ライフサイエンスコーパス: post injection

1 ocytes reaching the tumour (P < 0.001, day 7 post-injection).

2 nd ketamine restored pJAK2 levels within 2 h post injection.

3 body weight) and were sacrificed at 2 or 6 h post injection.

4 ed prior to LPS/placebo injection, 2 and 5 h post injection.

5 ociated retinal or anterior segment toxicity post injection.

6 njection, which were partially recovered 5 h post injection.

7 1 antibody concentrations in the plasma 24 h post injection.

8 temic exposure and increased local retention post injection.

9 %) achieved adhesion release at mean 10 days post injection.

10 istal tubules) and sustained beyond 2 months post injection.

11 essitating euthanasia of all mice by 15 days post injection.

12 o induction of neuropathology out to 6 weeks post injection.

13 Two eyes were enucleated 6, 24 and 72 hours post injection.

14 ery long with still 66% remained on 12th day post injection.

15 one significantly decreased T(IBAT) up to 3h post injection.

16 vel at 10 weeks, and lasted up to six months post injection.

17 ach conjugate per mouse) at 1, 7 and 13 days post-injection.

18 chronic-relapsing EAE followed for 120 days post-injection.

19 sion at 24 h, and an increase at 48 and 72 h post-injection.

20 g sc) failed to increase BLA CRF-BP mRNA 9 h post-injection.

21 uisition of a leverpress avoidance task 24 h post-injection.

22 g, im) evoked hypothermia that peaked 30 min post-injection.

23 bjective was safety and tolerability 28 days post-injection.

24 ation, one at 60 min and another at 180 min, post-injection.

25 hich was rapid in onset and peaked 45-60 min post-injection.

26 LS unit activity, peaking at about 15-20 min post-injection.

27 Food intake was determined at 2, 4, and 24 h post-injection.

28 spreading depression (SD) as early as 20 min post-injection.

29 aining diet showed an increase in intake 4 h post-injection.

30 Food intake was suppressed up to 4 days post-injection.

31 icle and were killed at 15-, 60- and 120-min post-injection.

32 e returned to baseline by approximately 24 h post-injection.

33 by 1 week, and persisted for up to 3 months post-injection.

34 ere taken at various times from 3 to 120 min post-injection.

35 al allodynia was observed between 3 and 12 h post-injection.

36 prived rats during the first and second hour post-injection.

37 latencies were attained between 10 and 12 h post-injection.

38 days after surgery and continued for 90 days post-injection.

39 howed significant differences up to 12 weeks post-injection.

40 duction of drusen-like deposits by 2 months' post-injection.

41 e products are present for at least 18 weeks post-injection.

42 le hyperphagia occurred sometimes after 48 h post-injection.

43 ent, that recovered by approximately 5 weeks post-injection.

44 group counted spike-waves over a 4 h period post-injection.

45 levels, with peak decreases occurring 2-3 h post-injection.

46 ression bilaterally in granule cells at 24 h post-injection.

47 bicin followed by LSFI at 3, 30, and 60 days post-injection.

48 motor impairment which lasted for 15-30 min post-injection.

49 SA/siRNA) compared to 4% (naked siRNA) 6days post-injection.

50 glioma brain compared to normal brain at 24h post-injection.

51 aged Ins2(Akita) mice even after 3months of post-injection.

52 per g of brain (%ID/g) within the first hour post-injection.

53 2, through endochondral ossification 6 weeks post-injection.

54 d FLLs were calculated and compared pre- and post-injection.

55 ction on the same day, between 1 and 14 days post-injection.

56 go in response to UV exposure up to 30 weeks post-injection.

57 ved wall motion relative to placebo 3 months post-injection.

58 ad accumulated in the U87MG tumor after 24 h post-injection.

59 s revealed stable gene expression at 7months post-injection.

60 with the greatest reduction observed at 12h post-injection.

61 RK was observed in glial cells as soon as 3h post-injection.

62 and total white blood cell count by 6 hours post-injection.

63 ells and macrophages) starting at around 24h post-injection.

64 triatum of active vector recipients 6 months post-injection.

65 ing programmed electrical stimulation 1 week post-injection.

66 n emission tomography images as late as 24 h post-injection.

67 creased shock-startle response 30 and 60 min post-injection.

68 cific imaging and renal clearance within 4 h post-injection.

69 efficiently cleared from the host by 20 days post-injection.

70 ssion of osseous metastases out to six weeks post-injection.

71 ebellar regions of interest (ROIs) 40-60 min post-injection.

72 ls could be visualized by SPECT up to 6 days post-injection.

73 After 1-3 days post-injection (1-3 dpi) of BAG into the deep cerebellar

74 e 33 genes were influenced by r-bursicon 3 h post-injection (24 up-regulated and 9 down-regulated gen

75 By 48 h post-injection, Abeta42-positive neurons were widespread

76 After approximately 30 days post-injection, aggregated A beta injection detrimentall

77 of a depot that released curcumin over 4days post-injection and decreased plasma AUC 7-fold.

78 m channel labeling began to increase at 23 h post-injection and reached maximum levels by 24 h.

79 ak responses (1.0 microgram) occurred 40 min post-injection and represented a 16-26-fold increase ove

80 y gene product expression peaked at 4 to 6 h post-injection and then declined to baseline.

81 centration of approximately 20 microM 15 min post-injection and was eliminated from plasma with a ter

82 This event began by 15 min post-injection and was maximal by 45 min.

83 gration was evident in some regions by 1 day post injection, and many newborn cells coexpressed the n

84 ailability in the infarct/border zone at 24h post-injection as compared with free AZ7379.

85 s including tremors, which lasted up to 24 h post injection, at submicrogram amounts.

86 Each mouse was sacrificed at 1-h post injection, at which time brains were removed, snap

87 k tissue, the HHR3 levels decline up to 21 h post-injection before rising to basal levels after 48 h.

88 e peptides bound amyloid deposits within 1 h post-injection, but the extent of the reactivity differe

89 and retinal expression was analyzed 4 weeks post-injection by qRT-PCR and histology.

90 inophil accumulation and preceded at 3 hours post-injection by significant increases in hepatic T hel

91 Twelve weeks post injection, cell-treated hearts showed preserved ECM

92 sufficient time for recovery (minimum of 3 h post injection), cellular activity was monitored for an

93 GdDOTA-CTB was visible for at least 1 month post-injection, clearing within 2 months.

94 old greater decrease in scar mass at 8 weeks post-injection compared to hCSCs (-29.2 +/- 2.7% vs. -8.

95 shrimp was extremely reduced at days 2 and 3 post-injection compared with uninfected shrimp but was f

96 pattern of binding proteins changed at 16 h post-injection, concurrent with enhanced estrogen recept

97 emonstrated that free DNR in the vitreous at post-injection day 14 was 66.52ng/mL for 95nm pore size

98 Even 68-h post-injection, decorin was found to reside within the t

99 lls arrive at the glomerular layer after day post injection (DPI) 7.

100 Spleen samples were collected at 40 days post injection (dpi), and sequenced.

101 naptic currents beginning approximately 14 d post-injection (dpi).

102 Forty-eight hours post injection, dramatic increases of TRH over control l

103 At 4 h post-injection, enzyme activity was retained in the live

104 ntly suppressed intake during the first hour post-injection following administration into six hypotha

105 such as good tumor uptake (3.69%ID/g at 2 h post-injection), high tumor contrast, and specificity we

106 We examined whether delayed post-injection imaging of a new ultrasound contrast agen

107 onal damage in KA-treated hippocampi at 16 h post-injection in both maternal and virgin rats.

108 2 mRNA expression appeared ex novo at 0.5-h post-injection in cells closely associated with blood ve

109 S and corner turn tests at 14, 21 and 28days post-injection in each treatment group (P<0.05) as compa

110 e brain 30 min post-injection ip. but not 2h post-injection in rats.

111 uptake in cardiac tissue (3.41%ID/g; 30 min post injection) in addition to favorable heart to nontar

112 thetic premotor areas were labeled by 6 days post-injection, including the rostral ventrolateral medu

113 red LPS bolus elicited an early (over 15 min post-injection) increase in brain ECF IL-1beta concentra

114 l and ratio of Ki67-positive CMs at 3 months post injection indicated engrafted CMs proliferated in t

115 GRP also stimulated feeding but only after a post-injection interval of 10 h.

116 ight, and water intake were measured at 24 h post-injection intervals.

117 jection increased levels in the brain 30 min post-injection ip. but not 2h post-injection in rats.

118 ls, and (iii) significant risk reduction for post-injection leakage by geological, gravitational, and

119 By 12h post-injection, LPS-treated mice exhibited activated as

120 In post-injection mice, we observed a strong inverse correl

121 Strikingly, by 3 months post injection, micro-dystrophin was still expressed to

122 ere euthanized at 1 h, 6 h, and 1, 3, 7 days post-injection (n=4 or 5/group/time point).

123 ve targeting alone (7.7 +/- 0.1%ID/g at 16 h post-injection; n = 3).

124 gnificantly abrogated (2.3+/-0.5%ID/g at 48h post-injection; n=3) when mice were pre-injected with a

125 XPC-3 tumors (peak at 31.5+/-6.0%ID/g at 48h post-injection; n=3), which was significantly abrogated

126 week and >10 nM in 3 of 4 patients 12 weeks post injection of 0.4 mg.

127 Subjects were imaged at 24 hr post injection of 360 MBq (9.7 mCi) of [123I]beta-CIT.

128 ich was confirmed after examination of somas post injection of a retrogradely transported antibody to

129 tal arrest with larval mortalities up to 73% post injection of dsRNA.

130 upper air way infection was observed 2 days post injection of the second eye.

131 ission tomography (PET) images were acquired post-injection of free (18)F-FDG/(18)F-FLT or (18)F-FDG/

132 dopamine turnover were increased immediately post-injection only by combined exposures, and returned

133 From approximately day 50 post-injection onward, A beta-injected subjects demonstr

134 As early as 90min post injection, ova-micelle conjugates were associated w

135 significantly higher than PYY3-36 up to 24 h post injection (p=0.0008 at 4 h, p=0.0028 at 24 h).

136 iata was observed to rise and peak at 30 min post-injection (p.i.) and declined with clearance half-l

137 eling was found in the spinal cord at 2 days post-injection (p.i.).

138 e obtained at 5 min, 0.5, 1, 2, 4, 8 and 24h post-injection (p.i.).

139 ated gastric contractility for up to 120 min post-injection, P < 0.05.

140 The increase was evident by 6 h post-injection, peaked at 24 h with a 4-fold increase, a

141 of BrdU labeling matched those of the 4 week post injection (pi) groups, suggesting that proliferatin

142 were killed at 12, 24, 36, 48, and 72 hours post injection (pi), the eyes were enucleated, and froze

143 nd 8 hours, and 1, 3, 7, 10, 14, and 60 days post injection (PI).

144 in an rpe65(-/-) model of LCA up to 6 months post-injection (PI), however the duration of this treatm

145 s of caspase activity than controls 24 hours post injection, providing biochemical evidence that inhi

146 jection and persisting to the end of the 6 h post-injection recording period.

147 re and self-reported fatigue, and depression post injection relative to baseline and placebo.

148 -to- cerebellum ratios (4.6 at 25 and 37 min post injection, respectively) and reversible binding in

149 lum ratios of 3.41, 3.24, and 3.00 at 85 min post-injection, respectively.

150 Controls and 1 year post-injection retinas were double labeled for protein k

151 Twenty-four hours post injection, saline maternal animals exhibited superi

152 DHS pups exhibited post-injection seizures, which were non-existent in sali

153 In all eyes there was post-injection spread of the retinal detachment within t

154 xpression involved retina beyond the initial post-injection subretinal bleb boundary.

155 ll molecule angiogenesis inhibitor, 10 weeks post-injection suppresses choroidal neovascularization (

156 Post-injection survival was 2, 4, 8 h, 2 days, 2 weeks,

157 t organs and background, such that by 10 min post-injection the pancreas is the most prominent organ

158 equences in TA muscle genomic DNA by 4 weeks post injection, the levels of which were found to increa

159 At 3h, 5h, and 12h post-injection, the morphology of the SN was assessed us

160 (10 mg/kg, s.c.), and sacrificed at various post-injection times to monitor the recovery of receptor

161 At 24 h post-injection, total enhanced green fluorescent protein

162 Although beta cell mass was preserved 8 days post-injection, total insulin content and insulin:chromo

163 o an injected protein or peptide during this post-injection transition period could affect the diffus

164 intense regions in the liver up to two weeks post injection using 9.4 T MRI.

165 tion of efferent nerve fibers began 3-7 days post-injection, versus 15-30 days for SGNs, and the loss

166 Fuel post-injection was conducted in some tests to investigat

167 nistered GDNF (0.1-100 micrograms) at 7 days post injection were also examined.

168 c.), and connectivity changes 15- and 30-min post-injection were studied.

169 ed and a 36-fold increase in CT contrast 4 h post injection, which makes it possible to acquire CT im

170 gnificantly affecting reward sensitivity 2 h post injection, which were partially recovered 5 h post

171 -acetate and is visualized as early as 2 min post-injection, with maximal activity achieved by 5 min.