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1 s not express p185 are strong suppressors of position effect variegation.
2 act in these domains, as shown by monitoring position effect variegation.
3 d by suppression of heterochromatin-mediated position effect variegation.
4 allelic to Lighten-up, a known suppressor of position effect variegation.
5 ression, a heterochromatin phenomenon called position effect variegation.
6 ncer (TE) and a region that protects against position effect variegation.
7 netic gene silencing classically observed as position effect variegation.
8 reminiscent of, but clearly different from, position effect variegation.
9 l involvement of the P0 protein in modifying position effect variegation.
10 In addition, Low weakly suppresses position effect variegation.
11 n, eye development, chromosomal proteins and position effect variegation.
12 yed by nuclear organization in cis and trans position-effect variegation.
13 acts as both an enhancer and a suppressor of position-effect variegation.
14 to further methylation, suggests a model for position-effect variegation.
15 cus, initially identified as a Suppressor of Position-Effect Variegation.
16 lls, an effect that is probably analogous to position-effect variegation.
17 nearby gene in cis, a hallmark of classical position-effect variegation.
18 9me3, and is a classic genetic suppressor of position-effect variegation.
19 a HP1, a heterochromatin protein involved in position-effect variegation.
20 By using a combination of two modifiers of position effect variegation, adding an extra Y chromosom
21 to demonstrate that dLDH and L-2HG influence position effect variegation and DNA methylation, suggest
23 natural or synthetic PATCs are resistant to position effect variegation and stochastic silencing in
24 ls of retinal degeneration and mechanisms of position-effect variegation and demonstrate the utility
26 gmentation patterns similar to those seen in position-effect variegation and yet most inserts were in
27 ivity to DNA-damaging agents, suppression of position-effect variegation, and female sterility in whi
28 L-1 histone H3S10 kinase act as enhancers of position-effect variegation at pericentric sites whereas
29 overed in HP2 act as dominant suppressors of position effect variegation, confirming a role in hetero
30 of silencers in trans or by the spreading of position effect variegation from rearrangements having h
31 in Drosophila melanogaster for modifiers of position-effect variegation have revealed the basis of m
32 a variety of biological processes including position-effect variegation, heterochromatin formation a
35 transcriptional silencing, as exemplified by position effect variegation in Drosophila melanogaster a
37 to the postulated role of this DNA repeat in position effect variegation in facio- scapulohumeral mus
38 as been described in diverse systems such as position effect variegation in insects, silencing near y
39 letes H3K9 methylation levels and suppresses position-effect variegation in various Drosophila tissue
40 presence of Su(z)12, a strong suppressor of position effect variegation, in PRC2 suggests that PRC2
41 y nor qualitatively affected by modifiers of position effect variegation including the Y chromosome,
43 tation in Drosophila PR-Set7 that suppresses position effect variegation, indicating that PR-Set7 ind
44 r full function of the AE1 promoter and that position effect variegation is associated with RNA trans
46 tic DNA instability described here underlies position effect variegation, molds the structure of poly
51 r the spread of inactivation associated with position-effect variegation or X chromosome inactivation
52 tic screens that relied on mosaic silencing (position-effect variegation, or PEV) of the yellow gene
53 d HSS3), which is required for prevention of position effect variegation (PEV) in transgenic mice.
54 l are lethal, heterozygotes display enhanced position effect variegation (PEV) indicative of the broa
57 some (Dp(1;f)1187) dramatically increase the position effect variegation (PEV) of a yellow(+) body-co
58 stone H3S10 kinase are strong suppressors of position effect variegation (PEV) of the wm4 allele and
59 d that loss of 8 out of 13 JmjC genes modify position effect variegation (PEV) phenotypes, consistent
60 able gene repression, such as is observed in position effect variegation (PEV) when the Drosophila me
61 Drosophila melanogaster chromosomes exhibit position effect variegation (PEV), a mosaic silencing ch
62 terochromatin-localized protein required for position effect variegation (PEV), colocalized with DmOR
64 a PcG gene and mutations in His2Av suppress position effect variegation (PEV), suggesting that this
65 uchromatin and heterochromatin can result in position effect variegation (PEV), the variable expressi
70 sed as the explanation for such phenomena as position-effect variegation (PEV) and control of segment
75 gaster, heterochromatin-induced silencing or position-effect variegation (PEV) of a reporter gene has
77 in into a euchromatic gene, which results in position-effect variegation (PEV), also causes the aberr
78 osome in regulating rRNA gene transcription, position-effect variegation (PEV), and the link among rD
79 iable, fertile, and recessive suppressors of position-effect variegation (PEV), indicating that, as i
80 eterochromatin and regulates heterochromatin position-effect variegation (PEV), organization of repet
81 in an understanding of aneuploid syndromes, position-effect variegation (PEV), quantitative traits,
82 , we found that dhtt acts as a suppressor of position-effect variegation (PEV), suggesting that it in
87 effect of both Su(var)3-9 and Su(var)2-5 on position-effect variegation, providing evidence that a f
91 Mutations in Nap-1 are shown to suppress position effect variegation, suggesting that Nap-1 funct
92 IR2 mutations were recently shown to perturb position effect variegation, suggesting that the role of
95 bed tyrosyl tRNA gene, SUP4-o, is subject to position effect variegation when located near a telomere
96 y RNA polymerase II (RNAP II) are subject to position effect variegation when located near yeast telo
97 is, and it decreased (but did not alleviate) position effect variegation within the expressing cell t
98 emplified by piRNAs (piwi-interacting RNAs), position effect variegation, X-chromosome inactivation,
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