ライフサイエンスコーパス: positive pressure

1 r exhibits a liquid-liquid critical point at positive pressure.

2 fluidic artificial muscles that operate with positive pressures.

3 At large positive pressure, a second phase of 2D monolayer ice, i

4 which a spring-loaded air cylinder generates positive pressure and flexible silica capillaries transf

5 activity back to either TRPV4, which sensed positive pressure and stimulated activity, or TRPV1, whi

6 Positive pressure applied through the pipette reversibly

7 ct to generate -2.5 cm H(2)O to trigger each positive pressure breath, and (4) added inspiratory resi

8 ubron (n=10) or volume controlled continuous positive pressure breathing (n=8) after acute lung injur

9 tory device failure (mechanical ventilators, positive pressure breathing assist devices, nebulizers,

10 llowing randomization, the volume controlled positive pressure breathing group developed a profound a

11 f the eight animals in the volume controlled positive pressure breathing group were alive at the end

12 unchanged over time in the volume controlled positive pressure breathing group.

13 Animals that received volume controlled positive pressure breathing remained hypoxic with no app

14 comprising the use of suction, intermittent positive-pressure breathing or bronchodilator treatments

15 ntricular stroke volume variation induced by positive-pressure breathing vary in proportion to preloa

16 Attempts to recruit lung with positive pressure constitute a major aim in the manageme

17 he pipette had no effect on the channel, but positive pressure could completely and reversibly close

18 onstrate that this extra thrust results from positive pressure created by a vortex ring underneath th

19 opic intubation supported with a noninvasive positive pressure delivery systems may be feasible alter

20 -5 GPa, above which it turns over to normal (positive) pressure dependence.

21 ro, and then revert to normal behaviour with positive pressure dependences.

22 K) at 175 GPa to 11.2 K at 250 GPa, giving a positive pressure derivative of 0.05 K/GPa.

23 During reperfusion, negative and positive pressure derivatives as well as developed press

24 viral strain, suggesting that the degree of positive pressure for HIV-1 amino acid change is host de

25 ains controversial and is thought to involve positive pressure generated by roots.

26 agm inhibition, esophageal shortening, and a positive pressure gradient between the stomach and the E

27 A positive pressure gradient between the stomach and the E

28 sure group (between -1 and +1 cm H(2)O), and positive pressure group (greater than +1 cm H(2)O).

29 hows that, during locomotion, varanids use a positive pressure gular pump to assist lung ventilation.

30 When a positive pressure is applied across the bilayer, from th

31 een the laser deflection length and the peak positive pressure is derived.

32 essure (LBNP; -10 mm Hg) and nonhypertensive positive pressure (LBPP; +10 mm Hg) in 11 treated HFrEF

33 (control) or inflated for 15 min (lower body positive pressure [LBPP]) in random order.

34 The experimental results show that the peak positive pressure measured by laser deflection method is

35 plete spinal cord injuries while on constant positive pressure mechanical ventilation (hence, respira

36 rtery pressures attributable to intermittent positive pressure mechanical ventilation formed more ede

37 Positive pressure mechanical ventilation has significant

38 y pressures arising from either intermittent positive pressure mechanical ventilation or from pulsati

39 iratory motor output as follows: (1) passive positive pressure mechanical ventilation, (2) voluntary

40 age than those occurring during intermittent positive pressure mechanical ventilation, suggesting tha

41 stitution of supplemental oxygen therapy and positive pressure mechanical ventilation.

42 Over 30% of critically ill patients on positive-pressure mechanical ventilation have difficulty

43 ilure, may compromise the general benefit of positive-pressure-mediated increases in intrapleural pre

44 is an intertwined chronicle of negative and positive pressure modes and their role in managing venti

45 sively and quantitatively measuring the peak positive pressure of HIFU fields.

46 GC interface can be fairly long, because the positive pressure of the carrier gas on the permeate sid

47 At 10 mm Hg positive pressure, peak calcium current increased from -

48 d with Na-pentobarbital were ventilated by a positive pressure respirator.

49 41.5% [95 of 229]) and the mean (SD) days of positive pressure respiratory support (55 [40] vs 54 [42

50 ed nitric oxide to preterm infants requiring positive pressure respiratory support on postnatal days

51 n 1250 g receiving mechanical ventilation or positive pressure respiratory support on postnatal days

52 The fluorescence images suggest that a positive pressure results in compression of the bilayer

53 and the flexible interface creates pulses of positive pressure rises, increase in temperature, stream

54 t (RS) (receiving supplemental oxygen and/or positive-pressure RS); among those, oxygen/RS at 36 week

55 low the transition, followed by a hardening (positive pressure shift) above it.

56 Such positive pressures should be tried in order to encourage

57 ebo until they no longer required oxygen and positive-pressure support or until they reached a postme

58 Noninvasive positive pressure techniques such as continuous and bile

59 indoor air measured during the negative and positive pressure test conditions was sufficient to dete

60 At positive pressure, the LMkappaT has escaped observation

61 d trials do not currently support a role for positive pressure therapies for reducing cardiovascular

62 Application of positive pressure through the patch pipette separated th

63 gion of the outer leaflet and increasing the positive pressure throughout the hydrocarbon core.

64 protective ventilation and an end-expiratory positive pressure titrated to a plateau pressure of 28-3

65 Application of positive pressure to cell-attached macropatch electrodes

66 ery were substantially increased by applying positive pressure to the patch electrode evoking membran

67 o "idle" at the genome end and to maintain a positive pressure towards the packaged state.

68 bitory influence of ACMV at increased VT and positive pressure upon the amplitude of respiratory moto

69 6 minutes at 100 compressions per minute and positive pressure ventilation (100% O2) with a compressi

70 ho progressed to acute lung injury requiring positive pressure ventilation (area under the receiver-o

71 with higher severity, defined by the use of positive pressure ventilation (i.e., continuous positive

72 these patients had not received inspiratory positive pressure ventilation (IPPV) despite having had

73 of conversion from conventional intermittent positive pressure ventilation (IPPV) to cuirass negative

74 of prolonged HFOV with low tidal volume (VT) positive pressure ventilation (LV-PPV) in an immature ba

75 Although noninvasive positive pressure ventilation (NIPPV) for patients with

76 Although noninvasive positive pressure ventilation (NPPV) is a widely accepte

77 Noninvasive positive pressure ventilation (NPPV) is usually applied

78 We used noninvasive positive pressure ventilation (NPPV) with a helmet-type

79 ly treated by using intermittent noninvasive positive pressure ventilation (NPPV).

80 to have required oxygen supplementation and positive pressure ventilation after birth than nonasthma

81 lmonary dysplasia between nasal intermittent positive pressure ventilation and CPAP, both when used a

82 support, most importantly nasal intermittent positive pressure ventilation and high flow nasal cannul

83 dministration is related to short periods of positive pressure ventilation and implies the risk of lu

84 Invasive positive pressure ventilation and renal replacement ther

85 piratory distress syndrome prior to need for positive pressure ventilation are required so that these

86 us positive airway pressure and non-invasive positive pressure ventilation are safe and efficacious.

87 = 4.7), history of bronchiolitis (OR = 4.7), positive pressure ventilation at birth (OR = 3.3), low m

88 s defined by the acute onset of the need for positive pressure ventilation by an endotracheal or trac

89 orse 24-hr neurologic outcomes compared with positive pressure ventilation cardiopulmonary resuscitat

90 uration (%) were significantly higher in the positive pressure ventilation compared with the no assis

91 Positive pressure ventilation exposes the lung to mechan

92 ve organ dysfunction (defined as: pulmonary, positive pressure ventilation for > 7 days; renal, incre

93 nt, and maintained on appropriate oxygen and positive pressure ventilation for at least 1 to 2 mo.

94 3, were intubated and initially managed with positive pressure ventilation for severe respiratory fai

95 ion pressure was significantly higher in the positive pressure ventilation group (33 +/- 15 vs. 14 +/

96 Paco2 was significantly lower in the positive pressure ventilation group (48 +/- 10 vs. 77 +/

97 rbations are becoming known, and noninvasive positive pressure ventilation has become an option for p

98 The uptake of noninvasive positive pressure ventilation has resulted in widespread

99 Both heliox and bilevel positive pressure ventilation have demonstrated clinical

100 ment may reduce lung parenchymal injury from positive pressure ventilation in ARDS.

101 ous positive airway pressure and noninvasive positive pressure ventilation in children with sleep-dis

102 ous positive airway pressure and noninvasive positive pressure ventilation in nonresponders has becom

103 I evidence supporting the use of noninvasive positive pressure ventilation in such critical care sett

104 ll designed studies suggest that noninvasive positive pressure ventilation is not an appropriate inte

105 Noninvasive positive pressure ventilation may be considered a first

106 CPAP with surfactant but without any positive pressure ventilation may work synergistically.

107 respirator was switched over to non-invasive positive pressure ventilation on 24th day.

108 terobserver variability was not explained by positive pressure ventilation or by the presence of (> 4

109 either promote lung healing and weaning from positive pressure ventilation or delay recovery because

110 supported with nasally delivered noninvasive positive pressure ventilation or high-flow nasal cannula

111 ive of seven alive and neurologically intact positive pressure ventilation pigs with a cerebral perfo

112 s with triggering and cycling of noninvasive positive pressure ventilation remain an issue in small o

113 ompelling, but evidence favors a noninvasive positive pressure ventilation trial.

114 ficantly enriched subpathway in infants with positive pressure ventilation use compared with those wi

115 uous positive airway pressure or noninvasive positive pressure ventilation use.

116 jury criteria to acute lung injury requiring positive pressure ventilation was 20 hours.

117 Nasal intermittent positive pressure ventilation was superior to continuous

118 ommended in 22% of patients; and noninvasive positive pressure ventilation was used by only 21% of pa

119 Positive pressure ventilation with large VTs has been sh

120 sitive pressure ventilation; b) intermittent positive pressure ventilation with tracheal insufflation

121 ositioned at the carina; and c) intermittent positive pressure ventilation with tracheal insufflation

122 lium-oxygen therapy (heliox), or noninvasive positive pressure ventilation within 24 hrs of extubatio

123 rtile versus 15% for the others; noninvasive positive pressure ventilation, 8% versus 19%; vasopresso

124 liox gaseous mixture and noninvasive bilevel positive pressure ventilation, are being utilized in the

125 er therapies for ARDS, including noninvasive positive pressure ventilation, inverse ratio ventilation

126 ventilator-induced lung injury (VILI) during positive pressure ventilation, mechanisms of normal alve

127 s rapidly developed requiring high levels of positive pressure ventilation.

128 pulmonary resuscitation (CPR) while allowing positive pressure ventilation.

129 d specificity (86%) in predicting the use of positive pressure ventilation.

130 design on CO2 rebreathing during noninvasive positive pressure ventilation.

131 ient's inspiratory effort during noninvasive positive pressure ventilation.

132 the lung damage associated with conventional positive pressure ventilation.

133 ary artery pressures in the absence of tidal positive pressure ventilation.

134 opulmonary bypass, aortic cross-clamping and positive pressure ventilation.

135 as instilled into the lung while maintaining positive pressure ventilation.

136 ients with lung injury prior to the need for positive pressure ventilation.

137 ssed to acute lung injury prior to requiring positive pressure ventilation.

138 %) progressed to acute lung injury requiring positive pressure ventilation.

139 ventilated by three methods: a) intermittent positive pressure ventilation; b) intermittent positive

140 rgical airway condition; chronic noninvasive positive pressure ventilation; the need to replace the e

141 < 0.001), with increased use of noninvasive positive-pressure ventilation (5% in 1998 to 14% in 2010

142 ), animals were randomized into intermittent positive-pressure ventilation (control group), bilevel,

143 Intermittent positive-pressure ventilation (IPPV) is the "gold standa

144 sive respiratory support--nasal intermittent positive-pressure ventilation (IPPV) or nasal continuous

145 of assisted ventilation, nasal intermittent positive-pressure ventilation (NIPPV) and synchronized i

146 The patterns and outcomes of noninvasive, positive-pressure ventilation (NIPPV) use in patients ho

147 Noninvasive positive-pressure ventilation (NPPV) has been shown to b

148 Noninvasive positive-pressure ventilation (NPPV) is increasingly use

149 We compared noninvasive positive-pressure ventilation (NPPV), using bilevel posi

150 f a perfluorocarbon liquid during continuous positive-pressure ventilation (partial liquid ventilatio

151 pressure [CPAP] or noninvasive intermittent positive-pressure ventilation [NIPPV]) appears to be of

152 diac output was measured during intermittent positive-pressure ventilation and after 15 minutes of ne

153 Both intermittent positive-pressure ventilation and bilevel provided simil

154 auses induced by means of a step increase in positive-pressure ventilation applied via a face mask.

155 e (Leycom) were measured over 8 intermittent positive-pressure ventilation breaths at tidal volume of

156 outcomes after continuous compressions with positive-pressure ventilation differed from those after

157 benefit of jet ventilation over conventional positive-pressure ventilation during heart failure.

158 rve pacemaker (PNP) and the reinstitution of positive-pressure ventilation for 8 mo.

159 ecially in severe patients, and non-invasive positive-pressure ventilation for treatment of acute ven

160 Role of noninvasive positive-pressure ventilation in acute lung injury/ARDS

161 demonstrate explicitly that lower-frequency positive-pressure ventilation not only preserves adequat

162 stroke volume variation during intermittent positive-pressure ventilation predict preload responsive

163 el, 261 (109/386) (p = 0.195 vs intermittent positive-pressure ventilation) and 236 (86/364) (p = 0.8

164 ation, 28 (27/32) (p = 0.001 vs intermittent positive-pressure ventilation) and 26 (18/29) (p = 0.004

165 32.7 (30.4/33.4) (p = 0.021 vs intermittent positive-pressure ventilation) and 27.0 (24.5/27.7) (p =

166 29.1 (25.6/37.1) (p = 0.574 vs intermittent positive-pressure ventilation) and 28.7 (24.2/36.5) (p =

167 level, 39 (35/41) (p = 0.574 vs intermittent positive-pressure ventilation) and 46 (42/49) (p = 0.798

168 on, 598 (471/650) (p < 0.001 vs intermittent positive-pressure ventilation) and 634 (115/693) (p = 0.

169 7.35 (7.29/7.37) (p = 0.645 vs intermittent positive-pressure ventilation) and 7.27 (7.17/7.31) (p =

170 7.34 (7.33/7.39) (p = 0.189 vs intermittent positive-pressure ventilation) and 7.35 (7.34/7.36) (p =

171 was achieved in five of eight (intermittent positive-pressure ventilation), six of eight (bilevel),

172 7.35 (7.34/7.36) (p = 0.006 vs intermittent positive-pressure ventilation).

173 27.0 (24.5/27.7) (p = 0.779 vs intermittent positive-pressure ventilation).

174 and 236 (86/364) (p = 0.878 vs intermittent positive-pressure ventilation); and chest compression sy

175 7.27 (7.17/7.31) (p = 0.645 vs intermittent positive-pressure ventilation); and chest compression sy

176 28.7 (24.2/36.5) (p = 0.721 vs intermittent positive-pressure ventilation); and chest compression sy

177 and 634 (115/693) (p = 0.054 vs intermittent positive-pressure ventilation); PaCO2 intermittent posit

178 es (torr) were as follows: PaO2 intermittent positive-pressure ventilation, 143 (76/256) and 262 (81/

179 inutes (mm Hg) were as follows: intermittent positive-pressure ventilation, 28.0 (25.0/29.6) and 27.9

180 ve-pressure ventilation); PaCO2 intermittent positive-pressure ventilation, 40 (38/43) and 45 (36/52)

181 9) (p = 0.004); mixed venous pH intermittent positive-pressure ventilation, 7.34 (7.31/7.35) and 7.26

182 y ventilated with volume-cycled intermittent positive-pressure ventilation, and negative-pressure ven

183 e investigated the influence of intermittent positive-pressure ventilation, bilevel ventilation, and

184 stroke volume variation during intermittent positive-pressure ventilation.

185 hodilators, corticosteroids, and noninvasive positive-pressure ventilation.

186 All were sedated and paralyzed and received positive-pressure ventilation.

187 elivered through a face mask, or noninvasive positive-pressure ventilation.

188 rest and decreased requirement for immediate positive-pressure ventilation.

189 with an inspiratory threshold valve between positive pressure ventilations.

190 either no assisted ventilation (n = 9) or 10 positive pressure ventilations/min (Smart Resuscitator B

191 The dogs were ventilated using a positive-pressure ventilator driven by phrenic neural ac

192 Likewise, the minute volume of positive pressure ventilatory support should be limited

193 The average percentage of time in which a positive pressure was recorded in the lungs was 47.3 +/-

194 provides a rapid way for measuring the peak positive pressure, without the scan time, which is requi