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1 premenopausal (age < 50 years) and 357 were post- menopausal.
3 ose-response associations in endometrial and post-menopausal breast cancer, and in degree and duratio
6 ncreased in patients with ABS, compared with post-menopausal controls, following acute mental stress
8 and PR isoform expression in normal pre- and post-menopausal endometrium, well-differentiated endomet
10 r being hospitalized or diagnosed with ABS), post-menopausal female controls (n = 12), and female pat
11 levels were low in the majority of males and post-menopausal females, but within normal limits for pr
13 y was a significant risk factor among women; post-menopausal hormones use was only associated with an
16 Grandmother Hypothesis to simulate how human post-menopausal longevity could have evolved as ancestra
17 icrobial-dose-doxycycline (SDD) treatment of post-menopausal osteopenic women significantly reduced p
19 above both eroded and formative surfaces in post-menopausal osteoporosis patients, and that this abs
26 usted p-value = 0.015) and more likely to be post-menopausal (p-value = 0.004; BH-adjusted p-value =
27 nificantly shorter survival, specifically in post-menopausal patients with advanced and terminal stag
30 gulates metabolic physiology, highlighted by post-menopausal temperature dysregulation (hot flashes),
32 Between 2001 and 2005, a total of 202 638 post-menopausal women aged 50-74 years were randomly ass
34 cardiomyopathy that occurs predominantly in post-menopausal women and may be triggered by acute ment
36 onary heart disease (CHD) risk prediction in post-menopausal women compared with assessment using tra
42 se, risk factors for sudden cardiac death in post-menopausal women include African-American race, hig
44 ex/hormone status was grouped as: 1) men; 2) post-menopausal women not receiving hormone replacement
45 d risk of future systemic bone loss in these post-menopausal women not yet on anti-osteoporotic drugs
47 in two measures of olfactory function in 14 post-menopausal women receiving estrogen replacement the
48 en not receiving hormonal contraceptives; 4) post-menopausal women receiving hormone replacement ther
52 window hypothesis, i.e., that the brains of post-menopausal women ultimately lose their ability to r
54 ouble-blind, double-dummy trial, we enrolled post-menopausal women with at least two moderate or one
55 tase inhibitor administered after surgery to post-menopausal women with hormonally responsive breast
57 d, double-blind, placebo-controlled trial of post-menopausal women with serum 25-hydroxyvitamin D con
59 n a preference-controlled trial involving 21 post-menopausal women, 16 weeks of supervised moderate i
60 with increased incidence of breast cancer in post-menopausal women, and with increased mortality from
61 n reduces the risk of Alzheimer's disease in post-menopausal women, beta-amyloid (Abeta) burden in an
62 ascular disease, the major cause of death in post-menopausal women, can be reduced by replacement of
63 n and -12.5 ml/yr (95% CI, -16.2 to -8.9) in post-menopausal women, compared with women menstruating
64 ssue, which are major sources of estrogen in post-menopausal women, could up-regulate hPRLR gene expr
65 was associated with decreasing HF risk among post-menopausal women, even in the absence of antecedent
66 declined more rapidly among transitional and post-menopausal women, in particular for FVC, beyond the
71 y benefits the outcome of cerebral stroke in post-menopausal women, we designed the present study to
72 cidence of HF hospitalization among healthy, post-menopausal women, whereas multivariable adjustment
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