ライフサイエンスコーパス: post-traumatic

1 3 multiple sclerosis [MS]; 6 post-stroke; 1 post-traumatic).

2 f proinflammatory mediators and inhibits the post-traumatic activation of JNK in a rodent model of TB

3 These changes were reversed with post-traumatic administration of recombinant human eryth

4 d further investigations were in favour of a post-traumatic aetiology.

5 Retrospective assessment of post-traumatic amnesia (PTA) must take into account fact

6 predominantly mild-to-moderate TBI (52% with post-traumatic amnesia (PTA)</=24 hours), but including

7 e novel insights into the pathophysiology of post-traumatic amnesia and evidence that memory impairme

8 traumatic brain injury were classified into post-traumatic amnesia and traumatic brain injury contro

9 the hypothesis that the mnemonic symptoms of post-traumatic amnesia are caused by functional disconne

10 pairments in associative memory, patients in post-traumatic amnesia demonstrated impairments in infor

11 Clinically, post-traumatic amnesia is an important predictor of func

12 unctional importance, the pathophysiology of post-traumatic amnesia is not understood.

13 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: Post-traumatic amnesia is very common immediately after

14 Patients in post-traumatic amnesia showed abnormal functional connec

15 Patients with post-traumatic amnesia showed evidence of widespread tra

16 normality was also observed in patients with post-traumatic amnesia.

17 ematic to generalize to disorders other than post-traumatic and acute-stress disorder.

18 Delayed presentation of post-traumatic aortic pseudoaneurysm and its fistulous c

19 n which we performed RPICIOL implantation in post-traumatic aphakia (141 eyes, group 1), post-catarac

20 evidence of IL-1 signaling as a mediator of post-traumatic astrogliosis and seizure susceptibility.S

21 ATP release, we here evaluated the impact on post-traumatic ATP release of deletion of connexins (Cx3

22 a contribution by astrocytic hemichannels to post-traumatic ATP release that aggravates secondary inj

23 for limiting the devastating consequences of post-traumatic autonomic hyperreflexia and post-injury i

24 lls by MHC-II on Schwann cells thus promotes post-traumatic axonal loss and neuropathic pain.

25 ts after repetitive surgery or who developed post-traumatic blepharoptosis.

26 cellular swelling and provide treatment for post-traumatic brain edema.

27 ntia, including Alzheimer's disease, and the post-traumatic brain frequently exhibits neurofibrillary

28 icacy of catecholaminergic drugs in treating post-traumatic brain injury cognitive impairments.

29 a cohort of 44 patients at least six months post-traumatic brain injury.

30 ter TBI may be a key factor that renders the post-traumatic brain vulnerable to tau aggregation and t

31 llary pathology and neurodegeneration in the post-traumatic brain.

32 n achieved despite repetitive surgery and in post-traumatic cases where levator muscle and aponeurosi

33 Imaging should be done in post-traumatic coma or when structural brain lesions are

34 To determine which post-traumatic complications impact these outcomes, we c

35 lly conscious state with language (n = 4) or post-traumatic confusional state (n = 4).

36 pression of the spinal cord and formation of post-traumatic cysts.

37 These findings indicate that post-traumatic decreases in miR-23a and miR-27a contribu

38 Our data show that cell-type-specific post-traumatic decreases in tonic GABA currents boost SG

39 Moreover, post-traumatic differences in SGC firing were abolished

40 ery during active treatment in patients with post-traumatic disorders of consciousness.

41 Depression is five times more common than is post-traumatic distress disorder after critical illness

42 1 at 12 months) had symptoms consistent with post-traumatic distress disorder.

43 spp. are common causes of post-operative and post-traumatic endophthalmitis.

44 ent nonconvulsive seizures characteristic of post-traumatic epilepsy (PTE).

45 the organotypic hippocampal culture model of post-traumatic epilepsy comprises a rapid assay of anti-

46 This study aimed to characterize post-traumatic epilepsy in a mouse model of pediatric br

47 t always model heritable absence epilepsy or post-traumatic epilepsy in humans, and may instead refle

48 Post-traumatic epilepsy is prevalent, often difficult to

49 rat hippocampal organotypic culture model of post-traumatic epilepsy was used to study the effects of

50 res (CPSs) following traumatic brain injury (post-traumatic epilepsy).

51 sy, but the mechanisms of mTOR activation in post-traumatic epileptogenesis are unknown.

52 pocampal in vitro preparations, during early post-traumatic epileptogenesis demonstrated rapid increa

53 of glia-mediated spatially localized HSP to post-traumatic epileptogenesis remains poorly understood

54 the organotypic hippocampal culture model of post-traumatic epileptogenesis.

55 e genotypes and chronicity of osteomyelitis, post-traumatic etiology, or with a specific bacterial et

56 Unresolved post-traumatic glial activation is a secondary injury me

57 rrence, satisfaction with facial appearance, post-traumatic growth, and post-traumatic stress symptom

58 Post-traumatic hypopituitarism (PTHP) can have major con

59 This post-traumatic immunosuppressed state has been attribute

60 romised fracture healing because of systemic post-traumatic inflammation.

61 nts, postoperative cardiac surgery patients, post-traumatic injury patients, and neurologic injury pa

62 in mice and investigated how this influenced post-traumatic intraneural inflammation and neuropathic

63 been described to enlarge in the presence of post-traumatic meningeal hemorrhages or deformities of t

64 adaptive circuit restructuring may mitigate post-traumatic morbidity.

65 lfonylureas, leading to the postulation that post-traumatic non-selective cation currents are determi

66 rgically induced knee OA in mice, a model of post-traumatic OA in humans, increased expression of VEG

67 ilage mechanotransduction in age-related and post-traumatic OA, and point to a novel disease-modifyin

68 of these cells attenuated the development of post-traumatic OA, reduced pain and increased cartilage

69 ed the spectrum of lesions characteristic of post-traumatic osteoarthritis (PTOA) across the knee joi

70 cytes and, therefore, may give rise to acute post-traumatic pain sensation via a yet elusive molecula

71 eceptors as valuable therapeutic targets for post-traumatic pain.

72 Patients who survive the initial trauma and post-traumatic resuscitation are at risk for immune dysr

73 tive deficits, and reduced the occurrence of post-traumatic seizure activity.

74 Early post-traumatic seizures are one potential mechanism for

75 rk events in the slice preparation and early post-traumatic seizures.

76 Participants in Balkan countries with post traumatic stress disorder (PTSD) had costs that wer

77 g disorders of anxiety and stress, including post traumatic stress disorder (PTSD).

78 last exposure had an antemortem diagnosis of post traumatic stress disorder.

79 sistence may be a model for some features of post traumatic stress disorder.

80 ng strategies and resources, and symptoms of post-traumatic stress after the hospitalization.

81 injury showed more severe neurobehavioural, post-traumatic stress and depression symptoms along with

82 t/hyperactivity disorder (ADHD), aggression, post-traumatic stress and substance abuse.

83 re those in support roles to report probable post-traumatic stress disorder (1.87, 1.26-2.78).

84 Depressive symptoms (17%) and post-traumatic stress disorder (18%) persisted at 1 year

85 4, p=0.011), higher risk of comorbidity with post-traumatic stress disorder (eight studies, 2494 part

86 Anxiety Disorder [GAD]-2 anxiety scale), and post-traumatic stress disorder (measured by the Post-Tra

87 thy control subjects (n = 20), patients with post-traumatic stress disorder (n = 25) demonstrated int

88 s were found to increase DES risk, including post-traumatic stress disorder (OR 1.92, 95% CI 1.91-1.9

89 Outcomes included depression, anxiety, and post-traumatic stress disorder (PTSD) among family 3 and

90 vestigated the pharmacological prevention of post-traumatic stress disorder (PTSD) and acute stress d

91 fghanistan are associated with high rates of post-traumatic stress disorder (PTSD) and comorbid alcoh

92 ys, and structured interview evaluations for post-traumatic stress disorder (PTSD) and depression.

93 s characterized by social withdrawal such as post-traumatic stress disorder (PTSD) and depression.

94 Post-traumatic stress disorder (PTSD) and generalized an

95 gests a link between early childhood trauma, post-traumatic stress disorder (PTSD) and higher risk fo

96 Post-traumatic stress disorder (PTSD) and other anxiety

97 Part of the symptomatology of post-traumatic stress disorder (PTSD) are alterations in

98 t, intrusive memories of traumatic events in post-traumatic stress disorder (PTSD) are an extreme exa

99 KEY POINTS: Patients with post-traumatic stress disorder (PTSD) are at a significa

100 Fear-related psychopathologies such as post-traumatic stress disorder (PTSD) are characterized

101 Viewing post-traumatic stress disorder (PTSD) as a disorder of e

102 e begin to elucidate the mechanisms by which post-traumatic stress disorder (PTSD) at a young age con

103 Stress modifies nociception, and humans with post-traumatic stress disorder (PTSD) exhibit co-morbid

104 Women are at increased risk of developing post-traumatic stress disorder (PTSD) following a trauma

105 Bipolar disorder (BD) and post-traumatic stress disorder (PTSD) frequently co-occu

106 xiety disorders such as major depression and post-traumatic stress disorder (PTSD) from normal subjec

107 Post-traumatic stress disorder (PTSD) has been an exempl

108 Prior structural imaging studies of post-traumatic stress disorder (PTSD) have observed smal

109 tudies investigating inflammatory markers in post-traumatic stress disorder (PTSD) have yielded mixed

110 regarding pharmacological interventions for post-traumatic stress disorder (PTSD) in children and ad

111 he present study investigates the effects of post-traumatic stress disorder (PTSD) in combat-exposed

112 pose that these mechanisms may contribute to post-traumatic stress disorder (PTSD) in humans.

113 There is increasing recognition of post-traumatic stress disorder (PTSD) in pediatric traum

114 Screen to assess for probable depression and post-traumatic stress disorder (PTSD) indicators.

115 Post-Traumatic Stress Disorder (PTSD) is a psychiatric d

116 Post-traumatic stress disorder (PTSD) is an anxiety diso

117 Post-traumatic stress disorder (PTSD) is an anxiety diso

118 ABSTRACT: Post-traumatic stress disorder (PTSD) is associated with

119 Post-traumatic stress disorder (PTSD) is associated with

120 e aim of this study was to determine whether post-traumatic stress disorder (PTSD) is associated with

121 Evidence suggests that post-traumatic stress disorder (PTSD) is associated with

122 Post-traumatic stress disorder (PTSD) is characterized b

123 Post-traumatic stress disorder (PTSD) is thought to deve

124 Post-traumatic stress disorder (PTSD) is twice as common

125 Post-traumatic stress disorder (PTSD) patients display p

126 isorder (MDD), Parkinson's Disease (PD), and Post-Traumatic Stress Disorder (PTSD) patients with inso

127 irst genome-wide association study (GWAS) of post-traumatic stress disorder (PTSD) performed using tr

128 cular factors involved in the development of Post-Traumatic Stress Disorder (PTSD) remain poorly unde

129 The genetic architecture of post-traumatic stress disorder (PTSD) remains poorly und

130 The neurobiology of post-traumatic stress disorder (PTSD) remains unclear.

131 Structural brain studies of adult post-traumatic stress disorder (PTSD) show reduced gray

132 predictors, and correlates of cancer-related post-traumatic stress disorder (PTSD) symptoms and diagn

133 Little is known about the trajectory of post-traumatic stress disorder (PTSD) symptoms in cancer

134 traumatic events (PTEs) of mass conflict and post-traumatic stress disorder (PTSD) symptoms to perina

135 We assessed patients' mood, post-traumatic stress disorder (PTSD) symptoms, and QOL

136 examines associations between war exposures, post-traumatic stress disorder (PTSD) symptoms, depressi

137 rstanding the evidence base for treatment of post-traumatic stress disorder (PTSD) today.

138 The definition of post-traumatic stress disorder (PTSD) underwent substant

139 ke each of whom was assessed for symptoms of post-traumatic stress disorder (PTSD) using the 17-item

140 Post-traumatic stress disorder (PTSD) was measured with

141 -wide differential gene expression survey of post-traumatic stress disorder (PTSD) with comorbid depr

142 f anxiety and fatigue (but not depression or post-traumatic stress disorder (PTSD)).

143 depressive disorder (MDD), 50 patients with post-traumatic stress disorder (PTSD), and 122 healthy c

144 iated with increased common mental disorder, post-traumatic stress disorder (PTSD), and poor general

145 ibing recovery from ICU included the risk of post-traumatic stress disorder (PTSD), anxiety, depressi

146 assess whether post-deployment screening for post-traumatic stress disorder (PTSD), depression, anxie

147 In adults with symptoms of post-traumatic stress disorder (PTSD), meta-analysis of

148 Study outcomes were presence of possible post-traumatic stress disorder (PTSD), psychological dis

149 dering the effect of traumatic experience on post-traumatic stress disorder (PTSD), this study aims t

150 e epigenetic response to traumatic stress on post-traumatic stress disorder (PTSD), this study examin

151 related neuropsychiatric conditions, such as post-traumatic stress disorder (PTSD), varies greatly am

152 al conduct, including combat experiences and post-traumatic stress disorder (PTSD), were assessed wit

153 eceptor type 2 (CRFR2) to be associated with post-traumatic stress disorder (PTSD)-like symptoms.

154 d point was the proportion of relatives with post-traumatic stress disorder (PTSD)-related symptoms o

155 nd prepulse inhibition) that are observed in post-traumatic stress disorder (PTSD).

156 stress-related psychopathologies, including post-traumatic stress disorder (PTSD).

157 f major depressive disorder (MDD) as well as post-traumatic stress disorder (PTSD).

158 ds threat is one of the defining features of post-traumatic stress disorder (PTSD).

159 elated conditions, such as mood disorders or post-traumatic stress disorder (PTSD).

160 sychiatric disorders, such as depression and post-traumatic stress disorder (PTSD).

161 ptor gene (NR3C1) are related to the risk of post-traumatic stress disorder (PTSD).

162 have high rates of depression, anxiety, and post-traumatic stress disorder (PTSD).

163 r the treatment of anxiety disorders such as post-traumatic stress disorder (PTSD).

164 tor have been implicated in animal models of post-traumatic stress disorder (PTSD).

165 underlie anxiety disorders and specifically post-traumatic stress disorder (PTSD).

166 uman psychological stress responses, such as post-traumatic stress disorder (PTSD).

167 phenotype is consistent with some aspects of post-traumatic stress disorder (PTSD).

168 r functional neurological disorder (FND) and post-traumatic stress disorder (PTSD).

169 tivity and is involved in the development of post-traumatic stress disorder (PTSD).

170 y adapt to the trauma is a characteristic of post-traumatic stress disorder (PTSD).

171 f depression (Beck Depression Inventory II), post-traumatic stress disorder (PTSD, Post-Traumatic Str

172 ions highlight a unique sensory pathology of post-traumatic stress disorder (ruling out effects merel

173 alcohol misuse; 2.16, 1.62-2.90; p<0.0001), post-traumatic stress disorder (violent offending in 25

174 eported symptoms of depression, anxiety, and post-traumatic stress disorder 1 year after flooding.

175 2.93), for anxiety 1.66 (1.12-2.46), and for post-traumatic stress disorder 1.70 (1.17-2.48) than peo

176 stress disorder (PTSD) was measured with the Post-traumatic Stress Disorder 8 items (PTSD-8) and seve

177 sease), mental health disorders (depression, post-traumatic stress disorder [PTSD], and panic disorde

178 (depression and anxiety disorders including post-traumatic stress disorder [PTSD], eating disorders,

179 sorders (major depression, bipolar disorder, post-traumatic stress disorder [PTSD], substance and alc

180 interventions that might offset the risk of post-traumatic stress disorder after cardiovascular dise

181 Interestingly, patients with post-traumatic stress disorder also showed heightened fr

182 Post-traumatic stress disorder and anxiety are more prev

183 redispose chronically stressed youth to both post-traumatic stress disorder and asthma.

184 ny candidate mechanisms for the link between post-traumatic stress disorder and cardiovascular diseas

185 rs of negative affect, such as depression or post-traumatic stress disorder and depression.

186 major depression and depressive symptoms in post-traumatic stress disorder and generalized anxiety d

187 ften seen in anxiety disorders, specifically post-traumatic stress disorder and panic disorder, and t

188 abis' effects on two psychiatric conditions: post-traumatic stress disorder and psychotic disorders.

189 ing or witnessing events related to 9/11 and post-traumatic stress disorder and respiratory illness,

190 the first recognition of what is now called post-traumatic stress disorder and the latter did not of

191 North Africa, major depressive disorders and post-traumatic stress disorder are a primary concern bec

192 Fear-related psychopathologies such as post-traumatic stress disorder are characterized by impa

193 rences were noted in attentional function or post-traumatic stress disorder between children with ser

194 ence of symptoms of depression, anxiety, and post-traumatic stress disorder between participants disp

195 may be effective in patients suffering from post-traumatic stress disorder by suppressing activity i

196 t-traumatic stress disorder (measured by the Post-Traumatic Stress Disorder Checklist [PCL]-6 scale).

197 y II), post-traumatic stress disorder (PTSD, Post-Traumatic Stress Disorder Checklist-Event Specific

198 Post-traumatic stress disorder develops in response to a

199 whom carried mild traumatic brain injury and post-traumatic stress disorder diagnoses.

200 is association, as well as the prevalence of post-traumatic stress disorder due to cardiovascular dis

201 ng to passive picture viewing, patients with post-traumatic stress disorder failed to demonstrate alp

202 io 1.22, 95% CI 1.02-1.46) and with probable post-traumatic stress disorder for reservists (2.83, 1.2

203 s to predict the emergence of depression and Post-Traumatic Stress Disorder in Twitter users.

204 Risk factors for post-traumatic stress disorder included proximity to the

205 We conclude that post-traumatic stress disorder is a risk factor for inci

206 Post-traumatic stress disorder is characterized by exagg

207 mited; however, prevalence of depression and post-traumatic stress disorder is high among trafficked

208 Post-traumatic stress disorder patients experience chron

209 Of the post-traumatic stress disorder symptoms, the hyperarousa

210 executive control, fuelling and perpetuating post-traumatic stress disorder symptoms.

211 s in four studies, more women presented with post-traumatic stress disorder than did men in two studi

212 berrations help construct a vicious cycle in post-traumatic stress disorder that is in action even at

213 ragmentation, may play a mechanistic role in post-traumatic stress disorder via an influence on safet

214 olent offending in 25 [8.6%] of 344 men with post-traumatic stress disorder vs 221 [3.0%] of 7256 wit

215 ersonnel, whereas the prevalence of probable post-traumatic stress disorder was low.

216 Scores for depression and post-traumatic stress disorder were higher in people who

217 e look at the evidence for an association of post-traumatic stress disorder with incident cardiovascu

218 sion with the Hopkins Symptoms Checklist and post-traumatic stress disorder with the Harvard Trauma Q

219 depression, generalized anxiety disorder and post-traumatic stress disorder) and leukocyte telomere l

220 flooding (p=0.04 for depression, p=0.01 for post-traumatic stress disorder), although the difference

221 e is linked to disorders from schizophrenia, post-traumatic stress disorder, and autism to cardiovasc

222 overlap between post-concussive syndrome and post-traumatic stress disorder, and blast-related mild T

223 Post-concussive syndrome, post-traumatic stress disorder, and chronic pain are a c

224 that examined the apolipoprotein E4 allele, post-traumatic stress disorder, and genetic risk for sch

225 is a disease associated with depression and post-traumatic stress disorder, and is prevalent among m

226 UXO victims reported a greater prevalence of post-traumatic stress disorder, anxiety, or depression t

227 ess-related psychiatric disorders, including post-traumatic stress disorder, are associated with disr

228 Other psychiatric disorders, such as post-traumatic stress disorder, bipolar disorder and dep

229 Alcoholism is frequently co-morbid with post-traumatic stress disorder, but it is unclear how al

230 alcohol and used a diagnostic tool to assess post-traumatic stress disorder, depression, and anxiety.

231 Six of the studies screened for post-traumatic stress disorder, depression, and anxiety;

232 target for neuropsychiatric diseases such as post-traumatic stress disorder, depression, and schizoph

233 iological disease states such as depression, post-traumatic stress disorder, hypertension, diabetes,

234 for understanding mental disorders, such as post-traumatic stress disorder, little is known about th

235 oradrenergic system, a known risk factor for post-traumatic stress disorder, modulates the stress-ind

236 controls, n=17; major depression, n=38; and post-traumatic stress disorder, n=50), we examine the di

237 e to the recurrent and intrusive memories of post-traumatic stress disorder, neuromodulatory receptor

238 Depression, post-traumatic stress disorder, or anxiety was present i

239 Post-traumatic stress disorder, panic disorder and phobi

240 ce extinction, a process that is impaired in post-traumatic stress disorder, schizophrenia, and addic

241 atric disorders, including major depression, post-traumatic stress disorder, schizophrenia, and addic

242 n, the most commonly recorded diagnoses were post-traumatic stress disorder, severe stress, or adjust

243 raging results as a treatment for refractory post-traumatic stress disorder, social anxiety in autist

244 nk between dreaming and emotional catharsis, post-traumatic stress disorder, supermemorization during

245 Post-traumatic stress disorder, though less prevalent, i

246 antial percentage of those deployed suffered post-traumatic stress disorder, traumatic brain injury,

247 To elucidate a sensory pathology of post-traumatic stress disorder, we examined intrinsic vi

248 tment of neuropsychiatric disorders, such as post-traumatic stress disorder, where a previous exposur

249 oad, threat-neutral sensory hyperactivity in post-traumatic stress disorder.

250 rders, such as depression, schizophrenia, or post-traumatic stress disorder.

251 disorders, including depression, anxiety and post-traumatic stress disorder.

252 anges in emotional arousal that characterize post-traumatic stress disorder.

253 ed protein kinase SGK1 in the development of post-traumatic stress disorder.

254 report symptoms of depression, anxiety, and post-traumatic stress disorder.

255 1), only five (1.1%) of 470 met criteria for post-traumatic stress disorder.

256 es associated with anxiety disorders such as post-traumatic stress disorder.

257 rs such as obsessive-compulsive disorder and post-traumatic stress disorder.

258 , and 38.9% (36.0-42.0) reported symptoms of post-traumatic stress disorder.

259 ores consistent with a probable diagnosis of post-traumatic stress disorder.

260 onditioning is considered an animal model of post-traumatic stress disorder.

261 ing an additional aetiological mechanism for post-traumatic stress disorder.

262 A2B) has been linked to emotional memory and post-traumatic stress disorder.

263 ctive disorders, such as chronic anxiety and post-traumatic stress disorder.

264 lated mild TBI seems to increase the risk of post-traumatic stress disorder.

265 disorders, including depression, anxiety and post-traumatic stress disorder.

266 iety disorders, such as major depression and post-traumatic stress disorder.

267 ers including depression, schizophrenia, and post-traumatic stress disorder.

268 and its levels are reduced in patients with post-traumatic stress disorder.

269 associated with excessive stress, including post-traumatic stress disorder.

270 development of anxiety disorders, including post-traumatic stress disorder.

271 for surgery, and the development of acute or post-traumatic stress disorder.

272 rders, such as depression, schizophrenia, or post-traumatic stress disorder.

273 atric disorders such as major depression and post-traumatic stress disorder.

274 er vs 221 [3.0%] of 7256 with no symptoms of post-traumatic stress disorder; 2.20, 1.36-3.55; p=0.001

275 One study reported reduced post-traumatic stress symptomatology in family members o

276 tudy has described their potential to reduce post-traumatic stress symptomatology in family members.

277 here was no evidence of difference in median post-traumatic stress symptomatology scores (diaries 24,

278 ess disorder (PTSD), anxiety, depression and post-traumatic stress symptomatology, health-related qua

279 d Post Concussive Symptom Questionnaire) and post-traumatic stress symptoms (Posttraumatic Stress Dis

280 ntensive care) wards experienced significant post-traumatic stress symptoms after their child's disch

281 study was to identify predictors of parental post-traumatic stress symptoms following child hospitali

282 ough RB survivors were more likely to report post-traumatic stress symptoms of avoidance and/or hyper

283 tic brain injury, older age, and more severe post-traumatic stress symptoms provided a good predictio

284 and self-blame, were associated with higher post-traumatic stress symptoms scores at three months po

285 nd coping strategies and resources predicted post-traumatic stress symptoms three months after the ch

286 e negative affectivity (anxiety, depression, post-traumatic stress symptoms) in mothers of newly diag

287 7% of parents (n=35) reported some degree of post-traumatic stress symptoms, and 21.5% (n=23) had ele

288 nship between negative coping strategies and post-traumatic stress symptoms.

289 ure, and this effect was also independent of post-traumatic stress symptoms.

290 fiable factors that most strongly influenced post-traumatic stress symptoms.

291 acial appearance, post-traumatic growth, and post-traumatic stress symptoms; noncancer CCSS siblings

292 ssments revealed heightened post-concussive, post-traumatic stress, and depressive symptoms along wit

293 of cued and contextual fear conditioning to post-traumatic stress, by extension we propose that thes

294 gnificant improvements in mood, anxiety, and post-traumatic stress; mothers in the NDS group showed n

295 Health Administration (VA) have a history of post-traumatic-stress-disorder (PTSD), and there exists

296 Patients with post-traumatic-stress-disorder reported higher pain scor

297 Our data suggests that a history of post-traumatic-stress-disorder was correlated with highe

298 Secondary outcomes included history of post-traumatic-stress-disorder, anesthesia type, first o

299 increasingly recognized as a cause of acute post-traumatic sudden death.

300 strate that CN2097 significantly reduces the post-traumatic synthesis of proinflammatory mediators an