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1 recently was likely considered idiopathic or postinfectious.
2 iology was found in 76%: 37% infectious, 16% postinfectious, and 23% noninfectious.
3 ng diseases with a wide array of infectious, postinfectious, and noninfectious causes.
4 d by etiological classification: infectious, postinfectious, and noninfectious.
5 e possibility that this phenotype could be a postinfectious autoimmune CNS disorder, and therefore si
6     These findings support a new subgroup of postinfectious autoimmune inflammatory disorders associa
7           Guillain-Barre syndrome (GBS) is a postinfectious autoimmune neuropathy and anti-gangliosid
8  virulence and provide a molecular basis for postinfectious autoimmunity associated with M. pneumonia
9 c fever (ARF) is one of the best examples of postinfectious autoimmunity due to molecular mimicry bet
10 asma adhesins may play in molecular mimicry, postinfectious autoimmunity, and immune-mediated damage.
11                                Patients with postinfectious bronchiolitis obliterans generally have c
12                               Infectious and postinfectious cases had higher median cerebrospinal flu
13 demiology, pathophysiology, and treatment of postinfectious chronic fatigue syndrome (CFS) in adolesc
14 lar hemolysis, initially attributed to acute postinfectious cold hemagglutinin (immunoglobulin M) dis
15 (143 +/- 62 cells/microl); and patients with postinfectious complications showed low EBV load (3.0 +/
16 f human enterocolitis and is associated with postinfectious complications, including irritable bowel
17 otrienes are involved in the pathogenesis of postinfectious cough and whooping cough (pertussis).
18                       However, the burden of postinfectious cough in primary care is high, making it
19                                              Postinfectious cough is common in primary care, but has
20 al, non-smoking adults aged 16-49 years with postinfectious cough of 2-8 weeks' duration were recruit
21 ene receptor antagonist, in the treatment of postinfectious cough.
22 ontelukast is not an effective treatment for postinfectious cough.
23 s, acute disseminated encephalomyelitis, and postinfectious disorders should be exclusions in the NMO
24 om keratoplasty restores vision in eyes with postinfectious, full-thickness, central corneal scars.
25                                              Postinfectious glomerulonephritis (PIGN) is a rare etiol
26                                              Postinfectious glomerulonephritis (PIGN) is primarily a
27 s, membranoproliferative glomerulonephritis, postinfectious glomerulonephritis, thin basement membran
28 ase and C3 GN), or clinical course (atypical postinfectious GN).
29                               With regard to postinfectious GN, C4d staining was negative in six (46%
30 d in human diseases presenting as nephritis (postinfectious GN, IgA nephropathy, antiglomerular basem
31 ecimens of C3 GN, and 13 biopsy specimens of postinfectious GN.
32 tients with sepsis, frequently included in a postinfectious hemophagocytic lymphohistiocytosis settin
33                                              Postinfectious hydrocephalus in infants is a major healt
34 -center study involving Ugandan infants with postinfectious hydrocephalus showed no significant diffe
35 operitoneal shunting in Ugandan infants with postinfectious hydrocephalus.
36     Recent data developing from the study of postinfectious IBS has challenged the belief that IBS is
37 ardiasis patients and the pathophysiology of postinfectious IBS is needed.
38 esponsible for diarrheal disease and chronic postinfectious illness.
39    Guillain-Barre syndrome (GBS) is an acute postinfectious immune-mediated polyneuropathy.
40  appears to be a significant risk factor for postinfectious irritable bowel and chronic fatigue syndr
41  and Campylobacter, are at increased risk of postinfectious irritable bowel syndrome and inflammatory
42 their impact on long-term effects, including postinfectious irritable bowel syndrome and inflammatory
43 luoroquinolone antibiotics and concurrently, postinfectious irritable bowel syndrome has been associa
44                                              Postinfectious irritable bowel syndrome may occur in 3%
45 lain-Barre syndrome, reactive arthritis, and postinfectious irritable bowel syndrome) contribute cons
46 whether rifaximin is effective in preventing postinfectious irritable bowel syndrome.
47 s' diarrhea causes substantial morbidity and postinfectious irritable bowel syndrome.
48 nt (30-011) who exhibited the characteristic postinfectious mononucleosis phenotype of XLP with hypog
49  with CNSL, 10 with encephalitis, and 4 with postinfectious neurological complications.
50           Guillain-Barre syndrome (GBS) is a postinfectious neuropathy most frequently caused by Camp
51 iated by multiple etiologies but it is often postinfectious or due to persistent cellular immune-medi
52 y hereditary, idiopathic, paraneoplastic, or postinfectious pancerebellitis.
53 o glycans present on glycolipids mediate the postinfectious paralytic disease, Guillain-Barre syndrom
54 bacter infection has also been linked to the postinfectious sequelae of reactive arthritis and Guilla
55 ation of the role of Campylobacter jejuni in postinfectious sequelae, a broadened understanding of Ca
56 imicrobial therapy is recommended to prevent postinfectious sequelae.
57 e of diarrhea and is associated with serious postinfectious sequelae.
58 unity and identification of risk factors for postinfectious sequelae.
59 s, providing a new paradigm for the study of postinfectious symptoms in a subset of patients with Lym

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