ライフサイエンスコーパス: postexercise

1 eased plasma IL-6 levels (peaked immediately postexercise).

2 xercise, and 19.5 +/- 1.7 cm H(2)O at 60 min postexercise.

3 as measured before and at 10, 30, and 60 min postexercise.

4 ements at baseline and 5, 15, 30, and 60 min postexercise.

5 ied by 24-h room calorimetry at baseline and postexercise.

6 s assessed pre- and at 2.5 through to 70 min postexercise.

7 ls of NF-kappaB binding were observed at 2 h postexercise.

8 and 17.3 +/- 6% on normal diet at 15 minutes postexercise.

9 ter 20 and 45 min of exercise, and at 30 min postexercise.

10 etected by a fall in quadriceps twitch force postexercise.

11 ficantly different from baseline at any time postexercise.

12 ficantly different from baseline at any time postexercise.

13 at baseline, 19.6 +/- 2.0 cm H(2)O at 10 min postexercise, 18.6 +/- 2.0 cm H(2)O at 30 min postexerci

14 At 3 h postexercise (3hPEX), isolated epitrochlearis muscles we

15 confidence interval [CI], -0.345 to -0.001), postexercise (6 hours: mean, -0.173%; 95% CI, -0.332 to

16 TwQ fell significantly postexercise; 79.2 +/- 5.4% of baseline value at 10 min

17 An increased postexercise (99m)Tc-sestamibi L/H adds significant diag

18 Group 1 had better ICD, ACD, and resting and postexercise ABI (P < 0.01) than Group 2.

19 At 5/12 median ICD, ACD, resting and postexercise ABI had increased by 197%, 212%, 17%, and 6

20 e) and 8 monkeys survived 27 weeks (12 weeks postexercise) after initial BrdU injections.

21 pothesis that amino acid availability limits postexercise anabolism in older individuals.

22 Sixteen monkeys survived 15 weeks (5 weeks postexercise) and 8 monkeys survived 27 weeks (12 weeks

23 e in the morning (after a 12-h fast and 12 h postexercise) and in the afternoon (after a 4-h fast and

24 ostexercise, 18.6 +/- 2.0 cm H(2)O at 30 min postexercise, and 19.5 +/- 1.7 cm H(2)O at 60 min postex

25 Animals were sacrificed 16 h postexercise, and gastrocnemius protein synthesis, mTOR

26 here was no significant change in resting or postexercise ankle/brachial indexes.

27 nfidence interval 0.93 to 0.97) between mean postexercise ankle:brachial systolic blood pressure indi

28 ths with type 1 diabetes demonstrated slowed postexercise ATP resynthesis and were more insulin resis

29 Postrehabilitation, TwQp fell significantly postexercise but the fall in TwQp with exercise was sign

30 Changes in pre-exercise and postexercise challenge values; percentage inhibition in

31 Pre- to postexercise changes were significantly greater during c

32 d a higher insulin-stimulated glucose uptake postexercise compared with wild-type mice.

33 t of mild to moderate exercise can lead to a postexercise decrease in blood pressure in hypertensive

34 , prerehabilitation, TwQp fell significantly postexercise down to a minimum value of 73.9 +/- 3.9% of

35 Prerehabilitation, TwQu fell significantly postexercise down to a minimum value of 82.5 +/- 3.1% of

36 1 METs; 95% CI, -0.70 to -0.11; P = .007) or postexercise E/e' of 15 or greater (-0.41 METs; 95% CI,

37 ls underwent moderate exercise with pre- and postexercise echocardiography.

38 With a need to control the postexercise energy balance, appetite responses after me

39 were no differences between E45 and E65 for postexercise energy substrate turnover or oxidation in m

40 occur during exercise are necessary for the postexercise enhancement of insulin-stimulated net hepat

41 to reduce the insulin dose administered with postexercise foods to further combat hypoglycemia.

42 ion (DFx) evaluation using standard 16-frame postexercise gated (99m)Tc-sestamibi myocardial perfusio

43 cance of differences between preexercise and postexercise grading for each reader.

44 led pAS160 as a possible mediator of greater postexercise GU of insulin-stimulated muscles from the i

45 od pressure in hypertensive subjects, namely postexercise hypotension (PEH).

46 In normally active individuals, postexercise hypotension after a single bout of aerobic

47 In sedentary individuals, postexercise hypotension following a single bout of aero

48 pha-adrenergic agonists is maintained during postexercise hypotension in humans.

49 opathy but can also suggest the diagnosis of postexercise hypotension in which an abnormality in auto

50 Thus, while postexercise hypotension is associated with increased va

51 ces vasodilatation after exercise and blunts postexercise hypotension.

52 eptor-mediated vasodilatation contributes to postexercise hypotension.

53 of an H1 receptor-mediated vasodilatation to postexercise hypotension.

54 rearm and leg vasculatures is blunted during postexercise hypotension.

55 he number of abnormal Tl-201 segments on the postexercise image was the only variable in the multivar

56 t side-by-side comparison of preexercise and postexercise images.

57 ficantly different from baseline at any time postexercise in either the fatiguers or nonfatiguers.

58 ude was unchanged from baseline at all times postexercise indicating that the fall in TwQ was due to

59 f maximal voluntary contraction) followed by postexercise ischaemia in normothermia and during heat s

60 During postexercise ischaemia, CVC and MAP returned to pre-exer

61 luated during ischemic handgrip exercise and postexercise ischemia, and it was defined as the slope r

62 though the ECMO group exhibited baseline and postexercise lung function abnormalities, there were no

63 ons of matched insulinemia and fiber, an HGI postexercise meal suppresses feelings of hunger and augm

64 ic index (LGI) and high-glycemic index (HGI) postexercise meals in type 1 diabetes patients.

65 Postexercise measurements are unreliable because of the

66 ffer between the study arms, and neither did postexercise measurements of left ventricular ejection f

67 e damage and also prevented DMD hallmarks of postexercise muscle damage, hypoxia, and fatigue in mdx

68 HG) at 30% maximum voluntary contraction and postexercise muscle ischaemia (PEMI).

69 consumed after resistance exercise increases postexercise muscle protein synthesis rates.

70 city of other protein-dense foods to augment postexercise muscle protein synthesis rates.

71 Both milk and beef ingestion augment the postexercise myofibrillar protein synthetic response in

72 However, whole-egg ingestion increased the postexercise myofibrillar protein synthetic response to

73 Postexercise nutrition is paramount to the restoration o

74 A persistent fall in Pdi,tw postexercise of >/= 10% was considered potentially indic

75 At 30 min postexercise on the control day, femoral vascular conduc

76 In contrast, at 30 min postexercise on the fexofenadine day, femoral vascular c

77 in the afternoon (after a 4-h fast and 12 h postexercise) on 2 separate days with the ventilated-hoo

78 d before treatment and at 10, 30, and 60 min postexercise or after the rest period.

79 mpairment due to arterial disease (ABI < 1.0 postexercise) or unrelated causes and those thrombectomi

80 at baseline to 19.7 +/- 1.6 cm H2O at 10 min postexercise (p < 0.0001, ANOVA).

81 xercise (p < 0.005), 75.7 +/- 4.8% at 30 min postexercise (p < 0.001), and 84.0 +/- 5.0% at 60 min po

82 e; 79.2 +/- 5.4% of baseline value at 10 min postexercise (p < 0.005), 75.7 +/- 4.8% at 30 min postex

83 ise (p < 0.001), and 84.0 +/- 5.0% at 60 min postexercise (p < 0.005).

84 L, preexercise, vs 2.42 +/- 2.27 micromol/L, postexercise, P < .001), an effect that was not related

85 -0.014), with a trend toward decrease during postexercise paced breathing (6 hours: mean, -0.142%; 95

86 s without angina and a normal image or small postexercise perfusion defect versus 71% for patients wi

87 availability of phenylalanine during the 5-h postexercise period tended to be higher after beef (64%

88 n with an increase in lipid oxidation in the postexercise period that is significantly more pronounce

89 sponsiveness to a bacterial challenge in the postexercise period.

90 than did beef ingestion during the 0- to 2-h postexercise phase (P = 0.013).

91 d beef ingestion during the entire 0- to 5-h postexercise phase (P = 0.114).

92 Mitochondrial capacity was assessed as the postexercise phosphocreatine recovery time constant (tau

93 ents had a persistent >/= 10% fall in Pdi,tw postexercise, potentially indicative of contractile fati

94 We conclude that postexercise protein supplementation does not increase r

95 ubjects with EIB, the n-3 PUFA diet improved postexercise pulmonary function compared with the normal

96 ise pulmonary function in either group or on postexercise pulmonary function in control subjects.

97 Both pre- and postexercise pulmonary function tests revealed air trapp

98 Despite abnormalities in baseline and postexercise pulmonary functions, ECMO graduates have si

99 The postexercise rate of glycogen resynthesis was nonlinear.

100 x 100 ml(-1) leg volume) but not during the postexercise recovery (64+/-9 nmol x min(-1) x 100 ml(-1

101 esis and degradation were greater during the postexercise recovery (65+/-10 and 74+/-10 nmol x min(-1

102 nitiation factor 4E-binding protein 1 during postexercise recovery (all P < 0.05).

103 < 0.05), and remained elevated during 3 h of postexercise recovery in both sexes (P < 0.05), but with

104 ties, and it remained elevated during 3 h of postexercise recovery in both sexes (P < 0.05).

105 ained elevated above the control over 3 h of postexercise recovery in men after exercise in E45 and E

106 did not remain significantly elevated during postexercise recovery in women, although MCR did remain

107 es (P < 0.05), but more in men during 3 h of postexercise recovery on D1 (P < 0.05) and remained elev

108 y exercise bout on a treadmill; and a 45-min postexercise recovery period (in reclining position) in

109 e uptake were three times greater during the postexercise recovery than at rest (P<0.05).

110 ns were not depressed in women during 3 h of postexercise recovery, and in contrast with that in men,

111 During 3 h of postexercise recovery, Ra(GL) remained significantly ele

112 plex 1 and 70-kDa S6 protein kinase 1 during postexercise recovery.

113 es net muscle protein accretion during acute postexercise recovery.

114 ring a protocol of rest, standing, exercise, postexercise rest, and 20 cycles of slow, paced breathin

115 survival periods, monkey age, and possibly a postexercise sedentary period but no direct effect of ex

116 ate steady state (ie, endurance) exercise on postexercise skeletal muscle metabolism are not well des

117 e use was prohibited for > or =24 h pre- and postexercise study days.

118 ks on each treatment to assess the degree of postexercise stunning with simultaneous sestamibi single

119 bide mononitrate (ISMN, 50 mg once daily) on postexercise stunning.

120 to groups by age (10-12 years, 15-17 years), postexercise survival periods, and controls, received 10

121 ase in plasma IL-10 levels (peaked at 1 hour postexercise), that was most likely mediated by increase

122 Postexercise, the maximal decreases in FEV(1) (mean +/-

123 tition of energy stored as IMCLs or glycogen postexercise.The purpose of this study was to compare th

124 and E- mice demonstrated a markedly reduced postexercise urinary nitrate excretion, aerobic capacity

125 ing recovery, tryptophan:LNAA increased from postexercise values in fasting trials.

126 ased during exercise and could contribute to postexercise vasodilatation via H1 receptors in the peri

127 ine and clonidine were similar (or enhanced) postexercise vs. preexercise.

128 ine and clonidine were similar (or enhanced) postexercise vs. preexercise.

129 susceptible to fatigue, and exhibited marked postexercise weakness.

130 osphor-IkappaBalpha content were found 0-1 h postexercise whereas P65 reached peak levels at 2-4 h.

131 nstrated a > or = 10% decrease in twitch Pdi postexercise, which was considered indicative of diaphra

132 adductor pollicis twitch force was unchanged postexercise while twitch Pdi fell, changes in milieu ca