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1 he left side of the mouse embryo by 8.0 days post coitum.
2 ryonic lethal, dying between 8 and 11.5 days post coitum.
3 early as the endogenous gene, at day 7.5-8.0 post coitum.
4 uring embryonic development at 9.5-14.5 days post coitum.
5 xpression in the gonad at 12.5 and 13.5 days post-coitum, after overt gonad differentiation, by compa
6 and renal cysts if they survive to day 15.5 post coitum and die in either the fetal or the perinatal
7 during embryonic development until 13.5 days post-coitum and in the adult heart, kidney, brain, teste
8 the developing heart at E10.5 and E11.5 days post-coitum and in the musculoskeletal system from E13.5
9 ession in the branchial arches from 9.5 days post-coitum and show that its activity in the context of
10 causes embryonic lethality at day 8.5 p.c. (post coitum) before establishment of a functional cardio
11 the time of PGC allocation around 7.25 days post coitum, Blimp1 heterozygous embryos exhibit decreas
12 lity in Cbfb(-/-) mouse embryos at 12.5 days post coitum (d.p.c.) from hemorrhages and lack of defini
13 se embryos deficient in Gata3 die by 11 days post coitum (d.p.c.) from pathology of undetermined orig
14 e results in embryonic lethality at 5.5 days post coitum (d.p.c.) which can be overcome by simultaneo
15 These embryos develop normally to 7.5 days post coitum (d.p.c.), but their growth is severely retar
16 d in pancreatic endocrine cells at 18.5 days post coitum (d.p.c.), when definitive islets first begin
20 mouse embryos ranging from 8.5 to 11.5 days post-coitum (d.p.c.), Pitx3 mRNA was seen only in the de
21 us mutation is embryonic lethal by 13.5 days post coitum, demonstrating the importance of Opa1 during
22 n of FGF signalling between 5.5 and 7.5 days post-coitum does not block neural differentiation in the
23 Lhx4 expression in RP epithelium at 9.0 days post coitum (dpc) and total loss of pituitary tissue by
24 able from wild-type embryos through 6.5 days post coitum (dpc) and were able to establish all three g
25 The mouse germ line originates at 6.5 days post coitum (dpc) in the proximal epiblast, apparently i
27 correctly induced and patterned at 7.5 days post coitum (dpc), but subsequently fails to develop.
28 isplay profound yolk sac defects at 9.5 days post coitum (dpc), including disrupted angiogenesis in m
29 expression of Gm114 begins at 12.5-13.5 days post coitum (dpc), the stage in mice when germ cells cea
30 omparable in size with controls at 13.5 days post coitum (dpc), their placentas were significantly la
31 XY gonad decline in numbers after 11.5 days post coitum (dpc), while germ cell numbers in XX gonads
42 njection of PAF or SP-A into AF at 17.5 days post coitum enhanced uterine NF-kappaB activation and co
44 (+)Thy1(-)) that, when sorted from 15.5 days post-coitum fetal bones and transplanted under the adult
45 The transgene was expressed from day 7.5 post coitum forward, resulting in activation of skeletal
46 transfer, but none developed beyond 8.5 days post coitum; however, a high percentage of enucleated oo
47 nt tissues was observed beginning at 16 days post coitum in developing brain neurons, primitive inner
48 n on mouse embryos ranging from 9 to 16 days post-coitum localized murine Og12x mRNA in the heart, ot
50 ions transiently, and declined from 6.5 days post coitum onward, as the cells underwent apoptosis dur
52 tion within the cardiogenic plate of 7.5-day post coitum (p.c.) embryos, and in 8.5-day p.c. embryos
53 rominent through the looping stage (day 12.5 post coitum [pc]) but fell below the threshold of detect
54 mical analysis of murine embryos at 7.8 days post coitum revealed that Csx protein is strongly expres
56 rly anterior neural plate, but from 8.5 days post coitum they developed severe forebrain and midbrain
57 Rent1(-/-) blastocysts isolated at 3.5 days post-coitum undergo apoptosis in culture following a bri
58 culoskeletal system from E13.5 to E15.5 days post-coitum, where it was co-localized with hyaluronan.
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