ライフサイエンスコーパス: positive cell

1 DNA double-strand breaks (>/=5% gamma-H2A.X-positive cells).

2 c markers (p53, cleaved caspase-7, and TUNEL-positive cells).

3 groups according to the percentage of PD-L1-positive cells.

4 creased number of BrdU- and beta-III tubulin-positive cells.

5 , which colocalized with megalin, in podocin-positive cells.

6 origenic than the freshly isolated Aldefluor-positive cells.

7 genome amplification in differentiating HPV-positive cells.

8 tive (HBsA-positive) and HBV DNA- and cccDNA-positive cells.

9 as shown by increased presence of caspase-3-positive cells.

10 an form a complex with LANA and PCNA in KSHV-positive cells.

11 red for activation of the ATM pathway in HPV-positive cells.

12 tured human colon CD3-negative, IL7-receptor-positive cells.

13 chromatin foci and increased numbers of Ki67-positive cells.

14 3beta mediate the activation of Tip60 in HPV-positive cells.

15 e Bim and higher frequency of active caspase-positive cells.

16 proliferation of immature (MafA(-)) insulin-positive cells.

17 with absent magnetic exposure showed no GFP-positive cells.

18 -associated beta-galactosidase (SA-beta-Gal)-positive cells.

19 or histocompatibility complex (MHC) class II-positive cells.

20 uman embryonic stem cells (hESCs) to insulin-positive cells.

21 n developing and adult insulin- and glucagon-positive cells.

22 uction and ALA-PDT in normal and ER- or HER2-positive cells.

23 co-localized with smooth muscle alpha-actin-positive cells.

24 as positively correlated with TCTP in HpslyD-positive cells.

25 lower PpIX level than MCF10A and ER- or HER2-positive cells.

26 targeting of IGF1R selectively impacts LMP1-positive cells.

27 levels by inducing apoptosis of Fas receptor-positive cells.

28 cleotidyl transferase dUTP nick-end labeling-positive cells.

29 teral to the central rosette of exorhodopsin-positive cells.

30 aminotransferase levels and numbers of TUNEL-positive cells.

31 rkedly reduced cyclobutane pyrimidine dimers-positive cells.

32 cient differentiation of CD4(+)CD8(+) double positive cells.

33 et-like structures containing MAFA/C-peptide-positive cells.

34 with camptothecin while sparing isogenic p53-positive cells.

35 minimal changes were seen in IL-4- and IL-5-positive cells.

36 ts accompanied by an expansion of Krt5/Krt14-positive cells.

37 N knockdown promotes PTK6 activation in PTEN positive cells.

38 increased 6-fold, protein 3-fold, and Cela1-positive cells 2-fold.

39 (1) increased Lgr5(+), without altering Bmi1 positive cells; (2) increased levels of proliferation ma

40 latelet-derived growth factor receptor-alpha-positive cells, 4) alpha-smooth muscle actin-positive bl

41 Collagen I-positive cells (89.43% +/- 6.53%) in day 28 allografts w

42 estimated that there are five effective Lgr5-positive cells able to give rise to monoclonal glands by

43 ed growth factor receptor alpha (PDGFRalpha) positive ( + ) cells accumulate in CTS SSCT and that the

44 During skin wound healing, CD26-positive cells accumulated over time and persisted in fo

45 ucleated tartrate-resistant acid phosphatase-positive cells along the alveolar bone surface was signi

46 Submucosal and epithelial IL-17A-positive cells and BAL IL-17A and IL-22 levels were simi

47 ing SOX5 gene significantly diminished RANKL positive cells and bone erosion in CIA mice.

48 and tartrate-resistant alkaline phosphatase-positive cells and Calcr mRNA expression.

49 nt protein or red fluorescent protein TagRFP positive cells and counted the percentage of positive ce

50 dysregulation restricted to such parvalbumin-positive cells and examined the impact on critical perio

51 ccumulation on autoradiography, with insulin-positive cells and GLP-1R expression on immunohistochemi

52 governing entry of measles virus into SLAMF1-positive cells and identified endocytic uptake of viral

53 eta-cell mass, decreased the number of TUNEL positive cells and improved glucose tolerance after gluc

54 BRCA1 are expressed at high levels in HPV31-positive cells and localize to sites of viral replicatio

55 ynthesis and to increased frequencies of HbF-positive cells and mature erythrocytes, as well as fewer

56 idine triphosphate nick end-labeling (TUNEL)-positive cells and mortality compared with vehicle-injec

57 This uptake pathway is specific to SLAMF1-positive cells and occurs within 60 min of viral attachm

58 dth of VEGF's influence extends beyond VEGFR-positive cells and propose a plausible mechanistic expla

59 P-BMSCs differentiated into type II collagen-positive cells and reversed cartilage degradation and su

60 By analyzing telomerase-positive cells and their human TERC knockout-derived ALT

61 t on the presence of both folate-FITC and FR-positive cells and was dose titratable with folate-FITC

62 latelet-derived growth factor receptor-alpha-positive cells, and alpha-smooth muscle actin-positive b

63 amma antibody led to a decrease in IFN-gamma-positive cells, and an increase in IL-5-positive cells,

64 were associated with increased HA, vimentin-positive cells, and fibrosis in Hyal2(-/-) compared with

65 ion of interleukin-1beta by CD11b- and Iba-1-positive cells, and loss of olfactory sensory neurons (O

66 c increase in valvular cell activation, CD45-positive cells, and matrix turnover.

67 local muscle cell compartments, such as PAX7 positive cells, and recruited macrophages during skeleta

68 In juveniles only, BrdU-positive cells appeared in contact with vimentin-labeled

69 Furthermore, we show that Ntsr1-Cre-positive cells are immuno-positive for the nuclear trans

70 eling showed that two clusters of melanopsin-positive cells are located lateral to the central rosett

71 ic neurons possess an AIS and that these AIS-positive cells are morphologically and functionally dist

72 These CK5-positive cells are therapy resistant and have increased

73 ndicates that Cytokeratin-Synaptophysin dual positive cells arise from the exocrine compartment.

74 d intestinal epithelial cells (IEC) and Hopx-positive cells as predominant fecal miRNA sources.

75 t was associated with reduced number of Drd1-positive cells at birth, in contrast to normal numbers o

76 ch were replaced by a notable number of IL-6-positive cells at day 14.

77 age, with few mature CD4(+) or CD8(+) single-positive cells being produced.

78 owing MSN-UPA nanoparticle treatment in UPAR positive cells blocked with a UPAR-blocking antibody.

79 ibers form perisomatic clusters around CCKBR-positive cell bodies in the BLA.

80 focus formation, and Akt activation in LMP1-positive cells but did not impair LMP1-induced cell migr

81 tage IV COPD, characterized by abundant BAFF-positive cells but few apoptotic cells (mostly B cells).

82 ycle arrest and eliminated Ki-67 mRNA in RB1-positive cells but had no effect in RB1-negative cells,

83 amma-positive cells, and an increase in IL-5-positive cells, but did not impact clearance of Pneumocy

84 ctivated for infection of CD4-negative, CCR5-positive cells, but the infection of CD4-positive, CCR5-

85 Upregulation of CK5-positive cells by 3-ketosteroids required induction of t

86 r- and progesterone receptor-negative (< 10% positive cells by immunohistochemistry) early breast can

87 We analyzed the expression profile of ASGR1-positive cells by microarray, and tested their ability t

88 ies of reports document induction of the CK5-positive cells by progestins, it is unknown if other 3-k

89 In this study, we demonstrate that insulin-positive cells can be generated in vitro from human indu

90 However, Snf5 ablation in GFAP-positive cells caused a neurodegenerative phenotype exac

91 Here, we investigate the role of CDH2 positive cell clusters in preserving healthy, biosynthet

92 rdal origin, and reside in N-cadherin (CDH2) positive cell clusters in vivo.

93 gy is preserved only when NP cells form CDH2 positive cell clusters.

94 These PDGF receptor alpha-positive cells co-localized with PDGF receptor beta, pro

95 eta) resulted in increased numbers of IL-17A-positive cells compared with wild-type controls.

96 These CCKBR-positive cells comprise a molecularly heterogenous popul

97 diminished the procoagulant activity of EMT-positive cells, confirming a functional role for TF in t

98 on of class-switched antibodies from Ara h 2-positive cells confirms enrichment for Ara h 2 specifici

99 elanocytes, immunotargeting of the chemokine-positive cells, continuous loss of the pigment-producing

100 ls; mixed epithelium: cK12-positive and MUC1-positive cells; corneal epithelium: cK12-positive and MU

101 GFP-positive cells could be detected in the cochlea for at l

102 as defined similarly, using the dates of CD4-positive cell counts or HIV-1 RNA measurements.

103 T-bet-positive cell counts per high-power field (hpf) were (a)

104 aracterized by the up-regulation of numerous positive cell cycle regulators.

105 CTGF treatment upregulates positive cell-cycle regulators and factors involved in b

106 The clearance of p16(Ink4a)-positive cells delayed tumorigenesis and attenuated age-

107 y this, we inoculated 30 nude rats with HER2-positive cells derived from a brain metastasis of a brea

108 ate a reliable transcriptional outcome in AR positive cells, despite their low genome-wide abundance.

109 The CD20-positive cells did not display the typical B cell morpho

110 This suggests that the majority of Lgr5-positive cell divisions serve to renew epithelial cells

111 t trigger ALT, as FANCD2 depleted telomerase positive cells do not acquire ALT-like phenotypes.

112 was stronger for microorganisms with a Gram-positive cell envelope compared to those with a Gram-neg

113 rminant of functional properties of the Gram-positive cell envelope.

114 High-risk human papillomavirus 31 (HPV31)-positive cells exhibit constitutive activation of the AT

115 Accordingly, EMT-positive cells exhibited a higher persistance/survival i

116 note, large microtumors of hormone receptor-positive cells exhibited an aggressive phenotype charact

117 elomeric lagging strands, whereas telomerase positive cells exhibited similar elongation between lead

118 Hox-positive cells express PDGFRalpha and CD51, are marked b

119 the arcopallium (RA), about half of the BrdU-positive cells expressed BDNF across sexes and ages.

120 Less than 10% of the BDNF-positive cells expressed BrdU, but this percentage was g

121 an average efficiency of 55% into C-peptide-positive cells, expressing markers of mature beta-cells,

122 numbers of antigen-specific CD8(+) IFN-gamma-positive cells following injection into BALB/c mice.

123 ates (ADC) target cytotoxic drugs to antigen-positive cells for treating cancer.

124 However, the purity of the UEA-I-positive cell fraction has not been fully evaluated.

125 by capturing green fluorescent protein (GFP)-positive cells from blood.

126 tinib resulted in a rapid reduction of T315I-positive cells from bone marrow.

127 ocess analysis for the generation of insulin-positive cells from hESCs.

128 erentiation markers, expansion of keratin-15-positive cells from localization within the bulge to the

129 in probe incubation, ready discrimination of positive cells from negative cells is achieved.

130 levels of CCL20 and to attract CD4/IL17/CCR6-positive cells, generated in vitro, in a CCL20/CCR6-depe

131 nd by 4 days colonies of Oct4-positive, Sox2-positive cells had formed.

132 d insulin independence, all examined insulin-positive cells had lost expression of the end-differenti

133 Additionally, the IL-33-positive cells had low expression of PCNA.

134 d large populations of EMT-derived, vimentin-positive cells having spindle-like morphology.

135 ion for why this microRNA is targeted in HPV-positive cells.IMPORTANCE We describe here for the first

136 r tartrate-resistant acid phosphatase (TRAP)-positive cells in alveolar bone.

137 ced the number of CD31(+) vessels and VEGF-A-positive cells in fibrotic peritoneum.

138 ot alter the numerical density of surfactant positive cells in lung tissue.

139 The percentage of CXCR3 expression in CD3-positive cells in PBMCs was inversely correlated with se

140 also found increased numbers of Ki67 (MKI67)-positive cells in regions of enhanced ILK expression in

141 diated dUTP-biotin nick end labeling (TUNEL)-positive cells in several regions of the brain.

142 In contrast with LacZ-positive cells in the afferent arterioles, LacZ-positive

143 increased numbers of CD25-, FoxP3-, and CD4-positive cells in the CNS.

144 Four cases (12%) showed a few melan-A-positive cells in the dermis, which was insufficient for

145 d to a 45% loss of smooth muscle alpha-actin positive cells in the eye drainage structure of 10- to 1

146 t the large majority (80%) of the calretinin positive cells in the ganglion cell layer are ganglion c

147 itive cells in the afferent arterioles, LacZ-positive cells in the glomerular tuft did not express re

148 iments yielded no glycinergic or cholinergic positive cells in the IC, and descending projections to

149 n addition, a small proportion of calretinin-positive cells in the inner nuclear layer and in the gan

150 endocrine cells are generated, such as POMC-positive cells in the intermediate lobe.

151 is active in a distinct population of Notch-positive cells in the intestinal epithelium.

152 ments demonstrated a specific region of Tbx1-positive cells in the labial cervical loop (LaCL, stem c

153 the DDC-stimulated number of cytokeratin-19-positive cells in the liver of wild-type animals was sli

154 on, there were fewer 5-bromo-2'-deoxyuridine-positive cells in the LK population in Chd7(f/f)Mx1-CreC

155 e in proliferative alpha-smooth muscle actin-positive cells in the lungs of ITSN-deficient mice, tran

156 timal hyperplasia (alpha-smooth muscle actin positive cells in the neointima) and endothelial activat

157 Paternal UBE3A-positive cells in the SCN show partial colocalization wi

158 There were fewer TRAP-positive cells in the SRP/SA group than in the NT group

159 wever, there were significantly more IL-17RA-positive cells in the submucosa and epithelium in childr

160 The fraction of glucagon-positive cells in the total endocrine mass was increased

161 onfirmed the presence of alphaFAP- and FSP-1-positive cells in the tumor stroma, and their presence c

162 gnosis (based on CD20-positive and cyclin D1-positive cells in tissue biopsy specimens), no upper lim

163 in tissue lysates and the number of SIV RNA-positive cells in tissue sections.

164 erial and assessed the frequency of BCR-ABL1-positive cells in various hematopoietic subpopulations;

165 Human monocytes give rise to langerin-positive cells in vitro, suggesting a potential precurso

166 human intestinal CD3-negative, IL7-receptor-positive cells, in a dose-dependent manner.

167 cyclooxygenase-2, and the number of vimentin-positive cells increased.

168 In JAK2V617F-positive cells, increased ROS levels results from aberra

169 CK5-positive cells induced by 3-ketosteroids lacked ERalpha

170 s tartrate-resistant acid phosphatase (TRAP)-positive cell induction than M0 or M2 macrophage transfe

171 and cystic index and markedly reducing CD45-positive cell infiltration.

172 our samples revealed that the number of ODZ1-positive cells inversely correlated with overall and pro

173 A heterogeneous population of CNA-positive cells is present in the bone marrow of non-meta

174 rate that disruption of aggregation in ErbB2-positive cells is sufficient to induce anoikis and that

175 esults in elevated AKT signaling in mutation-positive cells, is responsible for the mosaic overgrowth

176 mutant HCMV and increased the number of IE2-positive cells, it could not compensate for IE1 in full

177 scripts temporarily induces apoptosis in HPV-positive cells, it does not eliminate viral DNA within t

178 ed hosts were comprised of both K18- and K14-positive cells (K14/18) while those tumors arising in ir

179 Cell proliferation of the fusion-positive cell line was inhibited by knocking down the ex

180 ted HLA-B*51:08 subtype expressed in a Hap10-positive cell line was isolated, characterized by mass s

181 ased HIV-1 infection of a CD4-negative, CCR5-positive cell line.

182 and cytotoxicity studies in the alphavbeta6 positive cells line A375Pbeta6.

183 SR1 (Y537S) mutations in MCF7 and SUM44 ESR1-positive cell lines after acquisition of resistance to l

184 telomerase RNA) gene knockouts in telomerase positive cell lines that resulted in long-term surviving

185 BiFab-BCMA lysed target BCMA-positive cell lines up to 20-fold more potently than a C

186 ell cycle arrest and apoptosis of human EVI1-positive cell lines, and prolonged survival in both orth

187 th of TNBC cells to a greater extent than ER-positive cell lines.

188 ts growth, and reduces tumorigenicity in HPV-positive cell lines.

189 e dependent on TMEM16A for survival than HPV-positive cell lines.

190 er, IL-33 improved the survival of JAK2V617F-positive cell lines.

191 During this process, agonist-selected double-positive cells lose CD4/8 coreceptor expression and masq

192 significant proportion of hepatocyte marker-positive cells maintaining a less well-differentiated ph

193 ize, irregular shape, inflammation with CD68-positive cells, marked fibrosis, and hemosiderosis.

194 eratin 12 [cK12]-negative and mucin 1 [MUC1]-positive cells; mixed epithelium: cK12-positive and MUC1

195 00 +/- 81 vs. 2630 +/- 390 bromodeoxyuridine-positive cells mm(-2) in controls, P < 0.0001).

196 s were supported by an observation of an EBV-positive cell model in which silencing of endogenous EBV

197 acterized by abundant apoptotic but few BAFF-positive cells (mostly B cells); and (2) type B, the mai

198 Hnf4a, leading to an increase of both GLP-1-positive cell number and basal and stimulated GLP-1 plas

199 s reduced caspase-3/7 activity and annexin V-positive cell number upon induction of apoptosis.

200 d tartrate-resistant acid phosphatase (TRAP)-positive cell number, and enhanced osteoclast activity i

201 BMP-2-induced 5-bromo-2'-deoxyuridine (BrdU)-positive cell numbers at the injected site on day 7 and

202 ed basal Lgr5-positive and luminal keratin-8-positive cells of the adult mouse mammary gland evokes c

203 body clock require clock function in the PDF-positive cells of the fly brain.

204 eotidyl transferase dUTP nick end labeling])-positive cells) of NPIs compared with AIs.

205 assays revealed a strong dependence of HER2-positive cells on MUCL1 for cell proliferation.

206 h was suppressed with S3I-201 (percentage of positive cells per field: 31.7% +/- 3.4 vs 3.8% +/- 1.7;

207 histochemical staining and the number of HCV-positive cells per focus was assessed to determine focus

208 ignificant differences in the number of VEGF-positive cells per square millimeter (P = 0.07) and VEGF

209 iency was estimated as the percentage of GFP-positive cells per total cells in a microscopic field.

210 165.15 and 230.4 cyclin-dependent kinase 47-positive cells per x20 field, respectively, at day 7; P

211 esistance in mice with Mig-6 ablation in Pgr-positive cells (Pgr(cre/+)Mig-6(f/f)).

212 In HPV-positive cells, phosphorylation of p38 and MK2 proteins

213 macologic inhibition of HDAC elevated a SOX9-positive cell population from SOX2-positive cells, where

214 nished oncogenic activity, reduced the ALDH1-positive cell population, and increased reactive oxygen

215 hage and decreased alpha-smooth muscle actin-positive cell population, fibrous cap thinning, and decr

216 signaling inhibits the slow cycling JARID1B-positive cell population, which is critical for long-ter

217 Lfng also resulted in accumulation of Aldh1-positive cell population.

218 ineralocorticoids effectively expand the CK5-positive cell population.

219 lerated cell proliferation or expanded Aldh1-positive cell population.

220 shrinkage and efficient elimination of CD44-positive cell populations following irradiation.

221 f FITC-labeled Ki-67 antibody TuBB-9 in EGFR-positive cells pre-loaded with the photoactive dye BPD.

222 late cell signaling or migration of EGFRvIII-positive cells, probably because cell signaling was alre

223 d at synapses within human leukocyte antigen-positive cell processes and lysosomes, suggesting engulf

224 , we show that knockdown of KDM3A reduces ER-positive cell proliferation and demonstrate that KDM3A i

225 -negative cells significantly attenuates A23-positive cell proliferation and invasion.

226 repair, CHK1 and Wee1, are suppressed in HPV-positive cells, providing an explanation for why this mi

227 tic drop in the number of Pax7- and myogenin-positive cells relative to WT muscles, suggesting that d

228 Although the number of SIV-DNA-positive cells remained unchanged after CD8 depletion an

229 opulations of OPN5-positive and cryptochrome-positive cells reside within the caudal diencephalon.

230 mportant subset of alphabeta T cell receptor-positive cells residing in mouse kidney.

231 kat lymphoma cells (CD20-positive and TAG-72-positive cells, respectively).

232 ith short hairpin RNA in differentiating HPV-positive cells resulted in diminished levels of viral ge

233 Snf5 ablation in nestin-positive cells resulted in early lethality that could no

234 In vivo passaging of Aldefluor-positive cells resulted in the growth of larger, more ag

235 neurons and lead to decreased number of Drd1-positive cells retaining BrdU in postnatal day (P) 0 Rar

236 th conditional activation of KRAS in the PGR positive cells reveal an increase of SIRT1 expression in

237 We also showed that Aldefluor-positive cells revealed significantly higher expression

238 nization of calbindin-negative and calbindin-positive cells showed marked differences in entorhinal s

239 ingle-positive and CD3(+)CD4(+)CD8(+) double-positive cells showed severe restriction of repertoire d

240 NA sequencing comparison of VEC-null and VEC-positive cells suggested a more general role of VEC in a

241 mpanied by a decrease in the number of c-Fos-positive cells surrounded by PNNs.

242 of pancreatic islets and the area of insulin-positive cells tended to be higher in BEZ-treated mice.

243 ut also to maintain higher cell uptake (>90% positive cells) than the most densely PEGylated particle

244 from an ER/PR-negative cell or from an ER/PR-positive cell that later lost ER/PR.

245 Thus, p16(Ink4a)-positive cells that accumulate during adulthood negative

246 oplastic growth of smooth muscle-alpha-actin-positive cells that destroy lung parenchyma and by the f

247 positive cells and counted the percentage of positive cells that migrated to each region from at leas

248 ortas showed fewer alpha-smooth muscle actin positive cells that were not coimmunolocalized with mous

249 filtration of Ly-6G-, CD11b-, Iba-1- and CD3-positive cells, the production of interleukin-1beta by C

250 e proliferation and invasion of adjacent A23-positive cells through the production of LGI4 (Leucine-r

251 eurons, the parvalbumin and the somatostatin positive cells, tightly control both up-to-down and down

252 lass of inhibitory interneurons-somatostatin-positive cells-to the generation of slow waves during NR

253 e to loss of Atoh1 function than other Atoh1-positive cell types and that heterozygous mice actually

254 get for molecular imaging of different CXCR4-positive cell types in cardiovascular diseases such as a

255 lammatory cytokine secretion from other ROCK-positive cell types, corroborating the selective in vivo

256 BNA1 ChIP-Seq patterns in four different EBV-positive cell types, including Burkitt lymphoma (BL) cel

257 lecular analysis of fractionated VE-cadherin-positive cells uncovered copy-number alterations and mut

258 However, not all Ngn1-positive cells undergo delamination, nor has the mechani

259 urification of KSP (kidney specific protein)-positive cells using anti-KSP antibody.

260 e examined the effect of hyperthermia on HPV-positive cells using cervical cancer cell lines infected

261 d cell lines (LCLs), the proportion of ITPR1-positive cells using immunofluorescence was significantl

262 rison of cytosolic fluorescence intensity in positive cells versus background in negative cells yield

263 bears striking resemblance to a related Gram-positive cell-wall remodeling strategy that also promote

264 ition from CD4/CD8 double-negative to double-positive cells was blocked, and lck-cre(+) double-positi

265 Numerical density of surfactant protein positive cells was determined by immunohistochemistry.

266 alysis showed that the percentage of the Edu positive cells was increased in the treated cells.

267 lls, but the infection of CD4-positive, CCR5-positive cells was inhibited.

268 alue <0.0001), and positivity (percentage of positive cells) was significantly greater in cirrhosis c

269 ing these ALT cells with parental telomerase positive cells, we observed that ALT cells possess exces

270 by using a model of in vivo ablation of PAX7 positive cells, we show that this radiosensitive skeleta

271 tral fovea and found evidence that rod opsin positive cells were absent and violet-sensitive cone and

272 Across both brain regions, more BDNF-positive cells were detected in males compared with fema

273 Moesin-positive cells were embedded within the fibrotic areas b

274 Although Pcdh19-positive cells were evenly distributed in layer V of wil

275 ctive within glial fibrillary acidic protein-positive cells were generated and this allowed for selec

276 In all experiments, HCV-positive cells were identified by immunohistochemical st

277 IL-1alpha-positive cells were identified in the epithelium in huma

278 with NC/02 or MN/99 plus TX/96, H1/H3 double-positive cells were identified.

279 ged with angiotensin II, PDGF receptor alpha-positive cells were increased in the skin and heart.

280 ive cells was blocked, and lck-cre(+) double-positive cells were more prone to apoptosis and showed h

281 atic increase in alpha-SMA(+), EP4(+) double-positive cells were observed in EP4cKO(S100a4) suggestin

282 ng hypertrophic-like scars, whereas few CD26-positive cells were present in the regenerated gingival

283 Also, US28-positive cells were present within arterial neointima.

284 ensitive cone and green-sensitive cone opsin positive cells were present.

285 abeta transgene, normal levels of CD4 single-positive cells were produced.

286 orescent channel intensity and number of GFP-positive cells were recorded.

287 ng studies confirmed that the alpha-syn::GFP-positive cells were retinal ganglion cells containing al

288 egative, stem cell antigen-1-positive, c-Kit-positive) cells were quantified and proliferation assess

289 pen probabilities were reduced by 80% in GFP-positive cells; Western blots also showed significant re

290 ed a SOX9-positive cell population from SOX2-positive cells, whereas ectopic expression of SOX2 inhib

291 MSPCs are observed as leptin receptor (LepR)-positive cells, whereas osteoblasts can be classified as

292 eased the number of MUC5AC(+) and involucrin-positive cells, which were blocked with the DP2-selectiv

293 nding protein 1 foci, after incubating SSTR2-positive cells with (177)Lu-diethylene triamine pentaace

294 We sorted TERT-positive cells with cytogenetic changes and followed the

295 a significant correlation of clustered CD147 positive cells with largest basal diameter (p = 0.039),

296 ystem was shown to be highly cytotoxic to FR-positive cells with no activity against FR-negative cell

297 eriostin and transforming growth factor beta-positive cells within Asm bundles, in addition to lower

298 on therapy (ADT), destroy the bulk of the AR-positive cells within the tumor, eradicating this popula

299 Cytokeratin 19-, A6- and alpha-fetoprotein-positive cells within tumors were hepatocyte derived.

300 tive immunohistochemical scoring (percentage positive cells x intensity) were calculated.