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1 bule), insula, and limbic lobe (anterior and posterior cingulate gyrus).
2 evoked responses in the hippocampus and the posterior cingulate gyrus.
3 white matter of the basal ganglia and right posterior cingulate gyrus.
4 showed disorder-specific dysfunction in the posterior cingulate gyrus.
5 tices (PMCs), primarily in the precuneus and posterior cingulate gyrus.
6 nd left lateral prefrontal cortices, and the posterior cingulate gyrus.
7 Controls had greater activation in the right posterior cingulate gyrus.
8 rontal gyrus, posterior parietal cortex, and posterior cingulate gyrus.
9 later accumulation of cNFT pathology in the posterior cingulate gyrus.
10 tex, the left middle temporal gyrus, and the posterior cingulate gyrus.
11 reactivity and both NAA/Cr and NAA/mI in the posterior cingulate gyrus.
12 ominent deactivation foci bilaterally in the posterior cingulate gyrus (+/-8, -48, 26; left P < 0.001
13 ignificantly lower glucose metabolism in the posterior cingulate gyrus and cuneus, whereas the nonhoa
14 amygdala, corpus callosum, and anterior and posterior cingulate gyrus and from automatically defined
15 his study revealed that the network from the posterior cingulate gyrus and the semiology of PCE (moto
16 0, whereas most of the medial surface of the posterior cingulate gyrus and the ventral bank of the po
17 parietal region (supramarginal gyri), right posterior cingulate gyrus, and left occipital lobe (ling
18 he frontal and parietal cortex, anterior and posterior cingulate gyrus, and left temporal cortex.
19 on in SI, anterior and posterior cerebellum, posterior cingulate gyrus, and superior and inferior fro
20 es) who underwent antemortem (1)H-MRS of the posterior cingulate gyrus at 3 tesla were included in th
21 : controls 0.89 +/- 0.04, ADs 0.72 +/- 0.04; posterior cingulate gyrus: controls 1.05 +/- 0.09, ADs 0
22 s, entorhinal cortex, parahippocampal gyrus, posterior cingulate gyrus, cortex of the temporal lobes
24 th human brains, the monkey frontal lobe and posterior cingulate gyrus had significantly less metabol
25 relative to control participants, within the posterior cingulate gyrus, hippocampus, and other region
26 late gyrus, caudal anterior cingulate gyrus, posterior cingulate gyrus, hippocampus, basal forebrain,
28 patient groups shared underactivation in the posterior cingulate gyrus in relation to comparison subj
29 or cingulate gyrus) and parietal (precuneus, posterior cingulate gyrus, inferior parietal lobule (IPL
31 nd greater grey matter loss over time in the posterior cingulate gyrus, lateral and medial temporal l
32 ex, left posteroinferior temporal lobe, left posterior cingulate gyrus, left frontal lobe expressive
33 CI and AD patients had decreased rCBF in the posterior cingulate gyrus (P = .01) with extension to th
34 anterior ventral precuneus (BA7), along with posterior cingulate gyrus (PCC, BA23, sad condition) and
35 rietal, and temporal cortices as well as the posterior cingulate gyrus, precuneus, and mesial tempora
36 r temporal gyri; superior parietal lobe; and posterior cingulate gyrus, resulted in a fitted accuracy
37 /myoinositol (mI), and mI/Cr measured in the posterior cingulate gyrus reveal evidence of disease pro
38 d), supplementary motor cortex, anterior and posterior cingulate gyrus, right superior parietal lobe,
39 ng electrophysiological connections from the posterior cingulate gyrus to parietal, temporal, mesial
40 istochemical evaluation was performed on the posterior cingulate gyrus using antibodies to synaptic v
41 as deactivated in patients; in contrast, the posterior cingulate gyrus was activated in patients and
42 cortex, right inferior parietal cortex, and posterior cingulate gyrus when compared with control sub
43 ed areas 26, 29, 30, 23, and 31 on the human posterior cingulate gyrus without marking sulcal areas.
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