ライフサイエンスコーパス: postjunctional

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1 ypothesis that this is due to a reduction in postjunctional alpha-adrenergic responsiveness to endoge

2 ng is associated with a reduction in forearm postjunctional alpha-adrenergic responsiveness to endoge

3 ndings indicate that the ability to modulate postjunctional alpha-adrenergic vasoconstriction during

4 pothesis that exogenous ATP can blunt direct postjunctional alpha-adrenergic vasoconstriction in huma

5 we tested the hypothesis that modulation of postjunctional alpha-adrenergic vasoconstriction to exog

6 exogenous ATP is capable of blunting direct postjunctional alpha-adrenergic vasoconstriction, that t

7 present study, we tested the hypothesis that postjunctional alpha-adrenergic vasoconstrictor responsi

8 ageing is associated with a reduction in leg postjunctional alpha-adrenoceptor responsiveness to endo

9 Whether this involves direct modulation of postjunctional alpha-adrenoceptor responsiveness, or is

10 ists have prejunctional (alpha 2) as well as postjunctional (alpha 1) effects.

11 ch occurred ahead of other prejunctional and postjunctional components, suggesting that LRP4 may regu

12 cles, but not the p1 muscle, by activating a postjunctional conductance increase that was blocked by

13 -evoked enhancement but did not increase the postjunctional conductance.

14 compared beta-NAD(+) and ATP metabolism and postjunctional effects of the primary extracellular meta

15 n prejunctionally without significant direct postjunctional effects.

16 ucturally simplified and the organization of postjunctional folds is aberrant in mice lacking tyrosin

17 istributions of acetylcholine receptors, and postjunctional folds that are generally less organized a

18 it is most concentrated in the depths of the postjunctional folds.

19 eatic ganglia, and that they mediate pre-and postjunctional inhibition.

20 adenosine receptors are exerted not only on postjunctional M1/M3 receptors but also at M2 presynapti

21 an inhibitory neurotransmitter rather than a postjunctional mediator; (b) VIP is a prejunctional neur

22 eptor tyrosine kinases, are localized at the postjunctional membrane presumably to ensure localized s

23 tylcholine receptor (AChR) clustering at the postjunctional membrane.

24 s to maintain a high density of AChRs at the postjunctional membrane.

25 pse that is formed by motor nerve terminals, postjunctional muscle membranes, and terminal Schwann ce

26 Autoantibodies with reactivity against the postjunctional muscle receptor for acetylcholine recepto

27 A postjunctional negative inotropism was also shown, media

28 In the absence of ICC-IM, postjunctional neural responses are compromised.

29 Spontaneous electrical activity and postjunctional neural responses in hyperplastic ICC tiss

30 P from nerve terminal varicosities acting at postjunctional P2X receptors.

31 ooth muscle is consistent with its role as a postjunctional receptor in autonomic transmission, while

32 this does not appear to be due to changes in postjunctional receptors, or to a depletion of transmitt

33 Postjunctional responses to nerve stimulation, beta-NAD

34 through gap junctions conditionally trigger postjunctional spikes, depending on both neurons being c

35 n in isolated PDGFRalpha(+) cells, which are postjunctional targets for purinergic neurotransmission.

36 gh actions at alpha 2 A-adrenergic receptors postjunctional to noradrenergic terminals.

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