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1 ualize rolling of endogenous Tregs in dermal postcapillary venules.
2 pial vessels and in deep ascending cortical postcapillary venules.
3 nd pericytes (PCs) that form the wall of the postcapillary venules.
4 y channels connecting terminal arterioles to postcapillary venules.
5 displayed on endothelial cells in intestinal postcapillary venules.
6 shear stress levels similar to that found in postcapillary venules.
7 tructure and increases leukocyte adhesion in postcapillary venules.
8 sults in vasculitic lesions predominantly in postcapillary venules.
9 enous malformations, and focal dilatation of postcapillary venules.
10 gap formation, and protein extravasation in postcapillary venules.
11 deficient in in vivo crawling and TEM in the postcapillary venules.
12 small lymphocytes for the endothelium of the postcapillary venules.
13 rolling effector and central memory cells in postcapillary venules.
14 e and continue to roll as elongated cells in postcapillary venules.
15 es as the main L-selectin ligand in inflamed postcapillary venules.
16 minants of the initiation of cell rolling in postcapillary venules.
17 o the relatively low-oxygen and low-velocity postcapillary venules.
18 eukocyte rolling and adherence in mesenteric postcapillary venules.
19 and E-selectin-dependent adhesion in airway postcapillary venules.
20 ma that tumor vessels arise exclusively from postcapillary venules.
21 erial/venous characteristics and derive from postcapillary venules.
22 re weeks exhibit an inflammatory response in postcapillary venules.
23 occur primarily via capillaries rather than postcapillary venules.
24 nomethyl-L-arginine (NMA) on skeletal muscle postcapillary venules.
25 rolling eosinophils, but not neutrophils, to postcapillary venules.
26 y designed to mimic physiologic flow through postcapillary venules.
27 ophages and erythrocytes) in capillaries and postcapillary venules.
28 leukocyte rolling and adhesion in mesenteric postcapillary venules.
29 rteries and veins, most arterioles, and some postcapillary venules.
30 utrophil emigration across murine mesenteric postcapillary venules.
31 ophil but not eosinophil rolling in inflamed postcapillary venules.
32 of 478 microbubbles (13.6%) observed in six postcapillary venules 11 to 30 microm in diameter and 24
33 increased in the superficial arterioles and postcapillary venules, 2 weeks after the onset of diabet
34 sed luminally by endothelial cells that line postcapillary venules, a primary site of leukocyte recru
35 s and vascular permeability were measured in postcapillary venules after 4-hour and 1-hour reperfusio
36 TDLN cells began migrating across pulmonary postcapillary venules and first appeared within metastas
37 e blood were investigated in mouse cremaster postcapillary venules and in flow chambers coated with P
39 ity begins in the superficial arterioles and postcapillary venules and progresses to the capillary be
40 ross HEVs is faster than across conventional postcapillary venules and requires a unique set of adhes
42 ed adhesion of leukocytes in capillaries and postcapillary venules, but no such adhesion in arteriole
43 about midway between terminal arterioles and postcapillary venules, challenging the classical concept
44 We constructed composite models of the human postcapillary venule, combining ECs with PCs or PC-depos
46 adhesion of leukocytes to discrete sites on postcapillary venules, followed by upregulation of adhes
47 ting leukocytes were labeled and observed in postcapillary venules for adhesion before and up to 120
49 in tissues that possess receptors for SP in postcapillary venules; (ii) liposome material in these t
50 theless, the number of leukocytes rolling on postcapillary venules in an E-selectin-dependent manner
51 bone marrow (BM) endothelium and to inflamed postcapillary venules in an E-selectin-dependent manner.
52 phocyte-associated Ag tether on the walls of postcapillary venules in inflamed skin via interaction w
53 othelial venules (HEV) are specialized plump postcapillary venules in lymphoid tissues that support h
54 es can be found marginalized in the lumen of postcapillary venules in postmortem brain tissue derived
56 ) increases clearance of macromolecules from postcapillary venules in the in situ oral mucosa and, if
57 duced neutrophil rolling and adhesion to the postcapillary venules in the mouse ears is significantly
61 arkedly reduced (>60%) leukocyte adhesion to postcapillary venules in wild type and Fpr1(-/-), but no
62 mbranes that arise from inflamed venules and postcapillary venules, increase in size as the disease p
63 Transendothelial migration of neutrophils in postcapillary venules is a key event in the inflammatory
64 d that the abundance of leukocyte rolling in postcapillary venules is due to interactions between red
66 f leukocyte-endothelial cell interactions in postcapillary venules, leading to leukocyte recruitment
69 ncreased expression of P-selectin protein in postcapillary venules of all vital organs after trauma.
72 ng and adhesion were measured in cremasteric postcapillary venules of septic and control rats using i
73 med soluble ICs are rapidly deposited in the postcapillary venules of the cremaster microcirculation,
74 nt microbeads flowing within mildly inflamed postcapillary venules of the cremaster muscle in vivo.
75 in vivo, studied by intravital microscopy in postcapillary venules of the cremaster muscle, was marke
77 s leukocyte-endothelial cell interactions in postcapillary venules of the mouse cremaster muscle.
78 yte-endothelial cell adhesion in cremasteric postcapillary venules of wild-type (WT) mice, CuZn-super
79 sed to monitor L/E and P/E adhesion in brain postcapillary venules of wild-type (WT), SOD1 transgenic
80 mber of adherent and emigrated leukocytes in postcapillary venules of WT HCD mice was significantly h
81 n is important in inflammation and occurs in postcapillary venules over a wide range of wall shear st
82 se model suggest that adherent leukocytes in postcapillary venules play a critical role in vaso-occlu
84 that P-selectin and E-selectin expressed on postcapillary venules play overlapping roles in the recr
85 d leukocyte-endothelial cell interactions in postcapillary venules revealed that CXCL1-induced neutro
87 including endothelial cells of capillary and postcapillary venules, the epithelial cell of kidney col
88 thelium may regulate neutrophil migration in postcapillary venules through the presentation of variou
89 ltered adhesive interactions within inflamed postcapillary venules under conditions of blood flow by
90 reveal that the basement membrane of dermal postcapillary venules undergoes changes in structure and
91 d cells as they flow from a capillary into a postcapillary venule using a lattice Boltzmann approach.
94 designed to approximate physiologic flow in postcapillary venules, we have characterized a rolling i
98 ill offer an effective, direct access to the postcapillary venules, where the target event (leukocyte
99 hodamine-stained leukocytes were observed in postcapillary venules with analysis for adhesion and rol
101 ntibody fluorescence intensity in submucosal postcapillary venules with the use of intravital microsc
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