ライフサイエンスコーパス: posterior wall

1 ng to the formation of tail somites from the posterior wall.

2 thickness of the interventricular septum and posterior wall.

3 r septum, 12+/-4 [7-23] mm; left ventricular posterior wall, 11+/-4 [7-21] mm; left ventricular mass,

4 ssed systolic thickening of the noninfarcted posterior wall (3% +/- 3% vs, 65 +/- 9%, p < 0.05).

5 (93%), the appendage ridge (100%), the high posterior wall (84%), and the anterior wall and septal r

6 final location of the needle was through the posterior wall and deep to the venous lumen.

7 s associated with increased left ventricular posterior wall and interventricular septal and relative

8 using on components of LV mass revealed that posterior wall and interventricular septal thickness, bu

9 ventricular septum and left ventricular (LV) posterior wall and its prognostic value was compared wit

10 symptoms of cardiomyopathy and reductions of posterior wall and septal thickening, reversal of polyne

11 nsistently showed the largest increase in LV posterior wall and septal thickness and LV mass index, a

12 ssue Doppler echograms (lateral, septal, and posterior walls), and WMSI were assessed at rest and at

13 enlarged ventricular chamber diameter, thin posterior wall, and decreased contractility.

14 ntricle, reduced systolic strain rate of the posterior wall, and ECG abnormalities.

15 on over bolus injection was the abolition of posterior wall attenuation and the ability to quantify M

16 PV antrum isolation extended to the posterior wall between PVs plus empirical isolation of t

17 Anterior and posterior wall calcific deposits in the aorta at the lev

18 change from baseline to final visit in mean posterior-wall CIMT of the left and right common carotid

19 Irregular air collections or posterior wall defects were suggestive of dehiscence at

20 eckle tracking radial strain anteroseptal-to-posterior wall delay >/=130 milliseconds, or pulsed Dopp

21 (MVG), as measured from the left ventricular posterior wall during the predetermined phases of the ca

22 atic fringe and Notch, and a ventral domain (posterior wall) expressing the Notch ligand Delta.

23 Posterior wall first by using interrupted suture techniq

24 In duct-to-duct reconstruction, posterior wall first technique by using interrupted sutu

25 he right-anterior walls for RL-BAV and right-posterior walls for RN-BAV in comparison with aorta size

26 nctional conduction block, descending on the posterior wall from the roof, passing between the ostia

27 left pulmonary veins in 31 (38.3%), and mid-posterior wall in 27 (33%).

28 all thickening and delayed relaxation in the posterior wall in control dogs but was relatively ineffe

29 d MVG, as measured from the left ventricular posterior wall in early diastole during both isovolumic

30 evealed proportionately thicker anterior and posterior walls in females.

31 uced diastolic thickness of left ventricular posterior wall, increased ejection fraction and fraction

32 ts with cardiac amyloidosis, CV-IB at the LV posterior wall is a powerful predictor of clinical outco

33 ip between the esophagus and the left atrial posterior wall is variable, and the esophagus is most su

34 PV antrum isolation (paroxysmal AF) and posterior wall isolation with complex fractionated atria

35 on, in addition to pulmonary vein antrum and posterior wall isolation, ablation of nonpulmonary vein

36 CA included pulmonary vein isolation and posterior wall isolation.

37 The LA roof and the anterior, septal, and posterior wall LA were most often affected.

38 test the hypothesis that a longer septal-to-posterior wall motion delay (SPWMD) would predict greate

39 shortening delay [Strain-SL], and septal-to-posterior wall motion delay [SPWMD]) were quantified at

40 Principal methods include M-mode septal to posterior wall motion delay, pulsed Doppler measures of

41 obe was withdrawn along the left ventricular posterior wall of excised hearts.

42 (64%) residents accidentally penetrated the posterior wall of the internal jugular vein during cannu

43 onses were found to be located mainly in the posterior wall of the LA.

44 anterior wall of the pseudoaneurysm and the posterior wall of the right renal vein.

45 Tumor adherence to the lateral of posterior wall of the SMPV confluence often represents t

46 show that BMP signaling is activated in the posterior wall of the tail bud and is involved in the fo

47 bipotential neuromesodermal cells along the posterior wall of the tailbud that make a germ layer dec

48 ne bridge located between the stapes and the posterior wall of the tympanum, which affects the normal

49 mean distances from the IRE electrode to the posterior wall of the vertebral body or the exiting nerv

50 tricular septum and from 10 to 13 mm for the posterior wall (p<0.001).

51 number of skin breaks and redirections, and posterior wall penetration of the long axis and short ax

52 The prevalence of posterior wall penetration was internal jugular short ax

53 The odds of posterior wall penetration were significantly less in th

54 ion to monitor the needle path and determine posterior wall penetration.

55 catheters placed were associated with fewer posterior wall penetrations (p = .04).

56 central venous catheterization and avoiding posterior wall penetrations may result in fewer central

57 The median number of posterior wall penetrations was 1.0 for all residents.

58 atheterization is also associated with fewer posterior wall penetrations.

59 Septal (SW) and posterior wall (PW) thickness, thickening, quantitative

60 T by multivariable analysis: anteroseptal to posterior wall radial strain dyssynchrony >200 ms, lack

61 PV antrum and posterior wall remained isolated in 82% of the cases, an

62 direct current cardioversion before PVAI and posterior wall/septum ablation while in sinus rhythm (gr

63 rony (time difference in peak septal wall-to-posterior wall strain > or =130 ms) to predict response

64 ated antral surface area and the left atrial posterior wall surface area, while considering the indiv

65 aptopril MI vs. untreated MI]) and unchanged posterior wall thickening (49 +/- 12% vs. 37 +/- 3%, p =

66 ction of noninfarcted myocardium (% systolic posterior wall thickening 37+/-4 versus 62+/-10, P:<0.01

67 ategory (5); ratio of maximal wall thickness:posterior wall thickness <1.46 (0), 1.47 to 1.70 (1), 1.

68 on; P = 0.006), and a greater increase in LV posterior wall thickness (from 0.13 +/- 0.003 cm in age-

69 LV dimension, with much lower agreement for posterior wall thickness (ICC=0.65), fractional shorteni

70 ressure therapy resulted in reduction of the posterior wall thickness (P=0.02) and improvement in LV

71 ), left atrial diameter (r = 0.25 and 0.28), posterior wall thickness (r = 0.20 and 0.26), septal thi

72 ameter (RHO = 0.87), septal (RHO = 0.85) and posterior wall thickness (RHO = 0.83).

73 t ventricular (LV) mass and LV end-diastolic posterior wall thickness 4 years later (P < .01); increa

74 increase in ventricle/body weight ratio and posterior wall thickness and a selective up-regulation o

75 wer heart and lung weights, reduced LV mass, posterior wall thickness and end diastolic pressures, an

76 Posterior wall thickness and left ventricular mass were

77 Posterior wall thickness and LV mass index were signific

78 nth (40% vs 36%; P = .008), and an increased posterior wall thickness at 1 year (5.4 mm vs 4.4 mm; P

79 nterventricular septal thickness by 21%, and posterior wall thickness by 13%.

80 ic function and a reduction in LV septal and posterior wall thickness by echocardiography compared wi

81 that CNTF(Ax15) reduced cardiac hypertrophy [posterior wall thickness decreased by 29 +/- 8% (P < 0.0

82 , vs. control subjects) in the patients, but posterior wall thickness did not differ from that in con

83 e wall thickness (RWT) defined as septal and posterior wall thickness divided by LVID.

84 ive wall thickness (RWT), defined as 2 times posterior wall thickness divided by the left ventricular

85 SD decrease in z score, P=0.014), and higher posterior wall thickness in the RCM/HCM group only (haza

86 ng, or higher left ventricular end-diastolic posterior wall thickness or end-diastolic ventricular se

87 m vs. 28+/-8 mm, p < 0.001), a higher septum:posterior wall thickness ratio (2.7+/-1.2 vs. 1.8+/-0.9,

88 iagnosis younger than 14.3 years, and the LV posterior wall thickness to end-diastolic dimension rati

89 e had an increased ratio of left ventricular posterior wall thickness to internal dimensions but did

90 Mean posterior wall thickness was 0.02 cm larger in central m

91 Posterior wall thickness was significantly increased in

92 inflow, M-mode measurements of LV septal and posterior wall thickness were normal before PTE and did

93 iography demonstrated increased anterior and posterior wall thickness with normal left ventricular fu

94 sk of SCD in patients with DCM, and a higher posterior wall thickness Z-score was the sole risk facto

95 mass index (BMI), male sex, left ventricular posterior wall thickness, and left ventricular ejection

96 ventricular (LV) end-diastolic diameter, LV posterior wall thickness, LV mass (LVM), and LV mass ind

97 examination for the assessment of septal and posterior wall thickness, LV mass index, LV volumes and

98 se women had higher end-diastolic septal and posterior wall thickness, LV mass, and relative wall thi

99 K abolished the increase in left ventricular posterior wall thickness, myocyte cross-sectional area,

100 bnormal LV mass index, relative, septal, and posterior wall thickness, respectively.

101 rventricular septum and the left ventricular posterior wall thickness.

102 Relative wall thickness ratio (2 [Diastolic posterior wall thickness]/Diastolic LV internal chamber

103 Two players had an increased septal-to-posterior-wall-thickness ratio (> or =1.3), although no

104 Septal and posterior wall thicknesses increased with training, and

105 Left ventricular (LV) diastolic anterior and posterior wall thicknesses were greater in SHR than WKY

106 ass, end-diastolic dimension, and septal and posterior wall thicknesses were positively related to lo

107 Septal and posterior wall thicknesses were significantly and equall

108 ar diameters and interventricular septal and posterior wall thicknesses.

109 younger than 14.3 years, LV dilation, and LV posterior wall thinning.

110 ate analysis showed that the CV-IB at the LV posterior wall was the best predictor of cardiac death (

111 variate analysis showed that CV-IB at the LV posterior wall was the only independent predictor of bot

112 All 4 pulmonary veins and the left atrium posterior wall were found isolated in 69% and 23% of pat

113 The anterior, lateral, and posterior walls were the most common regions in which MP