270 Results Occlusion was identified in 39.7% of portal veins (29 of 73), 15.0% of hepatic veins (six of
271 f 21 patients with target tumors adjacent to portal veins developed mild to moderate cholestasis 2-6
272 resistant to vessel occlusion compared with portal veins, and only arterial patency within an ablati
273 immediately adjacent to major hepatic veins, portal veins, or both; thus, they were not considered su
274 covered extrinsic compression of hepatic and portal veins, resulting in functional Budd-Chiari syndro
275 how well-developed portal triads around most portal veins, with no elevation of serum bilirubin.
279 or more of pneumoperitoneum, fixed loop, and portal venous gas were present, and 1 point was assigned
280 ase states, minimally invasive transcatheter portal venous interventions have been developed to impro
281 dual-energy CT and arterial perfusion (AP), portal venous perfusion, and total perfusion (TP) from p
285 was associated with increased postoperative portal venous pressure and von Willebrand factor antigen
286 ostpreservation, as well as 30 minutes after portal venous reperfusion and hepatic arterial reperfusi
288 lular carcinoma (HCC) cells often invade the portal venous system and subsequently develop into porta
289 on (PVH) in liver cirrhosis complicated with portal venous thrombosis (PVT) has been mainly treated w
292 ex assembles at a special vertex called the 'portal vertex' and consists of a portal, a DNA packaging
293 atible with DNA coaxially spooled around the portal vertex, suggesting that newly packaged DNA trigge
294 ter closure of the umbilical inlet at birth, portal vessels undergo a transition from Neuropilin-1(+)
298 icipants from the clinic and from the online portal were significantly different in age (mean [SD] ag
299 Chemical In vitro-In vivo Profiling (CIIPro) portal, which can automatically extract in vitro biologi
300 h that of the P22 homolog complexed with the portal, which is achieved by repositioning of two consec
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