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1 not comprise tetramerization domains, our results provide a possible explanation as to the reason that RUNX1 is recurrent
3 may provide the mechanisms underlying Pitx2-induced AF and possible explanation behind the AAD effects of flecainide in
6 socio-economic position (SEP) to premature ageing and is a possible explanation for cross-national variation in patterns
8 Our systems biology approach offers a possible explanation for increased COVID-19 severity in patie
11 H gene expression than its own gene expression, providing a possible explanation for survival differences based on HIF1A
12 -2)levels were increased at the time of injury suggesting a possible explanation for the altered responses.
13 The coalescence of a member of this population offers a possible explanation for the anomalous properties of GW170817
14 axis is fundamental to host antimicrobial defense, offer a possible explanation for the clinical observation that coagul
15 n BALB/c(nu/nu) mice, the described mechanism may provide a possible explanation for the clinical phenomenon of hyperprog
16 ch highlights the role of epigenetics in WS, and provides a possible explanation for the complex phenotypes observed in p
18 Hedgehog signaling increases Wnt/Notch signaling provides a possible explanation for the failure of Smoothened antagonist
19 as downstream effectors in the same processes, offering one possible explanation for the high comorbidity rate of the two
22 That sympatric predators learn to ignore the defense is a possible explanation for the mountain katydid's counter-intui
24 compensation response (GCR) has recently been proposed as a possible explanation for the phenotypic discrepancies between
25 ieve element interface in mutant phloem tissue, providing a possible explanation for the reduced sucrose export in mutant
29 evealed transcriptional differences among HSCs, providing a possible explanation for their functional heterogeneity(12-17
33 al to those found in the respective EC tissues, providing a possible explanation for TZ tissue being the primary target f
45 rolongation, which may lead to ventricular arrhythmias as a possible explanation of this increased mortality.
48 are weakly sensitive for the detection of GORD, providing a possible explanation to discrepancies in AET and impedance in