ライフサイエンスコーパス: postimplantation

1 ilaments penetrated through the mesh by 3 mo postimplantation.

2 re scheduled at 1, 2, 3, 6, 9, and 12 months postimplantation.

3 biomicroscopy and histologically for 8 weeks postimplantation.

4 d in uncoated alginate spheres after 2 weeks postimplantation.

5 1 and 6 months, and 1, 2, 3, 4, and 5 years postimplantation.

6 dard parameters during the first five months postimplantation.

7 sed HLA class I antibody response at 2 years postimplantation.

8 ested from adult rat recipients 1 to 8 weeks postimplantation.

9 arge tumors (ca. 2000 mm(3)) treated 66 days postimplantation.

10 nse and seamless interface with brain tissue postimplantation achieved by ultraflexible open mesh ele

11 ser refractive surgery, LAL implantation and postimplantation adjustment provide a precise refractive

12 other atrial tachyarrhythmias (AF/AT), or if postimplantation AF/AT modulate the benefits of CRT-D, r

13 Catheters were removed at 7 and 14 days postimplantation and analyzed for hydroxyproline (OHP, n

14 Corresponding image slices on postimplantation and follow-up IVUS studies of 11 malapp

15 re TGF-beta family members that regulate the postimplantation and midgestation stages of pregnancy.

16 f the defect sites were harvested at week 16 postimplantation and processed for histometric analysis

17 ear singularly in some of the cysts by day 8 postimplantation and were observed in approximately 85%

18 ICP (39+/-4 vs. 19+/-5 cm H2O at seven days postimplantation) and corresponding accelerated neurolog

19 designed algorithm based on timing (pre- and postimplantation) and location (central or peripheral) o

20 mbryonic and extraembryonic tissues at early postimplantation (approximately E5.5-6.5), coincident wi

21 e suture contamination and biofilm formation postimplantation are warranted.

22 Postimplantation bacterial meningitis was strongly assoc

23 ged with magnetic resonance (MR; 3.0 T) 11 d postimplantation before and after intravenous administra

24 ion development, but become fully methylated postimplantation by de novo methylation of the paternal

25 tocyst axis of symmetry into the axes of the postimplantation conceptus involves asymmetric visceral

26 Postimplantation, CoreValve was associated with a larger

27 At postimplantation day 12, the recipients were evaluated f

28 r infiltration of the graft when examined at postimplantation day 12.

29 g/day, respectively, for 5 days beginning on postimplantation day 7.

30 Implanted EVPOMEs' histology on the seventh postimplantation day was used to correlate outcomes of g

31 sive mutation, 94-A/K, that results in early postimplantation death of the embryos, and we have mappe

32 on of Fgf4, which plays an important role in postimplantation development and growth and patterning o

33 is globally expressed at high levels during postimplantation development and its conditional ablatio

34 methylation of the mouse genome during early postimplantation development and of maternally imprinted

35 It follows that FGFR2 is required for early postimplantation development between implantation and th

36 ernal-fetal interface is essential for early postimplantation development in mice.

37 c growth through a surge of VIP during early postimplantation development in the rodent.

38 Postimplantation development is characterized by stage-

39 The transition from preimplantation to postimplantation development leads to the initiation of

40 In a model for postimplantation development, addition of FGF-4 to blast

41 PGCs are specified during early mammalian postimplantation development, and are uniquely programme

42 -wide accumulation of H3K9me2 is crucial for postimplantation development, and coincides with redistr

43 of DNA methylation patterns during pre- and postimplantation development.

44 the earliest differentiation events of human postimplantation development.

45 le in all adult somatic tissues and in early postimplantation development.

46 maternal-fetal interface of mice throughout postimplantation development.

47 d shape were found throughout pre- and early postimplantation development.

48 ice and may exert a critical function during postimplantation development.

49 ic and extraembryonic tissue lineages during postimplantation development.

50 enesis and leading to differentiation during postimplantation development.

51 ities in both late preimplantation and early postimplantation development.

52 rtilized mouse eggs disturbed their pre- and postimplantation development.

53 r, less is known about the role of NO during postimplantation development.

54 o be visualized in situ to examine the early postimplantation distribution of clones obtained by tran

55 Primate ESCs correspond to the postimplantation embryo and fail to resume development i

56 ognize imprinted genes but are absent in the postimplantation embryo and in ES cells.

57 of cells in the extraembryonic region of the postimplantation embryo and in trophoblast stem cells.

58 We discuss that although polarity of the postimplantation embryo can be traced back to the 8-cell

59 iblast cells and thus the development of the postimplantation embryo in mice.

60 he blastocyst, stem cell populations and the postimplantation embryo provides new insights into early

61 y all trophectoderm descendants in the early postimplantation embryo through E8.5, then disappears ex

62 ld provide cues that predict the axes of the postimplantation embryo, we have used the strategy of in

63 late with the anterior-posterior axis of the postimplantation embryo.

64 nt trophoblast cells that surround the early postimplantation embryo.

65 n of these cells to distinct lineages of the postimplantation embryo.

66 n in the blastocyst are also utilized in the postimplantation embryo.

67 Mbd3 is known to be essential for postimplantation embryogenesis in mice, but the function

68 l peptide, a neuropeptide regulator of early postimplantation embryonic growth, and the neuroprotecti

69 PECAM expression is next documented in the postimplantation embryonic yolk sac, where clumps of mes

70 Igf2 monoallelic expression is initiated in postimplantation embryos and that the single remaining C

71 nt epiblast stem cells (EpiSCs) derived from postimplantation embryos exhibit properties that are cha

72 sts derived from medulloblastoma nuclei form postimplantation embryos with typical cell layers.

73 DeltaDMD and H19Delta3.8kb-5'H19 in pre- and postimplantation embryos, we show that the DMD exhibits

74 methylation in embryonic stem (ES) cells and postimplantation embryos.

75 germ cell, in a few epiblast cells of early postimplantation embryos.

76 l myostatin deficiency were conferred by the postimplantation environment.

77 onship between sister cells in non-chimaeric postimplantation epiblast by ionophoretic injection of a

78 of investigation because PGCs originate from postimplantation epiblast cells in vivo.

79 cell fate decisions in rapidly proliferating postimplantation epiblast cells.

80 le with Prdm14 during PGC specification from postimplantation epiblast cells.

81 r the derivation and maintenance of ESCs and postimplantation epiblast stem cells (epiSCs).

82 c stem cell lines (ES cells), as well as the postimplantation epiblast, which gives rise to epiblast

83 ing pluripotent cell lines equivalent to the postimplantation epiblast.

84 c and distinct enhancer elements that govern postimplantation expression in the somitic myotomes and

85 occurs towards the region where the earlier postimplantation expression of Cer1 was strongest.

86 the secondary end point of mortality during postimplantation follow-up were compared in PFO versus n

87 ptide (VIP) has been shown to regulate early postimplantation growth in rodents through central nervo

88 onality was an independent associate of poor postimplantation health status for 6 of 8 of the SF-36 s

89 DX2(+)/p63(+) CTB subpopulation in the early postimplantation human placenta that is significantly re

90 tinue to use the HA in the contralateral ear postimplantation in order to determine whether or not th

91 gh this methylation is not maintained during postimplantation in the absence of the DMD.

92 ct of the uterine decidua and a regulator of postimplantation intrauterine events.

93 g) behind the strut, and where the immediate postimplantation IVUS revealed complete apposition of th

94 on mouse chromosome 8 associated with early postimplantation lethality in homozygotes and abnormal d

95 hatase, resulted in developmental arrest and postimplantation lethality.

96 ortion results from disrupted imprinting and postimplantation loss of mutant embryos.

97 the successful progression of both pre- and postimplantation mammalian development.

98 e the first cell type to be specified in the postimplantation mammalian embryo and serve highly speci

99 repair pathway is essential for normal early postimplantation mouse development and implicate an endo

100 he blastocyst stage and is ubiquitous during postimplantation mouse development, while the maternal E

101 rm (VE) is an epithelial tissue in the early postimplantation mouse embryo that encapsulates the plur

102 In the early postimplantation mouse embryo the ECM component laminin

103 the mouse, is essential for survival of the postimplantation mouse embryo.

104 n methylomes for early lineages in peri- and postimplantation mouse embryos.

105 brane grafting was performed in all cases of postimplantation necrosis (n = 10), but 8 eyes required

106 I) was performed in both preimplantation and postimplantation nuclear transfer embryos.

107 red healing extraction sockets 6 to 8 months postimplantation of a bioactive glass (BG) or deminerali

108 I) at day (d) 18 (preimplantation) and d 34 (postimplantation) of gestation.

109 , vascular measurements for access planning, postimplantation paravalvular regurgitation (PVR), and t

110 focused on attitudes toward hearing aid use postimplantation, patterns of usage, and perceived bimod

111 y day 11 after pump implantation; by 25 days postimplantation, PC2(-/-) islets were indistinguishable

112 sion with increasing angiogenesis during the postimplantation period (days 5-8).

113 sm by which the embryos survive at the early postimplantation period by pooling maternal blood in the

114 al and trophoblast ADA died during the early postimplantation period, whereas expression in trophobla

115 s well as resorption of embryos in the early postimplantation period.

116 pilot study was to evaluate the findings of postimplantation PET/CT of 90Y glass microspheres.

117 ein receptor type 2 (BMPR2) is essential for postimplantation physiology and fertility.

118 Postimplantation predictors were VVIR cumulative percent

119 Lymph node dissection and postimplantation prostatic biopsies were not routinely p

120 ist, and cellular distribution and viability postimplantation remain key issues.

121 Low baseline ejection fraction (EF) and postimplantation RVA-paced or spontaneous QRSd predicted

122 oscopy of the T9/mM-CSF tumor site, 2-4 days postimplantation, showed marked infiltration by macropha

123 ine non-redundant roles for TET1 at an early postimplantation stage of the mouse embryo, when its par

124 ing mouse embryo results in lethality at the postimplantation stage, demonstrating that it is an esse

125 From the two-cell to the postimplantation stage, methylation of the paternal geno

126 errant and restricted lineage priming at the postimplantation stage, which leads to early embryonic l

127 ull embryos exhibited lethality at the early postimplantation stage.

128 nt in maintaining IAP methylation during the postimplantation stage.

129 During early postimplantation stages Ada is highly expressed in both

130 ias observed in the blastocyst persists into postimplantation stages and therefore has relevance for

131 and myometrial smooth muscle at even earlier postimplantation stages.

132 methylation during preimplantation and early postimplantation stages.

133 inactive in the extraembryonic structures at postimplantation stages.

134 Leaching potentials peak at 5-30 days postimplantation, suggesting that targeted timing of imp

135 Rapid blood perfusion is critical for postimplantation survival of thick, prevascularized bioa

136 e grafts increased with progressively longer postimplantation survival times.

137 The postimplantation syndrome after EVAR may delay recovery

138 fect, with tumor growth inhibition at day 28 postimplantation (the day control animals began to requi

139 At 24.1 +/- 6.5 months postimplantation, the keratoprosthesis was retained in a

140 Commonly, acute, postimplantation thrombocytopenia causes significant ble

141 located away from the suture strut at a mean postimplantation time of 77.4 months (range, 33 to 132 m

142 eic VM/Dk mice, analyzing animals at various postimplantation time points using dynamic microPET imag

143 escent, and adult mice and were evaluated at postimplantation times up to 15 months.

144 The greatest increase was seen from postimplantation to 6 months.

145 Impressions were obtained pre- and postimplantation to determine changes in alveolar ridge

146 From postimplantation to follow-up, arterial curvature and an

147 use development from the eight-cell stage to postimplantation using lineage-specific RNA sequencing.

148 n, fertilization, and implantation; however, postimplantation uterine decidual cells showed terminal

149 an average 4.5-h period showed satisfactory postimplantation valve function.

150 Baseline and postimplantation variables were used to predict HFH in t

151 he progeny of inner cell mass cells into the postimplantation visceral endoderm.

152 The main reasons for postimplantation vision loss was glaucoma (12/31, 39%),

153 ng rabbits were treated on days 6, 9, and 12 postimplantation with alpha(nu)beta(3)-targeted fumagill

154 is presumed that impedance fluctuates early postimplantation, with implications for variations in cu

155 tly inhibited tumor growth for up to 50 days postimplantation, with negligible animal body weight los

156 changes in impedance within the first month postimplantation, with no further variation.