ライフサイエンスコーパス: positive end-expiratory pressure

1 for each individual lung (optimum-selective positive end-expiratory pressure).

2 rotocol (at clinical, 5 cm H2O, or 15 cm H2O positive end-expiratory pressure).

3 re in caudal regions at end-expiration (best-positive end-expiratory pressure).

4 is would imply an "atypical" response to the positive end-expiratory pressure.

5 xpiratory pressure and the optimum-selective positive end-expiratory pressure.

6 assessed at clinical, 5 cm H2O, or 15 cm H2O positive end-expiratory pressure.

7 emodynamics were measured at 5 and 15 cm H2O positive end-expiratory pressure.

8 daily minimum fraction of inspired oxygen or positive end-expiratory pressure.

9 change, lung recruitability, and response to positive end-expiratory pressure.

10 t with the use of low tidal volumes and high positive end-expiratory pressure.

11 ances may vary unpredictably with changes in positive end-expiratory pressure.

12 t ventilation (12 +/- 3 cm H2O), at clinical positive end-expiratory pressure.

13 er the reliability of this test depends upon positive end-expiratory pressure.

14 l volume and increasing respiratory rate and positive end-expiratory pressure.

15 as still significantly reduced by increasing positive end-expiratory pressure.

16 tive dependence between mechanical power and positive end-expiratory pressure.

17 trolled trials with differential response to positive end-expiratory pressure.

18 au and driving pressures, and high levels of positive end-expiratory pressure.

19 ) with high-strain (VT = 18.2 +/- 6.5 mL/kg, positive end-expiratory pressure = 0), high-strain plus

20 lipopolysaccharide (VT = 18.4 +/- 4.2 mL/kg, positive end-expiratory pressure = 0), or low-strain plu

21 ve end-expiratory transpulmonary pressure at positive end-expiratory pressure 1 cm H2O (mean -3.5 +/-

22 t also results in a significant drop in auto-positive end-expiratory pressure (1.2 [0.6-4] vs 10 [5-1

23 nd mainly in lungs prone to collapse (at low positive end-expiratory pressure), 1) the expiratory tra

24 5 +/- 0.4 cm H2O) into the positive range at positive end-expiratory pressure 10 cm H2O (mean 0.58 +/

25 %] in controls; p < 0.0001) and greater auto-positive end-expiratory pressure (10 [5-12.5] vs 0.7 [0.

26 e) and were mechanically ventilated for 4 h (positive end-expiratory pressure, 10 cm H2O; plateau pre

27 At clinical positive end-expiratory pressure (11+/-3 cm H2O), patien

28 8 airways (6.3%; radius, 0.35 +/- 0.08 mm at positive end-expiratory pressure 12) at baseline and fiv

29 with a 47.5 x 47.5 x 47.5 mum voxel size, at positive end-expiratory pressure 12, 9, 6, 3, and 0 cm H

30 At higher positive end-expiratory pressure (15 cm H2O [interquarti

31 ntrols; p < 0.001), required higher level of positive end-expiratory pressure (15 vs 8 cm H2O in cont

32 mm Hg; p = 0.045) despite of using a higher positive end-expiratory pressure (17.4 +/- 0.7 vs 9.5 +/

33 lipopolysaccharide (VT = 8.1 +/- 0.2 mL/kg, positive end-expiratory pressure = 17 +/- 3 cm H2O).

34 eceived high tidal volume (30-32 mL/kg) with positive end-expiratory pressure (3 cm H2O) and sustaine

35 FIO2, 0.6; inspiratory:expiratory, 1:2; and positive end-expiratory pressure, 3 cm H2O) at baseline.

36 tive tidal volume, 2) appropriate setting of positive end-expiratory pressure, 3) oxygen weaning, and

37 and positive end-expiratory pressure = best-positive end-expiratory pressure + 4 cm H2O, 3) spontane

38 and positive end-expiratory pressure = best-positive end-expiratory pressure + 4 cm H2O, 4) spontane

39 and positive end-expiratory pressure = best-positive end-expiratory pressure - 4 cm H2O, 2) no spont

40 and positive end-expiratory pressure = best-positive end-expiratory pressure - 4 cm H2O.

41 s per day (pressure support level, 8 cm H2O; positive end-expiratory pressure, 4 cm H2O; FiO2, 50%) (

42 essure of 5 and pressure support of 10 above positive end-expiratory pressure 5 cm H2O, as well as 5

43 lation (control group, tidal volume 9 mL/kg, positive end-expiratory pressure 5 cm H2O, n = 15), high

44 T scan and lung ultrasound were performed at positive end-expiratory pressure 5 cm H2O.

45 Two levels of positive end-expiratory pressure (5 and 15 cm H2O) were

46 oup received low tidal volume (7 mL/kg) with positive end-expiratory pressure (5 cm H2O) and regular

47 sed cardiac index to a larger extent than at positive end-expiratory pressure = 5 cm H2O (19% [interq

48 At positive end-expiratory pressure = 5 cm H2O, 29% of pati

49 iratory pressure support level, 5-15 cm H2O; positive end-expiratory pressure, 5-10 cm H2O; fraction

50 y distress syndrome patients required higher positive end-expiratory pressure (7 vs 6 vs 10 vs 15 cm

51 tive end-expiratory pressure (i.e., clinical positive end-expiratory pressure = 7 +/- 2 cm H2O) with

52 dex 26 +/- 6 kg/m, PaO2/FIO2 147 +/- 42, and positive end-expiratory pressure 9.3 +/- 1.4 cm H2O) wer

53 p) with identical tidal volume (7 mL/kg) and positive end-expiratory pressure (9 cm H2O) after induci

54 lung injury could be re-inflated by applying positive end expiratory pressure, although at the expens

55 At higher positive end-expiratory pressure, an end-expiratory occl

56 received maximal lung recruitment, titrated positive end expiratory pressure and further Vt limitati

57 reas of poorer management included levels of positive end expiratory pressures and timing of introduc

58 after a recruitment maneuver and decremental positive end-expiratory pressure and corresponded to a p

59 We hypothesized that higher positive end-expiratory pressure and enhanced spontaneou

60 d pressure with 82 centers (68.9%) employing positive end-expiratory pressure and FIO2 to optimize ox

61 f injurious mechanical ventilation using low positive end-expiratory pressure and high inspiratory pr

62 ergoing pressure support ventilation, higher positive end-expiratory pressure and lower support level

63 EFL) is not completely avoidable by applying positive end-expiratory pressure and may cause respirato

64 Lung recruitment by higher positive end-expiratory pressure and surfactant administ

65 Application of positive end-expiratory pressure and surfactant reduced

66 ch): ventilation applying the optimum global positive end-expiratory pressure and the optimum-selecti

67 rdistension with nonaerated regions at lower positive end-expiratory pressures and with hyperaerated

68 Increased positive-end expiratory pressure and reduced time at low

69 pliance was defined globally (optimum global positive end-expiratory pressure) and for each individua

70 ry mechanics included plateau pressure, auto-positive end-expiratory pressure, and flow-limited volum

71 ow Coma Scale, spontaneous respiratory rate, positive end-expiratory pressure, and systolic blood pre

72 he individual tidal cycle (plateau pressure, positive end-expiratory pressure, and their difference [

73 rs, as were peak airway pressures, intrinsic positive end-expiratory pressure, and use of vasopressor

74 The level of positive end-expiratory pressure applied may greatly aff

75 Our data suggest that the "higher" positive end-expiratory pressure approach in patients wi

76 We compared a "lower" and a "higher" positive end-expiratory pressure approach, respectively,

77 r size of normal mice in respiration without positive end expiratory pressure as 58 +/- 14 (mean +/-

78 ilator-induced lung injury (with low Vt/high positive end-expiratory pressure as the main pillars), i

79 mized cross-over design to find the level of positive end-expiratory pressure associated with: 1) pos

80 tory pressure set by the clinicians, at zero positive end-expiratory pressure, at best positive end-e

81 eled with restricted cubic spline), baseline positive end-expiratory pressure, baseline tidal volume,

82 r): 1) no spontaneous breathing activity and positive end-expiratory pressure = best-positive end-exp

83 2O, 2) no spontaneous breathing activity and positive end-expiratory pressure = best-positive end-exp

84 m H2O, 3) spontaneous breathing activity and positive end-expiratory pressure = best-positive end-exp

85 m H2O, 4) spontaneous breathing activity and positive end-expiratory pressure = best-positive end-exp

86 determine which bedside method would provide positive end-expiratory pressure better related to lung

87 as decreased with higher compared with lower positive end-expiratory pressure both without spontaneou

88 his study evaluated two methods of titrating positive end-expiratory pressure; both trials were done

89 In contrast, independent of positive end-expiratory pressure but dependent on lung c

90 Commonly used positive end-expiratory pressure by clinicians is inadeq

91 ured ventilation-perfusion mismatch at lower positive end-expiratory pressure by electrical impedance

92 of time, tidal volume, respiratory rate, and positive end-expiratory pressure can guide mechanical ve

93 and Tierney reported that high Vt with zero positive end-expiratory pressure caused overwhelming lun

94 ed lung injury, high peak pressure (and zero positive end-expiratory pressure) causes respiratory swi

95 pressure optimization strategies recommended positive end-expiratory pressure changes in opposite dir

96 pressure optimization strategies recommended positive end-expiratory pressure changes in opposite dir

97 lung recruitability and edema than at higher positive end-expiratory pressure clinically set.

98 t the end of the 15-minute optimum-selective positive end-expiratory pressure, compared with the opti

99 To test whether positive end-expiratory pressure consistent with an open

100 In this experimental setting, positive end-expiratory pressure consistent with the ope

101 th a duty cycle (TITTOT) of 0.33 and without positive end-expiratory pressure (control); 2) as in the

102 The positive end-expiratory pressure corresponding to maximu

103 ratory mechanics (peak inspiratory pressure, positive end-expiratory pressure, DeltaP [PIP minus PEEP

104 At 24 hours, peak inspiratory pressure, positive end-expiratory pressure, DeltaP were higher, an

105 Peak inspiratory pressure, positive end-expiratory pressure, DeltaP, tidal volume,

106 Removal of positive end-expiratory pressure did not result in abrup

107 +/-114 mL, corresponding to 19+/-1 cm H2O of positive end-expiratory pressure) did (from 314+/-55 to

108 ay pressures alone (plateau airway pressure--positive end-expiratory pressure) did not equate to thos

109 rtile range, 13-15]), the plateau pressure - positive end-expiratory pressure difference did not chan

110 lation strategy of low tidal volume and high positive end-expiratory pressure, does not reduce, and m

111 or detecting preload dependence whatever the positive end-expiratory pressure during acute respirator

112 intra-abdominal hypertension and changes in positive end-expiratory pressure during different models

113 n initial escalating/de-escalating (dynamic) positive end-expiratory pressure (DynPEEP; n = 26).

114 Positive end-expiratory pressure exerts its effects keep

115 pressure-expiratory-CT and optimum-selective positive end-expiratory pressure-expiratory-CT (3.7 +/-

116 t/dependent ratio between the optimum global positive end-expiratory pressure-expiratory-CT and optim

117 spontaneous breathing trials, used a common positive end-expiratory pressure-FI(O(2)) chart, sedatio

118 tilatory maneuvers (increase and decrease in positive end-expiratory pressure from 0 to 15 and back t

119 exclusion gave Frecruited values of zero at positive end-expiratory pressure greater than or equal t

120 nal distension, absence of bowel sounds, and positive end-expiratory pressure greater than or equal t

121 mbined with daily positive fluid balance and positive end-expiratory pressure greater than or equal t

122 ))/Fi(O(2)) < 200 mm Hg received helmet NIV (positive end-expiratory pressure >= 10 cm H(2)O, pressur

123 ed as Vt <=8 ml/kg predicted body weight and positive end-expiratory pressure >=8 cm H(2)O.

124 We compared the physiologic effects of positive end-expiratory pressure guided by electrical im

125 Tidal volume and positive end-expiratory pressure had no impact on mortal

126 The driving pressure (plateau pressure minus positive end-expiratory pressure) has been suggested as

127 ure 24 cm H2O and low pressure 5 cm H2O (low positive end-expiratory pressure-high driving pressure);

128 per airway obstruction, higher preextubation positive end-expiratory pressure, higher postextubation

129 pressure support ventilation level, a lower positive end-expiratory pressure (i.e., clinical positiv

130 The two methods of evaluating positive end-expiratory pressure identified similar opti

131 nd after a recruitment maneuver, and at best positive end-expiratory pressure identified through a be

132 ro positive end-expiratory pressure, at best positive end-expiratory pressure identified through esop

133 reanalysis of randomized clinical trials of positive end-expiratory pressure in ARDS that support th

134 The approach to applying positive end-expiratory pressure in morbidly obese patie

135 rent techniques exist to select personalized positive end-expiratory pressure in patients affected by

136 ystem elastances increased with increases in positive end-expiratory pressure in patients with positi

137 ory system elastances grew with increases in positive end-expiratory pressure in patients with positi

138 n predominates over expiratory flow bias and positive end-expiratory pressure in the prevention of gr

139 ular mechanics compared with higher or lower positive end-expiratory pressures in experimental acute

140 verdistension increases both at high and low positive end-expiratory pressures in nondependent region

141 space, increases in respiratory rate, higher positive end-expiratory pressures in patients who recrui

142 The use of higher positive end-expiratory pressures in the open lung appro

143 mm Hg [14-17] vs 15 mm Hg [13-18] before the positive end-expiratory pressure increase).

144 trical impedance tomography-derived maps and positive end-expiratory pressure indicate that, expected

145 elpful in explaining how different levels of positive end-expiratory pressure influence recruitment a

146 ressure-inspiratory-CT and optimum-selective positive end-expiratory pressure-inspiratory-CT (2.8 +/-

147 t/dependent ratio between the optimum global positive end-expiratory pressure-inspiratory-CT and opti

148 lation with the use of low tidal volumes and positive end-expiratory pressure is considered best prac

149 etween the plateau pressure and the level of positive end-expiratory pressure is not known.

150 preceded by lung recruitment identified the positive end-expiratory pressure level (17.4 +/- 2.1 cm

151 The positive end-expiratory pressure level resulting in high

152 Each positive end-expiratory pressure level was maintained fo

153 At each positive end-expiratory pressure level, we assessed arte

154 ry system elastances were calculated at each positive end-expiratory pressure level.

155 piratory pressure identified similar optimal positive end-expiratory pressure levels (20.7 +/- 4.0 vs

156 increasing intra-abdominal pressure at both positive end-expiratory pressure levels (p </= 0.0001) w

157 Lower positive end-expiratory pressure levels (until day 7) an

158 alveoli over short-time scales in all tested positive end-expiratory pressure levels and despite stab

159 is, preventing hyperinflation.Methods: Three positive end-expiratory pressure levels and four externa

160 Lower positive end-expiratory pressure levels and significantl

161 In multivariate analysis, higher positive end-expiratory pressure levels during the first

162 However, higher positive end-expiratory pressure levels during the first

163 ion method was the only one which gave lower positive end-expiratory pressure levels in mild and mode

164 Application of higher positive end-expiratory pressure levels increased Vt%dep

165 Even for positive end-expiratory pressure levels minimizing globa

166 The ideal positive end-expiratory pressure levels recommended by t

167 hereas the oxygenation-based method provided positive end-expiratory pressure levels related with lun

168 Personalized positive end-expiratory pressure levels selected by elec

169 Positive end-expiratory pressure levels selected by the

170 unrelated with lung recruitability, whereas positive end-expiratory pressure levels selected by the

171 The positive end-expiratory pressure levels set by the clini

172 oderate acute respiratory distress syndrome, positive end-expiratory pressure levels that stabilize d

173 ics or absolute esophageal pressures provide positive end-expiratory pressure levels unrelated to lun

174 Thereafter, three positive end-expiratory pressure levels were applied in

175 Positive end-expiratory pressure levels were higher in t

176 In patients who were ventilated at two positive end-expiratory pressure levels, chest wall and

177 ventilation with higher tidal volumes, lower positive end-expiratory pressure levels, or both, than p

178 At both positive end-expiratory pressure levels, pulmonary acute

179 ntilation, high FiO2 concentration, and high positive end-expiratory pressure levels.

180 ure 17 cm H2O and low pressure 5 cm H2O (low positive end-expiratory pressure-low driving pressure).

181 stress syndrome severity at standardized low positive end-expiratory pressure may improve the associa

182 iology Score II, maximum blood lactate, mean positive end-expiratory pressure, mean cumulative fluid

183 effect estimates) and conditional for higher positive end-expiratory pressure (moderate confidence in

184 with a body mass index greater than 30 kg/m, positive end-expiratory pressure more than 10 cm H2O, Pa

185 al volume group, tidal volume 25 mL/kg, zero positive end-expiratory pressure, n = 14), or high tidal

186 the same tidal volume as control but neither positive end-expiratory pressure nor sustained inflation

187 y after adjusting for Pa(O(2))/Fi(O(2)), and positive end-expiratory pressure (odds ratio, 1.51; P =

188 ibiotics for possible VAP with daily minimum positive end-expiratory pressure of </=5 cm H2O and frac

189 ra-abdominal pressures of 0 and 20 cm H2O at positive end-expiratory pressure of 1 and 10 cm H2O, und

190 minal hypertension increased both DP(AW) (at positive end-expiratory pressure of 1 cm H2O, p < 0.0001

191 ure of 10 cm H2O, p = 0.0091) and DP(TP) (at positive end-expiratory pressure of 1 cm H2O, p = 0.0510

192 ory pressure of 1 cm H2O, p < 0.0001; and at positive end-expiratory pressure of 10 cm H2O, p = 0.009

193 ory pressure of 1 cm H2O, p = 0.0510; and at positive end-expiratory pressure of 10 cm H2O, p = 0.033

194 % received mechanical ventilation, with mean positive end-expiratory pressure of 14 cm H2O at the ons

195 In both mechanical ventilation groups, positive end-expiratory pressure of 2 cm H2O was applied

196 ssure (control); 2) as in the control group, positive end-expiratory pressure of 5 cm H2O and TITTOT

197 n or equal to 6 were placed on FIO2 of 0.50, positive end-expiratory pressure of 5 cm H2O, and pressu

198 n 150 mm Hg, with an FiO2 of at least 0.6, a positive end-expiratory pressure of at least 5 cm of wat

199 using phase-contrast synchrotron imaging, at positive end-expiratory pressures of 12, 9, 6, 3, and 0

200 end-expiratory pressure with optimum global positive end-expiratory pressure on regional collapse an

201 gs (i.e. plateau pressure, tidal volume, and positive end-expiratory pressure) on ICU mortality using

202 The two positive end-expiratory pressure optimization strategies

203 evaluate the agreement between two published positive end-expiratory pressure optimization strategies

204 evaluate the agreement between two published positive end-expiratory pressure optimization strategies

205 The two positive end-expiratory pressure optimization strategies

206 remained virtually unaffected by changes in positive end-expiratory pressure or intra-abdominal pres

207 creases in dependent regions with decreasing positive end-expiratory pressure (p < 0.001) and suggest

208 d with lower tidal volume (P < 0.01), higher positive end-expiratory pressure (P < 0.01), and higher

209 %nondep/Vt%dep ratio, as compared with lower positive end-expiratory pressure (p < 0.01).

210 ng distensibility) decreased with increasing positive end-expiratory pressure (p = 0.001) independent

211 Both at 5 and 15 cm H2O of positive end-expiratory pressure PaO2/FIO2, respiratory

212 At high positive end-expiratory pressure, passive leg raising in

213 l volume of 6 mL/kg and progressively higher positive end-expiratory pressure (PEEP) (5, 10, 16, 20,

214 All patients received positive end-expiratory pressure (PEEP) at 5 cm H2O.

215 on of inspired oxygen (FiO(2)) by >/= 15% or positive end-expiratory pressure (PEEP) by >/= 2.5 cm H(

216 In this study, we examined whether (1) positive end-expiratory pressure (PEEP) has a protective

217 Positive end-expiratory pressure (PEEP) has been used du

218 ies using low tidal volume or high levels of positive end-expiratory pressure (PEEP) improve outcomes

219 Rationale: Response to positive end-expiratory pressure (PEEP) in acute respira

220 application of at least a minimal amount of positive end-expiratory pressure (PEEP) in ARDS (1B); hi

221 n whether invasive ventilation can use lower positive end-expiratory pressure (PEEP) in critically il

222 mes in both cohorts and with the response to positive end-expiratory pressure (PEEP) in the ALVEOLI c

223 RATIONALE: Optimal positive end-expiratory pressure (PEEP) is unknown in pa

224 The effects of recruitment maneuvers and positive end-expiratory pressure (PEEP) titration on cli

225 Adjusting positive end-expiratory pressure (PEEP) to offset pleura

226 The median positive end-expiratory pressure (PEEP) was 14 (IQR, 12-

227 dely accepted cutoff values of PaO2/FIO2 and positive end-expiratory pressure (PEEP) would identify s

228 datory ventilation group, multiple levels of positive end-expiratory pressure (PEEP; 5, 10, 16, 20, a

229 en to fractional inspired oxygen <27 kPa and positive end-expiratory pressure [PEEP] >=8 cm H(2)O) in

230 sed as mL/kg predicted bodyweight [PBW], and positive end-expiratory pressure [PEEP] expressed as cm

231 tion of inspired oxygen of <150 mm Hg with a positive end-expiratory pressure [PEEP] of >=8 cm of wat

232 domized into three groups: 1) nonprotective (positive end-expiratory pressure [PEEP], 5 cm H(2)O; Vt,

233 lues, supine and prone positions and various positive end-expiratory pressures (PEEPs) and tidal volu

234 ssures, lower tidal volumes (VT), and higher positive end-expiratory pressures (PEEPs) can improve su

235 al death based on quantiles of tidal volume, positive end-expiratory pressure, plateau pressure, and

236 Ventilation at lowest elastance positive end-expiratory pressure preceded by a recruitme

237 Combining sitting position and applied positive end-expiratory pressure provides the best strat

238 lower (r = 0.850; p = 0.032) but not higher positive end-expiratory pressure (r = 0.018; p = 0.972).

239 ures and with hyperaerated regions at higher positive end-expiratory pressures (r >/= 0.72; p < 0.003

240 in patients who recruit lung in response to positive end-expiratory pressure, recruitment maneuvers,

241 The application of positive end-expiratory pressure reduced the extent of l

242 At higher positive end-expiratory pressure, respiratory mechanics

243 changes (p < 0.01 for all) in tidal volume, positive end-expiratory pressure, respiratory rate, oxyg

244 chanics, lung recruitment, gas exchange, and positive end-expiratory pressure response.

245 Higher positive end-expiratory pressure resulted in a more homo

246 dex, 48 +/- 11 kg/m), 21.7 +/- 3.7 cm H2O of positive end-expiratory pressure resulted in the lowest

247 y pressure, compared with the optimum global positive end-expiratory pressure, resulted in 1) decreas

248 relieved expiratory flow limitation and auto-positive end-expiratory pressure resulting in a dramatic

249 Applied positive end-expiratory pressure reverses expiratory flo

250 The positive end-expiratory pressure selected by the differe

251 electrical impedance tomography could guide positive end-expiratory pressure selection based on opti

252 Bedside positive end-expiratory pressure selection methods based

253 volume of approximately 3 mL/kg and 1) high positive end-expiratory pressure set above the level whe

254 at zero end-expiratory pressure, and then at positive end-expiratory pressure set at the level of aut

255 Measurements were obtained at the baseline positive end-expiratory pressure set by the clinicians,

256 espiratory distress syndrome network and the positive end-expiratory pressure setting in adults with

257 in mechanically ventilated patients to guide positive end-expiratory pressure setting, assess the eff

258 ular mechanics compared with higher or lower positive end-expiratory pressure settings.

259 Increasing positive end-expiratory pressure shifted the predominant

260 psed areas of the lung; consequently, higher positive end-expiratory pressure should be limited to pa

261 Increasing positive end-expiratory pressure significantly reduced c

262 esophageal pressure, and oxygenation (higher positive end-expiratory pressure table of lung open vent

263 nstillation, animals underwent a decremental positive end-expiratory pressure titration (steps of 2 c

264 ry pressure; both trials were done utilizing positive end-expiratory pressure titration and recruitme

265 The use of Pes for positive end-expiratory pressure titration may help impr

266 l impedance tomography monitor underwent two positive end-expiratory pressure titration trials by ran

267 er maximum-recruitment maneuver, decremental positive end-expiratory pressure titration was performed

268 omography-derived maps were computed at each positive end-expiratory pressure-titration step, and who

269 atory pressure trial (volutrauma); or 2) low positive end-expiratory pressure to achieve driving pres

270 lation strategy, characterized by sufficient positive end-expiratory pressure to avoid atelectasis, a

271 stensibly to avoid volutrauma, together with positive end-expiratory pressure to increase the fractio

272 mic compliance increased more than 5% during positive end-expiratory pressure trial (volutrauma); or

273 g in highest compliance during a decremental positive end-expiratory pressure trial after lung recrui

274 nd to quantify the benefits of a decremental positive end-expiratory pressure trial preceded by a rec

275 A decremental positive end-expiratory pressure trial preceded by a rec

276 he development of atelectasis, a decremental positive end-expiratory pressure trial preceded by lung

277 We performed a two-step positive end-expiratory pressure trial with change of 10

278 Following lung recruitment and decremental positive end-expiratory pressure trial, animals were ran

279 essure identified through a best decremental positive end-expiratory pressure trial.

280 ssure, tidal volume, or plateau pressure and positive end-expiratory pressure, V(RM) remained indepen

281 tration and application of optimum-selective positive end-expiratory pressure values for the dependen

282 n enabled the titration of optimum-selective positive end-expiratory pressure values for the dependen

283 and esophageal pressure methods gave similar positive end-expiratory pressure values in mild, moderat

284 cm H2O and low pressure 20 cm H2O (very high positive end-expiratory pressure-very low driving pressu

285 idal volume (VT) and volume of gas caused by positive end-expiratory pressure (VPEEP) generate dynami

286 Positive end-expiratory pressure was increased from 9 +/

287 Positive end-expiratory pressure was increased to a plat

288 release ventilation with low tidal volumes, positive end-expiratory pressure was set 4 cm H2O above

289 needed to obtain viable oxygenation at lower positive end-expiratory pressure was significantly corre

290 Positive end-expiratory pressure was varied between 0 an

291 To select individual positive end-expiratory pressure, we applied bedside met

292 lung recruitment maneuvers and titration of positive end-expiratory pressure were both necessary to

293 Recruitment maneuvers followed by titrated positive end-expiratory pressure were effective at incre

294 e patients, lung recruitability and clinical positive end-expiratory pressure were higher than in pat

295 te respiratory distress syndrome at clinical positive end-expiratory pressure were reclassified to ei

296 for estimating pleural pressure and setting positive end-expiratory pressure were retrospectively ap

297 for estimating pleural pressure and setting positive end-expiratory pressure were retrospectively ap

298 at compare the effects of optimum-selective positive end-expiratory pressure with optimum global pos

299 dex and delivered ventilation increased with positive end-expiratory pressure, without affecting intr

300 estimated in cm H2O as (peak airway pressure-positive end-expiratory pressure)x[(100-gain)/gain], was