ライフサイエンスコーパス: postcoitum

1 oitum, rising progressively to term (19 days postcoitum).

2 6-somite-pair stage (approximately 8.5 days postcoitum).

3 neural plate stage (approximately 7.75 days postcoitum).

4 s embryos; thus, the mice die before day 8.5 postcoitum.

5 amniotic fluid between E14.5 and E16.5 days postcoitum.

6 comes critical in the chorion after 7.5 days postcoitum.

7 dow of sensitivity between 8.5 and 10.5 days postcoitum.

8 GCNF(lox/lox) mice die at 9.5-10.5 days postcoitum.

9 studied in embryonic heart tubes at day 9.5 postcoitum.

10 use of fetal death between 8.5 and 15.5 days postcoitum.

11 lay and embryonic lethality at about 10 days postcoitum.

12 Cox7ah mRNA was not detectable until day 17 postcoitum.

13 ostcoitum followed by resorption by 9.5 days postcoitum.

14 -)(/)(-)) embryos surviving beyond 12.5 days postcoitum.

15 ne thymus was observed as early as 13.5 days postcoitum.

16 At day 9.5 postcoitum, 2-kinase was expressed in the notochord, the

17 GATA-4 knockout mice die by 9.5 days postcoitum and exhibit profound defects in ventral morph

18 n mutation fail to progress beyond 10.5 days postcoitum and fail to form mature blood vessels in the

19 oughout the embryonic region at 5.5-6.5 days postcoitum and later in the node, midbrain, spinal cord,

20 of developing mouse fetuses (15.5-17.5 days postcoitum) and their nuclei were transferred into enucl

21 formed in the fetus (approximately 8.25 days postcoitum), and vascular continuity with the yolk sac a

22 i1-null embryos die between 3.5 and 5.5 days postcoitum, and Ini1-null blastocysts fail to hatch, for

23 placental defects are manifest at 10.5 days postcoitum as nearly complete loss of the labyrinth laye

24 by immunostaining in the placenta at 18 days postcoitum as well as in the small intestine.

25 opment until midgestation (12.5 to 13.5 days postcoitum), at which time they undergo a dramatic loss

26 heart tubes from Ncx1(-/-) mice at 9.5 days postcoitum but is activated in heart tubes from Ncx1(+/+

27 re of cranial closure between 9 and 9.5 days postcoitum coincided with increased apoptosis in the mid

28 amination of embryos between 8 and 10.5 days postcoitum confirmed that lethality was due to a failure

29 ted in transgenic mice as early as 13.5 days postcoitum, consistent with a defect of preplate develop

30 ene was expressed in rhombomere 4 in 9.5 day postcoitum (d.p.c.) embryos.

31 yos developed beyond 7.5 and up to 10.5 days postcoitum, demonstrating a requirement for SMAD2 in ext

32 Csn3(-/-) embryos arrested after 5.5 days postcoitum (dpc) and resorbed by 8.5 dpc.

33 inheritance was followed for up to 18.5 days postcoitum (dpc) and that approximately 90% of GYS1-null

34 omes of gonadal germ cells at 11.5-19.5 days postcoitum (dpc) are incompetent to support full-term de

35 mice was confirmed by genotyping of 12.5-day-postcoitum (dpc) embryos.

36 n situ RNA hybridization as early as 10 days postcoitum (dpc) in developing gut, as early as 14.5 dpc

37 detected by immunofluorescence at 13.5 days postcoitum (dpc) in the mesenchyme surrounding the ductu

38 onad begins to form shortly before 10.5 days postcoitum (dpc) on the ventromedial side of the mesonep

39 Embryos cloned from migrating 10.5-days-postcoitum (dpc) primordial germ cells (PGCs) showed nor

40 ells of the developing gonads from 10.5 days postcoitum (dpc) to 12.5 dpc.

41 mouse inner ears ranging from 10.25 to 17 d postcoitum (dpc) were filled with paint to reveal their

42 mozygous mutant embryos as early as 7.5 days postcoitum (dpc) were recovered.

43 development is retarded globally by 7.5 days postcoitum (dpc), and all the null embryos die before 9.

44 onic developmental process stops at 8.5 days postcoitum (dpc), and excessive cell death occurs at 9.5

45 transgenic mouse embryos commencing at 10 d postcoitum (dpc), beyond the period of primary dorsal-ve

46 in XX and XY genital ridges until 11.5 days postcoitum (dpc), by 12.5 dpc the XY gonad develops a di

47 At E12.5 days postcoitum (dpc), FVII(-/-)/PC(-/-) embryos demonstrated

48 -/-) embryos cannot survive beyond 10.5 days postcoitum (dpc), probably due to cardiovascular failure

49 oxm1b -/- embryos died in utero by 18.5 days postcoitum (dpc).

50 omatin, and G9a null embryos die at 8.5 days postcoitum (dpc).

51 e because of apoptosis initiated at 3.5 days postcoitum (dpc).

52 s were also absent between 6.5 and 12.5 days postcoitum (dpc).

53 al ridges isolated from embryos at 11.5 days postcoitum (dpc).

54 efects in neurulation and die before 11 days postcoitum (dpc).

55 eir normal d/v positions at 9.5 to 12.5 days postcoitum (dpc).

56 to middle gestation (approximately 9.5 days postcoitum [dpc]) and exhibited a number of novel phenot

57 ld-stage allantoises (approximately 8.0 days postcoitum; dpc) were subdivided into three proximodista

58 d1 mutant mice die at approximately 9.5 days postcoitum due to defects in allantois formation.

59 IP died between embryonic day 11.5 and 12.5 (postcoitum) due in most part to defects in the developme

60 ing that inhibiting p38 activity in 5.5 days postcoitum embryo cultures leads to a switch from AVE to

61 lectrical activation pattern of the 9.5-days postcoitum embryonic mouse heart and show that treatment

62 bridges in the ovaries of 11.5 to 17.5 days postcoitum embryos; microtubules and organelles have bee

63 Embryos lacking KIF3A die at 10 days postcoitum, exhibit randomized establishment of L-R asym

64 ith apparent developmental delay at 7.5 days postcoitum followed by resorption by 9.5 days postcoitum

65 mbryos die in utero between 4.0 and 4.5 days postcoitum, following the depletion of their CAN from ma

66 blasts were prepared from embryos (14.5 days postcoitum) for biochemical analysis.

67 expression is detectable as early as 9.5-day postcoitum in mouse embryos.

68 of high percentage null chimeras (8-10 days postcoitum) in which Gata4+/+ cells were restricted to v

69 thality between embryonic day 11.5 and 12.5 (postcoitum), indicating that PRIP and PBP are essential

70 tion of TM into a pregnant mouse at 8.5 days postcoitum leads to detectable recombination in the deve

71 tal for sustaining pregnancy beyond 7.5 days postcoitum, likely by regulating the balance of coagulat

72 end-sequenced cDNAs selected from a Day 10.5 postcoitum mouse embryo library were genetically mapped

73 expressed at relatively high levels in 7-day postcoitum mouse embryos and at much decreased levels at

74 on in the neural folds of embryos at day 8.5 postcoitum (p.c.).

75 Cdx2 protein expression was observed at 9.5 postcoitum (pc), whereas weak expression of Cdx1 protein

76 By 17.5 days postcoitum, preferential staining of superficial cortica

77 20alpha-HSD mRNA was decreased, but in 15 d postcoitum pregnant mice injected with the PR antagonist

78 t detected in amniotic fluid (AF) at 17 days postcoitum, rising progressively to term (19 days postco

79 bridization on mouse inner ears (from 8 days postcoitum to postnatal day 5) to establish the expressi

80 s detect Plac1 expression from 7.5 dpc (days postcoitum) to 14.5 dpc in ectoplacental cone, giant cel

81 expression is detected in 9.5 and 10.5 days postcoitum transgenic embryos in a manner consistent wit

82 cells of GCNF(lox/lox) embryos at 8.25 days postcoitum was not silenced as in the GCNF(+/+) embryos.

83 ty-four percent of live embryos at 10.5 days postcoitum were morphologically normal when premature an

84 after implantation at approximately 6.5 days postcoitum with a loss of epiblast cells, expansion of p

85 We report that 84% (21/25) of 13 days postcoitum XXSxr fetuses on the B6 inbred genomic backgr