ライフサイエンスコーパス: post infection

1 (DEGs) was highest during late mycosis (72 h post-infection).

2 de follicles throughout the follow-up (240 d post-infection).

3 for ROCK-mediated cell contraction from 2 hr post infection.

4 related with decreased lung function 21 days post infection.

5 pression and cell surface markers 8-16 hours post infection.

6 ely, CFU was significantly lower by 48 hours post infection.

7 etioles of wild-type and coi1 plants at 10 d post infection.

8 acked vasculitis and fibrosis 15 to 120 days post infection.

9 ia-infected mice lived until at least day 30 post infection.

10 ant compared with control strains at 21 days post infection.

11 extravasating pial vessels as early as 1 day post infection.

12 ymal vessels were not observed until 5 weeks post infection.

13 vival) when administered between day 0 and 3 post infection.

14 n C. jeuni numbers in the ceca by nine weeks post infection.

15 ge of all IBVs tested when administered 72 h post infection.

16 TmCactin expression were detected at 9 hours post infection.

17 ed from VV-infected cells at 1 hr and 3.5 hr post infection.

18 a slight reduction in activity over a decade post infection.

19 pecimens from inoculated bats from 5-19 days post infection.

20 id not affect viral contents at 4 and 8 days post infection.

21 neutrophil trafficking to the brain at day 8 post infection.

22 pha, and cyclooxygenase-2 expression at 24 h post infection.

23 animals is tracked as a function of the time post infection.

24 gained a numerical advantage as early as 8 h post infection.

25 ressed both PRRSV and PCV2 infection at 12 h post infection.

26 d only from donor hosts between 3 and 9 days post infection.

27 ] vs 413.5 [70.5], p=0.0476) in BAL at day 7 post infection.

28 arly between the groups of 4, 7, and 60 days post infection.

29 H1N1 and were followed up for up to 21 days post infection.

30 ncentration significantly decreased at day 3 post infection.

31 er survival and less weight loss at 24 hours post infection.

32 (TLR) expression were measured at 6-20 weeks post-infection.

33 expression was completely absent by 6 months post-infection.

34 ly reached the titre of wild-type virus late post-infection.

35 7 was silenced by approximately 85% on day 3 post-infection.

36 ining the bacterial loads at different times post-infection.

37 rnea when applied topically starting 8 hours post-infection.

38 Both models are monitored for up to 5 d post-infection.

39 sacs infected with R. prowazekii at ten days post-infection.

40 iminated from peripheral blood within 3 days post-infection.

41 l activation and decreased humoral responses post-infection.

42 orders of magnitude above base-line 24-48 h post-infection.

43 observed only when BIP was added within 3 h post-infection.

44 y, we observed an increase in apoptosis 48 h post-infection.

45 P. berghei-infected mice on days 3, 4, and 5 post-infection.

46 became predominantly hypophosphorylated 48 h post-infection.

47 igens and the antibodies lasted out to 52 wk post-infection.

48 clining to 50% of control levels by 16 weeks post-infection.

49 ame rounded within 1-2 h and detached by 5 h post-infection.

50 but the expression was downregulated by 12 h post-infection.

51 ered by virus and dsRNA at three time-points post-infection.

52 acquire influenza antigen in the lungs early post-infection.

53 k-out mutant for analyses at different times post-infection.

54 NAs) of ileal tissues collected at one-month post-infection.

55 tly reduced by 6.5-11.4% beginning in week 3 post-infection.

56 Fusobacteria was only present 12 h post-infection.

57 after therapeutic administration up to 24 h post-infection.

58 ange was seen when IBRV-A4 was added 2 hours post-infection.

59 tralization titers at day one, three and six post-infection.

60 of measuring virus neutralization at 6 hours post-infection, a duration likely confined to a single v

61 ar in both study groups, peaking by 10 weeks post-infection, a higher plateau (set-point) being achie

62 ion when treatments were initiated nine days post-infection, a time when animals were demonstrating a

63 genes, such as Irf7 and Cxcl9; 7 and 60 days post infection, acquired immunity-related genes, such as

64 Finally, topical and post-infection administration of rolipram into the middl

65 recovery of infectious virus 28 days or more post infection and has been a useful construct for exper

66 related host response and elevated SA levels post infection and in the down-regulation of jasmonic ac

67 vel of PML protein was expressed within 24 h post-infection and a detectable level remained at day 16

68 become committed to a B-blast fate <12 days post-infection and are unable to de-repress p18INK4c or

69 rhans cells were first detected at about 2 y post-infection and comprised about one-third of the Lang

70 pacts proIL-1beta expression as early as 2 h post-infection and delays activation of AIM2 and NLRP3-i

71 infection activates PKR at very early times post-infection and despite the presence of E3 and K3.

72 cally treated with MAb F429 starting 8 hours post-infection and evaluated for bacterial levels and co

73 s of gene expression were detected at 3 days post-infection and increased over time, suggesting this

74 atrilysin mRNA levels were detectable at 3 h post-infection and peaked at a 25-fold induction between

75 Protein synthesis ceases at late times post-infection and the translation initiation factor eIF

76 conditions (mouse and bacterial strain, time post infection) and was validated in outbred mice (AUC>0

77 activation during progression (days 6 and 12 post-infection) and regression (days 20-34 post-infectio

78 s were seen at the earliest time points (4 h post-infection) and the number of differentially express

79 rus and persists at least through day eleven post infection; and (4) the transient nature of the infe

80 fection/disease via immunization and less on post-infection antibiotics.

81 tor T cell phenotypes at various time points post infection as promising indicators of infection outc

82 increase in fungal burden beginning 2 weeks post-infection (as compared with mice infected with urea

83 % probability of presentation within 3 hours post-infection, as observed experimentally.

84 PRV-IR neurons were first observed on day 3 post-infection at L6 in the SPN.

85 Compared to day 1 post infection, bacterial colony counts, BALF total cell

86 Our results demonstrated that at 48 hours post-infection Brucella adjusts its metabolism in order

87 splayed minimal bacterial clearance for 1 mo post-infection but eventually resolved genital tract inf

88 irus decayed significantly from 1 to 2 weeks post infection, but decreased minimally thereafter.

89 t 5 weeks post infection followed at 9 weeks post infection by infusions of a primatized monoclonal a

90 y expressed genes at one or more time points post infection compared to the day 0 baseline.

91 ificant attenuation in BALB/c mice at 1 week post infection compared with the virulent parental strai

92 were not statistically different at 18 days post-infection compared to uninfected animals indicating

93 strate increased PKR expression through 96 h post-infection, concomitant with an increase in eIF-2alp

94 Gross findings post-infection confirmed extensive hemorrhage in the lun

95 lectron microscopy structures of the pre and post-infection conformations of the virus.

96 nd DbpB were only seen at the 1 h time point post infection, consistent with its low infectivity phen

97 rpoD transcripts were detected at all times post-infection, consistent with their expected function

98 03 expression in the crypts at days 6 and 12 post infection correlated with reduced levels of its tar

99 riend retrovirus (FV) replication at 2 weeks post-infection correlated with stronger natural killer,

100 lation, ZIKV RNA is detected in plasma 1 day post infection (d.p.i.) in all animals (N=8, including 2

101 e and analyzed at multiple time points up to post-infection day 20.

102 ype (WT) and Tetherin KO mice at 3 to 7 days post-infection despite removal of a potent restriction f

103 dissected syncytial cells at 3, 6 and 9 days post infection (dpi) during the course of the resistant

104 m samples were collected at 7, 14 and 21 day post infection (DPI) from infected and control mice and

105 n H129, and at daily intervals up to 16 days post infection (dpi) rose bengal or lissamine green B wa

106 levels of host immune-related genes at 1 day post infection (dpi) were higher in H5N8-infected than H

107 At 3, 4, or 5 days post infection (dpi), both optic tracts (OT) were dissec

108 enza A virus at 0, 6, 10, 14, 21 and 28 days post infection (dpi), representing the major stages of I

109 isolated from draining lymph nodes at 2 days post infection (dpi).

110 tion, the tissue FA depots depleted at 3days post-infection (DPI) and were then restored to their pre

111 d from infected rats were elevated by 4 days post-infection (dpi), but were not elevated in serum and

112 isera per animal, at 30 minutes, 1 or 2 days post-infection (dpi), in which all animals survived.

113 ng FcgammaRs were expressed maximally at 5 d post-infection (dpi), while the inhibitory receptor (Fcg

114 entical adsorption kinetics in the first 2 h post infection, EhV-207 gained a numerical advantage as

115 l of 62 miRNAs were differentially expressed post infection (false discovery rate <0.1).

116 At 24 h post infection, fluticasone treatment suppressed rhinovi

117 p study and who had a median of 24 months of post-infection follow-up.

118 a 90-day course of ART initiated at 5 weeks post infection followed at 9 weeks post infection by inf

119 ted the highest titers at three and six days post-infection for the type A viruses and lower titers f

120 es for prophylaxis, and 24, 48, and 72 hours post-infection for treatment.

121 ta), Wnt and Notch pathways on 12 or 34 days post infection formed monolayers in vitro, and underwent

122 hallenge (day 0) and at 4, 7, 11 and 14 days post infection from 44 pigs revealed 6,430 differentiall

123 re sampled and cultured at 6, 13 and 20 days post-infection from the 4 groups.

124 inning at 2 weeks and continuing for 6 weeks post infection, however, GR106 and 2308 displayed equiva

125 By 3-4 wk post infection, however, iNOS knockout mice did succumb

126 llowed in intact leaf tissue from 12 to 36 h post infection (hpi) by using confocal microscopy.

127 ins (ICPs) synthesized between 3 and 6 hours post infection (hpi).

128 ed with 25 mg/kg of either L or V at 2 hours post infection (hpi).

129 nduced > or =2-fold in HFF between 2 and 6 h post-infection (hpi) in repeated experiments while immed

130 gnal response detected as early as ~30 hours post-infection (hpi).

131 ft towards stromal staining at peak (12 days post infection) hyperplasia, whereas staining for Lgr5 a

132 ly infected with HSV-1 and surveyed at times post infection in the nervous system and lymph node for

133 al plasma samples collected over three years post-infection in a cohort of MSM HIV-1 seroconvertors,

134 ore, expression of Stmn2 was reduced 30-fold post-infection in a mouse cellular model of prion diseas

135 d therapeutic efficacy when dosed up to 18 h post-infection in the pneumonia model.

136 breadth of an infant at approximately 1 year post-infection, including one with cross-clade breadth.

137 ted in Arabidopsis root tips as early as 6 h post infection, indicating that ET signaling was activat

138 the separation were as follows: 4 and 7 days post infection, innate immunity-related genes, such as I

139 Analysis of shorter post-infection intervals showed that levels of viral RNA

140 Three weeks post infection, intestinal tissue samples were collected

141 mes are completely converted, within 2 weeks post-infection, into non-rearranged circular concatamers

142 Five days post-infection, labeling was seen in the IML and lamina

143 Three days post-infection, mice were given saline or ethanol (5 g/k

144 animals sacrificed between 16 and 44 months post infection (mpi) were examined for the presence of t

145 ognized for its association with the serious post-infection neurological complications of the Miller-

146 In the lumbosacral cord, 3 days post-infection, neurons labeled by virus from the extern

147 We show that at later times post-infection NF-kappaB activation is blocked in reovir

148 s constructed from Arabidopsis at 6 and 14 h post infection of non-pathogenic, virulent and avirulent

149 er liter of culture medium collected at 72 h post-infection of BTI-Tn-5B1-4 cells with the basic prot

150 cellular bacteria during the first few hours post-infection of macrophages but the expression was dow

151 Ninety-six h post-infection of Sf9 insect cells with recombinant bacu

152 By 7 days post-infection of sponge implants in Flk-1(LacZ) knock-i

153 m Legionella pneumophila were induced at 4 h post-infection of the U937 macrophage-like cells.

154 h the largest difference of 100-fold at 72 h post infection (p.i.) and peak titers decreased by appro

155 our group II animals died by 8 and 10 months post infection (p.i.), all four group III animals remain

156 The mice were euthanized 10 days post infection (p.i.), and the urethra, bladder, epididi

157 ice exhibited reduced weight loss at 15 days post-infection (p < 0.01) and demonstrated reduced histo

158 ghly discriminatory for sera taken from pigs post-infection (P < 0.05) indicating that these peptides

159 imary HIV infection (PHI) and up to one year post-infection (p.i).

160 r C. trachomatis and harvested at t = 0-48 h post-infection (p.i; active).

161 Variation in post-infection parasite growth and killing time depended

162 2 post-infection) and regression (days 20-34 post-infection) phases of TMCH.

163 a persistent cytopathic infection at 21-day post infection (PI) based upon the quantitative detectio

164 oducible distinct succession of peaks at 12h post infection (PI), 23h PI and 31h PI and local minima

165 By day 28 post infection (PI), 5-HT turnover had normalized, but 5

166 ected in a patchy fashion beginning on day 7 post infection (pi), and the infection progressed to inv

167 e were elevated significantly at 1 to 3 days post infection (PI), peaked at 3 and decreased at 5 days

168 diographically and grossly apparent by day 7 post infection (PI).

169 ory pathways was examined following 2-5 days post-infection (PI) and the virus was located immunocyto

170 experiments, vancomycin was injected 4 hours post-infection (PI) for each passively immunized group.

171 infection were observed as early as 12 hours post-infection (PI) in osteoblasts, skeletal muscle myoc

172 on of neoplastic lesions in vitro at 27 days post-infection (PI), providing new evidence of the role

173 at HC-23 mRNA levels decreased from 6 to 12h post-infection (pi), were maximally expressed at 24h pi,

174 tarted on day 15 and was completed by day 35 post-infection (pi), whereas the KO mice remained partia

175 male and female mice at days 90, 150 and 180 post-infection (PI).

176 ation, there is a transient period (day 1-20 post-infection) prior to the establishment of persistenc

177 activation of mTORC1 occurs as early as 8 h post-infection, prior to AC formation.

178 eloped severe colitis and hepatitis by 10 wk post-infection, progressing into colon carcinoma by 20 w

179 that the qRT-PCR signal detected at 6 hours post-infection reflected an amplification of at least 10

180 296, 400, to 1086 at 24, 48, 72 and 96 hours post infection, respectively).

181 ype B viruses in MDCK cells before day three post-infection, resulting in the systematic underestimat

182 sessment of the LCMV-specific memory at 65 d post infection revealed many more LCMV-specific WT memor

183 O and WT CD4 and CD8 T cell responses at 8 d post infection revealed modest but significant differenc

184 d nucleotide reductase NrdEF was observed in post-infection samples compared to the pre-infection sta

185 necrotizing fasciitis and myositis, and the post-infection sequelae glomerulonephritis and rheumatic

186 sults observed for neutralization at one day post-infection showed MDCK cells were similar (<1 log(2)

187 etion was noted for periods of up to 6 weeks post-infection, significantly longer than that which occ

188 By six weeks post infection, SIV-infected cells were no longer detect

189 At 0, 24, 72, and 144 hours post-infection, spleen and lung samples were removed for

190 epresents the pre-infection state, while the post-infection state is thick.

191 III secretion (T3S) in the RB stage at 18 h post infection, suggesting a reduced T3S capacity or a l

192 res unable to transmit malaria within 5 days post-infection, surpassing the World Health Organization

193 ls to neonatal CNS infection with PVC-211 at post-infection survival times 7, 14, 21, and 28 days.

194 ated from rotavirus-infected cells six hours post-infection (termed in vivo 6 hr large RNAs), also ac

195 are induced but rapidly wane and by 3 months post-infection the bats are seronegative.

196 he A protein is absent from the virus shell (post-infection), the process of crystallization exerts s

197 After either 4- or 5-day survival period post-infection, the rats were euthanized by transcardial

198 ced its presence in the sera, over ten years post infection; the prevalence of other GP-specific IgG

199 -infected cells were detectable at two weeks post infection, their abundance increased with time, and

200 were infected with Mp and, twenty-four hours post infection, their host defense responses were compar

201 By day 5 post infection, there was a significant loss in T, NK, a

202 the lymph node of both species at two weeks post infection, they were confined to the lymph node par

203 force to convert the protein shell from the post-infection (thick) state to the pre-infection (thin)

204 ared whole-blood RNA-seq profiles at pre-and post-infection time points from Malian adults who were e

205 ase, but generally remaining elevated at all post-infection time points.

206 ency virus (SIV)mac239 genomes at four weeks post-infection to determine the extent of acute CTL esca

207 At 6 days post infection, transcription of viral genes was detecte

208 herapy, such as pre-exposure prophylaxis and post infection treatments.

209 e vs. early chronic infection (7 vs. 28 days post-infection), using neurological and behavioral pheno

210 tinct immunological correlates compared with post-infection virological control and required the incl

211 V-1) vaccine candidates have typically shown post-infection virological control, but protection again

212 ly distributed within the cytoplasm even 6 h post-infection, VLPs comprising both L1 and L2 exhibited

213 FTL_0325 mutant-infected macrophages at 24 h post-infection was independent of both AIM2 and NLRP3.

214 encephalomyelitis virus on 4, 7, and 60 days post infection, we conducted bioinformatics analyses, in

215 e expression changes over the first 12 hours post-infection, we developed a statistically rigorous en

216 peak infection density in gut at 10-12 days post-infection when blood viral loads were low.

217 icles per Coxiella call was observed at 12 h post-infection when compared with the starting inoculum.

218 ASC response was obviously detected on day 7 post-infection when the virus was completely cleared fro

219 were not readily detectable by ET at 5-days post-infection, whereas HIV-1-infected cells surrounded

220 oHV-1 infected MDBK cells at 0 and 0.5 hours post-infection, whereas no change was seen when IBRV-A4

221 M1 classical macrophage activation two days post infection, which has the collateral effect of suppr

222 at the wild type persists in mouse lung 24 h post infection while the mutant strain is cleared by hos

223 monary CD11c(+) cells revealed that at day 7 post infection, wild-type cells up-regulated type-I IFN-

224 replication is readily detectable by one day post infection with HBV baculovirus and persists at leas

225 specimens from rhesus macaques prior to and post infection with pathogenic SIVmac239.

226 mutated ancestor emerged between weeks 30-38 post-infection with a 35-residue CDR H3, and neutralized

227 ssue were assessed over a period of 16 weeks post-infection with ASRV.

228 he culture medium of BTI-Tn-5B1-4 cells 72 h post-infection with the basic protein promoter-uPA-IgG v

229 ntigen levels increase up to 2-fold at day 3 post-infection with toxigenic Sterne 34F(2) strain, wher

230 n, progressing into colon carcinoma by 20 wk post-infection, with pronounced pathology in the cecum a

231 1 in early infection and 0 around two years post infection, yielding MDRI of 420 [361, 467] days.