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1 t hypotheses to account for the mechanism of post transcriptional gene silencing.
2 ) proteins that mediate RNA interference and post-transcriptional gene silencing.
3 oRNAs are small non-coding RNAs that mediate post-transcriptional gene silencing.
4 ory processes, including transcriptional and post-transcriptional gene silencing.
5 e cleavage of complementary mRNA, leading to post-transcriptional gene silencing.
6 nificant contribution to transcriptional and post-transcriptional gene silencing.
7 NA (siRNA) molecules are potent effectors of post-transcriptional gene silencing.
8 scriptional activator and/or a suppressor of post-transcriptional gene silencing.
9 of the transcribed region is associated with post-transcriptional gene silencing.
10 A interference (RNAi) is a powerful tool for post-transcriptional gene silencing.
11 rocessing, transcriptional co-activation and post-transcriptional gene silencing.
12 zoa, animals, plants and fungi, resulting in post-transcriptional gene silencing.
13 synthase, a process called cosuppression or post-transcriptional gene silencing.
14 that the DNA itself is a potent activator of post-transcriptional gene silencing.
15 nce to further infection, factors typical of post-transcriptional gene silencing.
16 ayed several phenotypes that we attribute to post-transcriptional gene silencing.
17 ) of the N gene displayed resistance through post-transcriptional gene silencing.
18 NA interference (RNAi) reagents for directed post- transcriptional gene silencing.
19 ns in eukaryotes are known as key players in post-transcriptional gene silencing(1), while recent stu
20 NAs (siRNA) are potent reagents for directed post-transcriptional gene silencing and a major new gene
22 genes implicate cellular energy homeostasis, post-transcriptional gene silencing and fatty-acid amide
24 of endogenous, non-coding RNAs that mediate post-transcriptional gene silencing by inhibiting mRNA t
25 l and evolutionarily conserved phenomenon of post-transcriptional gene silencing by means of sequence
26 ssues (with the probable involvement of some post-transcriptional gene silencing by pathways related
28 of transformants spontaneously triggers Nia post-transcriptional gene silencing (class II) whereas a
29 ile microRNAs (miRNAs) generally function in post-transcriptional gene silencing in both somatic and
31 , in addition to well-characterized roles in post-transcriptional gene silencing in the cytoplasm.
36 d with the potential of transgenes to confer post-transcriptional gene silencing, is a cause or a con
37 ), the double-stranded RNA (dsRNA) triggered post-transcriptional gene silencing, is becoming a power
38 of "sensing" activates meiotic silencing, a post-transcriptional gene silencing-like mechanism that
39 These responses, called RNA interference or post-transcriptional gene silencing, may provide anti-vi
40 and consequent gene silencing, and (ii) the post-transcriptional gene silencing mechanism can trans-
41 n evolutionarily conserved sequence-specific post-transcriptional gene silencing mechanism that is we
45 nown variously as RNA interference (RNAi) or post-transcriptional gene silencing, mediates resistance
47 ion of miRNA, molecular silencers that enact post-transcriptional gene silencing of coding genes.
48 MA) tumor model was used to demonstrate that post-transcriptional gene silencing of dicer, the enzyme
49 stability and translation, and siRNAs cause post-transcriptional gene silencing of transposons, viru
50 Our results define the core components of a post-transcriptional gene silencing pathway in Arabidops
54 with a transgene and the ability to promote post-transcriptional gene silencing (PTGS) and transgene
55 tion can be caused by many factors including post-transcriptional gene silencing (PTGS) and/or rapid
56 In plants, both transcriptional (TGS) and post-transcriptional gene silencing (PTGS) can be self-r
57 on') reproducibly and consistently activates post-transcriptional gene silencing (PTGS) in every plan
58 ediated defence mechanism that is related to post-transcriptional gene silencing (PTGS) in transgenic
65 the basis of genetic studies appears to be a post-transcriptional gene silencing (PTGS) mechanism.
68 ic tobacco plants expressing a suppressor of post-transcriptional gene silencing (PTGS) with a PVX am
69 st suppressors identified that could inhibit post-transcriptional gene silencing (PTGS), but with lit
70 ith inverted repeats can efficiently trigger post-transcriptional gene silencing (PTGS), presumably v
72 d small-interfering RNA (siRNA) pathways for post-transcriptional gene silencing (PTGS), whereas AGO4
79 results strongly suggest that miRNA-mediated post-transcriptional gene silencing relies primarily on
81 relate the processes of transcriptional and post-transcriptional gene silencing (TGS, PTGS) in persp
83 nce (RNAi) is an intracellular mechanism for post-transcriptional gene silencing that is frequently u
85 Argonaute (Ago) proteins, where they direct post-transcriptional gene silencing via base-pairing wit
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