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1 trial (Prediction of Arrhythmic Events with Positron Emission Tomography).
2 n were measured using 18F-fluorodeoxyglucose positron emission tomography.
3 scular magnetic resonance, and (11)C-acetate positron emission tomography.
4 e to that achieved by expert assessment with positron emission tomography.
5 ostic index, and CFR was quantified by using positron emission tomography.
6 [(18)F]fluorodeoxyglucose ([(18)F]FDG) with positron emission tomography.
7 in the murine brain for up to one week using positron emission tomography.
8 preparation of potential tracers for use in positron emission tomography.
9 new radiotracers for molecular imaging with positron emission tomography.
10 metabolic response by 18F-fluorodeoxyglucose positron-emission tomography.
11 s of 1344 pixel range of perfusion in paired positron emission tomographies.
13 ion delivered to the liver utilizing an oral positron emission tomography (18) F-isotopologue validat
15 using fluorine-18-labeled fluorodeoxyglucose positron emission tomography ([(18)F]FDG PET), [(18)F]FD
17 f 10 minutes between (18)F-FCH injection and positron emission tomography acquisition is appropriate
18 arbon 11-labeled Pittsburgh Compound B (PiB) positron emission tomography after long-term prospective
19 3) or elevated (n = 202) brain amyloid using positron emission tomography amyloid imaging or a cerebr
21 inical evaluation and imaging at enrollment (positron emission tomography and 2-dimensional echo).
22 ers, and 18 nonsmokers who were scanned with positron emission tomography and [(11)C]raclopride, afte
23 erebral glucose metabolism was assessed with positron emission tomography and [F-18]fluorodeoxyglucos
24 14 healthy individuals using [(11)C]-acetate positron emission tomography and cardiovascular magnetic
25 disease, thanks to advances in MRI, amyloid positron emission tomography and cerebrospinal fluid bio
26 nt for pathogenesis and treatment.IMPORTANCE Positron emission tomography and computed tomography (PE
27 luorodeoxyglucose [FDG] avidity) measured by positron emission tomography and computed tomography at
29 essment in patients with lymphoma, including positron emission tomography and computed tomography sca
30 stic accuracy of Fluor-18-fluorodeoxyglucose positron emission tomography and computed tomography, la
31 d 77 years underwent 11C-(R)PK11195, 11C-PIB positron emission tomography and magnetic resonance imag
32 d the activity of the PSMA-PI3K axis through positron emission tomography and magnetic resonance imag
33 tients underwent (11)C-Pittsburgh compound B positron emission tomography and magnetic resonance imag
34 MENT We here show that combined simultaneous positron emission tomography and magnetic resonance imag
38 ects and 23 healthy controls participated in positron emission tomography and structural magnetic res
39 agnetic resonance imaging, amyloid (11C-PiB) positron emission tomography and tau (18F-AV-1451) posit
40 he results of their florbetapir F-18 (Abeta) positron emission tomography and their Alzheimer disease
41 ndication of neuroinflammation in vivo using positron emission tomography and TSPO-specific radioliga
43 ter stress testing with myocardial perfusion positron emission tomography and with left ventricular e
45 ned by fluorodeoxyglucose F 18 [FDG]-labeled positron emission tomography and/or hippocampal volume [
46 amyloid burden (measured by florbetapir F-18 positron emission tomography) and cognitive performance
47 on emission tomography and tau (18F-AV-1451) positron emission tomography, and episodic and semantic
48 beta-cells in pigs and nonhuman primates by positron emission tomography as well as in immunodeficie
49 d potentially have changed 332 of 1732 (19%) positron emission tomographies at low-risk physiological
52 amyloid-beta plaques measured as florbetapir positron emission tomography binding antecedent to 18F-A
53 ng cerebrospinal fluid (CSF) or imaging (tau positron emission tomography) biomarkers for Alzheimer d
54 dementia) and 12 healthy controls underwent positron emission tomography brain imaging with [(18)F]A
55 tomography, magnetic resonance imaging, and positron emission tomography can be used to assess pulmo
57 rticipants underwent 18-F Fluorodeoxyglucose Positron Emission Tomography Computed Tomography (18-FDG
58 cted by (18)F-fluorodeoxyglucose ((18)F-FDG) positron emission tomography computed tomography (PET/CT
59 Purpose Magnetic resonance imaging (MRI) and positron emission tomography-computed tomography (PET-CT
60 ained from baseline (18)F-fluorodeoxyglucose positron emission tomography-computed tomography (PET-CT
61 l present the analyses of centrally reviewed positron emission tomography-computed tomography (PET-CT
62 TV0 was measured by (18)F-fluorodeoxyglucose-positron emission tomography-computed tomography in 108
63 cation, adenocarcinoma histology, and higher positron emission tomography-computed tomography N stage
64 r extracranial metastatic lesions on choline positron emission tomography-computed tomography, and se
66 stenosis underwent (18)F-fluorodeoxyglucose-positron emission tomography/computed tomographic imagin
67 med to determine whether 18F-fludeoxyglucose-positron emission tomography/computed tomography (FDG-PE
68 ng of surveillance [(18)F]fluorodeoxyglucose-positron emission tomography/computed tomography (FDG-PE
70 ings from dual-energy spectral CT(DEsCT) and positron emission tomography/computed tomography (PET/CT
73 duals who underwent (18)F-fluorodeoxyglucose positron emission tomography/computed tomography imaging
74 ascular inflammation by 18fluorodeoxyglucose positron emission tomography/computed tomography in vivo
75 ned in a single hybrid imaging session using positron emission tomography/computed tomography or sing
76 rom 31 patients and results of early interim positron emission tomography/computed tomography scans i
77 ascular inflammation by 18fluorodeoxyglucose positron emission tomography/computed tomography, with g
81 s an insufficient explanation of 18F-AV-1451 positron emission tomography data in vivo, at least in t
83 and insulin sensitivity were measured using positron emission tomography during an isoglycemic clamp
84 ose concentrations) with 1-[(11)C]-d-glucose positron emission tomography during hyperinsulinemic glu
85 ical assessment, brain 18-fluorodeoxyglucose positron emission tomography, electroneurography, and EL
86 ontribute to disease profiles of 18F-AV-1451 positron emission tomography, especially in primary tauo
88 l that used early interim fluorodeoxyglucose positron emission tomography (FDG-PET) imaging to determ
89 t and high-resolution (18)fluorodeoxyglucose positron emission tomography (FDG-PET) imaging to unders
92 together to enable simultaneous tetramodal (positron emission tomography/fluorescence/Cerenkov lumin
93 nts underwent magnetic resonance imaging and positron emission tomography for amyloid-beta ((11) C-Pi
95 pir F 18 (previously known as AV 1451, T807) positron emission tomography (FTP-PET) imaging for tau a
96 erebral Abeta on Pittsburgh Compound B (PiB) positron emission tomography, gait speed over 4.57 m (15
97 the clinical use of RHL30 in the context of positron emission tomography-guided response assessment
100 aphy with iron oxide particles, and targeted positron emission tomography imaging are currently under
103 nflammation, were measured with [(11)C]PBR28 positron emission tomography imaging in 15 healthy contr
106 We assessed the radiotracer 18F-AV-1451 with positron emission tomography imaging to compare the dist
107 had available plasma total tau levels, Abeta positron emission tomography imaging, and a complete neu
108 l and structural magnetic resonance imaging, positron emission tomography imaging, and behavioral dat
109 structural MRI, [(18)F]flutemetamol amyloid positron emission tomography imaging, apolipoprotein E g
110 d quantitative techniques, including in vivo positron emission tomography imaging, gamma counting, an
114 etic resonance imaging and spectroscopy, and positron emission tomography in these areas and discusse
118 take with [(18) F]2-fluoro-2-deoxy-D-glucose/positron emission tomography, lipolysis (RaGly) with [U-
121 netic melanin nanoparticles is developed for positron-emission tomography/magnetic resonance/photoaco
122 ing fundamentals necessary to understand how positron emission tomography makes robust, quantitative
123 e in PCC fludeoxyglucose F 18 ([(18) F] FDG) positron emission tomography measured in Alzheimer's Dis
124 pleasant, and neutral images), and underwent positron emission tomography measurements of dopamine D2
125 latform for magnetic resonance/photoacoustic/positron emission tomography multimodal imaging and ligh
126 t state-of-the-art hardware and software for positron emission tomography myocardial perfusion imagin
127 lume of denervated myocardium (defect of the positron emission tomography norepinephrine analog (11)C
128 [(11)C]5-hydroxy-tryptophan ([(11)C]5-HTP) positron emission tomography of the pancreas has been sh
129 etic resonance imaging, nuclear imaging, and positron emission tomography) performed on an outpatient
132 single-photon emission tomography (SPECT) or positron emission tomography (PET) and coronary computed
133 hcare, Shanghai, China) followed by combined positron emission tomography (PET) and CT (hereafter, PE
135 y menstruating, asymptomatic women completed positron emission tomography (PET) and functional magnet
136 fluorine 18 ((18)F) fluorodeoxyglucose (FDG) positron emission tomography (PET) and hyperpolarized ca
137 18 ((18)F) fluorodeoxyglucose (FDG) combined positron emission tomography (PET) and magnetic resonanc
138 h-resolution neuroimaging data consisting of positron emission tomography (PET) and magnetic resonanc
140 vity at fluorine 18 fluorodeoxyglucose (FDG) positron emission tomography (PET) and survival in patie
141 22 healthy recreationally active males using positron emission tomography (PET) and the MOR-selective
142 is to synthesise current evidence on amyloid-positron emission tomography (PET) burden and presumed p
143 racy of fluorine 18 fluorodeoxyglucose (FDG) positron emission tomography (PET) combined with diagnos
144 d mass spectrometric detection to quantify a positron emission tomography (PET) detection tracer for
145 fluorine 18 ((18)F) fluorodeoxyglucose (FDG) positron emission tomography (PET) has added value over
150 ing (FL) and photodynamic therapy (PDT) with positron emission tomography (PET) imaging and internal
152 exploited the potential targeting of TF for positron emission tomography (PET) imaging of pancreatic
156 ized male nonhuman primates (n = 3), we used positron emission tomography (PET) imaging with the radi
157 employed in the realm of nanoparticle-based positron emission tomography (PET) imaging, whereas its
159 ased attenuation correction (ATAC) for brain positron emission tomography (PET) in an integrated time
161 surgery on the human brain immune system by positron emission tomography (PET) in relation to blood
166 tem (CNS) disorders, we sought to identify a positron emission tomography (PET) ligand to enable targ
168 ed that fluorine 18 fluorodeoxyglucose (FDG) positron emission tomography (PET) may detect the inflam
169 tional magnetic resonance imaging (fMRI) and positron emission tomography (PET) multimodal imaging wi
170 DPGM) is developed for MR/photoacoustic (PA)/positron emission tomography (PET) multimodal imaging-gu
171 Here we report a non-invasive quantitative positron emission tomography (PET) nanoreporter technolo
175 by analyzing motor defects and binding of a positron emission tomography (PET) radioligand to the ve
176 ation is possible with the recent advance in positron emission tomography (PET) radioligands that bin
177 ch toward the radiosynthesis of heterocyclic positron emission tomography (PET) radioligands using th
179 (CCR2)-binding peptide adapted for use as a positron emission tomography (PET) radiotracer for nonin
184 nonsmokers participated in two [(11)C]ABP688 positron emission tomography (PET) scans on the same day
185 had magnetic resonance imaging (MRI) scans, positron emission tomography (PET) scans with carbon 11-
187 ur objective was to determine the pattern of positron emission tomography (PET) tau tracer AV-1451 up
188 racer [(11)C]DAA1106 (a ligand for TSPO) and positron emission tomography (PET) to determine the effe
189 a radioligand that binds to the mGluR5, and positron emission tomography (PET) to quantify in vivo m
190 calisation and magnitude of the presumed tau Positron Emission Tomography (PET) tracer [(18)F]Flortau
192 individuals: (1) Tau, detected with a novel positron emission tomography (PET) tracer known as (18)F
194 )-3 ([(11)C]-(R)-IPMICF16), a first-in-class positron emission tomography (PET) TrkB/C-targeting radi
195 They came to an academic research center for positron emission tomography (PET) using [(18)F]fallypri
196 duced D2R internalization can be imaged with positron emission tomography (PET) using D2R radiotracer
198 ents of the New York metropolitan area using Positron Emission Tomography (PET) with [(11)C]racloprid
199 n of dopamine to value-based attention using positron emission tomography (PET) with [(11)C]racloprid
201 d tumor cytopenia on repeat (68)Ga-DOTA-TATE positron emission tomography (PET) within 6 months, sugg
202 d included magnetic resonance imaging (MRI), positron emission tomography (PET), and single-photon em
203 sease (PD) with 18F-fluorodeoxyglucose (FDG) positron emission tomography (PET), and their associatio
205 nitive continuum aged >60 years with amyloid positron emission tomography (PET), tau PET, and magneti
206 g a combination of functional MRI (fMRI) and positron emission tomography (PET), we investigated whet
207 ANCE STATEMENT: We present a high-resolution positron emission tomography (PET)- and magnetic resonan
209 stine, prednisone (R-CHOP14) induction and a positron emission tomography (PET)-driven ASCT or standa
222 Purpose To assess the accuracy of staging positron emission tomography (PET)/computed tomography (
223 time using a combination of autoradiography, positron emission tomography (PET)/computed tomography (
224 eath hold (DIBH) in fluorodeoxyglucose (FDG) positron emission tomography (PET)/computed tomography (
227 e of flourine 18 ((18)F) fluorocholine (FCH) positron emission tomography (PET)/magnetic resonance (M
231 ecombinant cellular systems, and critically, positron-emission-tomography (PET) studies with a specif
235 id (defined by cerebrospinal fluid assays or positron emission tomography regional summaries) can be
236 ated variability; and (iii) this 18F-AV-1451 positron emission tomography retention pattern significa
238 ed with age, and cross-sectional florbetapir positron emission tomography retention, but not with yea
241 sion phenotyping approaches using (11)C with positron emission tomography, root autoradiography, and
243 dard for reporting the results of an amyloid positron emission tomography scan is to assign a dichoto
245 tently associated with an Abeta+ florbetapir positron emission tomography scan, not all Abeta+ subjec
246 = 306) and (18) fluorodeoxyglucose (n = 305) positron emission tomography scanning to assess amyloid
248 F]fluoro-levo-dihydroxyphenylalanine dynamic positron emission tomography scans and striatal regions
249 resonance imaging and Pittsburgh compound B-positron emission tomography scans enrolled in the Mayo
250 3)I-MIBG scans, or [(18)F]fluorodeoxyglucose-positron emission tomography scans for MIBG-nonavid dise
251 eferred for rest/stress myocardial perfusion positron emission tomography scans from January 2006 to
252 ional resting-state (18)F-fluorodeoxyglucose positron emission tomography scans from VPA-exposed and
253 ne (MIBG) scans or [(18)F]fluorodeoxyglucose-positron emission tomography scans if the tumor is MIBG
254 atients were evaluated by fluorodeoxyglucose-positron emission tomography scans performed at baseline
255 DS AND In 127 volunteers, serial rest-stress positron emission tomography scans using rubidium-82 wit
257 D and 12 healthy controls (HC) completed two positron emission tomography scans with [(11)C]-(+)-PHNO
258 tomography binding antecedent to 18F-AV-1451 positron emission tomography scans, and to what extent t
259 betapir retention, antecedent to 18F-AV-1451 positron emission tomography scans, in the parieto-tempo
260 information that could be available from tau positron-emission tomography scans and its use to determ
262 on between 18 kDa translocator protein brain positron emission tomography signal, which arises largel
263 years), response (PR v CR) after R-CHOP, and positron emission tomography status at assignment (negat
265 usion, this is one of the first longitudinal positron emission tomography studies evaluating longitud
268 study and invited these individuals back for positron emission tomography study with [(18)F]-fluorode
271 ulti-session [(15)O]-water and [(18)F]-FDOPA positron emission tomography to determine striatal blood
272 fluorodihydroxyphenyl-l-alanine ([18F]-DOPA) positron emission tomography to examine dopamine synthes
274 hy age-matched control individuals underwent positron emission tomography to measure cerebral metabol
277 labeled with fluorine-18 ((18)F) are used in positron emission tomography to visualize, characterize
278 n tau tangle accumulation (measured with the positron emission tomography tracer 18F-AV-1451) associa
280 evelopment of tools such as radioligands and positron emission tomography tracers that are not curren
282 brain amyloid burden, as detected by amyloid positron emission tomography using 11C-Pittsburgh B comp
283 ith [(11)C]carfentanil and [(18)F]fluorodopa positron emission tomography using a high-resolution sca
284 x-matched control subjects had (18)F-AV-1451 positron emission tomography using a Siemens high-resolu
286 nonoffenders were included and examined with positron emission tomography, using the radioligand [(11
287 1C-Pittsburgh compound B and 11C-(R)-PK11195 positron emission tomography was used to determine the a
291 glucose uptake, assessed through noninvasive positron emission tomography, was an effective predictiv
293 dy, flortaucipir tau and florbetapir amyloid positron emission tomography were obtained for 217 subje
294 perfusion and coronary flow reserve (CFR) by positron emission tomography, where submaximal stress pr
296 f functional SGLT2 proteins in rodents using positron emission tomography with 4-[(18)F]fluoro-dapagl
297 iatal D2R binding potential (D2R BPND) using positron emission tomography with a D2R-selective radiol
299 e acquired in 14 long-term ex-smokers, using positron emission tomography with radiolabeled [11C]ABP6
300 onal neuroimaging ([(18)F]fluorodeoxyglucose positron emission tomography) with a fear-regulating ext
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