ライフサイエンスコーパス: positive end-expiratory pressure

1 for each individual lung (optimum-selective positive end-expiratory pressure).

2 rotocol (at clinical, 5 cm H2O, or 15 cm H2O positive end-expiratory pressure).

3 au and driving pressures, and high levels of positive end-expiratory pressure.

4 emodynamics were measured at 5 and 15 cm H2O positive end-expiratory pressure.

5 daily minimum fraction of inspired oxygen or positive end-expiratory pressure.

6 change, lung recruitability, and response to positive end-expiratory pressure.

7 t with the use of low tidal volumes and high positive end-expiratory pressure.

8 ances may vary unpredictably with changes in positive end-expiratory pressure.

9 t ventilation (12 +/- 3 cm H2O), at clinical positive end-expiratory pressure.

10 er the reliability of this test depends upon positive end-expiratory pressure.

11 when ceilings exist on the level of desired positive end-expiratory pressure.

12 trolled trials with differential response to positive end-expiratory pressure.

13 xpiratory pressure and the optimum-selective positive end-expiratory pressure.

14 assessed at clinical, 5 cm H2O, or 15 cm H2O positive end-expiratory pressure.

15 rate adjusted to normocapnia) at low (n = 2, positive end-expiratory pressure = 0) and high (n = 2, p

16 ) with high-strain (VT = 18.2 +/- 6.5 mL/kg, positive end-expiratory pressure = 0), high-strain plus

17 lipopolysaccharide (VT = 18.4 +/- 4.2 mL/kg, positive end-expiratory pressure = 0), or low-strain plu

18 ve end-expiratory transpulmonary pressure at positive end-expiratory pressure 1 cm H2O (mean -3.5 +/-

19 t also results in a significant drop in auto-positive end-expiratory pressure (1.2 [0.6-4] vs 10 [5-1

20 a-abdominal hypertension negates most of the positive end-expiratory pressure 10 benefit in reversing

21 5 +/- 0.4 cm H2O) into the positive range at positive end-expiratory pressure 10 cm H2O (mean 0.58 +/

22 al hypertension were simultaneously applied, positive end-expiratory pressure 10 failed to improve ti

23 sitive end-expiratory pressure of 10 cm H2O (positive end-expiratory pressure 10) increased mean func

24 %] in controls; p < 0.0001) and greater auto-positive end-expiratory pressure (10 [5-12.5] vs 0.7 [0.

25 e) and were mechanically ventilated for 4 h (positive end-expiratory pressure, 10 cm H2O; plateau pre

26 At clinical positive end-expiratory pressure (11+/-3 cm H2O), patien

27 in which a recruitment maneuver followed by positive end-expiratory pressure (110 cm H2O) had no eff

28 to either a recruitment maneuver (RM) group (positive end-expiratory pressure 15 cm H2O, peak inspira

29 At higher positive end-expiratory pressure (15 cm H2O [interquarti

30 mm Hg; p = 0.045) despite of using a higher positive end-expiratory pressure (17.4 +/- 0.7 vs 9.5 +/

31 lipopolysaccharide (VT = 8.1 +/- 0.2 mL/kg, positive end-expiratory pressure = 17 +/- 3 cm H2O).

32 n (peak inspiratory pressure 22 cm H(2)O and positive end-expiratory pressure 2 cm H(2)O for 2 hrs);

33 Duty cycle of 0.75 resulted in intrinsic positive end-expiratory pressure (2.1 +/- 1.1 cm H2O [p

34 eceived high tidal volume (30-32 mL/kg) with positive end-expiratory pressure (3 cm H2O) and sustaine

35 tive tidal volume, 2) appropriate setting of positive end-expiratory pressure, 3) oxygen weaning, and

36 s per day (pressure support level, 8 cm H2O; positive end-expiratory pressure, 4 cm H2O; FiO2, 50%) (

37 essure of 5 and pressure support of 10 above positive end-expiratory pressure 5 cm H2O, as well as 5

38 lation (control group, tidal volume 9 mL/kg, positive end-expiratory pressure 5 cm H2O, n = 15), high

39 Two levels of positive end-expiratory pressure (5 and 15 cm H2O) were

40 oup received low tidal volume (7 mL/kg) with positive end-expiratory pressure (5 cm H2O) and regular

41 esponding intra-abdominal pressure: baseline positive end-expiratory pressure (= 5 cm H2O), moderate

42 sed cardiac index to a larger extent than at positive end-expiratory pressure = 5 cm H2O (19% [interq

43 At positive end-expiratory pressure = 5 cm H2O, 29% of pati

44 At positive end-expiratory pressure = 5 cm H2O, we measured

45 iratory pressure support level, 5-15 cm H2O; positive end-expiratory pressure, 5-10 cm H2O; fraction

46 y distress syndrome patients required higher positive end-expiratory pressure (7 vs 6 vs 10 vs 15 cm

47 tive end-expiratory pressure (i.e., clinical positive end-expiratory pressure = 7 +/- 2 cm H2O) with

48 nd-expiratory pressure = 0) and high (n = 2, positive end-expiratory pressure adjusted to achieve a p

49 At higher positive end-expiratory pressure, an end-expiratory occl

50 after a recruitment maneuver and decremental positive end-expiratory pressure and corresponded to a p

51 We hypothesized that higher positive end-expiratory pressure and enhanced spontaneou

52 Alveolar recruitment was accomplished using positive end-expiratory pressure and exogenous surfactan

53 y was to assess the value of adding baseline positive end-expiratory pressure and Fio(2) to PaO(2)/Fi

54 d pressure with 82 centers (68.9%) employing positive end-expiratory pressure and FIO2 to optimize ox

55 f injurious mechanical ventilation using low positive end-expiratory pressure and high inspiratory pr

56 f injurious mechanical ventilation using low positive end-expiratory pressure and high inspiratory pr

57 ergoing pressure support ventilation, higher positive end-expiratory pressure and lower support level

58 setting of higher (but not lower) levels of positive end-expiratory pressure and reduced central ven

59 Lung recruitment by higher positive end-expiratory pressure and surfactant administ

60 Application of positive end-expiratory pressure and surfactant reduced

61 ch): ventilation applying the optimum global positive end-expiratory pressure and the optimum-selecti

62 rdistension with nonaerated regions at lower positive end-expiratory pressures and with hyperaerated

63 Increased positive-end expiratory pressure and reduced time at low

64 pliance was defined globally (optimum global positive end-expiratory pressure) and for each individua

65 ameters (tidal volume per ideal body weight, positive end-expiratory pressure, and airway pressure),

66 Po2 before ECLS, higher oxygen index, higher positive end-expiratory pressure, and development of ren

67 ry mechanics included plateau pressure, auto-positive end-expiratory pressure, and flow-limited volum

68 ow Coma Scale, spontaneous respiratory rate, positive end-expiratory pressure, and systolic blood pre

69 The level of positive end-expiratory pressure applied may greatly aff

70 r size of normal mice in respiration without positive end expiratory pressure as 58 +/- 14 (mean +/-

71 tory pressure set by the clinicians, at zero positive end-expiratory pressure, at best positive end-e

72 determine which bedside method would provide positive end-expiratory pressure better related to lung

73 his study evaluated two methods of titrating positive end-expiratory pressure; both trials were done

74 In contrast, independent of positive end-expiratory pressure but dependent on lung c

75 ra-abdominal pressure compared with 5 cm H2O positive end-expiratory pressure) but led to a greater d

76 Commonly used positive end-expiratory pressure by clinicians is inadeq

77 and Tierney reported that high Vt with zero positive end-expiratory pressure caused overwhelming lun

78 ed lung injury, high peak pressure (and zero positive end-expiratory pressure) causes respiratory swi

79 stroke volume/cardiac index after a fluid or positive end-expiratory pressure challenge.

80 pressure optimization strategies recommended positive end-expiratory pressure changes in opposite dir

81 pressure optimization strategies recommended positive end-expiratory pressure changes in opposite dir

82 lung recruitability and edema than at higher positive end-expiratory pressure clinically set.

83 t the end of the 15-minute optimum-selective positive end-expiratory pressure, compared with the opti

84 To test whether positive end-expiratory pressure consistent with an open

85 In this experimental setting, positive end-expiratory pressure consistent with the ope

86 th a duty cycle (TITTOT) of 0.33 and without positive end-expiratory pressure (control); 2) as in the

87 The positive end-expiratory pressure corresponding to maximu

88 Positive end-expiratory pressure decreased the respirato

89 ratory mechanics (peak inspiratory pressure, positive end-expiratory pressure, DeltaP [PIP minus PEEP

90 At 24 hours, peak inspiratory pressure, positive end-expiratory pressure, DeltaP were higher, an

91 Peak inspiratory pressure, positive end-expiratory pressure, DeltaP, tidal volume,

92 fter controlling for PaO(2)/Fio(2), baseline positive end-expiratory pressure did not predict mortali

93 Removal of positive end-expiratory pressure did not result in abrup

94 +/-114 mL, corresponding to 19+/-1 cm H2O of positive end-expiratory pressure) did (from 314+/-55 to

95 ay pressures alone (plateau airway pressure--positive end-expiratory pressure) did not equate to thos

96 rtile range, 13-15]), the plateau pressure - positive end-expiratory pressure difference did not chan

97 lation strategy of low tidal volume and high positive end-expiratory pressure, does not reduce, and m

98 or detecting preload dependence whatever the positive end-expiratory pressure during acute respirator

99 intra-abdominal hypertension and changes in positive end-expiratory pressure during different models

100 Positive end-expiratory pressure exerts its effects keep

101 pressure-expiratory-CT and optimum-selective positive end-expiratory pressure-expiratory-CT (3.7 +/-

102 t/dependent ratio between the optimum global positive end-expiratory pressure-expiratory-CT and optim

103 spontaneous breathing trials, used a common positive end-expiratory pressure-FI(O(2)) chart, sedatio

104 dominal pressure was an effective adjunct to positive end-expiratory pressure for recruiting atelecta

105 al pressure/volume curves show that adequate positive end-expiratory pressure for severe acute lung i

106 tilatory maneuvers (increase and decrease in positive end-expiratory pressure from 0 to 15 and back t

107 High positive end-expiratory pressure further increased end-e

108 exclusion gave Frecruited values of zero at positive end-expiratory pressure greater than or equal t

109 iratory system compliance (</=40 mL/cm H2O), positive end-expiratory pressure (>/=10 cm H2O), and cor

110 Tidal volume and positive end-expiratory pressure had no impact on mortal

111 d-expiratory pressure (= 5 cm H2O), moderate positive end-expiratory pressure (= half intra-abdominal

112 The driving pressure (plateau pressure minus positive end-expiratory pressure) has been suggested as

113 per airway obstruction, higher preextubation positive end-expiratory pressure, higher postextubation

114 pressure support ventilation level, a lower positive end-expiratory pressure (i.e., clinical positiv

115 The two methods of evaluating positive end-expiratory pressure identified similar opti

116 nd after a recruitment maneuver, and at best positive end-expiratory pressure identified through a be

117 ro positive end-expiratory pressure, at best positive end-expiratory pressure identified through esop

118 Neither moderate nor high positive end-expiratory pressure improved PaO2 (p = .7).

119 application of at least a minimal amount of positive end-expiratory pressure in acute lung injury (1

120 ung mechanical heterogeneity with increasing positive end-expiratory pressure in an animal model of a

121 reanalysis of randomized clinical trials of positive end-expiratory pressure in ARDS that support th

122 The approach to applying positive end-expiratory pressure in morbidly obese patie

123 ystem elastances increased with increases in positive end-expiratory pressure in patients with positi

124 ory system elastances grew with increases in positive end-expiratory pressure in patients with positi

125 n predominates over expiratory flow bias and positive end-expiratory pressure in the prevention of gr

126 minal pressure may be a potential adjunct to positive end-expiratory pressure in the recruitment of d

127 trical impedance tomography can help titrate positive end-expiratory pressure in these regions, there

128 ular mechanics compared with higher or lower positive end-expiratory pressures in experimental acute

129 verdistension increases both at high and low positive end-expiratory pressures in nondependent region

130 space, increases in respiratory rate, higher positive end-expiratory pressures in patients who recrui

131 The use of higher positive end-expiratory pressures in the open lung appro

132 latory occlusions performed at two levels of positive end-expiratory pressure, in view of widening th

133 mm Hg [14-17] vs 15 mm Hg [13-18] before the positive end-expiratory pressure increase).

134 level of intra-abdominal pressure, moderate positive end-expiratory pressure increased end-expirator

135 trical impedance tomography-derived maps and positive end-expiratory pressure indicate that, expected

136 elpful in explaining how different levels of positive end-expiratory pressure influence recruitment a

137 ressure-inspiratory-CT and optimum-selective positive end-expiratory pressure-inspiratory-CT (2.8 +/-

138 t/dependent ratio between the optimum global positive end-expiratory pressure-inspiratory-CT and opti

139 nal pressure in cm H2O + 5 cm H2O), and high positive end-expiratory pressure (= intra-abdominal pres

140 lation with the use of low tidal volumes and positive end-expiratory pressure is considered best prac

141 etween the plateau pressure and the level of positive end-expiratory pressure is not known.

142 preceded by lung recruitment identified the positive end-expiratory pressure level (17.4 +/- 2.1 cm

143 The positive end-expiratory pressure level resulting in high

144 uted tomography scans were performed at each positive end-expiratory pressure level to quantify tidal

145 At any positive end-expiratory pressure level, electrical imped

146 ry system elastances were calculated at each positive end-expiratory pressure level.

147 piratory pressure identified similar optimal positive end-expiratory pressure levels (20.7 +/- 4.0 vs

148 increasing intra-abdominal pressure at both positive end-expiratory pressure levels (p </= 0.0001) w

149 Lower positive end-expiratory pressure levels (until day 7) an

150 alveoli over short-time scales in all tested positive end-expiratory pressure levels and despite stab

151 Lower positive end-expiratory pressure levels and significantl

152 However, higher positive end-expiratory pressure levels during the first

153 In multivariate analysis, higher positive end-expiratory pressure levels during the first

154 ion method was the only one which gave lower positive end-expiratory pressure levels in mild and mode

155 Application of higher positive end-expiratory pressure levels increased Vt%dep

156 Even for positive end-expiratory pressure levels minimizing globa

157 Positive end-expiratory pressure levels must be high eno

158 The ideal positive end-expiratory pressure levels recommended by t

159 hereas the oxygenation-based method provided positive end-expiratory pressure levels related with lun

160 Positive end-expiratory pressure levels selected by the

161 unrelated with lung recruitability, whereas positive end-expiratory pressure levels selected by the

162 The positive end-expiratory pressure levels set by the clini

163 aintain blood pressure (and higher FIO2) and positive end-expiratory pressure levels to maintain oxyg

164 ics or absolute esophageal pressures provide positive end-expiratory pressure levels unrelated to lun

165 Thereafter, three positive end-expiratory pressure levels were applied in

166 Positive end-expiratory pressure levels were higher in t

167 In patients who were ventilated at two positive end-expiratory pressure levels, chest wall and

168 ventilation with higher tidal volumes, lower positive end-expiratory pressure levels, or both, than p

169 ntilation, high FiO2 concentration, and high positive end-expiratory pressure levels.

170 , we do not recommend routine application of positive end-expiratory pressure matched to intra-abdomi

171 a pig model of intra-abdominal hypertension, positive end-expiratory pressure matched to intra-abdomi

172 stress syndrome severity at standardized low positive end-expiratory pressure may improve the associa

173 = 1.9] in 2010), and an increase in applied positive end-expiratory pressure (mean 4.2 cm H2O [SD =

174 iology Score II, maximum blood lactate, mean positive end-expiratory pressure, mean cumulative fluid

175 effect estimates) and conditional for higher positive end-expiratory pressure (moderate confidence in

176 with a body mass index greater than 30 kg/m, positive end-expiratory pressure more than 10 cm H2O, Pa

177 al volume group, tidal volume 25 mL/kg, zero positive end-expiratory pressure, n = 14), or high tidal

178 the same tidal volume as control but neither positive end-expiratory pressure nor sustained inflation

179 idence interval: 0.88; 0.78-0.99; p = .049), positive end-expiratory pressure (odds ratio per 5-cm H2

180 ibiotics for possible VAP with daily minimum positive end-expiratory pressure of </=5 cm H2O and frac

181 Animals were ventilated with positive end-expiratory pressure of 0, 5, 10, 15, 20, an

182 ra-abdominal pressures of 0 and 20 cm H2O at positive end-expiratory pressure of 1 and 10 cm H2O, und

183 minal hypertension increased both DP(AW) (at positive end-expiratory pressure of 1 cm H2O, p < 0.0001

184 ure of 10 cm H2O, p = 0.0091) and DP(TP) (at positive end-expiratory pressure of 1 cm H2O, p = 0.0510

185 Positive end-expiratory pressure of 10 cm H2O (positive

186 ory pressure of 1 cm H2O, p < 0.0001; and at positive end-expiratory pressure of 10 cm H2O, p = 0.009

187 ory pressure of 1 cm H2O, p = 0.0510; and at positive end-expiratory pressure of 10 cm H2O, p = 0.033

188 % received mechanical ventilation, with mean positive end-expiratory pressure of 14 cm H2O at the ons

189 In both mechanical ventilation groups, positive end-expiratory pressure of 2 cm H2O was applied

190 ssure (control); 2) as in the control group, positive end-expiratory pressure of 5 cm H2O and TITTOT

191 n or equal to 6 were placed on FIO2 of 0.50, positive end-expiratory pressure of 5 cm H2O, and pressu

192 stantially after mechanical ventilation with positive end-expiratory pressure of 5-10 cm H(2)O, and t

193 n 150 mm Hg, with an FiO2 of at least 0.6, a positive end-expiratory pressure of at least 5 cm of wat

194 using phase-contrast synchrotron imaging, at positive end-expiratory pressures of 12, 9, 6, 3, and 0

195 In a previous experimental study, positive end-expiratory pressures of up to 15 cm H2O did

196 y was to evaluate effects of duty cycles and positive end-expiratory pressure on mucus clearance in p

197 end-expiratory pressure with optimum global positive end-expiratory pressure on regional collapse an

198 gs (i.e. plateau pressure, tidal volume, and positive end-expiratory pressure) on ICU mortality using

199 The two positive end-expiratory pressure optimization strategies

200 evaluate the agreement between two published positive end-expiratory pressure optimization strategies

201 evaluate the agreement between two published positive end-expiratory pressure optimization strategies

202 The two positive end-expiratory pressure optimization strategies

203 41, 0.50, 0.60, and 0.75) with no associated positive end-expiratory pressure or 5 cm H2O of positive

204 ined by sustained increases in daily minimum positive end-expiratory pressure or FIO2 after either 2

205 tress syndrome include PaO(2)/Fio(2) but not positive end-expiratory pressure or Fio2.

206 remained virtually unaffected by changes in positive end-expiratory pressure or intra-abdominal pres

207 creases in dependent regions with decreasing positive end-expiratory pressure (p < 0.001) and suggest

208 d with lower tidal volume (P < 0.01), higher positive end-expiratory pressure (P < 0.01), and higher

209 %nondep/Vt%dep ratio, as compared with lower positive end-expiratory pressure (p < 0.01).

210 ng distensibility) decreased with increasing positive end-expiratory pressure (p = 0.001) independent

211 Both at 5 and 15 cm H2O of positive end-expiratory pressure PaO2/FIO2, respiratory

212 At high positive end-expiratory pressure, passive leg raising in

213 l volume of 6 mL/kg and progressively higher positive end-expiratory pressure (PEEP) (5, 10, 16, 20,

214 ecreased to 100-150 mm Hg on an FIO2 of 1.0, positive end-expiratory pressure (PEEP) 5 cm H2O, and ti

215 on of inspired oxygen (FiO(2)) by >/= 15% or positive end-expiratory pressure (PEEP) by >/= 2.5 cm H(

216 ntinuous positive airway pressure (CPAP) and positive end-expiratory pressure (PEEP) for the manageme

217 In this study, we examined whether (1) positive end-expiratory pressure (PEEP) has a protective

218 Positive end-expiratory pressure (PEEP) has been used du

219 ies using low tidal volume or high levels of positive end-expiratory pressure (PEEP) improve outcomes

220 application of at least a minimal amount of positive end-expiratory pressure (PEEP) in ARDS (1B); hi

221 mes in both cohorts and with the response to positive end-expiratory pressure (PEEP) in the ALVEOLI c

222 RATIONALE: Optimal positive end-expiratory pressure (PEEP) is unknown in pa

223 ction of inspired oxygen (Fio2) of 1.0 and a positive end-expiratory pressure (PEEP) of 20 cm of wate

224 Second, we evaluated the contribution of positive end-expiratory pressure (PEEP) on the relations

225 The effects of recruitment maneuvers and positive end-expiratory pressure (PEEP) titration on cli

226 dely accepted cutoff values of PaO2/FIO2 and positive end-expiratory pressure (PEEP) would identify s

227 We hypothesized that in patients on higher positive end-expiratory pressure (PEEP), sedatives, opio

228 tion with small tidal volumes and the use of positive end-expiratory pressure (PEEP); however, the op

229 ed, also to a variable extent, by increasing positive end-expiratory pressure (PEEP; 2-6 cmH(2)O).

230 datory ventilation group, multiple levels of positive end-expiratory pressure (PEEP; 5, 10, 16, 20, a

231 lt rats were ventilated with high (18 ml/kg, positive end-expiratory pressure [PEEP] 0) or low Vt (6

232 sed as mL/kg predicted bodyweight [PBW], and positive end-expiratory pressure [PEEP] expressed as cm

233 lues, supine and prone positions and various positive end-expiratory pressures (PEEPs) and tidal volu

234 ssures, lower tidal volumes (VT), and higher positive end-expiratory pressures (PEEPs) can improve su

235 al death based on quantiles of tidal volume, positive end-expiratory pressure, plateau pressure, and

236 Ventilation at lowest elastance positive end-expiratory pressure preceded by a recruitme

237 ion, including tidal volume limitation, high positive end-expiratory pressure, pressure-controlled in

238 Combining sitting position and applied positive end-expiratory pressure provides the best strat

239 ures and with hyperaerated regions at higher positive end-expiratory pressures (r >/= 0.72; p < 0.003

240 in patients who recruit lung in response to positive end-expiratory pressure, recruitment maneuvers,

241 The application of positive end-expiratory pressure reduced the extent of l

242 requiring high levels of inspired oxygen and positive end-expiratory pressures), reduced lung complia

243 changes (p < 0.01 for all) in tidal volume, positive end-expiratory pressure, respiratory rate, oxyg

244 Recruitment maneuvers and changes in positive end-expiratory pressure resulted in no improvem

245 dex, 48 +/- 11 kg/m), 21.7 +/- 3.7 cm H2O of positive end-expiratory pressure resulted in the lowest

246 y pressure, compared with the optimum global positive end-expiratory pressure, resulted in 1) decreas

247 relieved expiratory flow limitation and auto-positive end-expiratory pressure resulting in a dramatic

248 Applied positive end-expiratory pressure reverses expiratory flo

249 The positive end-expiratory pressure selected by the differe

250 Bedside positive end-expiratory pressure selection methods based

251 volume of approximately 3 mL/kg and 1) high positive end-expiratory pressure set above the level whe

252 at zero end-expiratory pressure, and then at positive end-expiratory pressure set at the level of aut

253 Measurements were obtained at the baseline positive end-expiratory pressure set by the clinicians,

254 ure/volume inflation curve plus 2 cmH2O as a positive end-expiratory pressure setting limits hyperinf

255 after each new intra-abdominal pressure and positive end-expiratory pressure setting.

256 ular mechanics compared with higher or lower positive end-expiratory pressure settings.

257 Increasing positive end-expiratory pressure shifted the predominant

258 psed areas of the lung; consequently, higher positive end-expiratory pressure should be limited to pa

259 gative abdominal pressure (-5 cm H2O) to the positive end-expiratory pressure significantly increased

260 Increasing positive end-expiratory pressure significantly reduced c

261 esophageal pressure, and oxygenation (higher positive end-expiratory pressure table of lung open vent

262 nstillation, animals underwent a decremental positive end-expiratory pressure titration (steps of 2 c

263 ry pressure; both trials were done utilizing positive end-expiratory pressure titration and recruitme

264 The use of Pes for positive end-expiratory pressure titration may help impr

265 er maximum-recruitment maneuver, decremental positive end-expiratory pressure titration was performed

266 omography-derived maps were computed at each positive end-expiratory pressure-titration step, and who

267 atory pressure trial (volutrauma); or 2) low positive end-expiratory pressure to achieve driving pres

268 lation strategy, characterized by sufficient positive end-expiratory pressure to avoid atelectasis, a

269 cute lung injury, but may not provide enough positive end-expiratory pressure to avoid cyclical recru

270 herefore, we examined the effect of matching positive end-expiratory pressure to the intra-abdominal

271 mic compliance increased more than 5% during positive end-expiratory pressure trial (volutrauma); or

272 g in highest compliance during a decremental positive end-expiratory pressure trial after lung recrui

273 nd to quantify the benefits of a decremental positive end-expiratory pressure trial preceded by a rec

274 A decremental positive end-expiratory pressure trial preceded by a rec

275 he development of atelectasis, a decremental positive end-expiratory pressure trial preceded by lung

276 Following lung recruitment and decremental positive end-expiratory pressure trial, animals were ran

277 essure identified through a best decremental positive end-expiratory pressure trial.

278 ssure, tidal volume, or plateau pressure and positive end-expiratory pressure, V(RM) remained indepen

279 tration and application of optimum-selective positive end-expiratory pressure values for the dependen

280 n enabled the titration of optimum-selective positive end-expiratory pressure values for the dependen

281 and esophageal pressure methods gave similar positive end-expiratory pressure values in mild, moderat

282 tify implementation of low tidal volume/high positive end-expiratory pressure ventilatory strategies

283 tify implementation of low tidal volume/high positive end-expiratory pressure ventilatory strategies,

284 idal volume (VT) and volume of gas caused by positive end-expiratory pressure (VPEEP) generate dynami

285 ressure was 27 cm H2O +/- 6 cm H2O, and mean positive end-expiratory pressure was 8.9 cm +/- 2.9 cm H

286 No effect of positive end-expiratory pressure was found; however, as

287 Positive end-expiratory pressure was increased to a plat

288 To select individual positive end-expiratory pressure, we applied bedside met

289 lung recruitment maneuvers and titration of positive end-expiratory pressure were both necessary to

290 Recruitment maneuvers followed by titrated positive end-expiratory pressure were effective at incre

291 e patients, lung recruitability and clinical positive end-expiratory pressure were higher than in pat

292 of intra-abdominal pressure, three levels of positive end-expiratory pressure were randomly applied w

293 itive end-expiratory pressure or 5 cm H2O of positive end-expiratory pressure were randomly applied.

294 te respiratory distress syndrome at clinical positive end-expiratory pressure were reclassified to ei

295 for estimating pleural pressure and setting positive end-expiratory pressure were retrospectively ap

296 for estimating pleural pressure and setting positive end-expiratory pressure were retrospectively ap

297 ra-abdominal pressure compared with 5 cm H2O positive end-expiratory pressure) while minimally decrea

298 at compare the effects of optimum-selective positive end-expiratory pressure with optimum global pos

299 Network hospitals, the addition of baseline positive end-expiratory pressure would not have increase

300 estimated in cm H2O as (peak airway pressure-positive end-expiratory pressure)x[(100-gain)/gain], was